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Journal Article
HUMAN MOLECULAR GENETICS, ISSN 0964-6906, 02/2014, Volume 23, Issue 4, pp. 889 - 905
Primary aldosteronism (PA) is the main cause of secondary hypertension, resulting from adrenal aldosterone-producing adenomas (APA) or bilateral hyperplasia.... 
CANCER-CELLS | FREQUENT EPIGENETIC INACTIVATION | ADRENAL-CORTEX | ZONA GLOMERULOSA | COLORECTAL-CANCER | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | KCNJ5 MUTATIONS | BETA-CATENIN | ADRENOCORTICAL TUMORS | SOMATIC MUTATIONS | Adrenal Cortex Neoplasms - complications | Aldosterone - biosynthesis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Hyperaldosteronism - metabolism | Mice, 129 Strain | Nuclear Receptor Subfamily 4, Group A, Member 2 - genetics | Hyperaldosteronism - etiology | Adult | Female | Membrane Proteins - metabolism | Adrenal Cortex Neoplasms - metabolism | Cytochrome P-450 CYP11B2 - metabolism | Wnt Signaling Pathway | Nuclear Receptor Subfamily 4, Group A, Member 1 - genetics | Membrane Proteins - genetics | Down-Regulation | Mice, Inbred C57BL | Aldosterone - blood | Aldosterone - secretion | Adrenocortical Adenoma - metabolism | Mice, Knockout | Animals | Cell Line, Tumor | Nuclear Receptor Subfamily 4, Group A, Member 1 - metabolism | Mice | Nuclear Receptor Subfamily 4, Group A, Member 2 - metabolism | Adrenocortical Adenoma - complications | Cytochrome P-450 CYP11B2 - genetics | Index Medicus | Animal genetics | Cytochrome P-450 CYP11B2 | Hyperaldosteronism | Genomics | Nuclear Receptor Subfamily 4, Group A, Member 1 | Nuclear Receptor Subfamily 4, Group A, Member 2 | Aldosterone | Embryology and Organogenesis | Life Sciences | Genetics | Endocrinology and metabolism | Biochemistry, Molecular Biology | Adrenocortical Adenoma | Membrane Proteins | Human health and pathology | Adrenal Cortex Neoplasms | Development Biology | Molecular biology | Cancer
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 02/2016, Volume 422, Issue C, pp. 57 - 63
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, pp. e0181055 - e0181055
The effects of retinoids on adrenal aldosterone synthase gene (CYP11B2) expression and aldosterone secretion are still unknown. We therefore examined the... 
SYNTHASE | ACUTE REGULATORY PROTEIN | ACTIVATED RECEPTOR-GAMMA | 3-BETA-HYDROXYSTEROID DEHYDROGENASE | ZONA GLOMERULOSA | MULTIDISCIPLINARY SCIENCES | ANGIOTENSIN-II | HYPERTENSION | DIFFERENTIAL REGULATION | CANCER | ADRENOCORTICAL H295R CELLS | Apoptosis - drug effects | Calcium - metabolism | Humans | Adrenal Cortex - metabolism | Hydrocortisone - secretion | Body Weight - drug effects | RNA, Messenger - metabolism | Heart Rate - drug effects | Point Mutation - genetics | Blood Pressure - drug effects | Adrenal Cortex - drug effects | Retinoid X Receptors - antagonists & inhibitors | Thiazolidinediones - pharmacology | Retinoid X Receptors - metabolism | Steroids - biosynthesis | Cytochrome P-450 CYP11B2 - metabolism | Promoter Regions, Genetic | 2-Naphthylamine - pharmacology | RNA, Messenger - genetics | Aldosterone - secretion | Ions | Mice, Transgenic | Pyrimidines - pharmacology | 2-Naphthylamine - analogs & derivatives | Animals | Cell Line, Tumor | Cell Proliferation - drug effects | Nuclear Receptor Subfamily 4, Group A, Member 2 - metabolism | Cytochrome P-450 CYP11B2 - genetics | Sequence Deletion - genetics | Hypertension | Care and treatment | Antihypertensive drugs | Dosage and administration | Blood pressure | Research | Aldosterone | Health aspects | Drugs | Pediatrics | Transcription factors | Dehydrogenases | Adrenal glands | Genes | Biosynthesis | Retinoids | Gene deletion | Drug development | Kinases | Blood | Proteins | Clonal deletion | Rodents | Deletion | Angiotensin II | University graduates | Secretion | Pioglitazone | Aldosterone synthase | Retinoid X receptors | Gene expression | Nuclear receptors | Mutants | Medicine | Nurr1 protein | Angiotensin | Peroxisome proliferator-activated receptors | Potassium | Endocrinology | Apoptosis | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 10/2016, Volume 163, pp. 68 - 76
Spironolactone and its major metabolite canrenone are potent mineralocorticoid receptor antagonists and are, therefore, applied as drugs for the treatment of... 
Hypertension | Xenobiotic metabolism | Aldosterone | Human CYP11B1/CYP11B2 | Mineralocorticoid receptor antagonists | MYOCARDIAL-INFARCTION | ADRENAL-CORTEX | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | HEART-FAILURE | LIVER MICROSOMES | MOLECULAR-LEVEL | SELECTIVE ALDOSTERONE BLOCKER | ADRENODOXIN REDUCTASE | CYTOCHROME-P450 ENZYMES | ENDOCRINOLOGY & METABOLISM | ELECTRON-TRANSPORT | Recombinant Proteins - metabolism | Receptors, Mineralocorticoid - genetics | Canrenone - pharmacology | Receptors, Mineralocorticoid - metabolism | Humans | Transcriptional Activation - drug effects | Recombinant Proteins - genetics | Spironolactone - metabolism | Steroid 11-beta-Hydroxylase - genetics | Mineralocorticoid Receptor Antagonists - metabolism | Cortodoxone - metabolism | Spironolactone - pharmacology | Steroid 11-beta-Hydroxylase - metabolism | Escherichia coli - genetics | Biotransformation | Canrenone - metabolism | Cloning, Molecular | Desoxycorticosterone - metabolism | Escherichia coli - metabolism | Mineralocorticoid Receptor Antagonists - pharmacology | Kinetics | Spironolactone - analogs & derivatives | Cytochrome P-450 CYP11B2 - metabolism | Cytochrome P-450 CYP11B2 - genetics | Physiological aspects | Metabolites | Spironolactone | Cytochrome P-450 | Steroids | Nuclear magnetic resonance spectroscopy | Xenobiotics | Index Medicus
Journal Article
Hypertension, ISSN 0194-911X, 01/2014, Volume 63, Issue 1, pp. 188 - 195
Journal Article