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Biological Psychiatry, ISSN 0006-3223, 2010, Volume 67, Issue 9, pp. 831 - 839
Journal Article
Cardiovascular Research, ISSN 0008-6363, 7/2009, Volume 83, Issue 2, pp. 213 - 225
Journal Article
Journal Article
Cardiovascular Research, ISSN 0008-6363, 2002, Volume 55, Issue 3, pp. 534 - 543
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 08/2015, Volume 464, Issue 1, pp. 286 - 291
In the current study, we studied the potential role of ABT-737, a novel Bcl-2 inhibitor, on curcumin-induced anti-melanoma cell activity . The associated... 
ABT-737 | Curcumin | Cell death and chemo-sensitization | Mitochondrial permeability transition pore (mPTP) | Melanoma | APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | REPERFUSION INJURY | MALIGNANT-MELANOMA | CANCER | JNK ACTIVATION | MITOCHONDRIAL PERMEABILITY TRANSITION | CYCLOPHILIN-D | BIOPHYSICS | EPIDERMAL-GROWTH-FACTOR | PORE | ADENINE-NUCLEOTIDE TRANSLOCASE | RNA, Small Interfering - genetics | Cyclosporine - pharmacology | Humans | Gene Expression Regulation, Neoplastic | Melanoma, Experimental - metabolism | Cyclophilins - metabolism | Membrane Potential, Mitochondrial - drug effects | Lactones - pharmacology | Mitochondrial Membrane Transport Proteins - genetics | Cyclophilins - antagonists & inhibitors | Proto-Oncogene Proteins c-bcl-2 - metabolism | Biphenyl Compounds - pharmacology | Nitrophenols - pharmacology | Adenine Nucleotide Translocator 1 - antagonists & inhibitors | Antineoplastic Agents - pharmacology | Cell Death - drug effects | Adenine Nucleotide Translocator 1 - genetics | Mitochondrial Membrane Transport Proteins - metabolism | Signal Transduction | Curcumin - pharmacology | Melanoma, Experimental - pathology | Adenine Nucleotide Translocator 1 - metabolism | Cyclophilins - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Sulfonamides - pharmacology | Piperazines - pharmacology | Drug Synergism | Animals | Melanoma, Experimental - genetics | Mitochondrial Membrane Transport Proteins - agonists | Cell Line, Tumor | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Mice | Proto-Oncogene Proteins c-bcl-2 - genetics | Spiro Compounds - pharmacology | RNA, Small Interfering - metabolism | Purines | Cyclosporine | Permeability | Ophthalmology
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 11/2000, Volume 20, Issue 22, pp. 8229 - 8237
Activation of the mitochondrial permeability transition may contribute to excitotoxic neuronal death (Ankarcrona et al., 1996; Dubinsky and Levi, 1998).... 
Mitochondria | Permeability transition | Cyclosporin A | Cyclophilin | Neurodegeneration | Excitotoxicity | GLUTAMATE NEUROTOXICITY | INNER MEMBRANE | permeability transition | mitochondria | cyclosporin A | neurodegeneration | cyclophilin | RAT-LIVER MITOCHONDRIA | CA2+ UNIPORTER | CA-2+ RELEASE | NEUROSCIENCES | CELL-DEATH | CA-2+-INDUCED MEMBRANE TRANSITION | CORTICAL-NEURONS | excitotoxicity | HEART-MITOCHONDRIA | ADENINE-NUCLEOTIDE TRANSLOCASE | Palmitic Acid - pharmacology | Mitochondrial Swelling - drug effects | Calcium - metabolism | Cyclosporine - pharmacology | Brain - metabolism | Cyclophilins - pharmacology | Onium Compounds - pharmacokinetics | Oligomycins - pharmacology | Polyethylene Glycols - pharmacology | Membrane Potentials - drug effects | Serum Albumin, Bovine - pharmacology | Permeability - drug effects | Calcium - pharmacology | Glutamic Acid - pharmacology | Enzyme Inhibitors - pharmacology | Rats | Magnesium - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Magnesium - pharmacology | Adenosine Diphosphate - pharmacology | Animals | Brain Chemistry | Carbonyl Cyanide p-Trifluoromethoxyphenylhydrazone - pharmacology | Glutamic Acid - metabolism | Intracellular Membranes - drug effects | Adenosine Diphosphate - metabolism | Organophosphorus Compounds - pharmacokinetics | Intracellular Membranes - metabolism | ARTICLE
Journal Article
Nature, ISSN 0028-0836, 12/2010, Volume 468, Issue 7324, pp. 696 - 700
Parkinson's disease is a pervasive, ageing-related neurodegenerative disease the cardinal motor symptoms of which reflect the loss of a small group of neurons,... 
PATHOGENESIS | CELLS | SUBSTANTIA-NIGRA NEURONS | MULTIDISCIPLINARY SCIENCES | DYSFUNCTION | MUTATIONS | MITOCHONDRIAL-DNA DELETIONS | CALCIUM-CHANNEL BLOCKERS | PARKINSONS-DISEASE | Oncogene Proteins - genetics | Oxidative Stress | Calcium - metabolism | Iridoid Glycosides - pharmacology | Male | Neurons - cytology | Cyclophilins - metabolism | Calcium Channels, L-Type - pharmacology | Brain - metabolism | Iridoids | Mitochondrial Proteins - metabolism | Gene Deletion | Superoxides - metabolism | Neurons - metabolism | Parkinson Disease - metabolism | Calcium Signaling | Dopamine - metabolism | Brain - cytology | Parkinson Disease - pathology | Purines - pharmacology | Mitochondrial Proteins - antagonists & inhibitors | Oncogene Proteins - metabolism | Mice, Transgenic | Mitochondria - metabolism | Calcium Channel Blockers - pharmacology | Ion Channels - antagonists & inhibitors | Peroxiredoxins | Protein Deglycase DJ-1 | Parkinson Disease - prevention & control | Animals | Biological Clocks - physiology | Ion Channels - metabolism | Dihydropyridines - pharmacology | Oncogene Proteins - deficiency | Mice | Calcium Channels, L-Type - metabolism | Uncoupling Protein 1 | Development and progression | Calcium channels | Parkinson's disease | Dopaminergic mechanisms | Health aspects | Green fluorescent protein | Proteins | Neurotransmitters | Neurons | Parkinsons disease
Journal Article
BBA - Bioenergetics, ISSN 0005-2728, 08/2016, Volume 1857, Issue 8, pp. 1197 - 1202
Journal Article
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, ISSN 0363-6135, 04/2015, Volume 308, Issue 7, pp. H749 - H758
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 146, Issue 7, pp. 1775 - 1783
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2010, Volume 49, Issue 12, pp. 1925 - 1936
Low levels of reactive oxygen species (ROS) can function as redox-active signaling messengers, whereas high levels of ROS induce cellular damage. Menadione... 
Reactive oxygen species | Mitochondria | RoGFP | Free radicals | Redox cycling agents | Apoptosis | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | STRESS-INDUCED APOPTOSIS | INJURY | FLUORESCENT PROTEIN INDICATORS | DEFICIENT MICE | CYTOCHROME-C | MITOCHONDRIAL PERMEABILITY TRANSITION | CYCLOPHILIN-D | REDOX GENE-THERAPY | ENDOCRINOLOGY & METABOLISM | ISCHEMIA-REPERFUSION | Fibroblasts - enzymology | Oxidants - pharmacology | Reactive Oxygen Species - metabolism | Oxidative Stress | Apoptosis - drug effects | Cyclophilins - metabolism | Myocytes, Cardiac - enzymology | Glutathione - pharmacology | Cell Death - drug effects | Fibroblasts - metabolism | Recombinant Proteins - metabolism | Green Fluorescent Proteins - metabolism | Signal Transduction | Cytochromes c - metabolism | Mice, Inbred C57BL | Cells, Cultured | Mitochondria - metabolism | Antioxidants - pharmacology | Chick Embryo | Mice, Knockout | Vitamin K 3 - pharmacology | Poly(ADP-ribose) Polymerases - metabolism | Animals | Myocytes, Cardiac - drug effects | Fibroblasts - drug effects | Acetylcysteine - pharmacology | Myocytes, Cardiac - metabolism | Mice | Enzyme Activation | Poly (ADP-Ribose) Polymerase-1 | Mitochondria - physiology | bcl-X Protein - metabolism | Staurosporine - pharmacology | Cytochrome c | Analysis | Genetically modified organisms | Biochemistry | Mitochondrial DNA | Superoxide | Permeability | apoptosis | redox cycling agents | mitochondria
Journal Article