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1. Full Text Developing Lymph Nodes Collect CD4+CD3− LTβ+ Cells That Can Differentiate to APC, NK Cells, and Follicular Cells but Not T or B Cells
by Mebius, Reina E and Rennert, Paul and Weissman, Irving L
Immunity, ISSN 1074-7613, 10/1997, Volume 7, Issue 4, pp. 493 - 504
For a brief period during fetal lymph node organogenesis in mice, lymph node postcapillary high endothelial venules surprisingly express the Peyer's patch... 
NATURAL-KILLER-CELLS | TUMOR-NECROSIS-FACTOR | DENDRITIC CELLS | ALPHA-DEFICIENT MICE | HEMATOPOIETIC STEM-CELLS | K NI IMMUNOLOGY | MONOCLONAL-ANTIBODY | GERMINAL-CENTERS | VASCULAR ADDRESSIN EXPRESSION | IMMUNOLOGY | PRECURSOR CELLS | RECEPTOR-GAMMA-CHAIN
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2. Full Text CD8+ T Cell Immunity Against a Tumor/Self-Antigen Is Augmented by CD4+ T Helper Cells and Hindered by Naturally Occurring T Regulatory Cells
by Antony, Paul A and Piccirillo, Ciriaco A and Akpinarli, Akgul and Finkelstein, Steven E and Speiss, Paul J and Surman, Deborah R and Palmer, Douglas C and Chan, Chi-Chao and Klebanoff, Christopher A and Overwijk, Willem W and Rosenberg, Steven A and Restifo, Nicholas P
The Journal of Immunology, ISSN 0022-1767, 03/2005, Volume 174, Issue 5, pp. 2591 - 2601
CD4(+) T cells control the effector function, memory, and maintenance of CD8(+) T cells. Paradoxically, we found that absence of CD4(+) T cells enhanced... 
IN-VITRO | IMMUNOTHERAPY | INTERLEUKIN-2 DEFICIENT MICE | IMMUNOLOGICAL SELF-TOLERANCE | IL-2 | IMMUNOLOGY | HOMEOSTATIC PROLIFERATION | AUTOIMMUNE-DISEASE | CUTTING EDGE | ESTABLISHED TUMORS | ACUTE INFECTION | Cell Proliferation | Self Tolerance - immunology | Membrane Glycoproteins - biosynthesis | Interleukin-2 - therapeutic use | Neoplasm Proteins - immunology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | gp100 Melanoma Antigen | Interleukin-2 - physiology | Lymphocyte Activation - immunology | Melanoma, Experimental - immunology | Membrane Glycoproteins - immunology | Transplantation Tolerance - immunology | Tumor Escape - immunology | Immunotherapy, Adoptive - adverse effects | Immunotherapy, Adoptive - methods | Receptors, Interleukin-2 - biosynthesis | Neoplasm Proteins - biosynthesis | Melanoma, Experimental - therapy | Mice, Inbred C57BL | Homeostasis - immunology | Melanoma, Experimental - pathology | Mice, Transgenic | Immunity, Innate | Mice, Knockout | Animals | CD4-Positive T-Lymphocytes - transplantation | Mice | T-Lymphocytes, Regulatory - transplantation | CD8-Positive T-Lymphocytes - immunology
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3. Full Text Invariant NKT Cells Suppress CD8+ T-Cell–Mediated Allergic Contact Dermatitis Independently of Regulatory CD4+ T Cells
by Goubier, Anne and Vocanson, Marc and Macari, Claire and Poyet, Gaelle and Herbelin, André and Nicolas, Jean- François and Dubois, Bertrand and Kaiserlian, Dominique
Journal of Investigative Dermatology, ISSN 0022-202X, 04/2013, Volume 133, Issue 4, pp. 980 - 987
Invariant natural killer T (iNKT) cells expressing a CD1d-restricted invariant αβTCR have key regulatory roles in autoimmunity, pathogen immunity, and tumor... 
DENDRITIC CELLS | SENSITIVITY | ORAL TOLERANCE | DEFICIENT MICE | INKT-CELLS | INDUCTION | IL-13 | SKIN INFLAMMATION | LANGERHANS CELLS | HYPERSENSITIVITY | DERMATOLOGY | CD8-Positive T-Lymphocytes - cytology | Cell Communication - immunology | Dermatitis, Allergic Contact - immunology | T-Lymphocytes, Regulatory - metabolism | Coculture Techniques | Interferon-gamma - metabolism | Dermatitis, Allergic Contact - pathology | Dermatitis, Allergic Contact - metabolism | T-Lymphocytes, Regulatory - immunology | Interleukin-13 - genetics | Flow Cytometry | Forkhead Transcription Factors - metabolism | Natural Killer T-Cells - metabolism | T-Lymphocytes, Regulatory - cytology | CD8-Positive T-Lymphocytes - metabolism | Female | Interleukin-4 - genetics | Interleukin-4 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Natural Killer T-Cells - cytology | Interleukin-13 - metabolism | Antigens, CD1d - metabolism | Mice, Knockout | Animals | Down-Regulation - immunology | Mice | CD8-Positive T-Lymphocytes - immunology | CD4 Antigens - metabolism | Natural Killer T-Cells - immunology
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4. Full Text IL-4-producing CD4+ T cells in reactive lymph nodes during helminth infection are T follicular helper cells
by King, Irah L and Mohrs, Markus
Journal of Experimental Medicine, ISSN 0022-1007, 05/2009, Volume 206, Issue 5, pp. 1001 - 1007
Interleukin (IL)-4 is the quintessential T helper type 2 (Th2) cytokine produced by CD4(+) T cells in response to helminth infection. IL-4 not only promotes... 
CXC CHEMOKINE RECEPTOR-5 | MEDICINE, RESEARCH & EXPERIMENTAL | IN-VIVO | IL-4 RECEPTOR | GENERATION | DEFICIENT MICE | DIFFERENTIATION | INTERLEUKIN-21 | IMMUNOLOGY | ICOS | TH2 | LYMPHOCYTES | CD4 Antigens - immunology | RNA, Messenger - genetics | Th2 Cells - immunology | Lymph Nodes - immunology | CD4-Positive T-Lymphocytes - immunology | Interleukin-4 - immunology | Animals | B-Lymphocytes - immunology | Immunoglobulin E - immunology | Immunoglobulin G - immunology | Antibody Formation | Helminthiasis - immunology | T-Lymphocytes - immunology | Transcription, Genetic | Mice | Interleukin-4 - genetics | Brief Definitive Report
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5. Full Text Vascular niche E-selectin regulates hematopoietic stem cell dormancy, self renewal and chemoresistance
by Winkler, Ingrid G and Barbier, Valérie and Nowlan, Bianca and Jacobsen, Rebecca N and Forristal, Catherine E and Patton, John T and Magnani, John L and Lévesque, Jean-Pierre
Nature Medicine, ISSN 1078-8956, 11/2012, Volume 18, Issue 11, pp. 1651 - 1657
The microenvironment, or niche, surrounding a stem cell largely governs its cellular fate. Two anatomical niches for hematopoietic stem cells (HSCs) have been... 
PROGENITOR CELLS | CANCER-CELLS | MEDICINE, RESEARCH & EXPERIMENTAL | MOBILIZATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW NICHE | DEFICIENT MICE | CELL BIOLOGY | ADHESION MOLECULE-1 | LIGANDS | IN-VIVO | P-SELECTIN | ENDOTHELIAL SELECTINS | Hydroxyurea - pharmacology | Neutrophils - cytology | Radiation | Cell Lineage - drug effects | Neutrophils - radiation effects | E-Selectin - genetics | Cell Differentiation | Neutrophils - metabolism | Cell Survival - drug effects | Bone Marrow Cells - cytology | Endothelial Cells - metabolism | Mice, Inbred C57BL | Neutrophils - drug effects | Hematopoietic Stem Cell Transplantation | E-Selectin - metabolism | Hematopoietic Stem Cells - metabolism | Cell Survival - radiation effects | Cell Lineage - radiation effects | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Endothelial Cells - cytology | Gene Expression Regulation - radiation effects | Ligands | Cell Proliferation - drug effects | Mice | Bone Marrow Cells - metabolism | Cell Proliferation - radiation effects | Physiological aspects | Research | Hematopoietic stem cells | Cell adhesion molecules | Chemotherapy | Drug resistance | Vascular endothelial growth factor | Rodents | Stem cells
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6. Full Text Yes T cells, but three different T cells (αβ, γδ and NK T cells), and also B‐1 cells mediate contact sensitivity
by Askenase, P. W
Clinical & Experimental Immunology, ISSN 0009-9104, 09/2001, Volume 125, Issue 3, pp. 345 - 350
Transfer of contact sensitivity (CS) responses by immune lymphoid cells was the first finding that distinguished cellular from humoral immunity. CS has... 
γδ T cells | NK T cells | contact hypersensitivity | αβ T cells | γσ T cells | Contact hypersensitivity | INITIATING CELLS | COLLAGEN-INDUCED ARTHRITIS | B-LYMPHOCYTES | gamma delta T cells | MAST-CELLS | DEFICIENT MICE | IMMUNOLOGY | DELAYED-TYPE HYPERSENSITIVITY | ADOPTIVE TRANSFER | ELICITATION | alpha beta T cells | TRANSGENIC MICE | ANTIGEN | Models, Immunological | Dermatitis, Contact - etiology | Humans | Receptors, Antigen, T-Cell, alpha-beta | B-Lymphocytes | Killer Cells, Natural - immunology | Receptors, Antigen, T-Cell, gamma-delta | T-Lymphocytes - immunology | Immunity, Cellular | Immunoglobulin M - biosynthesis | B-1 cells
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7. Full Text Differences in the importance of mast cells, basophils, IgE, and IgG versus that of CD4+ T cells and ILC2 cells in primary and secondary immunity to Strongyloides venezuelensis
by Mukai, Kaori and Karasuyama, Hajime and Kabashima, Kenji and Kubo, Masato and Galli, Stephen J
Infection and Immunity, ISSN 0019-9567, 05/2017, Volume 85, Issue 5
There is evidence that mast cells, basophils, and IgE can contribute to immune responses to parasites; however, the relative levels of importance of these... 
Immunoglobulin E | Mast cells | Parasites | Basophils | INFECTIOUS DISEASES | FC-EPSILON-RI | BONE-MARROW-TRANSPLANTATION | C-KIT | basophils | DEFICIENT MICE | IMMUNOLOGY | mast cells | PROTECTIVE IMMUNITY | INTESTINAL HELMINTHS | TARGETED DISRUPTION | IN-VIVO | parasites | STEM-CELL | MHC CLASS-II | CD4-Positive T-Lymphocytes - immunology | Strongyloides - immunology | Mast Cells - immunology | Animals | Antibodies, Helminth - blood | Immunoglobulin G - blood | Mice, Inbred C57BL | Basophils - immunology | Mice, Inbred BALB C | Strongyloidiasis - immunology | Immunoglobulin E - blood | Disease Models, Animal
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8. Full Text APRIL is critical for plasmablast survival in the bone marrow and poorly expressed by early-life bone marrow stromal cells
by Belnoue, Elodie and Pihlgren, Maria and McGaha, Tracy L and Tougne, Chantal and Rochat, Anne-Frangoise and Bossen, Claudia and Schneider, Pascal and Huard, Bertrand and Lambert, Paul-Henri and Siegrist, Claire-Anne
Blood, ISSN 0006-4971, 03/2008, Volume 111, Issue 5, pp. 2755 - 2764
The persistence of serum IgG antibodies elicited in human infants is much shorter than when such responses are elicited later in life. The reasons for this... 
BAFF | MEMORY B-CELLS | MEASLES-VACCINE | CONJUGATE VACCINE | RATE IMMUNOGLOBULIN SECRETION | MYELOMA CELLS | HUMORAL IMMUNITY | TERMINAL DIFFERENTIATION | DEFICIENT MICE | ANTIBODY-RESPONSES | HEMATOLOGY | Tumor Necrosis Factor Ligand Superfamily Member 13 - immunology | Cell Line | Bone Marrow Cells - cytology | Cell Survival | Humans | RNA, Messenger - genetics | Gene Expression Regulation | Proteoglycans - metabolism | Stromal Cells - immunology | RNA, Messenger - metabolism | Animals | B-Cell Activating Factor - metabolism | Bone Marrow Cells - immunology | Protein Binding | B-Cell Activating Factor - immunology | Tumor Necrosis Factor Ligand Superfamily Member 13 - genetics | Mice | Plasma Cells - cytology | Stromal Cells - cytology | Plasma Cells - immunology
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9. Full Text Protein Kinase C-β-Dependent Activation of NF-κB in Stromal Cells Is Indispensable for the Survival of Chronic Lymphocytic Leukemia B Cells In Vivo
by Lutzny, Gloria and Kocher, Thomas and Schmidt-Supprian, Marc and Rudelius, Martina and Klein-Hitpass, Ludger and Finch, Andrew J and Dürig, Jan and Wagner, Michaela and Haferlach, Claudia and Kohlmann, Alexander and Schnittger, Susanne and Seifert, Marc and Wanninger, Stefan and Zaborsky, Nadja and Oostendorp, Robert and Ruland, Jürgen and Leitges, Michael and Kuhnt, Toni and Schäfer, Yvonne and Lampl, Benedikt and Peschel, Christian and Egle, Alexander and Ringshausen, Ingo
Cancer Cell, ISSN 1535-6108, 01/2013, Volume 23, Issue 1, pp. 77 - 92
Tumor cell survival critically depends on heterotypic communication with benign cells in the microenvironment. Here, we describe a survival signaling pathway... 
APOPTOSIS | RECRUITMENT | CLL | ONCOLOGY | MOUSE | MALIGNANCIES | DEGRADATION | DEFICIENT MICE | MODEL | EXPRESSION | CANCER | CELL BIOLOGY | Protein Kinase C - genetics | Protein Kinase C beta | Protein Kinase C - physiology | Cytokines - metabolism | Signal Transduction | Humans | Stromal Cells - metabolism | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Mice, Transgenic | NF-kappa B - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - pathology | Mice, Knockout | Animals | NF-kappa B - genetics | Protein Kinase C - metabolism | Leukemia, Lymphocytic, Chronic, B-Cell - metabolism | Mice | B-Lymphocytes - pathology | B-Lymphocytes - metabolism
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10. Full Text Effector and regulatory B cells: modulators of CD4 + T cell immunity
by Randall, Troy D and Lund, Frances E
Nature Reviews Immunology, ISSN 1474-1733, 04/2010, Volume 10, Issue 4, pp. 236 - 247
B cells are essential for humoral immunity, but the role that they have in regulating CD4(+) T cell responses remains controversial. However, new data showing... 
IDIOPATHIC THROMBOCYTOPENIC PURPURA | IFN-GAMMA PRODUCTION | LYMPHOTOXIN-BETA-RECEPTOR | LUPUS NEPHRITIS | NONOBESE DIABETIC MICE | IN-VIVO | CD40 LIGAND | ANTIGEN-PRESENTING CELLS | DEPLETION THERAPY | DEFICIENT MICE | IMMUNOLOGY | CD4-Positive T-Lymphocytes - immunology | B-Lymphocyte Subsets - immunology | Lymphocyte Activation - immunology | Animals | B-Lymphocytes - immunology | Humans
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11. Full Text Smooth Muscle Cell–Derived Interleukin-17C Plays an Atherogenic Role via the Recruitment of Proinflammatory Interleukin-17A+ T Cells to the Aorta
by Butcher, Matthew J and Waseem, Tayab C and Galkina, Elena V
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2016, Volume 36, Issue 8, pp. 1496 - 1506
OBJECTIVE—Atherosclerosis is characterized by frequent communication between infiltrating leukocytes and vascular cells, through chemokine and cytokine... 
atherosclerosis | smooth muscle cells | leukocytes | cytokines | inflammation | IL-17C | RECEPTOR | IMMUNE | HOST-DEFENSE | PERIPHERAL VASCULAR DISEASE | INFECTION | ATHEROSCLEROTIC PLAQUE | MANNER | HEMATOLOGY | E-DEFICIENT MICE | NF-KAPPA-B | Chemotaxis, Leukocyte | Inflammation - pathology | T-Lymphocytes, Regulatory - metabolism | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Myocytes, Smooth Muscle - pathology | Male | Aorta - metabolism | Aortic Diseases - metabolism | Inflammation - metabolism | Th17 Cells - metabolism | Diet, High-Fat | Aortic Diseases - prevention & control | Female | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Fibroblasts - metabolism | Th17 Cells - pathology | Atherosclerosis - pathology | Genetic Predisposition to Disease | Collagen Type I - metabolism | Paracrine Communication | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Interleukin-17 - genetics | Interleukin-17 - deficiency | Plaque, Atherosclerotic | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Interleukin-17 - metabolism | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Inflammation - genetics | Inflammation - prevention & control | Atherosclerosis - prevention & control
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