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Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1625 - 1636
Signaling through the adaptor protein myeloid differentiation factor 88 (MyD88) promotes carcinogenesis in several cancer models. In contrast, MyD88 signaling... 
EXPRESSION PATTERNS | MEDICINE, RESEARCH & EXPERIMENTAL | CHRONIC INTESTINAL INFLAMMATION | MUTANT MICE | SODIUM-SULFATE COLITIS | CELL LINES | COLITIS-ASSOCIATED CANCER | IMMUNOLOGY | BETA-CATENIN | TUMORIGENESIS | INNATE IMMUNITY | BOWEL-DISEASE | Colonic Neoplasms - genetics | Intestinal Mucosa - metabolism | Adenocarcinoma - pathology | Azoxymethane - pharmacology | Epithelial Cells - metabolism | Gene Expression - drug effects | Apoptosis - drug effects | Gene Expression - genetics | Adenocarcinoma - chemically induced | Colonic Polyps - pathology | Epithelial Cells - drug effects | Colon - drug effects | DNA Repair Enzymes - genetics | Apoptosis - genetics | Colonic Neoplasms - chemically induced | Gene Expression Profiling | Inflammatory Bowel Diseases - metabolism | Colonic Neoplasms - metabolism | Intestinal Mucosa - drug effects | Adenocarcinoma - metabolism | Cyclooxygenase 2 - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Interleukin-18 Receptor alpha Subunit - genetics | Inflammatory Bowel Diseases - chemically induced | Adenocarcinoma - genetics | Phosphorylation - drug effects | STAT3 Transcription Factor - genetics | Dextran Sulfate - pharmacology | Genetic Predisposition to Disease - genetics | Specific Pathogen-Free Organisms | Colon - pathology | Mice, Inbred C57BL | Epithelial Cells - pathology | Mutation - genetics | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Animals | Colonic Neoplasms - pathology | Receptors, Interleukin-1 Type I - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | Interleukin-18 - genetics | Myeloid Differentiation Factor 88 - metabolism | Interleukin-18 - metabolism | Intestinal Mucosa - pathology | Index Medicus
Journal Article
Science, ISSN 0036-8075, 8/2013, Volume 341, Issue 6147, pp. 789 - 792
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 04/2006, Volume 207, Issue 1, pp. 261 - 270
Recent evidence indicates that cyclooxygenase‐2 (COX‐2) and epidermal growth factor receptor (EGFR) are involved in hepatocarcinogenesis. This study was... 
Proto-Oncogene Proteins c-met - metabolism | RNA, Small Interfering - genetics | src-Family Kinases | Gene Expression - genetics | Protein-Tyrosine Kinases - metabolism | Caproates - pharmacology | Humans | Receptors, Prostaglandin - metabolism | Receptors, Prostaglandin E - genetics | Alprostadil - analogs & derivatives | Cell Movement - physiology | Membrane Proteins - analysis | Receptor, Epidermal Growth Factor - metabolism | Receptors, Prostaglandin - genetics | Transfection | RNA Interference | Cyclooxygenase 2 - genetics | Carcinoma, Hepatocellular - genetics | Protein Binding - drug effects | Liver Neoplasms - pathology | Membrane Proteins - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins - metabolism | Dinoprostone - pharmacology | Bridged Bicyclo Compounds - pharmacology | Liver Neoplasms - genetics | Membrane Proteins - genetics | Neoplasm Invasiveness | Alprostadil - pharmacology | Cyclooxygenase 2 - analysis | Receptors, Prostaglandin E - physiology | Cell Movement - drug effects | Carcinoma, Hepatocellular - pathology | Cyclooxygenase 2 - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Receptors, Prostaglandin E, EP1 Subtype | Enzyme Activation | Carcinoma, Hepatocellular - metabolism | Index Medicus
Journal Article
Journal Article
FASEB Journal, ISSN 0892-6638, 07/2014, Volume 28, Issue 7, pp. 3183 - 3196
Journal Article
STEM CELLS, ISSN 1066-5099, 10/2012, Volume 30, Issue 10, pp. 2283 - 2296
Culturing cells in three dimension (3D) provides an insight into their characteristics in vivo. We previously reported that human mesenchymal stem/stromal... 
Sphere | Caspase | COX2 | MSC | LPS | Macrophage | PROTEIN TSG-6 | STEM-CELLS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | IN-VITRO | MSCS | BACTERIAL CLEARANCE | ONCOLOGY | HUMAN BONE-MARROW | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | INFLAMMATION | ENDOTHELIAL-CELLS | TNF-ALPHA | HEMATOLOGY | MARROW STROMAL CELLS | Mannose-Binding Lectins - metabolism | RNA, Small Interfering - genetics | Humans | NF-kappa B - metabolism | Spheroids, Cellular - cytology | Lectins, C-Type - genetics | Lectins, C-Type - metabolism | Dinoprostone - isolation & purification | Mesenchymal Stromal Cells - cytology | Caspases - metabolism | Cyclooxygenase 2 - genetics | Spheroids, Cellular - drug effects | Cell Culture Techniques | Fibroblasts - metabolism | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Receptors, Prostaglandin E, EP4 Subtype - genetics | Dinoprostone - pharmacology | Mesenchymal Stromal Cells - drug effects | Cyclooxygenase 2 Inhibitors - pharmacology | Caspases - genetics | Spheroids, Cellular - metabolism | Mesenchymal Stromal Cells - metabolism | Receptors, Cell Surface - metabolism | Antibodies - pharmacology | Gene Expression Regulation - drug effects | Culture Media, Conditioned | Macrophages - metabolism | Mannose-Binding Lectins - genetics | NF-kappa B - genetics | Signal Transduction - drug effects | Fibroblasts - drug effects | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Dinoprostone - antagonists & inhibitors | Macrophage Activation - drug effects | Cytokines - biosynthesis | Receptors, Cell Surface - genetics | Anti-inflammatory drugs | RNA | Analysis | Prostaglandins E | Stem cells | Genetic aspects | Information management | Macrophages | Cytokines | Genotype & phenotype | Molecular weight | Index Medicus | Antibodies | macrophage | spheroid | caspase
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 237 - 12
Journal Article
Stem Cells: the international journal of cell differentiation and proliferation, ISSN 1066-5099, 02/2013, Volume 31, Issue 2, pp. 305 - 316
textabstractHuman microvascular pericytes (CD1461/342/452/562) contain multipotent precursors and repair/regenerate defective tissues, notably skeletal muscle.... 
Myocardial infarction | Pericytes | Angiogenesis | Stem cell therapy | Immunomodulation | ACTIVATION | MECHANISMS | MESENCHYMAL STEM-CELLS | HUMAN SKELETAL-MUSCLE | TRANSPLANTATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | THERAPY | ONCOLOGY | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | IMMUNOSUPPRESSION | GROWTH-FACTOR | HEMATOLOGY | EXPRESSION | Myocardial Infarction - genetics | Heme Oxygenase-1 - metabolism | Humans | Transplantation, Heterologous | Leukemia Inhibitory Factor - genetics | Myocardial Infarction - diagnostic imaging | Vascular Endothelial Growth Factor A - metabolism | Pericytes - physiology | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - genetics | Antigens, CD - metabolism | Leukemia Inhibitory Factor - metabolism | Heme Oxygenase-1 - genetics | Myocardial Infarction - therapy | Cyclooxygenase 2 - genetics | Myocardial Infarction - pathology | Myocardium - metabolism | Pericytes - transplantation | Ultrasonography | Chemokine CCL2 - metabolism | Membrane Proteins - metabolism | Cell Culture Techniques | Fibrosis - prevention & control | Interleukin-6 - metabolism | Biomarkers - metabolism | Gene Expression | Proto-Oncogene Proteins c-sis - genetics | Interleukin-6 - genetics | Proto-Oncogene Proteins c-sis - metabolism | Membrane Proteins - genetics | Chemokine CCL2 - genetics | Myocardium - pathology | Regeneration - physiology | Animals | Cyclooxygenase 2 - metabolism | Mice | Neovascularization, Physiologic | Echocardiography | Developmental biology | Leukemia | Muscles | Fluorescence | Macrophages | Heart attack | Endothelium | Stem cell research | Orthopedic surgery | Interleukins | Ischemia | Analysis | Heme | Stem cells | Universities and colleges | Health aspects | Growth factors | Proteins | Musculoskeletal system | Hypoxia | Rodents | Index Medicus | immunomodulation | pericytes | angiogenesis | stem cell therapy | myocardial infarction
Journal Article
CANCER RESEARCH, ISSN 0008-5472, 12/2011, Volume 71, Issue 24, pp. 7463 - 7470
Journal Article
Journal of Clinical Endocrinology and Metabolism, ISSN 0021-972X, 05/2017, Volume 102, Issue 5, pp. 1468 - 1477
Context: Increasing evidences suggest a correlation between gut and type 1 diabetes (T1D). Objective: The objective of this study is to evaluate the gut... 
INCREASED INTESTINAL PERMEABILITY | HEALTHY-CHILDREN | DIFFERS | FECAL MICROBIOTA | DISEASE | ENDOCRINOLOGY & METABOLISM | CELL AUTOIMMUNITY | GUT MICROBIOTA | MELLITUS | ONSET | Receptors, CCR2 - genetics | Serum Amyloid P-Component - immunology | Receptors, CCR2 - immunology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Transcriptome | Child, Preschool | Male | Monocyte Chemoattractant Proteins - immunology | Vascular Endothelial Growth Factor A - genetics | Antigens, CD - genetics | Interleukin-4 Receptor alpha Subunit - genetics | Young Adult | Celiac Disease - microbiology | Cyclooxygenase 2 - genetics | Intestinal Mucosa - immunology | Diabetes Mellitus, Type 1 - microbiology | Tumor Necrosis Factor-alpha - immunology | Child | Real-Time Polymerase Chain Reaction | Duodenum - immunology | Vascular Endothelial Growth Factor A - immunology | C-Reactive Protein - genetics | Intestinal Mucosa - microbiology | Reverse Transcriptase Polymerase Chain Reaction | Monocyte Chemoattractant Proteins - genetics | Chemokine CCL22 - genetics | Chemokine CCL19 - genetics | Adolescent | RNA, Ribosomal, 16S - genetics | C-Reactive Protein - immunology | Antigens, CD - immunology | Infant | Case-Control Studies | Duodenum - microbiology | Chemokine CCL22 - immunology | Celiac Disease - immunology | Adult | Female | Diabetes Mellitus, Type 1 - immunology | Chemokine CCL19 - immunology | Cyclooxygenase 2 - immunology | Antigens, Differentiation, Myelomonocytic - immunology | Diabetes Mellitus, Type 1 - genetics | Gastrointestinal Microbiome - genetics | Interleukin-4 Receptor alpha Subunit - immunology | Antigens, Differentiation, Myelomonocytic - genetics | Serum Amyloid P-Component - genetics | Aged | Immunohistochemistry | CC chemokine receptors | rRNA 16S | CCL19 protein | Pathogenesis | Mucosa | Microbiomes | Diabetes mellitus (insulin dependent) | Macrophages | Small intestine | Gene sequencing | CCL22 protein | Microbiota | CCR2 protein | Intestine | Interleukin 4 receptors | Digestive tract | Duodenum | Digestive system | Abnormalities | Diabetes mellitus | Inflammation | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Disease control | Patients | Celiac disease | Biopsy | Infiltration | Diabetes | Cyclooxygenase-2 | Autoimmune diseases | Monocyte chemoattractant protein 1 | Index Medicus | Abridged Index Medicus
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 02/2016, Volume 25, Issue 3, pp. 584 - 596
Leber's hereditary optic neuropathy (LHON) is the most common mitochondrial disorder. Nuclear modifier genes are proposed to modify the phenotypic expression... 
OXIDATIVE STRESS | COMPLEX-I | CAUSES MYOPATHY | BIOCHEMISTRY & MOLECULAR BIOLOGY | HIGH PENETRANCE | GENETICS & HEREDITY | HAN CHINESE FAMILIES | LACTIC-ACIDOSIS | ND4 G11778A MUTATION | SIDEROBLASTIC ANEMIA | CELL-LINES | MTDNA MUTATIONS | Mitochondria - enzymology | Neurons - pathology | Humans | Molecular Sequence Data | NADH Dehydrogenase - genetics | Optic Atrophy, Hereditary, Leber - genetics | Mitochondrial Proteins - genetics | Electron Transport Complex I - metabolism | Case-Control Studies | Electron Transport Complex IV - metabolism | Exome | Optic Atrophy, Hereditary, Leber - pathology | DNA, Mitochondrial - genetics | Mitochondria - genetics | Optic Atrophy, Hereditary, Leber - enzymology | Cyclooxygenase 2 - genetics | Electron Transport Complex I - genetics | Mitochondrial Proteins - metabolism | Base Sequence | Tyrosine-tRNA Ligase - metabolism | Cell Line | Tyrosine-tRNA Ligase - chemistry | Genetic Predisposition to Disease | DNA, Mitochondrial - metabolism | Gene Expression Regulation | Models, Molecular | Tyrosine-tRNA Ligase - genetics | Electron Transport Complex IV - genetics | Mitochondria - pathology | NADH Dehydrogenase - metabolism | Homozygote | Phenotype | Pedigree | Neurons - enzymology | Alleles | Cyclooxygenase 2 - metabolism | Mitochondrial Proteins - chemistry | Heterozygote | Mutation | Index Medicus
Journal Article