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1. Cytokines, cellular & molecular therapy
Cytokines, cellular & molecular therapy, ISSN 1368-4736, 1997
Periodicals | Cytokines | Genetic regulation | Immunotherapy
Journal
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2. Full Text Interleukin-1 Beta as a Target for Atherosclerosis Therapy: Biological Basis of CANTOS and Beyond
by Libby, Peter
Journal of the American College of Cardiology, ISSN 0735-1097, 10/2017, Volume 70, Issue 18, pp. 2278 - 2289
Inflammatory pathways drive atherogenesis and link conventional risk factors to atherosclerosis and its complications. One inflammatory mediator has come to... 
high-sensitivity C-reactive protein | interleukin-1 | acute coronary syndromes | myocardial infarction | C-REACTIVE PROTEIN | ELEMENT-BINDING PROTEIN-2 | CARDIAC & CARDIOVASCULAR SYSTEMS | VASCULAR SMOOTH-MUSCLE | RECEPTOR ANTAGONIST | ENDOTHELIAL-CELLS | ACUTE MYOCARDIAL-INFARCTION | CARDIOVASCULAR-DISEASE | GENE-EXPRESSION | BLOCKING INTERLEUKIN-1 | Atherosclerosis - blood | Animals | Atherosclerosis - drug therapy | Humans | Anti-Inflammatory Agents - administration & dosage | Biomarkers - blood | Drug Delivery Systems - trends | Anti-Inflammatory Agents - metabolism | Drug Delivery Systems - methods | Interleukin-1beta - antagonists & inhibitors | Interleukin-1beta - blood | Medical colleges | Interleukins | Cardiac patients | Therapeutics | Atherosclerosis | Health aspects | Coronary heart disease | Cardiovascular agents | Homeopathy | Materia medica and therapeutics | Therapy | Atherogenesis | Heart attacks | Pathogenesis | Interleukin | Smooth muscle | Leukocytes | Inflammatory diseases | Risk factors | Cell adhesion & migration | Proteins | Interleukin 1 | Tumor necrosis factor-TNF | Enzymes | Cytokines | Complications | Inflammation | Risk analysis | Gene expression | Arteriosclerosis | Biomarkers | Mutation | Cardiovascular diseases | Cancer
Journal Article
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3. Biologic therapy of leukemia
2003, ISBN 9781588290717, xii, 269
Book
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4. Full Text Heart Failure Therapy–Induced Early ST2 Changes May Offer Long-Term Therapy Guidance
by Breidthardt, Tobias, MD and Balmelli, Cathrin, MD and Twerenbold, Raphael, MD and Mosimann, Tamina, MD and Espinola, Jaqueline, MD and Haaf, Philip, MD and Thalmann, Gregor, BSc and Moehring, Berit, MD and Mueller, Mira, MD and Meller, Bernadette, MD and Reichlin, Tobias, MD and Murray, Karsten, MD and Ziller, Ronny, MD and Benkert, Pascal, PhD and Osswald, Stefan, MD and Mueller, Christian, MD
Journal of Cardiac Failure, ISSN 1071-9164, 2013, Volume 19, Issue 12, pp. 821 - 828
Abstract Background Biomarkers may help to monitor and tailor treatment in patients with acute heart failure (AHF). Methods and Results Levels of ST2, a novel... 
Cardiovascular | ST2 | mortality | heart failure therapy | Acute heart failure | Heart failure therapy | Mortality | SURVIVAL | CARDIAC & CARDIOVASCULAR SYSTEMS | CYTOKINE | SOLUBLE ST2 | NATRIURETIC PEPTIDE | FAMILY-MEMBER ST2 | DIURETIC USE | IMPACT | INTERLEUKIN-1 RECEPTOR | OUTCOMES | Interleukin-1 Receptor-Like 1 Protein | Predictive Value of Tests | Follow-Up Studies | Practice Guidelines as Topic - standards | Humans | Male | Receptors, Cell Surface - blood | Biomarkers - blood | Heart Failure - blood | Heart Failure - therapy | Time Factors | Aged, 80 and over | Female | Aged | Heart Failure - diagnosis | Early Diagnosis | Cohort Studies | Heart failure | Heart | Medical colleges | Cardiac patients | Cardiovascular agents
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5. Cytokine therapy
by Cawley, J. C and Galvani, David W
1992, ISBN 0521423376, xii, 193
Cytokines | Therapeutic use
Book
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6. Full Text Chimeric antigen receptor T-cell therapy-assessment and management of toxicities
by Neelapu, Sattva S and Tummala, Sudhakar and Kebriaei, Partow and Wierda, William and Gutierrez, Cristina and Locke, Frederick L and Komanduri, Krishna V and Lin, Yi and Jain, Nitin and Daver, Naval and Westin, Jason and Gulbis, Alison M and Loghin, Monica E and De Groot, John F and Adkins, Sherry and Davis, Suzanne E and Rezvani, Katayoun and Hwu, Patrick and Shpall, Elizabeth J
Nature Reviews Clinical Oncology, ISSN 1759-4774, 01/2018, Volume 15, Issue 1, pp. 47 - 62
Immunotherapy using T cells genetically engineered to express a chimeric antigen receptor (CAR) is rapidly emerging as a promising new treatment for... 
C-REACTIVE PROTEIN | SIGNAL-TRANSDUCTION | ADULT PATIENTS | ADVERSE EVENT | SYNDROME CRS | B-CELL | ONCOLOGY | CYTOKINE RELEASE SYNDROME | SOLUBLE IL-6 | NONCONVULSIVE STATUS EPILEPTICUS | HEMOPHAGOCYTIC LYMPHOHISTIOCYTOSIS | Immunotherapy, Adoptive - adverse effects | Cytokines - metabolism | Brain Diseases - therapy | Humans | Receptors, Antigen, T-Cell - therapeutic use | Adult | Female | Receptors, Antigen, T-Cell - immunology | Brain Diseases - etiology | Syndrome | Complications and side effects | Cell receptors | Care and treatment | Oncology, Experimental | Cellular therapy | Research | Health aspects | Cancer | Antigens | Neurotoxicity | Hematology | Toxicity | Immunotherapy | Encephalopathy | T cell receptors | Genetic engineering | Lymphocytes T | Management | Morbidity | Index Medicus
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7. Full Text Pulmonary macrophage transplantation therapy
by Suzuki, Takuji and Suzuki, Takuji and Arumugam, Paritha and Arumugam, Paritha and Sakagami, Takuro and Sakagami, Takuro and Lachmann, Nico and Lachmann, Nico and Chalk, Claudia and Chalk, Claudia and Sallese, Anthony and Sallese, Anthony and Abe, Shuichi and Abe, Shuichi and Trapnell, Cole and Trapnell, Cole and Carey, Brenna and Carey, Brenna and Moritz, Thomas and Moritz, Thomas and Malik, Punam and Malik, Punam and Lutzko, Carolyn and Lutzko, Carolyn and Wood, Robert E and Wood, Robert E and Trapnell, Bruce C and Trapnell, Bruce C
Nature, ISSN 0028-0836, 10/2014, Volume 514, Issue 7253, pp. 450 - 454
Bone-marrow transplantation is an effective cell therapy but requires myeloablation, which increases infection risk and mortality. Recent lineage-tracing... 
RECEPTOR-DEFICIENT MICE | LUNG | STEM-CELLS | ALVEOLAR PROTEINOSIS | COLONY-STIMULATING FACTOR | GM-CSF | BONE-MARROW-TRANSPLANTATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | TISSUE MACROPHAGES | GENE-THERAPY | Proteins | Studies | Biomarkers | Histopathology | Surfactants | M-CSF | Alveolar Macrophage | Myeloid Cell Disorder | Gene Therapy | Genetic Disease | Surfactant Homeostasis
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8. Full Text Prognosis of metastatic renal cell carcinoma with first‐line interferon‐α therapy in the era of molecular‐targeted therapy
by Kawano, Yoshiaki and Takahashi, Wataru and Eto, Masatoshi and Kamba, Tomomi and Miyake, Hideaki and Fujisawa, Masato and Kamai, Takao and Uemura, Hirotsugu and Tsukamoto, Taiji and Azuma, Haruhito and Matsubara, Akio and Nishimura, Kazuo and Nakamura, Tsuyoshi and Ogawa, Osamu and Naito, Seiji
Cancer Science, ISSN 1347-9032, 07/2016, Volume 107, Issue 7, pp. 1013 - 1017
The RCC‐SELECT study showed the correlation between single nucleotide polymorphisms (SNP) in STAT3 gene and survival in metastatic renal cell carcinoma (mRCC)... 
overall survival | renal cell carcinoma | prognostic factors | interferon‐α | The Era of molecular targeted therapy | Interferon-α | Overall survival | Renal cell carcinoma | Prognostic factors | interferon-α | SURVIVAL | STAT3 POLYMORPHISM | FOLLOW-UP | SORAFENIB | PHASE-III TRIAL | RADICAL NEPHRECTOMY | HYPERCALCEMIA | INVASION | ONCOLOGY | SUNITINIB | interferon-alpha | JAPANESE PATIENTS | Kidney Neoplasms - genetics | Prognosis | Humans | Middle Aged | Carcinoma, Renal Cell - genetics | Male | Antineoplastic Agents - therapeutic use | Antineoplastic Agents - administration & dosage | Molecular Targeted Therapy | Case-Control Studies | Neoplasm Metastasis | Aged, 80 and over | Adult | Female | Carcinoma, Renal Cell - drug therapy | STAT3 Transcription Factor - genetics | Carcinoma, Renal Cell - pathology | Interferon-alpha - therapeutic use | Survival Rate | Treatment Outcome | Interferon-alpha - administration & dosage | Polymorphism, Single Nucleotide - genetics | Kidney Neoplasms - pathology | Aged | Kidney Neoplasms - drug therapy | Cohort Studies | Thrombocytopenia | Demography | Cell survival | Disease | Cytokines | Research funding | Stat3 protein | Histology | Single-nucleotide polymorphism | Metastasis | Multivariate analysis | Patients | Lymph nodes | Metastases | Hypercalcemia | Studies | Medical prognosis | Kidney cancer | Growth patterns | Interferon | Clear cell-type renal cell carcinoma | Original
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9. Full Text Immune therapy for cancer
by Dougan, Michael and Dranoff, Glenn
Annual Review of Immunology, ISSN 0732-0582, 2009, Volume 27, Issue 1, pp. 83 - 117
Over the past decade, immune therapy has become a standard treatment for a variety of cancers. Monoclonal antibodies, immune adjuvants, and vaccines against... 
Adjuvants | Monoclonal (antibody) | Inflammation | Vaccine | Immunotherapy | BACILLUS-CALMETTE-GUERIN | COLONY-STIMULATING FACTOR | IMMUNOLOGY | PHASE-III TRIAL | GROWTH-FACTOR RECEPTOR | vaccine | TUMOR-NECROSIS-FACTOR | inflammation | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | HELICOBACTER-PYLORI ERADICATION | immunotherapy | METASTATIC BREAST-CANCER | REGULATORY T-CELLS | monoclonal (antibody) | RECOMBINANT INTERLEUKIN-2 THERAPY | adjuvants | Immunotherapy, Adoptive - methods | Cytokines - metabolism | Adjuvants, Immunologic | Antigens, Neoplasm - immunology | Humans | T-Lymphocytes, Regulatory - immunology | Cancer Vaccines - immunology | Cancer Vaccines - therapeutic use | Neoplasms - therapy | Animals | Neoplasms - immunology | Bone Marrow Transplantation | Immunotherapy, Active - methods | Antibodies, Monoclonal - immunology | Cytokines - immunology | Care and treatment | Physiological aspects | Research | Methods | Cancer | Immunological research
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10. Full Text Immunological Responses Triggered by Photothermal Therapy with Carbon Nanotubes in Combination with Anti‐CTLA‐4 Therapy to Inhibit Cancer Metastasis
by Wang, Chao and Xu, Ligeng and Liang, Chao and Xiang, Jian and Peng, Rui and Liu, Zhuang
Advanced Materials, ISSN 0935-9648, 12/2014, Volume 26, Issue 48, pp. 8154 - 8162
Photothermal ablation of primary tumors with single‐walled carbon nanotubes is demonstrated to be able to trigger significant adaptive immune responses, which... 
anti‐CTLA‐4 | single‐walled carbon nanotubes | cancer metastasis inhibition | photothermal therapy | immunotherapy | Photothermal therapy | Single-walled carbon nanotubes | Anti-CTLA-4 | Immunotherapy | Cancer metastasis inhibition | NEAR-INFRARED LIGHT | PHYSICS, CONDENSED MATTER | ACTIVATION | PHYSICS, APPLIED | DENDRITIC CELLS | DRUG-DELIVERY | MATERIALS SCIENCE, MULTIDISCIPLINARY | CHEMISTRY, PHYSICAL | NANOSCIENCE & NANOTECHNOLOGY | BIODISTRIBUTION | TUMORS | CHEMISTRY, MULTIDISCIPLINARY | B-CELLS | THERMAL ABLATION | MICE | AGENTS | Antibodies - therapeutic use | Cytokines - metabolism | Lung Neoplasms - mortality | Antibodies - chemistry | Dendritic Cells - immunology | Lung Neoplasms - pathology | Polyethylene Glycols - chemistry | Survival Rate | Lung Neoplasms - therapy | CTLA-4 Antigen - immunology | Phototherapy | Animals | Nanotubes, Carbon - chemistry | Antibodies - immunology | Dendritic Cells - cytology | Mice | Mice, Inbred BALB C | Dendritic Cells - metabolism | Prevention | Care and treatment | Nanotubes | Metastasis | Health aspects | Cancer | Therapy | Carbon nanotubes | Antibodies | Death | Ablation | Single wall carbon nanotubes | Tumors
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11. Full Text Antiinflammatory Therapy with Canakinumab for Atherosclerotic Disease
by Ridker, Paul M and Everett, Brendan M and Thuren, Tom and MacFadyen, Jean G and Chang, William H and Ballantyne, Christie and Fonseca, Francisco and Nicolau, Jose and Koenig, Wolfgang and Anker, Stefan D and Kastelein, John J.P and Cornel, Jan H and Pais, Prem and Pella, Daniel and Genest, Jacques and Cifkova, Renata and Lorenzatti, Alberto and Forster, Tamas and Kobalava, Zhanna and Vida-Simiti, Luminita and Flather, Marcus and Shimokawa, Hiroaki and Ogawa, Hisao and Dellborg, Mikael and Rossi, Paulo R.F and Troquay, Roland P.T and Libby, Peter and Glynn, Robert J and CANTOS Trial Grp and CANTOS Trial Group and Sahlgrenska akademin and Institute of Medicine and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy and Institutionen för medicin
The New England Journal of Medicine, ISSN 0028-4793, 09/2017, Volume 377, Issue 12, pp. 1119 - 1131
Patients with myocardial infarction and a high-sensitivity CRP level of 2 mg or more per liter were assigned to one of three canakinumab doses or placebo. The... 
C-REACTIVE PROTEIN | MEDICINE, GENERAL & INTERNAL | HEART-DISEASE | MYOCARDIAL-INFARCTION | STATIN THERAPY | CLONAL HEMATOPOIESIS | INFLAMMATION | CARDIOVASCULAR-DISEASE | INTERLEUKIN-1-BETA INHIBITION | RISK | CORONARY-ARTERY-DISEASE | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Male | Secondary Prevention | Infection - etiology | Incidence | Dose-Response Relationship, Drug | Anti-Inflammatory Agents - adverse effects | C-Reactive Protein - metabolism | Lipids - blood | Cardiovascular Diseases - epidemiology | Cardiovascular Diseases - mortality | Anti-Inflammatory Agents - administration & dosage | Female | Neutropenia - chemically induced | Interleukin-1beta - antagonists & inhibitors | Stroke - prevention & control | Atherosclerosis - drug therapy | Double-Blind Method | Interleukin-1beta - immunology | Atherosclerosis - blood | Myocardial Infarction - drug therapy | Antibodies, Monoclonal - administration & dosage | Aged | Myocardial Infarction - prevention & control | Myocardial infarction | Cerebral infarction | Stroke | Heart attacks | C-reactive protein | Cytokines | Clinical trials | Cardiovascular disease | Innate immunity | Inflammation | Angina | Angina pectoris | Thrombosis | Cholesterol | Disease prevention | Proteins | Arteriosclerosis | Atherosclerosis | Cardiovascular diseases | Health risk assessment | Drug therapy | Drug dosages | Klinisk medicin | Clinical Medicine
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