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CANCER RESEARCH, ISSN 0008-5472, 12/2011, Volume 71, Issue 24, pp. 7463 - 7470
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 06/2014, Volume 306, Issue 12, pp. E1378 - E1387
Incomplete beta-oxidation of fatty acids in mitochondria is a feature of insulin resistance and type 2 diabetes mellitus (T2DM). Previous studies revealed that... 
Acylcarnitine | β-oxidation | Inflammation | TLR | Pattern recognition receptors | OXIDATION | PHYSIOLOGY | acylcarnitine | SATURATED FATTY-ACIDS | INDUCED INSULIN-RESISTANCE | pattern recognition receptors | TOLL-LIKE RECEPTOR-2 | OBESITY | inflammation | beta-oxidation | ENDOCRINOLOGY & METABOLISM | EXPRESSION | Phosphorylation | Toll-Like Receptor 2 - agonists | Toll-Like Receptor 2 - antagonists & inhibitors | Toll-Like Receptor 2 - genetics | Humans | Carnitine - metabolism | Diabetes Mellitus, Type 2 - metabolism | MAP Kinase Signaling System | Receptors, Pattern Recognition - antagonists & inhibitors | Receptors, Pattern Recognition - metabolism | Macrophages - secretion | Cyclooxygenase 2 - genetics | Diabetes Mellitus, Type 2 - immunology | Myeloid Differentiation Factor 88 - antagonists & inhibitors | Receptors, Pattern Recognition - agonists | Carnitine - analogs & derivatives | Macrophages - immunology | Recombinant Proteins - metabolism | Myristic Acids - metabolism | Cyclooxygenase 2 - chemistry | Cytokines - secretion | Gene Silencing | Myeloid Differentiation Factor 88 - genetics | Enzyme Induction | Recombinant Proteins - chemistry | Toll-Like Receptor 2 - metabolism | Macrophage Activation | Myeloid Differentiation Factor 88 - agonists | Macrophages - metabolism | Animals | Cyclooxygenase 2 - metabolism | Cell Line, Tumor | Receptors, Pattern Recognition - genetics | Mice | Protein Processing, Post-Translational | Myeloid Differentiation Factor 88 - metabolism | Cell Line, Transformed | Docosahexaenoic Acids - metabolism | Type 2 diabetes | Physiological aspects | Insulin resistance | Cellular signal transduction | Research | Diabetes | Oxidation | T cell receptors | Cytokines | Index Medicus
Journal Article
GLIA, ISSN 0894-1491, 04/2010, Volume 58, Issue 5, pp. 588 - 598
Neural injury leads to inflammation and activation of microglia that in turn may participate in progression of neurodegeneration. The mechanisms involved in... 
Neurodegenerative diseases | Innate immune system | Heme oxygenase-1 | OXIDATIVE STRESS | ACTIVATION | LEWY BODY DISEASE | heme oxygenase-1 | NEUROSCIENCES | innate immune system | CELL-DEATH | neurodegenerative diseases | TRANSCRIPTION FACTOR NRF2 | BRAIN INFLAMMATION | MOUSE MODEL | THERAPEUTIC TARGET | ENHANCES SUSCEPTIBILITY | Dopamine Plasma Membrane Transport Proteins - metabolism | Tyrosine 3-Monooxygenase - metabolism | Flow Cytometry - methods | Leukocyte Common Antigens - metabolism | Reactive Oxygen Species - metabolism | Culture Media, Conditioned - pharmacology | Male | Glial Fibrillary Acidic Protein - metabolism | Brain - metabolism | MPTP Poisoning - complications | Inflammation - metabolism | Microglia - physiology | MPTP Poisoning - pathology | Antigens, Differentiation - metabolism | Neurons - metabolism | Neurons - drug effects | Dopamine - metabolism | Gliosis - etiology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Cytokines - metabolism | Neurons - chemistry | Mice, Inbred C57BL | Gene Expression Regulation - physiology | NF-E2-Related Factor 2 - deficiency | Inflammation - etiology | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Analysis of Variance | Microfilament Proteins | NF-E2-Related Factor 2 - metabolism | Cyclooxygenase 2 - metabolism | Brain - pathology | Mice | CD11b Antigen - metabolism | Cell Line, Transformed | Nitric Oxide Synthase Type II - metabolism | Index Medicus
Journal Article
Journal Article
Breast Cancer Research, ISSN 1465-5411, 09/2011, Volume 13, Issue 5, pp. R87 - R87
Introduction: Some molecular subtypes of breast cancer have preferential sites of distant relapse. The protein expression pattern of the primary tumor may... 
MOLECULAR SUBTYPES | SURVIVAL | RELAPSE | CELLS | ONCOLOGY | ERBB2 | BRAIN METASTASES | MARKERS | PATTERNS | CARCINOMA | TUMORS | Nestin | Receptors, Estrogen - metabolism | Cadherins - metabolism | Follow-Up Studies | Humans | Lung Neoplasms - metabolism | Glycoproteins - metabolism | Receptor, ErbB-2 - metabolism | Bone Neoplasms - secondary | Proteins - analysis | Brain Neoplasms - metabolism | Antigens, CD - metabolism | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | Receptors, Progesterone - metabolism | Brain Neoplasms - secondary | Receptor, Epidermal Growth Factor - metabolism | Peptides - metabolism | Lung Neoplasms - secondary | Female | Liver Neoplasms - secondary | Snail Family Transcription Factors | AC133 Antigen | Skin Neoplasms - metabolism | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | Proteins - metabolism | Breast Neoplasms - pathology | Cyclooxygenase 2 - metabolism | Finland | Liver Neoplasms - metabolism | Skin Neoplasms - secondary | Keratin-5 - metabolism | Intermediate Filament Proteins - metabolism | Cohort Studies | Immunohistochemistry | Care and treatment | Estrogen | Development and progression | Breast cancer | Research | Gene expression | Keratin | Epidermal growth factor | Genetic aspects | Diagnosis | Progesterone | Tumor proteins | Index Medicus
Journal Article
Journal Article
European Journal of Pharmacology, ISSN 0014-2999, 2010, Volume 648, Issue 1, pp. 179 - 187
Previously, we have reported that Corni Fructus possessed hypoglycemic and hypocholesterolemic effects in streptozotocin-induced type 1 diabetic rats and... 
Oxidative stress | Hyperglycemia | Advanced glycation endproducts | Loganin | db/db mice | Dyslipidemia | Db/db mice | LIPID-METABOLISM | RATS | HACHIMI-JIO-GAN | KAPPA-B | MORRONISIDE | INSULIN-RESISTANCE | DISEASE | PHARMACOLOGY & PHARMACY | TISSUES | GLYCATION END-PRODUCTS | Gene Expression Regulation, Enzymologic - drug effects | Liver - pathology | Kidney - pathology | Iridoids - isolation & purification | Body Weight - drug effects | Male | Hematologic Tests | Diabetes Mellitus, Type 2 - metabolism | RNA, Messenger - metabolism | Inflammation - complications | Cornus - chemistry | Kidney - metabolism | Liver - drug effects | Inflammation - drug therapy | Lipid Metabolism - genetics | Lysine - metabolism | Sterol Regulatory Element Binding Protein 2 - metabolism | Fatty Acids - metabolism | Diabetes Mellitus, Type 2 - complications | Sterol Regulatory Element Binding Protein 1 - metabolism | Biomarkers - metabolism | Lysine - analogs & derivatives | Kidney - drug effects | Liver - metabolism | RNA, Messenger - genetics | Cholesterol - metabolism | Triglycerides - metabolism | Organ Size - drug effects | Animals | Transcription Factor RelA - metabolism | Iridoids - pharmacology | Glycation End Products, Advanced - metabolism | Cyclooxygenase 2 - metabolism | Lipid Metabolism - drug effects | Glucose - metabolism | Drinking - drug effects | Iridoids - therapeutic use | Mice | Diabetes Mellitus, Type 2 - pathology | Oxidative Stress - drug effects | PPAR alpha - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Nitric Oxide Synthase Type II - metabolism | Hypercholesterolemia | RNA | Type 1 diabetes | Analysis | Inflammation | Protein binding | Index Medicus
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 2011, Volume 192, Issue 5, pp. 767 - 780
The unique morphology of tuft cells was first revealed by electron microscopy analyses in several endoderm-derived epithelia. Here, we explore the relationship... 
PROGENITOR CELLS | MOUSE INTESTINE | STEM-CELLS | GOBLET CELLS | ENTEROENDOCRINE CELLS | BRUSH CELLS | ALPHA-GUSTDUCIN | PANETH CELLS | TRANSCRIPTION FACTOR | ADENOMATOUS POLYPOSIS | CELL BIOLOGY | Intestinal Mucosa - metabolism | Adenocarcinoma - pathology | Humans | Adenoma - metabolism | Intestinal Mucosa - cytology | Intestine, Large - metabolism | Adenocarcinoma - metabolism | Basic Helix-Loop-Helix Transcription Factors - metabolism | Cell Differentiation | Membrane Proteins - metabolism | Intestine, Large - cytology | Protein-Serine-Threonine Kinases - metabolism | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Basic Helix-Loop-Helix Transcription Factors - physiology | Nerve Tissue Proteins - physiology | Basic Helix-Loop-Helix Transcription Factors - genetics | Intestinal Neoplasms - metabolism | Mice, Inbred C57BL | Biomarkers - analysis | Nerve Tissue Proteins - genetics | Intestinal Neoplasms - pathology | Nerve Tissue Proteins - metabolism | Animals | Intestine, Small - cytology | Cyclooxygenase 2 - metabolism | Adenoma - pathology | Mice | Cyclooxygenase 1 - metabolism | Intestine, Small - metabolism | Isomerases - metabolism | Proteins | Intestines | Research | Chemical properties | Epithelium | Cells | Scanning electron microscopy | Enzymes | Identification | Anatomy & physiology | Stem cells | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2012, Volume 7, Issue 9, pp. e44789 - e44789
Increasing evidence supports the involvement of microRNAs (miRNA) in the regulation of inflammation in human neurological disorders. In the present study we... 
INTERLEUKIN-1 | CORTICAL DYSPLASIA | ACTIVATION | MALFORMATIONS | MULTIDISCIPLINARY SCIENCES | TOLL-LIKE RECEPTOR | DISEASE | EPILEPSY | NF-KAPPA-B | EXPRESSION | RAT MODEL | Brain Diseases - metabolism | Interleukin-1 Receptor-Associated Kinases - metabolism | Interleukin-1beta - pharmacology | Humans | Astrocytes - pathology | Brain Diseases - genetics | Epilepsy - metabolism | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Brain Diseases - immunology | Ganglioglioma - pathology | Inflammation Mediators - metabolism | Epilepsy - genetics | Female | Toll-Like Receptors - metabolism | Malformations of Cortical Development - immunology | Malformations of Cortical Development - pathology | Interleukin-6 - metabolism | Astrocytes - drug effects | Malformations of Cortical Development, Group I | Immunity, Innate - drug effects | Malformations of Cortical Development - metabolism | Malformations of Cortical Development - genetics | Receptors, Interleukin-1 - metabolism | Gene Expression Regulation - drug effects | Pregnancy | Ganglioglioma - genetics | Brain Diseases - pathology | Signal Transduction - drug effects | Cyclooxygenase 2 - metabolism | TNF Receptor-Associated Factor 6 - metabolism | Cell Line, Tumor | MicroRNAs - genetics | Ganglioglioma - metabolism | Epilepsy - pathology | Astrocytes - metabolism | Astrocytes - secretion | Medical research | Nervous system diseases | Immune response | MicroRNA | Cytokines | Epilepsy | Medicine, Experimental | Inflammation | Research | Enzyme-linked immunosorbent assay | Neurosciences | Downstream effects | Disorders | Inflammatory response | Nervous system | Kinases | Interleukin 6 | Transfection | Modulation | Lesions | IRAK protein | Encephalitis | Immunomodulation | Astrocytes | MiRNA | Cultures | Tumor necrosis factor-α | Gene expression | Ribonucleic acid--RNA | Neurological diseases | Immune systems | Pathology | Signaling | Brain research | MicroRNAs | Cyclooxygenase-2 | Index Medicus | RNA | Ribonucleic acid
Journal Article
STEM CELLS, ISSN 1066-5099, 10/2012, Volume 30, Issue 10, pp. 2283 - 2296
Culturing cells in three dimension (3D) provides an insight into their characteristics in vivo. We previously reported that human mesenchymal stem/stromal... 
Sphere | Caspase | COX2 | MSC | LPS | Macrophage | PROTEIN TSG-6 | STEM-CELLS | CELL & TISSUE ENGINEERING | CELL BIOLOGY | IN-VITRO | MSCS | BACTERIAL CLEARANCE | ONCOLOGY | HUMAN BONE-MARROW | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | INFLAMMATION | ENDOTHELIAL-CELLS | TNF-ALPHA | HEMATOLOGY | MARROW STROMAL CELLS | Mannose-Binding Lectins - metabolism | RNA, Small Interfering - genetics | Humans | NF-kappa B - metabolism | Spheroids, Cellular - cytology | Lectins, C-Type - genetics | Lectins, C-Type - metabolism | Dinoprostone - isolation & purification | Mesenchymal Stromal Cells - cytology | Caspases - metabolism | Cyclooxygenase 2 - genetics | Spheroids, Cellular - drug effects | Cell Culture Techniques | Fibroblasts - metabolism | Receptors, Prostaglandin E, EP4 Subtype - metabolism | Receptors, Prostaglandin E, EP4 Subtype - genetics | Dinoprostone - pharmacology | Mesenchymal Stromal Cells - drug effects | Cyclooxygenase 2 Inhibitors - pharmacology | Caspases - genetics | Spheroids, Cellular - metabolism | Mesenchymal Stromal Cells - metabolism | Receptors, Cell Surface - metabolism | Antibodies - pharmacology | Gene Expression Regulation - drug effects | Culture Media, Conditioned | Macrophages - metabolism | Mannose-Binding Lectins - genetics | NF-kappa B - genetics | Signal Transduction - drug effects | Fibroblasts - drug effects | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Dinoprostone - antagonists & inhibitors | Macrophage Activation - drug effects | Cytokines - biosynthesis | Receptors, Cell Surface - genetics | Anti-inflammatory drugs | RNA | Analysis | Prostaglandins E | Stem cells | Genetic aspects | Information management | Macrophages | Cytokines | Genotype & phenotype | Molecular weight | Index Medicus | Antibodies | macrophage | spheroid | caspase
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2012, Volume 32, Issue 3, pp. 669 - 676
OBJECTIVE—Nicotinic acid (NA) treatment has been associated with benefits in atherosclerosis that are usually attributed to effects on plasma lipoproteins. The... 
atherosclerosis | macrophages | vascular biology | receptors | cholesterol-lowering drugs | LONG-TERM | INTEGRIN VLA-4 | ACTIVATION | DENDRITIC CELLS | KAPPA-B | FOAM CELLS | ENDOTHELIAL-CELLS | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | EXTENDED-RELEASE NIACIN | Tumor Necrosis Factor-alpha - metabolism | Phosphorylation | Integrin alpha4beta1 - metabolism | Toll-Like Receptor 2 - agonists | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, G-Protein-Coupled - metabolism | Humans | Human Umbilical Vein Endothelial Cells - immunology | Monocytes - metabolism | Monocytes - immunology | Niacin - pharmacology | Receptors, Prostaglandin - metabolism | Receptors, Immunologic - antagonists & inhibitors | Inflammation - metabolism | Transfection | Chemotaxis, Leukocyte - drug effects | I-kappa B Kinase - metabolism | RNA Interference | Inflammation Mediators - metabolism | Toll-Like Receptor 4 - agonists | Chemokine CCL2 - metabolism | Receptors, G-Protein-Coupled - drug effects | Interleukin-6 - metabolism | Receptors, Prostaglandin - antagonists & inhibitors | Receptors, Nicotinic - drug effects | Receptors, Nicotinic - metabolism | Human Umbilical Vein Endothelial Cells - drug effects | Cyclooxygenase 2 Inhibitors - pharmacology | Anti-Inflammatory Agents - pharmacology | Cells, Cultured | Inflammation - immunology | Toll-Like Receptor 2 - metabolism | Cell Adhesion - drug effects | Toll-Like Receptor 4 - metabolism | Monocytes - drug effects | Transcription Factor RelA - metabolism | Lipopolysaccharides - pharmacology | Inflammation - genetics | Receptors, G-Protein-Coupled - genetics | Pyrazines - pharmacology | Vascular Cell Adhesion Molecule-1 - metabolism | Receptors, Nicotinic - genetics | Receptors, Immunologic - metabolism | Index Medicus
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2010, Volume 30, Issue 24, pp. 8285 - 8295
Toll-like receptors play an important role in the innate immune response, although emerging evidence indicates their role in brain injury and... 
ETHANOL TREATMENT | ALZHEIMERS-DISEASE | IN-VIVO | TOLL-LIKE RECEPTORS | INJURY | NEURODEGENERATION | ENHANCES INFLAMMATORY MEDIATORS | ASTROGLIAL CELLS | ASTROCYTES | NEUROSCIENCES | CELL-DEATH | Tumor Necrosis Factor-alpha - metabolism | Rats, Wistar | Lymphocyte Antigen 96 - metabolism | Tumor Necrosis Factor-alpha - genetics | Caspase 3 - metabolism | Ethanol - administration & dosage | Green Fluorescent Proteins - genetics | Brain Injuries - metabolism | Interleukin-1beta - genetics | Lipopolysaccharides - adverse effects | Lipopolysaccharide Receptors - metabolism | Lymphocyte Antigen 96 - genetics | Ethanol - blood | Time Factors | Toll-Like Receptor 4 - deficiency | Cyclooxygenase 2 - genetics | Interleukin-1beta - metabolism | Encephalitis - metabolism | Statistics, Nonparametric | Brain Injuries - blood | Female | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Astrocytes - drug effects | Mice, Inbred C57BL | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Brain Injuries - chemically induced | Encephalitis - pathology | Rats | Mice, Knockout | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Toll-Like Receptor 4 - physiology | Animals | Microfilament Proteins | Central Nervous System Depressants - administration & dosage | Cyclooxygenase 2 - metabolism | Mice | Encephalitis - chemically induced | Central Nervous System Depressants - blood | Brain Injuries - pathology | Encephalitis - blood | Astrocytes - metabolism | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 04/2006, Volume 207, Issue 1, pp. 261 - 270
Recent evidence indicates that cyclooxygenase‐2 (COX‐2) and epidermal growth factor receptor (EGFR) are involved in hepatocarcinogenesis. This study was... 
Proto-Oncogene Proteins c-met - metabolism | RNA, Small Interfering - genetics | src-Family Kinases | Gene Expression - genetics | Protein-Tyrosine Kinases - metabolism | Caproates - pharmacology | Humans | Receptors, Prostaglandin - metabolism | Receptors, Prostaglandin E - genetics | Alprostadil - analogs & derivatives | Cell Movement - physiology | Membrane Proteins - analysis | Receptor, Epidermal Growth Factor - metabolism | Receptors, Prostaglandin - genetics | Transfection | RNA Interference | Cyclooxygenase 2 - genetics | Carcinoma, Hepatocellular - genetics | Protein Binding - drug effects | Liver Neoplasms - pathology | Membrane Proteins - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins - metabolism | Dinoprostone - pharmacology | Bridged Bicyclo Compounds - pharmacology | Liver Neoplasms - genetics | Membrane Proteins - genetics | Neoplasm Invasiveness | Alprostadil - pharmacology | Cyclooxygenase 2 - analysis | Receptors, Prostaglandin E - physiology | Cell Movement - drug effects | Carcinoma, Hepatocellular - pathology | Cyclooxygenase 2 - metabolism | Liver Neoplasms - metabolism | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Protein Kinase Inhibitors - pharmacology | Receptors, Prostaglandin E, EP1 Subtype | Enzyme Activation | Carcinoma, Hepatocellular - metabolism | Index Medicus
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 02/2016, Volume 17, Issue 2, pp. 190 - 190
The pathogenesis of Parkinson's disease (PD) often involves the over-activation of microglia. Over-activated microglia could produce several inflammatory... 
Glucagon-like peptide-2 | NF-κB | MAPK | Parkinson’s disease | Microglia | ACTIVATION | Parkinson's disease | glucagon-like peptide-2 | ALZHEIMERS-DISEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | NF-B | ANTIINFLAMMATORY ACTIONS | NEURODEGENERATION | DEATH | DOPAMINERGIC-NEURONS | CHEMISTRY, MULTIDISCIPLINARY | NEUROINFLAMMATION | RECEPTORS | microglia | PARKINSONS-DISEASE |