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dahls.z-lepr/lepr (9) 9
index medicus (9) 9
animal-model (8) 8
animals (8) 8
heart-failure (8) 8
male (8) 8
oxidative stress (7) 7
rats (7) 7
rats, inbred dahl (7) 7
metabolic syndrome (6) 6
hypertension - physiopathology (5) 5
inflammation (5) 5
obesity (5) 5
cardiac & cardiovascular systems (4) 4
cardiac remodeling (4) 4
diastolic dysfunction (4) 4
disease models, animal (4) 4
hypertension (4) 4
metabolic syndrome - metabolism (4) 4
metabolic syndrome - physiopathology (4) 4
myocytes, cardiac - pathology (4) 4
rats, zucker (4) 4
adipose tissue - metabolism (3) 3
adipose tissue - pathology (3) 3
blood pressure - drug effects (3) 3
echocardiography (3) 3
fibrosis (3) 3
hypertensive-rats (3) 3
hypertrophy (3) 3
insulin-resistance (3) 3
medicine, research & experimental (3) 3
metabolic syndrome - drug therapy (3) 3
metabolic syndrome - pathology (3) 3
mineralocorticoid receptor (3) 3
myocytes, cardiac - metabolism (3) 3
oxidative stress - physiology (3) 3
peripheral vascular disease (3) 3
activated protein-kinase (2) 2
adipose tissue (2) 2
adipose tissue - drug effects (2) 2
adipose tissues (2) 2
blood pressure (2) 2
blood-pressure (2) 2
cardiovascular (2) 2
diet, sodium-restricted (2) 2
hypertension - drug therapy (2) 2
hypertension - metabolism (2) 2
hypertrophy, left ventricular - drug therapy (2) 2
inflammation mediators - metabolism (2) 2
insulin resistance (2) 2
mineralocorticoid receptor antagonists - pharmacology (2) 2
myocardium - metabolism (2) 2
myocardium - pathology (2) 2
myocytes, cardiac - drug effects (2) 2
obesity - metabolism (2) 2
obesity - pathology (2) 2
obesity - physiopathology (2) 2
original paper (2) 2
renin-angiotensin system (2) 2
renin-angiotensin system - drug effects (2) 2
sensitive hypertensive-rats (2) 2
sodium (2) 2
sodium chloride, dietary (2) 2
time factors (2) 2
activated-protein-kinase (1) 1
activation (1) 1
adipocytes - drug effects (1) 1
adipocytes - metabolism (1) 1
adipocytes - pathology (1) 1
adiponectin receptors (1) 1
adipose-tissue (1) 1
adrenalectomy (1) 1
adrenergic beta-antagonists - pharmacology (1) 1
adrenergic beta-antagonists - therapeutic use (1) 1
age (1) 1
age factors (1) 1
aldosterone (1) 1
aldosterone antagonism (1) 1
amp-activated protein kinase (1) 1
angiotensin system (1) 1
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atorvastatin (1) 1
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blood glucose - metabolism (1) 1
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cardiac remodeling ventricular (1) 1
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Journal Article
Life Sciences, ISSN 0024-3205, 08/2019, Volume 230, pp. 19 - 27
We investigated the effect of the selective sodium-dependent glucose cotransporter 2 (SGLT2) inhibitor ipragliflozin on cardiac dysfunction and histopathology... 
Ipragliflozin l-proline (PubChem CID: 57339444) | Cardio-protective effect | Sodium-dependent glucose cotransporter 2 | MEDICINE, RESEARCH & EXPERIMENTAL | HEART-FAILURE | COTRANSPORTER 2 INHIBITION | DIASTOLIC DYSFUNCTION | BIOMARKERS | Ipragliflozin L-proline (PubChem CID: 57339444) | DAHLS.Z-LEPR(FA)/LEPR(FA) RATS | INFLAMMATION | PHARMACOLOGY & PHARMACY | ANIMAL-MODEL | COMPLICATIONS | FAT MASS | EMPAGLIFLOZIN | Fibrosis - drug therapy | Sodium-Glucose Transporter 2 Inhibitors - pharmacology | Systole - drug effects | Male | Thiophenes - metabolism | Heart Rate - drug effects | Sodium-Glucose Transporter 2 Inhibitors - metabolism | Metabolic Syndrome - physiopathology | Blood Pressure - drug effects | Rats, Inbred Dahl | Hypertrophy, Left Ventricular - drug therapy | Fibrosis - prevention & control | Disease Models, Animal | Cardiomyopathies - drug therapy | Obesity | Glucosides - pharmacology | Sodium-Glucose Transporter 2 - drug effects | Echocardiography | Sodium-Glucose Transporter 2 - metabolism | Hypertrophy, Left Ventricular - prevention & control | Rats | Thiophenes - pharmacology | Cardiomyopathies - pathology | Glucosides - metabolism | Hypoglycemic Agents - pharmacology | Animals | Heart - drug effects | Heart beat | Cardiomyopathy | Proline | Drug discovery | Glucose | Heart diseases | Dextrose | Histochemistry | Urine | Excretion | Diabetes mellitus | Cardiomyocytes | Heart rate | Histopathology | Inhibitors | Sodium | Rodents | Fibrosis | Blood pressure | Diabetes | Ventricle | Age | Hypertrophy | Index Medicus
Journal Article
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2015, Volume 308, Issue 10, pp. H1275 - H1286
Restraint stress stimulates sympathetic nerve activity and can affect adiposity and metabolism. However, the effects of restraint stress on cardiovascular and... 
Restraint stress | Adipose tissue | Cardiac remodeling | Metabolic syndrome | Sympathetic nervous system | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | sympathetic nervous system | HEART-FAILURE | adipose tissue | NEUROPEPTIDE-Y | metabolic syndrome | DAHLS.Z-LEPR(FA)/LEPR(FA) RATS | HYPERTROPHY | OBESITY | INSULIN-RESISTANCE | cardiac remodeling | PERIPHERAL VASCULAR DISEASE | ANIMAL-MODEL | DYSFUNCTION | restraint stress | EXPRESSION | Oxidative Stress | Propranolol - therapeutic use | Norepinephrine - urine | Hypertension - drug therapy | Male | Metabolic Syndrome - metabolism | Metabolic Syndrome - drug therapy | Adipose Tissue - metabolism | Adrenergic beta-Antagonists - pharmacology | Glucose Intolerance - drug therapy | Propranolol - pharmacology | Metabolic Syndrome - physiopathology | Restraint, Physical | Rats, Inbred Dahl | Hypertrophy, Left Ventricular - drug therapy | Receptors, Adrenergic, beta - metabolism | Glucose Intolerance - physiopathology | Adrenergic beta-Antagonists - therapeutic use | Echocardiography | Signal Transduction | Adipose Tissue - pathology | Rats | Lipid Metabolism | Hypertension - physiopathology | Rats, Zucker | Myocytes, Cardiac - pathology | Animals | Receptors, Adrenergic, beta - genetics | Myocytes, Cardiac - metabolism | Hypertrophy, Left Ventricular - physiopathology | Stress, Psychological - etiology | Stress, Psychological - physiopathology | Index Medicus
Journal Article
International Journal of Cardiology, ISSN 0167-5273, 2017, Volume 240, pp. 332 - 338
Abstract Background Statins are strong inhibitors of cholesterol biosynthesis and help to prevent cardiovascular disease. They also exert additional... 
Cardiovascular | Atorvastatin | Inflammation | Nuclear factor–κB | Metabolic syndrome | AMP-activated protein kinase | Nuclear factor-kappa B | COA REDUCTASE INHIBITOR | CARDIAC & CARDIOVASCULAR SYSTEMS | HEART-FAILURE | ACTIVATED-PROTEIN-KINASE | DIASTOLIC DYSFUNCTION | OBESE MICE | DAHLS.Z-LEPR(FA)/LEPR(FA) RATS | INSULIN-RESISTANCE | ANIMAL-MODEL | NF-KAPPA-B | HYPERTENSIVE-RATS | Inflammation - pathology | Obesity - drug therapy | Atorvastatin Calcium - pharmacology | Oxidative Stress - physiology | Male | Metabolic Syndrome - metabolism | Metabolic Syndrome - drug therapy | Adipose Tissue - metabolism | Inflammation - metabolism | Inflammation - drug therapy | Inflammation Mediators - metabolism | Inflammation Mediators - antagonists & inhibitors | Rats, Inbred Dahl | Atorvastatin Calcium - therapeutic use | Adipose Tissue - pathology | Cells, Cultured | Rats | Random Allocation | Obesity - metabolism | Obesity - pathology | Myocytes, Cardiac - pathology | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Myocytes, Cardiac - drug effects | Hydroxymethylglutaryl-CoA Reductase Inhibitors - therapeutic use | Myocytes, Cardiac - metabolism | Metabolic Syndrome - pathology | Oxidative Stress - drug effects | Adipose Tissue - drug effects | Prevention | Adipose tissues | Cardiovascular agents | Antilipemic agents | Insulin resistance | Protein kinases | Index Medicus
Journal Article
Journal of the American Heart Association, ISSN 2047-9980, 12/2014, Volume 3, Issue 6, pp. e001312 - n/a
Background-Metabolic syndrome (MetS) enhances salt sensitivity of blood pressure and is an important risk factor for cardiovascular disease. The effects of... 
sodium | remodeling | diet | hypertension | obesity | Hypertension | Obesity | Diet | Sodium | Remodeling | WISTAR RATS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHOSPHORYLATION | HEART-FAILURE | MINERALOCORTICOID RECEPTORS | BLOOD-PRESSURE | DIASTOLIC DYSFUNCTION | DAHLS.Z-LEPR(FA)/LEPR(FA) RATS | INSULIN-RESISTANCE | ANIMAL-MODEL | Obesity - diet therapy | Age Factors | Oxidative Stress | Cardiomegaly - pathology | Male | Insulin - blood | Obesity - blood | Obesity - genetics | Hypertension - etiology | Hypertension - blood | Metabolic Syndrome - blood | Renin-Angiotensin System | Time Factors | Myocardium - metabolism | Diet, Sodium-Restricted | Inflammation Mediators - metabolism | Metabolic Syndrome - physiopathology | Hypertension - prevention & control | Metabolic Syndrome - diet therapy | Rats, Inbred Dahl | Disease Models, Animal | Cardiomegaly - blood | Insulin Resistance | Cardiomegaly - physiopathology | Myocardium - pathology | Obesity - physiopathology | Biomarkers - blood | Intra-Abdominal Fat - metabolism | Sodium Chloride, Dietary | Hypertension - pathology | Hypertension - physiopathology | Obesity - pathology | Rats, Zucker | Animals | Cardiomegaly - prevention & control | Metabolic Syndrome - genetics | Fibrosis | Hemodynamics | Intra-Abdominal Fat - pathology | Metabolic Syndrome - pathology | Blood Glucose - metabolism | Cardiomegaly - genetics | Index Medicus
Journal Article
Hypertension Research, ISSN 0916-9636, 02/2014, Volume 37, Issue 2, pp. 125 - 133
Journal Article
Nagoya journal of medical science, ISSN 0027-7622, 2014, Volume 76, Issue 1-2, pp. 59 - 72
We previously showed that selective mineralocorticoid receptor (MR) blockade by eplerenone is cardioprotective in Dahl salt-sensitive (DS) rats. To clarify the... 
MEDICINE, RESEARCH & EXPERIMENTAL | METABOLIC SYNDROME | INFLAMMATORY RESPONSES | HEART-FAILURE | mineralocorticoid receptor | VENTRICULAR MYOCYTES | DAHLS.Z-LEPR(FA)/LEPR(FA) RATS | HYPERTROPHY | diastolic function | inflammation | NITRIC-OXIDE | ANIMAL-MODEL | glucocorticoids | HYPERTENSIVE-RATS | oxidative stress | Receptors, Mineralocorticoid - agonists | Oxidative Stress | Receptors, Mineralocorticoid - metabolism | Collagen Type III - metabolism | NADPH Oxidases - metabolism | Hypertension - drug therapy | Male | RNA, Messenger - metabolism | Ventricular Dysfunction, Left - chemically induced | Corticosterone - toxicity | Collagen Type I - genetics | Spironolactone - pharmacology | Time Factors | Mineralocorticoid Receptor Antagonists - pharmacology | Glucocorticoids - toxicity | Blood Pressure - drug effects | Rats, Inbred Dahl | Disease Models, Animal | Collagen Type I - metabolism | Ventricular Function, Left - drug effects | Rats | Collagen Type III - genetics | Sodium Chloride, Dietary | Hypertension - physiopathology | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Hypertension - metabolism | Adrenalectomy | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Renin-Angiotensin System - drug effects | Ventricular Remodeling - drug effects | Index Medicus | Original Paper
Journal Article
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