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Journal of Clinical Immunology, ISSN 0271-9142, 6/2011, Volume 31, Issue 3, pp. 472 - 478
Leptin, one of the adipokines, functions as a hormone and a cytokine. In this investigation, we show for the first time that leptin, in a... 
Medical Microbiology | Biomedicine | Immunology | Internal Medicine | Infectious Diseases | activation markers | cytokines | B cells | PROTEIN-KINASE | TRANSDUCER | RECEPTOR | PROLIFERATION | IMMUNOLOGY | BLOOD MONONUCLEAR-CELLS | DEFICIENCY | PI3K/AKT | IMMUNE-RESPONSES | T-CELLS | Autoimmunity | Interleukin-6 - analysis | Janus Kinase 2 - biosynthesis | Mitogen-Activated Protein Kinase 3 - analysis | Humans | p38 Mitogen-Activated Protein Kinases - biosynthesis | p38 Mitogen-Activated Protein Kinases - analysis | Dose-Response Relationship, Drug | Signal Transduction - immunology | Inflammation - metabolism | Mitogen-Activated Protein Kinases - analysis | Mitogen-Activated Protein Kinase 3 - biosynthesis | Mitogen-Activated Protein Kinase 6 - analysis | Leptin - pharmacology | Phosphorylation - drug effects | Interleukin-10 - analysis | B-Lymphocytes - metabolism | Janus Kinase 2 - analysis | Enzyme-Linked Immunosorbent Assay | Lymphocyte Activation | Cells, Cultured | Mitogen-Activated Protein Kinases - biosynthesis | Receptors, Leptin - immunology | STAT3 Transcription Factor - analysis | Inflammation - immunology | Tumor Necrosis Factor-alpha - analysis | STAT3 Transcription Factor - biosynthesis | B-Lymphocytes - drug effects | B-Lymphocytes - immunology | Mitogen-Activated Protein Kinase 6 - biosynthesis | Interleukin-6 - biosynthesis | Interleukin-10 - biosynthesis | Receptors, Leptin - metabolism | Tumor Necrosis Factor-alpha - biosynthesis | Index Medicus
Journal Article
Science, ISSN 0036-8075, 5/2009, Volume 324, Issue 5927, pp. 651 - 654
The circadian clock is encoded by a transcription-translation feedback loop that synchronizes behavior and metabolism with the light-dark cycle. Here we report... 
RNA | Hepatocytes | Liver | Genes | Oscillation | Control loops | Reports | Biosynthesis | Gene expression regulation | Metabolism | Research universities | LIFE-SPAN | CELLS | GLUCOSE-HOMEOSTASIS | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | NICOTINAMIDE PHOSPHORIBOSYLTRANSFERASE | EXTENSION | COMPONENT | SIRT1 | BINDING | CALORIE RESTRICTION | Humans | Adipose Tissue, White - metabolism | Hepatocytes - metabolism | Nicotinamide Phosphoribosyltransferase - antagonists & inhibitors | Basic Helix-Loop-Helix Transcription Factors - metabolism | Nicotinamide Phosphoribosyltransferase - metabolism | Piperidines - pharmacology | Sirtuin 1 | NAD - biosynthesis | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Transcription, Genetic | Biological Clocks | Nuclear Proteins - genetics | Acrylamides - pharmacology | Cytokines - genetics | ARNTL Transcription Factors | Cell Line | Basic Helix-Loop-Helix Transcription Factors - genetics | Cytokines - metabolism | Liver - metabolism | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Transcription Factors - genetics | Circadian Rhythm | Period Circadian Proteins | Feedback, Physiological | Nicotinamide Phosphoribosyltransferase - genetics | Animals | CLOCK Proteins | Cell Line, Tumor | Protein Binding | Trans-Activators - metabolism | Mice | Cytokines - antagonists & inhibitors | Sirtuins - metabolism | Enzymes | Circadian rhythm | Cellular biology | Animal behavior | Rodents | Index Medicus
Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 01/2004, Volume 172, Issue 1, pp. 567 - 576
The signaling mechanism by which the anti-inflammatory cytokine IL-10 mediates suppression of proinflammatory cytokine synthesis remains largely unknown.... 
RHEUMATOID-ARTHRITIS | TUMOR-NECROSIS-FACTOR | DNA-BINDING | HUMAN NEUTROPHILS | TYROSINE PHOSPHORYLATION | INTERLEUKIN-10 RECEPTOR | GENE-EXPRESSION | KAPPA-B-ALPHA | IMMUNOLOGY | HUMAN MONOCYTES | MONONUCLEAR PHAGOCYTES | Protein Binding - genetics | Protein Biosynthesis | Interleukin-6 - antagonists & inhibitors | Humans | Tumor Necrosis Factor-alpha - genetics | Immunoglobulins - genetics | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - metabolism | Suppressor of Cytokine Signaling Proteins | Repressor Proteins - antagonists & inhibitors | Antigens, CD - metabolism | Trans-Activators - physiology | Protein Tyrosine Phosphatases - antagonists & inhibitors | RNA, Messenger - biosynthesis | Protein Tyrosine Phosphatases - genetics | Inflammation Mediators - physiology | Glycoproteins - genetics | DNA-Binding Proteins - physiology | Protein Tyrosine Phosphatases - biosynthesis | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction - genetics | DNA - metabolism | Down-Regulation - genetics | Macrophages - metabolism | Protein Tyrosine Phosphatase, Non-Receptor Type 2 | Repressor Proteins - biosynthesis | Up-Regulation - immunology | Interleukin-10 - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Adenoviruses, Human - genetics | Interleukin-10 - immunology | Tumor Necrosis Factor-alpha - biosynthesis | Phosphorylation | Tissue Inhibitor of Metalloproteinase-1 - biosynthesis | Antigens, CD - biosynthesis | Receptors, Cell Surface | Receptors, IgG - biosynthesis | Receptors, IgG - antagonists & inhibitors | Interleukin-10 - physiology | Signal Transduction - immunology | Tissue Inhibitor of Metalloproteinase-1 - metabolism | Signaling Lymphocytic Activation Molecule Family Member 1 | Receptors, Tumor Necrosis Factor - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Receptors, Tumor Necrosis Factor, Type II | Trans-Activators - genetics | Inflammation Mediators - antagonists & inhibitors | Trans-Activators - biosynthesis | Immunoglobulins - biosynthesis | Macrophages - immunology | Inflammation Mediators - immunology | Receptors, Tumor Necrosis Factor - metabolism | Immune Sera - pharmacology | Proteins - physiology | Cells, Cultured | Glycoproteins - antagonists & inhibitors | Histocompatibility Antigens Class II - biosynthesis | Tissue Inhibitor of Metalloproteinase-1 - antagonists & inhibitors | Transcription Factors - antagonists & inhibitors | Transcription Factors - biosynthesis | Up-Regulation - genetics | DNA-Binding Proteins - genetics | DNA - antagonists & inhibitors | Glycoproteins - biosynthesis | Suppressor of Cytokine Signaling 3 Protein | Down-Regulation - immunology | Interleukin-6 - biosynthesis | Receptors, Tumor Necrosis Factor - biosynthesis | STAT3 Transcription Factor | Trans-Activators - antagonists & inhibitors | Genetic Vectors | DNA-Binding Proteins - biosynthesis | Tumor Necrosis Factor-alpha - antagonists & inhibitors | SOCS-3 protein | Index Medicus | Abridged Index Medicus
Journal Article
Contact Dermatitis, ISSN 0105-1873, 10/2010, Volume 63, Issue 4, pp. 215 - 222
Objectives: To investigate epidermal mRNA expression of mediators implicated in the regulation of inflammation induced by tape stripping according to our... 
tumour necrosis factor-α | tape stripping | inflammation | strip patch test | interleukin-33 | interleukin-8 | epidermal barrier | heat shock proteins 70 and 90 | ALLERGIC CONTACT-DERMATITIS | HUMAN SKIN | STRATUM-CORNEUM | TIME QUANTITATIVE PCR | INDUCTION | DERMATOLOGY | HEAT-SHOCK PROTEINS | ELICITATION | ALLERGY | IMMUNE-RESPONSES | GENE-EXPRESSION | tumour necrosis factor-alpha | HYPERSENSITIVITY | Interleukin-8 - genetics | Up-Regulation | Cytoskeletal Proteins - genetics | NLR Family, Pyrin Domain-Containing 3 Protein | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Patch Tests - methods | Reverse Transcription | HSP27 Heat-Shock Proteins - genetics | Young Adult | Interleukins - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | HSP90 Heat-Shock Proteins - genetics | Interleukins - biosynthesis | Cytokines - genetics | HSP70 Heat-Shock Proteins - biosynthesis | Interleukin-33 | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Keratinocytes - metabolism | Intercellular Adhesion Molecule-1 - genetics | Adolescent | Chemokine CCL5 - genetics | Chemokine CCL2 - biosynthesis | RNA, Messenger | Tumor Necrosis Factor-alpha - biosynthesis | HSP90 Heat-Shock Proteins - biosynthesis | Ribosomal Proteins - biosynthesis | Surgical Tape | Intercellular Adhesion Molecule-1 - biosynthesis | HSP27 Heat-Shock Proteins - biosynthesis | Adult | Protein Biosynthesis - genetics | Interferon-gamma - genetics | Cytoskeletal Proteins - biosynthesis | Epidermis - metabolism | Interleukin-8 - biosynthesis | Ribosomal Proteins - genetics | Carrier Proteins - biosynthesis | Chemokine CCL2 - genetics | HSP70 Heat-Shock Proteins - genetics | Carrier Proteins - genetics | Chemokine CCL5 - biosynthesis | Epidermis - immunology | Caspase 1 - genetics | CARD Signaling Adaptor Proteins | Cytokines - biosynthesis | Interferon-gamma - biosynthesis | Caspase 1 - biosynthesis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 03/2016, Volume 531, Issue 7595, pp. 528 - 532
Journal Article
Cell Reports, ISSN 2211-1247, 09/2016, Volume 17, Issue 1, pp. 69 - 85
Journal Article
Journal Article
Journal of Neuroimmunology, ISSN 0165-5728, 2007, Volume 194, Issue 1, pp. 54 - 61
Abstract Interleukin (IL)-17-producing helper T cells may play a pivotal role in the pathogenesis of multiple sclerosis. Here, we examined the effects of IL-17... 
Neurology | Allergy and Immunology | EAE | Cytokine | IL-17 | Microglia | cytokine | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MULTIPLE-SCLEROSIS LESIONS | MESSENGER-RNA EXPRESSION | NEUROTROPHIC FACTOR | IMMUNOLOGY | NEUROSCIENCES | NERVE GROWTH-FACTOR | INTERLEUKIN-17 | NEURONAL DEATH | NITRIC-OXIDE | TNF-ALPHA | microglia | T-CELLS | Brain-Derived Neurotrophic Factor - genetics | Microglia - metabolism | Humans | Tumor Necrosis Factor-alpha - genetics | Interleukin-17 - physiology | Receptors, Interleukin-17 - genetics | Interleukin-17 - pharmacology | Interleukin-1beta - genetics | Interleukin-23 - genetics | RNA, Messenger - biosynthesis | Chemokine CXCL2 - genetics | Brain-Derived Neurotrophic Factor - biosynthesis | Interleukin-1beta - biosynthesis | Multiple Sclerosis - metabolism | Autocrine Communication | Nitric Oxide - biosynthesis | Cells, Cultured - drug effects | Interleukin-6 - genetics | Microglia - drug effects | Receptors, Interleukin-17 - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - biosynthesis | Glial Cell Line-Derived Neurotrophic Factor - genetics | Interleukin-17 - biosynthesis | Recombinant Proteins - pharmacology | Gene Expression Regulation - drug effects | Up-Regulation - drug effects | Animals | Interleukin-23 - biosynthesis | Interleukin-6 - biosynthesis | Cells, Cultured - metabolism | Chemokine CXCL2 - biosynthesis | Mice | Tumor Necrosis Factor-alpha - biosynthesis | Multiple sclerosis | Interleukins | T cells | Nitric oxide | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2013, Volume 288, Issue 36, pp. 25938 - 25949
NAD(+) is mainly synthesized in human cells via the "salvage" pathways starting from nicotinamide, nicotinic acid, or nicotinamide riboside (NR). The... 
NAMPT INHIBITION | IN-VITRO | CYCLIC-ADP-RIBOSE | CHS 828 | ENZYME | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH | NICOTINAMIDE PHOSPHORIBOSYLTRANSFERASE | CD38 | FK866 | LYMPHOCYTES | 5'-Nucleotidase - genetics | Gene Expression Regulation, Enzymologic - drug effects | Humans | Membrane Glycoproteins - biosynthesis | GPI-Linked Proteins - biosynthesis | Neoplasm Proteins - antagonists & inhibitors | Neoplasm Proteins - metabolism | Nicotinamide Phosphoribosyltransferase - antagonists & inhibitors | Neoplasms - genetics | Nicotinamide Phosphoribosyltransferase - metabolism | Piperidines - pharmacology | NAD - biosynthesis | Cell Death - drug effects | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Acrylamides - pharmacology | Cytokines - genetics | Gene Expression Regulation, Neoplastic - genetics | 5'-Nucleotidase - biosynthesis | NAD - genetics | ADP-ribosyl Cyclase 1 - genetics | Cytokines - metabolism | Gene Silencing | Neoplasms - enzymology | Down-Regulation - drug effects | Down-Regulation - genetics | Membrane Glycoproteins - genetics | Neoplasms - drug therapy | Nicotinamide Phosphoribosyltransferase - genetics | Gene Expression Regulation, Enzymologic - genetics | ADP-ribosyl Cyclase 1 - biosynthesis | Cell Line, Tumor | Cytokines - antagonists & inhibitors | Nicotinamide Mononucleotide - biosynthesis | Nicotinamide Mononucleotide - genetics | GPI-Linked Proteins - genetics | Index Medicus | NAD | Cancer Therapy | CD73 | Nicotinamide Riboside | NAD Biosynthesis | Signal Transduction | Cell Death | Nicotinamide Mononucleotide | Nicotinamide
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 06/2006, Volume 79, Issue 6, pp. 1279 - 1285
Dendritic cells (DCs) play an important role in innate and adaptive immune responses. In addition to their phagocytic activity, DCs present foreign antigens to... 
human | cytokines | cell activation | Human | Cytokines | Cell activation | SIGNAL-TRANSDUCTION PATHWAYS | ACTIVATION | RECEPTOR | ANTIGEN PRESENTATION | IMMUNOLOGY | CUTTING EDGE | CELL BIOLOGY | B-CELLS | HUMAN NK | COMMON GAMMA-CHAIN | HEMATOLOGY | T-CELLS | IFN-ALPHA | Dendritic Cells - immunology | Humans | Membrane Glycoproteins - biosynthesis | Receptors, Interleukin-1 - genetics | Tumor Necrosis Factor-alpha - genetics | Suppressor of Cytokine Signaling 1 Protein | Gene Expression Profiling | Lipopolysaccharides - antagonists & inhibitors | RNA, Messenger - biosynthesis | Interleukins - physiology | T-Lymphocytes - metabolism | Chemokines, CC - biosynthesis | Cells, Cultured - immunology | Chemokines, CXC - biosynthesis | Cytokines - genetics | Intracellular Signaling Peptides and Proteins - genetics | Interleukin-12 - biosynthesis | Chemokine CCL5 | Cells, Cultured - drug effects | Receptors, Interleukin-21 | Lymphocyte Activation | Repressor Proteins - genetics | Suppressor of Cytokine Signaling Proteins - genetics | Toll-Like Receptor 4 - genetics | Receptors, Interleukin-1 - biosynthesis | Receptors, Interleukin - genetics | Dendritic Cells - secretion | Feedback, Physiological | Macrophages - metabolism | Repressor Proteins - biosynthesis | T-Lymphocytes - immunology | Cells, Cultured - metabolism | HLA-DR Antigens - biosynthesis | Suppressor of Cytokine Signaling Proteins - biosynthesis | Tumor Necrosis Factor-alpha - biosynthesis | Receptors, Interleukin - biosynthesis | Toll-Like Receptor 4 - biosynthesis | Interleukin-12 - genetics | Dendritic Cells - drug effects | Interleukins - pharmacology | Receptors, Interferon - genetics | Dendritic Cells - metabolism | RNA, Messenger - genetics | Cell Communication | Recombinant Proteins - pharmacology | Chemokines, CXC - genetics | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | Suppressor of Cytokine Signaling 3 Protein | Chemokines, CC - genetics | Macrophages - drug effects | Interleukin-21 Receptor alpha Subunit | Receptors, Interferon - biosynthesis | Cytokines - biosynthesis | Interferon-gamma - pharmacology | Index Medicus
Journal Article
Cellular Immunology, ISSN 0008-8749, 01/2013, Volume 281, Issue 1, pp. 51 - 61
Journal Article