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Bioorganic & Medicinal Chemistry, ISSN 0968-0896, 02/2020, Volume 28, Issue 3, p. 115274
[Display omitted] •BPE-X, bisphenol E analogs containing CX3 (CF3 or CBr3), were newly synthesized.•Bisphenol AF (BPAF), BPE-F, BPE-Cl, and BPE-Br were assayed... 
Estrogen receptor | Agonist | Halogen bonding | Antagonist | Structure-activity relationship | Bisphenol | ESTROGEN-RECEPTORS | CHEMISTRY, MEDICINAL | ENDOCRINE DISRUPTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | CHEMISTRY, ORGANIC | BINDING CHARACTERISTICS | DDT
Journal Article
Human & Experimental Toxicology, ISSN 0960-3271, 1/2014, Volume 33, Issue 1, pp. 41 - 53
Journal Article
Toxicology Letters, ISSN 0378-4274, 2008, Volume 183, Issue 1, pp. 65 - 71
Some organochlorine pesticides (OCPs) are suspected of modulating the endocrine systems of humans. Aspects of neuro-endocrine system modulation include... 
Estrogen receptor | Endocrine disruptor | Progesterone receptor | Estrogen-related receptor | Organochlorine pesticides | Androgen receptor | TRANSCRIPTIONAL ACTIVATION | ASSAY | PROGESTERONE | ESTROGEN-RELATED-RECEPTOR | YEAST | CHEMICALS | TRANSACTIVATION DOMAIN | BISPHENOL-A | TOXICOLOGY | VINCLOZOLIN | Androgen Receptor Antagonists | Hexachlorobenzene - pharmacology | Humans | Receptors, Progesterone - antagonists & inhibitors | Hexachlorobenzene - chemistry | Dichlorodiphenyl Dichloroethylene - chemistry | Endocrine Disruptors - pharmacology | Estrogen Receptor alpha - agonists | Plasmids - genetics | beta-Galactosidase - metabolism | Estrogen Receptor alpha - physiology | Receptors, Androgen - physiology | Yeasts - metabolism | Lindane - pharmacology | Receptors, Estrogen - agonists | Endocrine Disruptors - chemistry | Hydrocarbons, Chlorinated - chemistry | Hydrocarbons, Chlorinated - pharmacology | Dichlorodiphenyl Dichloroethylene - pharmacology | Lindane - chemistry | Receptors, Estrogen - antagonists & inhibitors | DDT - chemistry | Yeasts - genetics | DDT - pharmacology | Receptors, Progesterone - physiology | Pesticides - chemistry | beta-Galactosidase - genetics | Pesticides - pharmacology | Receptors, Estrogen - physiology | Endocrine disruptors | Progesterone | Bisphenol-A | Organochlorine compounds | Estrogen | Pesticides | Index Medicus
Journal Article
Journal of Steroid Biochemistry and Molecular Biology, ISSN 0960-0760, 07/2017, Volume 171, pp. 94 - 109
•Prenatal exposures to DDT induce depressive-like behavior in adolescent mice.•DDT accumulates in mouse brain and leads to global DNA hypomethylation.•p,p’-DDT... 
DNA methylation | Estrogen receptors | Aryl hydrocarbon receptor | Brain | Depression | Pesticide | ESTROGEN-RECEPTORS | MALE RATS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROPROTECTIVE ACTION | X-RECEPTOR | ENDOCRINE DISRUPTORS | ARYL-HYDROCARBON RECEPTOR | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | NEURONAL CELLS | AHR FUNCTION | ORGANOCHLORINE PESTICIDES | Prenatal Exposure Delayed Effects | Receptors, Estrogen - metabolism | Receptors, G-Protein-Coupled - metabolism | Male | Antidepressive Agents, Second-Generation - therapeutic use | Isomerism | Neurotoxicity Syndromes - drug therapy | Brain - metabolism | DDT - toxicity | Depression - drug therapy | Depression - metabolism | Depression - chemically induced | Behavior, Animal - drug effects | Estrogen Receptor alpha - metabolism | Female | Neurons - metabolism | Neurons - drug effects | Endocrine Disruptors - chemistry | Receptors, Estrogen - antagonists & inhibitors | Estrogen Receptor alpha - antagonists & inhibitors | Random Allocation | Sex Characteristics | DDT - chemistry | Endocrine Disruptors - toxicity | Brain - drug effects | Pregnancy | Animals | Signal Transduction - drug effects | Receptors, G-Protein-Coupled - antagonists & inhibitors | Neurotoxicity Syndromes - metabolism | Citalopram - therapeutic use | Mice | DNA Methylation - drug effects | Bisphenol-A | Pregnant women | DNA | Antidepressants | Depression, Mental | Estrogen | Methylation | Mass spectrometry
Journal Article
Toxicological Sciences, ISSN 1096-6080, 3/2008, Volume 102, Issue 1, pp. 33 - 41
In previous studies, we detected a dichlorodiphenyltrichloroethane (DDT) derivative in the serum of children with sexual precocity after migration from... 
Glutamate receptors | Estrogen receptors | Aryl hydrocarbon receptor | Gonadotropin-releasing hormone | Endocrine-disrupting chemicals | ESTROGEN-RECEPTORS | endocrine-disrupting chemicals | estrogen receptors | SEXUAL PRECOCITY | aryl hydrocarbon receptor | EXCITATORY AMINO-ACIDS | gonadotropin-releasing hormone | IN-VITRO | ESTRADIOL | NEURONS | BISPHENOL-A | TOXICOLOGY | GONADAL-STEROIDS | ADULT MALE | EXPOSURE | glutamate receptors | Hypothalamus - growth & development | Receptors, Estrogen - metabolism | Rats, Wistar | Phenols - toxicity | Estrogen Antagonists - pharmacology | Sexual Maturation - drug effects | DDT - toxicity | Dose-Response Relationship, Drug | Time Factors | Protein Kinase C - metabolism | Injections, Subcutaneous | Receptors, Aryl Hydrocarbon - metabolism | Female | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Hypothalamus - drug effects | Receptors, AMPA - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Gonadotropin-Releasing Hormone - secretion | Estradiol - metabolism | Benzhydryl Compounds | Tissue Culture Techniques | Rats | Endocrine Disruptors - blood | Excitatory Amino Acid Antagonists - pharmacology | Protein Kinase C - antagonists & inhibitors | Receptors, Aryl Hydrocarbon - drug effects | DDT - administration & dosage | Endocrine Disruptors - administration & dosage | Endocrine Disruptors - toxicity | Methoxychlor - toxicity | Receptors, Estrogen - drug effects | Animals | Hypothalamus - metabolism | Mitogen-Activated Protein Kinases - antagonists & inhibitors | Signal Transduction - drug effects | Hypothalamus - enzymology | Glutamic Acid - metabolism | Protein Kinase Inhibitors - pharmacology | DDT - blood | Receptors, AMPA - drug effects | Mitogen-Activated Protein Kinases - metabolism
Journal Article
Biochemistry (Moscow), ISSN 0006-2979, 10/2017, Volume 82, Issue 10, pp. 1118 - 1128
The insecticide dichlorodiphenyltrichloroethane (DDT) is a nonmutagenic xenobiotic compound able to exert estrogen-like effects resulting in activation of... 
Life Sciences | Biochemistry, general | Biomedicine, general | microRNAs | Microbiology | hormonal carcinogenesis | DDT | ERα | Bioorganic Chemistry | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | INCREASES | MIR-205 | CANCER | TUMORS | ROLES | ESTROGEN-RECEPTOR-ALPHA | AROMATASE | ER alpha | Rats, Wistar | Uterus - drug effects | Uterus - metabolism | Gene Expression Regulation, Neoplastic | Aromatase - metabolism | RNA, Messenger - metabolism | DDT - toxicity | PTEN Phosphohydrolase - antagonists & inhibitors | Aromatase - genetics | DEAD-box RNA Helicases - antagonists & inhibitors | Estrogen Receptor alpha - metabolism | Female | Ovary - drug effects | DEAD-box RNA Helicases - metabolism | Ovary - metabolism | PTEN Phosphohydrolase - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | RNA, Messenger - genetics | Carcinogenesis - genetics | PTEN Phosphohydrolase - metabolism | Rats | Ribonuclease III - antagonists & inhibitors | DEAD-box RNA Helicases - genetics | Animals | Estrogen Receptor alpha - genetics | MicroRNAs - genetics | MicroRNA | Uterus | Genetic aspects | Ovaries | Gene expression | Genetic regulation | Health aspects | Carcinogenesis | Genes | Estrogens | Pesticides | Estrogen receptors | Activation | mRNA | Carcinogens | MicroRNAs | Rodents | Tumor suppressor genes | miRNA | PTEN protein
Journal Article
International Journal of Andrology, ISSN 0105-6263, 02/2006, Volume 29, Issue 1, pp. 96 - 104
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 07/2014, Volume 392, Issue 1-2, pp. 90 - 105
•DDT-induced apoptosis of neuronal cells depends on caspase-9, caspase-3, and GSK3β.•DDT-induced apoptosis involves p,p’-DDT-specific impairment of classical... 
Bcl-2 | Estrogen receptors | GSK-3β | Neurotoxicity | Caspases | Primary neuronal cell cultures | ACTIVATION | GPR30 | 17-BETA-ESTRADIOL | ER-ALPHA | PESTICIDES | CELL BIOLOGY | BREAST-CANCER CELLS | EPITHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | ESTROGEN | ENDOCRINE DISRUPTION | GSK-3 beta | L-Lactate Dehydrogenase - metabolism | Cytochrome P-450 CYP1A1 - genetics | Caspase Inhibitors - pharmacology | Receptors, Estrogen - metabolism | Receptors, Aryl Hydrocarbon - antagonists & inhibitors | Receptors, G-Protein-Coupled - metabolism | Apoptosis - drug effects | Caspase 3 - metabolism | Glycogen Synthase Kinase 3 beta | Neurons - cytology | Receptors, G-Protein-Coupled - agonists | RNA, Messenger - metabolism | Isomerism | Quinolines - pharmacology | Time Factors | beta-Naphthoflavone - pharmacology | Receptors, Aryl Hydrocarbon - metabolism | Neurons - metabolism | Neurons - drug effects | Pyrazoles - pharmacology | Receptors, Estrogen - genetics | RNA, Messenger - genetics | Cells, Cultured | Receptors, Aryl Hydrocarbon - genetics | Receptors, Estrogen - antagonists & inhibitors | Pyrimidines - pharmacology | Cytochrome P-450 CYP1A1 - metabolism | Glycogen Synthase Kinase 3 - metabolism | DDT - chemistry | DDT - pharmacology | Animals | Benzoflavones - pharmacology | Signal Transduction - drug effects | Neurons - enzymology | Mice | Receptors, G-Protein-Coupled - genetics | Benzodioxoles - pharmacology | Bisphenol-A | RNA | Neurons | Apoptosis | Index Medicus | Reduction | Cellular | Receptors | DDT | Toxicity | Impairment | Stimulation
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