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PLoS Pathogens, ISSN 1553-7366, 02/2008, Volume 4, Issue 2, pp. e1000003 - e1000003
Journal Article
Infection and Immunity, ISSN 0019-9567, 09/2013, Volume 81, Issue 9, pp. 3139 - 3147
Classifications Services IAI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit... 
CYCLOPHILIN-A | INFECTIOUS DISEASES | INVASION | PROTEIN | TYROSINE PHOSPHORYLATION | NEISSERIA-MENINGITIDIS | BARRIER | INFECTION | DEATH | IMMUNOLOGY | EXPRESSION | STATISTICAL-MODEL | Cytoskeleton - microbiology | Up-Regulation | Cryptococcus neoformans - physiology | Brain - microbiology | Cryptococcosis - microbiology | Humans | Cell Membrane - genetics | Blood-Brain Barrier - microbiology | HMGB1 Protein - genetics | Brain - metabolism | Endothelial Cells - microbiology | Inflammation - metabolism | S100 Proteins - genetics | HMGB1 Protein - metabolism | Muscle Proteins - metabolism | Myosins - metabolism | Cell Membrane - metabolism | Cryptococcosis - genetics | Cyclophilin A - genetics | Microfilament Proteins - metabolism | Microfilament Proteins - genetics | Cyclophilin A - metabolism | Brain - cytology | Inflammation - microbiology | Endothelial Cells - metabolism | Cells, Cultured | Cytoskeleton - genetics | S100 Proteins - metabolism | Signal Transduction - genetics | Cryptococcosis - metabolism | Myosins - genetics | Blood-Brain Barrier - metabolism | Cryptococcus neoformans - metabolism | Muscle Proteins - genetics | Annexin A2 - genetics | Cytoskeleton - metabolism | Inflammation - genetics | Cell Membrane - microbiology | Annexin A2 - metabolism | Index Medicus | Cellular Microbiology | Pathogen-Host Cell Molecular Interactions
Journal Article
Journal of Virology, ISSN 0022-538X, 09/2012, Volume 86, Issue 18, pp. 9875 - 9887
Journal Article
Journal Article
Journal Article
PLoS Pathogens, ISSN 1553-7366, 09/2010, Volume 6, Issue 9, pp. e1001118 - e1001118
Since the advent of genome-wide small interfering RNA screening, large numbers of cellular cofactors important for viral infection have been discovered at a... 
RNA REPLICATION | ACTIVE-SITE | POLYMERASE | MICROBIOLOGY | ISOMERASE ACTIVITY | IN-VITRO | INHIBITION | VIROLOGY | NS5A PROTEIN | SPECTROSCOPY | RESISTANCE | INFECTION | PARASITOLOGY | Hepatitis C - drug therapy | Cyclosporine - pharmacology | Humans | Protein Conformation - drug effects | Drug Resistance, Viral | Hepacivirus - genetics | Molecular Sequence Data | Hepacivirus - metabolism | Immunoenzyme Techniques | RNA, Viral - genetics | Cyclophilin A - genetics | Circular Dichroism | Immunosuppressive Agents - pharmacology | Viral Nonstructural Proteins - immunology | Cyclophilin A - metabolism | Hepacivirus - drug effects | Amino Acid Sequence | Cell Line | Virus Replication - drug effects | Mutagenesis, Site-Directed | Enzyme-Linked Immunosorbent Assay | Magnetic Resonance Spectroscopy | RNA, Messenger - genetics | RNA, Small Interfering - pharmacology | Viral Nonstructural Proteins - genetics | Hepatitis C - genetics | Mutation - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Sequence Homology, Amino Acid | Gene Expression Regulation, Viral | Hepatitis C - virology | Viral Nonstructural Proteins - metabolism | Replicon - genetics | DNA replication | Gene mutations | Cyclophilin | Dosage and administration | Genetic aspects | Research | Cyclosporine | Properties | Hepatitis C virus | Identification and classification | Index Medicus | Proteins | Hepatitis | Cell culture | Dependence | Clinical trials | Viruses | Genomes | Mutation | Experiments | Viral infections | Binding sites
Journal Article
Oncogene, ISSN 0950-9232, 08/2009, Volume 28, Issue 31, pp. 2784 - 2795
Signal transducer and activator of transcription 3 (Stat3) is the major mediator of interleukin-6 (IL-6) family cytokines. In addition, Stat3 is known to be... 
Cyclosporine A | Stat3 | Cyclophilin B | Cyclophilin A | Multiple myeloma | Interleukin-6 | RHEUMATOID-ARTHRITIS | TRANSDUCER GP130 | multiple myeloma | cyclosporine A | cyclophilin B | cyclophilin A | CONSTITUTIVE ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION | interleukin-6 | MASS-SPECTROMETRY | CELL BIOLOGY | ONCOLOGY | IN-VIVO | GENETICS & HEREDITY | PROTEIN EXPRESSION PROFILES | UP-REGULATION | BINDING | RNA, Small Interfering - genetics | Luciferases - metabolism | Humans | Transcriptional Activation - drug effects | Immunoblotting | Cyclophilins - metabolism | Recombinant Fusion Proteins - metabolism | Luciferases - genetics | Flow Cytometry | Transfection | Chromatin Immunoprecipitation | Cyclophilin A - genetics | Phosphorylation - drug effects | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Cyclophilin A - metabolism | Cell Line | Signal Transduction | Cell Survival | Cyclophilins - genetics | Multiple Myeloma - metabolism | Multiple Myeloma - pathology | Cell Cycle | Interleukin-6 - pharmacology | Cell Line, Tumor | Protein Binding | Recombinant Fusion Proteins - genetics | Luminescent Proteins - genetics | Microscopy, Fluorescence | Multiple Myeloma - genetics | Apoptosis | Luminescent Proteins - metabolism | Physiological aspects | Genetic aspects | Research | Health aspects | STAT proteins | Signal transduction | Genetics | Cytokines | Index Medicus
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2017, Volume 140, Issue 5, pp. 1299 - 1309
Background Atopic eczema (AE) is characterized by skin barrier and immune dysfunction. Null mutations in filaggrin (FLG), a key epidermal barrier protein,... 
Allergy and Immunology | dermatitis | cyclophilin A | skin | proteomic | kallikrein-7 | filaggrin | Atopic eczema | BARRIER | STRATUM-CORNEUM | IMMUNOLOGY | GENE | ALLERGY | STAPHYLOCOCCUS-AUREUS | MUTATIONS | PROTEASE ACTIVITY | EXPRESSION | CYCLOPHILIN | CYCLOSPORINE | Cyclophilin A - metabolism | RNA, Small Interfering - genetics | Dermatitis, Atopic - genetics | Humans | Cells, Cultured | Gene Expression Regulation | Kallikreins - genetics | Transcriptome | Proteome | Inflammation - immunology | Cofilin 1 - genetics | Keratinocytes - pathology | Cofilin 1 - metabolism | Keratinocytes - metabolism | Proteolysis | Mass Spectrometry | Dermatitis, Atopic - immunology | Intermediate Filament Proteins - genetics | Chromatography, Liquid | Cytoskeleton - metabolism | Inflammation - genetics | Cyclophilin A - genetics | Kallikreins - metabolism | Loss of Function Mutation - genetics | Proteins | RNA | Eczema | Kallikrein | Skin | Liquid chromatography | Mass spectrometry | Transcription | Pathogenesis | Cytology | Filaggrin | Kinases | Dermatitis | Western blotting | Atopy | Protein folding | Consent | Cofilin | Skin diseases | Mass spectroscopy | Inflammation | Gene expression | Ribonucleic acid--RNA | Patients | Polymerase chain reaction | Studies | Biopsy | Cytoskeleton | Mutation | Immunofluorescence | Health risk assessment | Binding sites | Index Medicus | Abridged Index Medicus | LSE, Living skin equivalent | shFLG, Filaggrin knockdown short hairpin RNA | shNT, Nontargeting control short hairpin RNA | ANXA3, Annexin A3 | CFL1, Cofilin-1 | eIF, Eukaryotic initiation factor | IPA, Ingenuity Pathway Analysis | TXN, Thioredoxin | FLG, Filaggrin | KLK7, Kallikrein-7 | PPIA, Cyclophilin A | AE, Atopic eczema | RT-qPCR, Quantitative RT-PCR | mTOR, Mammalian target of rapamycin | VIM, Vimentin | CTSV, Cathepsin V
Journal Article
11.