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Cell Metabolism, ISSN 1550-4131, 07/2011, Volume 14, Issue 1, pp. 80 - 90
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 04/2014, Volume 23, Issue 8, pp. 2106 - 2119
Journal Article
Orphanet Journal of Rare Diseases, ISSN 1750-1172, 2012, Volume 7, Issue 1, pp. 21 - 21
Journal Article
Brain Research, ISSN 0006-8993, 2001, Volume 922, Issue 2, pp. 282 - 292
The neuroprotective effect of Ginkgo biloba extract (EGb 761) against ischemic injury has been demonstrated in animal models. In this study, we compared the... 
Selective vulnerability | Cytochrome oxidase | Histology | Mitochondrial DNA | In situ hybridization | Hippocampus | hippocampus | PROTECTION | mitochondrial DNA | INJURY | selective vulnerability | NEUROSCIENCES | DAMAGE | DELAYED NEURONAL DEATH | histology | TRANSIENT FOREBRAIN ISCHEMIA | cytochrome oxidase | EGB 761 | RAT-BRAIN | CEREBRAL-ISCHEMIA | in situ hybridization | CONSTITUENTS | Cytochrome-c Oxidase Deficiency - physiopathology | Gene Expression Regulation, Enzymologic - drug effects | DNA, Mitochondrial - drug effects | Plant Extracts - pharmacology | Cell Count | Furans - pharmacology | Nerve Degeneration - physiopathology | Reperfusion Injury - drug therapy | Brain Ischemia - metabolism | Male | Hippocampus - drug effects | RNA, Messenger - metabolism | Electron Transport Complex IV - metabolism | Brain - metabolism | Oxidative Phosphorylation - drug effects | DNA, Mitochondrial - genetics | Neuroprotective Agents - pharmacology | Cell Death - genetics | Nerve Degeneration - prevention & control | Neurons - metabolism | Cell Death - drug effects | Energy Metabolism - genetics | Neurons - drug effects | Reperfusion Injury - metabolism | RNA, Messenger - drug effects | DNA, Mitochondrial - metabolism | Brain - physiopathology | Cytochrome-c Oxidase Deficiency - drug therapy | Brain Ischemia - physiopathology | Ginkgo biloba - chemistry | Cyclopentanes - pharmacology | Electron Transport Complex IV - genetics | Ginkgolides | Brain - drug effects | Hippocampus - metabolism | Animals | Gerbillinae | Gene Expression Regulation, Enzymologic - genetics | Brain Ischemia - drug therapy | Electron Transport Complex IV - drug effects | Reperfusion Injury - physiopathology | Diterpenes | Cytochrome-c Oxidase Deficiency - genetics | Hippocampus - physiopathology | Energy Metabolism - drug effects | Nerve Degeneration - drug therapy
Journal Article
Journal Article
Journal Article
EMBO Molecular Medicine, ISSN 1757-4676, 04/2009, Volume 1, Issue 1, pp. 30 - 36
Cytochrome c oxidase (COX) deficiency is associated with a wide spectrum of clinical conditions, ranging from early onset devastating encephalomyopathy and... 
cytochrome c oxidase | superoxides | alternative oxidase | human skin fibroblasts | Cytochrome c oxidase | Alternative oxidase | Human skin fibroblasts | Superoxides | MEDICINE, RESEARCH & EXPERIMENTAL | TRANSDUCTION | PATHOLOGY | LENTIVIRAL VECTORS | B-CELLS | PATHWAY | DISEASE | GENE-THERAPY | MUTATIONS | PLANT-MITOCHONDRIA | ELECTRON-TRANSPORT | Fibroblasts - enzymology | Electron Transport - drug effects | Humans | Alkyl and Aryl Transferases - metabolism | Electron Transport Complex IV | Mitochondrial Proteins | Genetic Complementation Test | Antimycin A - pharmacology | Gene Knockdown Techniques | Membrane Proteins - deficiency | Membrane Proteins - metabolism | Cytochrome-c Oxidase Deficiency - enzymology | Alkyl and Aryl Transferases - deficiency | Skin - pathology | Cell Survival - drug effects | Cytochrome-c Oxidase Deficiency - pathology | Oxidoreductases - metabolism | Antimycin A - analogs & derivatives | Hydrogen Peroxide - pharmacology | Mutation - genetics | Ciona intestinalis - enzymology | Animals | Fibroblasts - drug effects | Plant Proteins | Oxidative Stress - drug effects | Cell Line, Transformed | Cytochrome | Cyanide | Oxidative stress | Disease | Cardiomyopathy | Genes | Cytochrome-c oxidase | Pyruvic acid | Mammals | Metabolism | Defects | Neurological diseases | Mitochondria | COX10 protein | Fibroblasts | Uridine | Oxidation | Mutation | Respiration | Geriatrics | Report
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 08/2018, Volume 115, Issue 32, pp. 8161 - 8166
Copper is an essential cofactor of cytochrome c oxidase (CcO), the terminal enzyme of the mitochondrial respiratory chain. Inherited loss-of-function mutations... 
elesclomol | cytochrome c oxidase | Copper | mitochondria | MATRIX | MULTIDISCIPLINARY SCIENCES | BINDING PROTEIN | CYTOCHROME-C-OXIDASE | HYPERTROPHIC CARDIOMYOPATHY | DRUG ELESCLOMOL | BIOGENESIS | SCO2 | DISEASE | copper | MUTATIONS | COA6 | Drugs, Investigational - pharmacology | Humans | Drugs, Investigational - therapeutic use | Antineoplastic Agents - therapeutic use | Mitochondrial Proteins - genetics | Electron Transport Complex IV - metabolism | Membrane Transport Proteins - genetics | Coenzymes - deficiency | Fibroblasts | Antineoplastic Agents - pharmacology | Biological Transport - genetics | Copper - deficiency | Disease Models, Animal | Cell Line | Hydrazines - pharmacology | Hydrazines - therapeutic use | Mutagenesis, Site-Directed | Metabolism, Inborn Errors - metabolism | Rats | Zebrafish | Mitochondria - metabolism | Drug Repositioning | Mitochondria - drug effects | Metabolism, Inborn Errors - genetics | Carrier Proteins - genetics | Animals | Copper - therapeutic use | Metabolism, Inborn Errors - drug therapy | Mutation | Zebrafish Proteins - genetics | Dietary Supplements | Saccharomyces cerevisiae | Antimitotic agents | Copper in the body | Mitochondria | Physiological aspects | Genetic aspects | Minerals in nutrition | Models | Dosage and administration | Drug therapy | Antineoplastic agents | Biological Sciences
Journal Article
Nature Medicine, ISSN 1078-8956, 10/2008, Volume 14, Issue 10, pp. 1097 - 1105
Journal Article