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The Journal of infectious diseases, ISSN 1537-6613, 2015, Volume 211, Issue 1, pp. 156 - 165
Background. A predominantly T-helper type 2 (Th2) immune response is critical in the prognosis of pulmonary Pseudomonas aeruginosa infection. But the mucosal... 
human-milk | cytokine production | butyrate | in-vitro | chain fatty-acids | galacto-oligosaccharides | t-cells | lactobacillus | virus-infection | cystic-fibrosis patients | intestinal microbiota | prebiotics | Th1 and M1 polarization | P. aeruginosa lung infection | INFECTIOUS DISEASES | VIRUS-INFECTION | GALACTO-OLIGOSACCHARIDES | MICROBIOLOGY | IMMUNOLOGY | CYTOKINE PRODUCTION | HUMAN-MILK | METABOLISM | CYSTIC-FIBROSIS PATIENTS | CHAIN FATTY-ACIDS | BUTYRATE | T-CELLS | LACTOBACILLUS | Intestines - drug effects | Lung Diseases - microbiology | GATA3 Transcription Factor - immunology | Pseudomonas aeruginosa - growth & development | Microbiota - drug effects | Male | T-Box Domain Proteins - immunology | Lung Diseases - drug therapy | Th1 Cells - immunology | Intestines - immunology | Leukocytes - immunology | Oligosaccharides - pharmacology | Leukocytes - microbiology | Tumor Necrosis Factor-alpha - immunology | Keratinocytes - microbiology | Macrophages - immunology | Macrophages - microbiology | Chemotactic Factors - immunology | Microbiota - immunology | Pseudomonas Infections - drug therapy | Pseudomonas aeruginosa - drug effects | Pseudomonas Infections - microbiology | Keratinocytes - immunology | Pectins - chemistry | Pseudomonas Infections - immunology | Lung Diseases - immunology | Interleukin-4 - immunology | Th1 Cells - microbiology | Animals | Intestines - microbiology | Interferon-gamma - immunology | Mice | Mice, Inbred BALB C | Interleukin-10 - immunology | Pseudomonas Infections/microbiology | Macrophages/microbiology | Oligosaccharides/pharmacology | Macrophages/immunology | Lung Diseases/immunology | Lung Diseases/microbiology | Interferon-gamma/immunology | Keratinocytes/microbiology | Microbiota/drug effects | Life Sciences | Leukocytes/microbiology | Pseudomonas Infections/immunology | Interleukin-10/immunology | Lung Diseases/drug therapy | T-Box Domain Proteins/immunology | Leukocytes/immunology | Pseudomonas aeruginosa/drug effects | Th1 Cells/microbiology | GATA3 Transcription Factor/immunology | Interleukin-4/immunology | Intestines/drug effects | Intestines/microbiology | Keratinocytes/immunology | Chemotactic Factors/immunology | Tumor Necrosis Factor-alpha/immunology | Microbiota/immunology | Pseudomonas aeruginosa/growth & development | Pectins/chemistry | Pseudomonas Infections/drug therapy | Th1 Cells/immunology | Intestines/immunology
Journal Article
PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 11, p. e1003773
Interferons (IFNs) are a group of cytokines with a well-established antiviral function. They can be induced by viral infection, are secreted and bind to... 
CELLS | DEFENSE | VIRUS-INFECTION | DISTINCT | RIG-I | MICROBIOLOGY | IRF-7 | ANTIVIRAL RESPONSE | VIROLOGY | GENE | SIGNALING PATHWAY | ADAPTER PROTEIN | PARASITOLOGY | Epithelial Cells - metabolism | Influenza A virus - genetics | Respiratory Mucosa - virology | Interferon Regulatory Factor-7 - genetics | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | Interleukins - metabolism | Orthomyxoviridae Infections - genetics | Respiratory Mucosa - pathology | Interleukins - genetics | Interleukins - immunology | Respiratory Mucosa - immunology | Adaptor Proteins, Signal Transducing - immunology | Interferon Type I - metabolism | Influenza A virus - immunology | Membrane Proteins - metabolism | Interferon Regulatory Factor-3 - immunology | Nerve Tissue Proteins - immunology | Membrane Proteins - genetics | Orthomyxoviridae Infections - metabolism | Epithelial Cells - pathology | Membrane Proteins - immunology | Interferon Regulatory Factor-7 - immunology | Interferon Regulatory Factor-7 - metabolism | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Influenza A virus - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Adaptor Proteins, Signal Transducing - genetics | Interferon Regulatory Factor-3 - metabolism | Interferon Type I - genetics | Mice | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Orthomyxoviridae Infections - immunology | Epithelial cells | Influenza | Physiological aspects | Host-parasite relationships | Interferon | Genetic aspects | Genetic transcription | Research | Health aspects | Airway (Medicine) | Interleukins/metabolism | Nerve Tissue Proteins/immunology | Orthomyxoviridae Infections/genetics | Orthomyxoviridae Infections/metabolism | Membrane Proteins/genetics | Adaptor Proteins, Signal Transducing/genetics | Interferon Regulatory Factor-7/genetics | Interleukins/immunology | Membrane Proteins/immunology | Life Sciences | Orthomyxoviridae Infections/immunology | Influenza A virus/genetics | Nerve Tissue Proteins/metabolism | Respiratory Mucosa/immunology | Immunology | Interferon Type I/immunology | Epithelial Cells/immunology | Epithelial Cells/virology | Interferon Regulatory Factor-7/immunology | Epithelial Cells/metabolism | Interferon Regulatory Factor-3/immunology | Respiratory Mucosa/pathology | Influenza A virus/immunology | Interleukins/genetics | Interferon Regulatory Factor-3/genetics | Influenza A virus/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Epithelial Cells/pathology | Adaptor Proteins, Signal Transducing/immunology | Interferon Type I/genetics | Interferon Regulatory Factor-3/metabolism | Membrane Proteins/metabolism | Nerve Tissue Proteins/genetics | Interferon Regulatory Factor-7/metabolism | Interferon Type I/metabolism | Respiratory Mucosa/metabolism | Respiratory Mucosa/virology | Cytokines | Genes | Rodents | Genomics | Genomes | Kinases | Experiments | Viral infections
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2014, Volume 193, Issue 2, pp. 961 - 969
Immunotherapy aiming at enhancing innate and acquired host immunity is a promising approach for cancer treatment. The invariant NKT (iNKT) cell ligand... 
IMMUNITY | ACTIVATION | CANCER-PATIENTS | VIVO | KRN7000 | KILLER T-CELLS | CYTOKINE BIAS | ANTIGEN PRESENTATION | ANERGY | IMMUNOLOGY | TLR LIGANDS | Antigens, CD - immunology | Nanoparticles - chemistry | Minor Histocompatibility Antigens | Antibodies - chemistry | Dendritic Cells - immunology | Lectins, C-Type - immunology | Male | Galactosylceramides - administration & dosage | Neoplasms, Experimental - pathology | Lymphocyte Activation - immunology | T-Lymphocytes - metabolism | Natural Killer T-Cells - metabolism | Neoplasms, Experimental - immunology | Antibodies - immunology | CD8-Positive T-Lymphocytes - metabolism | Dendritic Cells - metabolism | T-Lymphocytes, Cytotoxic - immunology | Receptors, Antigen, T-Cell, gamma-delta - metabolism | Mice, Inbred C57BL | Neoplasms, Experimental - therapy | Mice, Transgenic | Receptors, Antigen, T-Cell, gamma-delta - immunology | Antigen Presentation - immunology | Galactosylceramides - chemistry | Receptors, Cell Surface - immunology | CD8 Antigens - immunology | Mice, Knockout | CD8 Antigens - metabolism | Animals | Galactosylceramides - immunology | T-Lymphocytes, Cytotoxic - metabolism | Cell Line, Tumor | T-Lymphocytes - immunology | Mice | Nanoparticles - administration & dosage | CD8-Positive T-Lymphocytes - immunology | Drug Delivery Systems - methods | Natural Killer T-Cells - immunology | Tumor Burden - immunology | Nanoparticles/administration & dosage | Receptors, Cell Surface/immunology | Neoplasms, Experimental/therapy | CD8 Antigens/metabolism | Dendritic Cells/metabolism | Innate immunity | Galactosylceramides/immunology | Antigen Presentation/immunology | Natural Killer T-Cells/metabolism | Receptors, Antigen, T-Cell, gamma-delta/metabolism | Life Sciences | Galactosylceramides/chemistry | Lectins, C-Type/immunology | CD8-Positive T-Lymphocytes/immunology | Antigens, CD/immunology | Immunology | Antibodies/chemistry | Antibodies/immunology | T-Lymphocytes, Cytotoxic/metabolism | T-Lymphocytes/metabolism | Drug Delivery Systems/methods | T-Lymphocytes/immunology | CD8-Positive T-Lymphocytes/metabolism | Galactosylceramides/administration & dosage | Natural Killer T-Cells/immunology | Receptors, Antigen, T-Cell, gamma-delta/immunology | CD8 Antigens/immunology | Tumor Burden/immunology | Neoplasms, Experimental/immunology | Lymphocyte Activation/immunology | Nanoparticles/chemistry | Dendritic Cells/immunology | T-Lymphocytes, Cytotoxic/immunology | Neoplasms, Experimental/pathology
Journal Article
eJournal
The Journal of immunology (1950), ISSN 1550-6606, 2008, Volume 180, Issue 9, pp. 6139 - 6148
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e50385
ATP released in the early inflammatory processes acts as a danger signal by binding to purinergic receptors expressed on immune cells. A major contribution of... 
Chemokines, CXC - immunology | Receptors, Purinergic P2Y2 - immunology | Interleukin-12 - genetics | Th2 Cells - virology | Dendritic Cells - immunology | Bronchoalveolar Lavage Fluid - immunology | Th2 Cells - immunology | Th1 Cells - immunology | Murine pneumonia virus - immunology | Pneumovirus Infections - metabolism | Pneumonia, Viral - metabolism | Th1 Cells - virology | Chemokine CCL20 - immunology | Inflammation - metabolism | Lung - virology | Adenosine Triphosphate - metabolism | Chemokine CXCL10 - immunology | Chemokine CCL20 - genetics | Female | Lung - metabolism | Dendritic Cells - virology | Receptors, Purinergic P2Y2 - genetics | Gene Expression | Interleukin-6 - genetics | Inflammation - virology | Neutrophils - immunology | Survival Rate | Inflammation - immunology | Chemokines, CXC - genetics | Pneumovirus Infections - virology | Neutrophils - virology | Pneumonia, Viral - immunology | Receptors, Purinergic P2Y2 - deficiency | Mice, Knockout | Pneumovirus Infections - immunology | Animals | CD8-Positive T-Lymphocytes - virology | Chemokine CXCL10 - genetics | Interleukin-12 - immunology | Interleukin-6 - immunology | Mice | Bronchoalveolar Lavage Fluid - chemistry | CD8-Positive T-Lymphocytes - immunology | Pneumonia, Viral - virology | Lung - immunology | Pneumonia | Purine receptors | CD8 antigen | Immune clearance | Viruses | Cytotoxicity | Lymphocytes T | Infections | Recruitment | Interleukin 6 | Respiratory tract | Airway management | Receptors | Lymphocytes | Rodents | IP-10 protein | Respiratory tract diseases | Alveoli | Immune system | Dendritic cells | Mortality | Cloning | Bronchus | Cystic fibrosis | Interleukin 12 | Inflammation | Hazards | Interdisciplinary aspects | Morbidity | Asthma | CD4 antigen | Respiratory syncytial virus | Lungs | Fibrosis | Signal processing | Infiltration | ATP | Chemokines | Viral infections
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 11, p. e113101
Background: Tuberculosis-associated immune reconstitution inflammatory syndrome (TB-IRIS) remains a poorly understood complication in HIV-TB patients receiving... 
MYCOBACTERIUM-TUBERCULOSIS | CELLS | RISK-FACTORS | PROFILE | RECONSTITUTION INFLAMMATORY SYNDROME | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | ANTIRETROVIRAL THERAPY | IMMUNE RESTORATION DISEASE | EPIDEMIOLOGY | HIV-INFECTED PATIENTS | Prospective Studies | Immune Reconstitution Inflammatory Syndrome - immunology | Humans | Antigens, Bacterial - immunology | Mycobacterium tuberculosis - immunology | Tuberculosis - complications | Male | Cytomegalovirus Infections - complications | Cytomegalovirus Infections - immunology | Influenza, Human - complications | Interferon-gamma - metabolism | Receptors, Interleukin-12 | Lipopolysaccharides - immunology | HIV Infections - immunology | Tuberculosis - drug therapy | Interleukin-10 - metabolism | Adult | Anti-HIV Agents - therapeutic use | Female | Antitubercular Agents - immunology | Interleukin-6 - metabolism | Antitubercular Agents - therapeutic use | Cytokines - immunology | Cytokines - metabolism | Immune Reconstitution Inflammatory Syndrome - complications | CD4 Lymphocyte Count | Enzyme-Linked Immunospot Assay | Interleukin-12 - metabolism | Tuberculosis - immunology | Interferon-gamma - immunology | HIV Infections - complications | Interleukin-12 - immunology | Interleukin-6 - immunology | HIV Infections - drug therapy | Interleukin-10 - immunology | Anti-HIV Agents - immunology | Influenza, Human - immunology | Highly active antiretroviral therapy | Antiviral agents | Influenza | HIV patients | Interferon | Comparative analysis | Immunity | Health sciences | Pathogenesis | Infections | Epidemiology | Lipopolysaccharides | Interleukin 6 | Sexually transmitted diseases | Acquired immune deficiency syndrome--AIDS | Antiretroviral agents | Human immunodeficiency virus--HIV | Enzyme-linked immunosorbent assay | Immune system | Antigens | Cytomegalovirus | Immune response | Cytokines | Blood & organ donations | Immune reconstitution | Interleukin 12 | Inflammation | Tumor necrosis factor-α | Patients | Antiretroviral therapy | CD4 antigen | Medicine | Infectious diseases | Tuberculosis | Production methods | γ-Interferon | Interleukin 10 | Acquired immune deficiency syndrome | AIDS | HIV | Human immunodeficiency virus
Journal Article
Nature (London), ISSN 1476-4687, 2016, Volume 529, Issue 7585, pp. 226 - 230
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2009, Volume 183, Issue 10, pp. 6469 - 6477
Alternatively activated macrophages (AAM) play a crucial role in type 2 immunity. Mice deficient in ST2, a receptor for the latest member of the IL-1 family,... 
IL-1-LIKE CYTOKINE IL-33 | IMMUNE-RESPONSE | ST2 RECEPTOR | EFFECTOR FUNCTION | MAST-CELLS | IN-VIVO | INDUCED ARTHRITIS | ALVEOLAR MACROPHAGES | INTERLEUKIN-1 RECEPTOR | IMMUNOLOGY | T-CELLS | Interleukin-1 Receptor-Like 1 Protein | Asthma - metabolism | Epithelial Cells - metabolism | Ovalbumin - immunology | Humans | Middle Aged | Chemokine CCL24 - metabolism | Interleukin-13 - immunology | Male | Interleukins - metabolism | Receptors, Interleukin - metabolism | Signal Transduction - immunology | Inflammation - metabolism | Interleukins - immunology | Asthma - immunology | Chemokine CCL17 - immunology | Adult | Female | Interleukin-4 - genetics | Macrophages, Alveolar - immunology | Recombinant Proteins - metabolism | Cell Polarity - immunology | Lung - pathology | Receptors, Interleukin - immunology | Interleukin-33 | Macrophage Activation - immunology | Receptors, Interleukin-1 - immunology | Interleukin-4 - metabolism | Receptors, Interleukin-4 - metabolism | Inflammation - immunology | Receptors, Interleukin-4 - immunology | Interleukin-13 - metabolism | Chemokine CCL24 - immunology | Receptors, Interleukin - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Interleukin-4 - immunology | Animals | Eosinophilia - immunology | Recombinant Proteins - immunology | Epithelial Cells - immunology | Receptors, Interleukin-4 - genetics | Mice | Mice, Inbred BALB C | Macrophages, Alveolar - metabolism | Chemokine CCL17 - metabolism | Lung - immunology
Journal Article
Journal of autoimmunity, ISSN 0896-8411, 2015, Volume 60, pp. 1 - 11
Journal Article
Journal Article