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by Jiang, Y and Li, WB and Lu, J and Zhao, X and Li, L
MEDICINE, ISSN 0025-7974, 06/2019, Volume 98, Issue 25, pp. e16067 - e16067
Journal Article
Cancer, ISSN 0008-543X, 08/2017, Volume 123, Issue 15, pp. 2927 - 2935
The results of the current study suggest that the B‐cell chronic lymphocytic leukemia/lymphoma (Bcl‐2)‐like 11 ( BCL2L11 ) (Bim ) deletion polymorphism is... 
ALK | crizotinib | BCL2L11 | B‐cell chronic lymphocytic leukemia/lymphoma (Bcl‐2)‐like 11 | anaplastic lymphoma kinase | Bim | non–small cell lung cancer | ROS proto‐oncogene 1 | receptor tyrosine kinase | ROS1 | receptor tyrosine kinase (ROS1) | B-cell chronic lymphocytic leukemia/lymphoma (Bcl-2)-like 11 (BCL2L11) (Bim) | ROS proto-oncogene 1 | anaplastic lymphoma kinase (ALK) | non-small cell lung cancer | SURVIVAL | APOPTOSIS | CHEMOTHERAPY | ALK REARRANGEMENT | TYROSINE KINASE INHIBITORS | GENE | ONCOLOGY | ASSOCIATION | EXPRESSION | DRIVER MUTATIONS | Lung Neoplasms - drug therapy | Pyrazoles - therapeutic use | Prognosis | Humans | Middle Aged | Asian Continental Ancestry Group - genetics | Male | Young Adult | Protein-Tyrosine Kinases - genetics | Gene Deletion | Aged, 80 and over | Adult | Female | Adenocarcinoma - genetics | Retrospective Studies | Pyridines - therapeutic use | Lung Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Proto-Oncogene Proteins - genetics | Bcl-2-Like Protein 11 - genetics | Adenocarcinoma - drug therapy | Polymorphism, Genetic | Disease-Free Survival | Drug Resistance, Neoplasm - genetics | Receptor Protein-Tyrosine Kinases - genetics | Protein Kinase Inhibitors - therapeutic use | Aged | Carcinoma, Non-Small-Cell Lung - drug therapy | Reactive oxygen species | Bcl-2 protein | Leukemia | Lung | Lung cancer | Cell fusion | Gene deletion | Multivariate analysis | Risk factors | Epidermal growth factor | Clonal deletion | Deletion | Protein-tyrosine kinase receptors | Protein-tyrosine kinase | Chromosomes | Tyrosine | Chronic lymphatic leukemia | Cell survival | Epidermal growth factor receptors | Non-small cell lung carcinoma | Lymphatic leukemia | Patients | Survival | Lymphoma | Polymerase chain reaction | Studies | Lymphocytes B | Response rates | Lymphomas | Mutation | BIM protein | Cancer | Polymorphism | Index Medicus | Abridged Index Medicus
Journal Article
The Plant Journal, ISSN 0960-7412, 09/2012, Volume 71, Issue 5, pp. 750 - 762
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 06/2017, Volume 23, Issue 12, pp. 3139 - 3149
Purpose: The BIM deletion polymorphism is associated with apoptosis resistance to EGFR tyrosine kinase inhibitors (EGFR-TKI), such as gefitinib and erlotinib,... 
NSCLC | ONCOLOGY | MESSENGER-RNA EXPRESSION | MUTATION | GROWTH | ACQUIRED-RESISTANCE | DIFFERENTIATION | ERLOTINIB | AZD9291 | CHEMOTHERAPY | TKI RESISTANCE | Carcinoma, Non-Small-Cell Lung - pathology | Piperazines - administration & dosage | Receptor, Epidermal Growth Factor - genetics | Sequence Deletion | Histone Deacetylases - genetics | Apoptosis - drug effects | Carcinoma, Non-Small-Cell Lung - genetics | Humans | Hydroxamic Acids - adverse effects | Histone Deacetylase Inhibitors - administration & dosage | Drug Resistance, Neoplasm | Bcl-2-Like Protein 11 - genetics | Piperazines - adverse effects | Protein Kinase Inhibitors - administration & dosage | Cell Line, Tumor | Hydroxamic Acids - administration & dosage | Histone Deacetylases - administration & dosage | Carcinoma, Non-Small-Cell Lung - drug therapy | Alternative splicing | Biotechnology | Histone deacetylase | Lung | Lung cancer | mRNA | Gene deletion | Kinases | Gene polymorphism | Risk factors | Western blotting | Clonal deletion | Xenografts | Deletion | Inhibition | Gefitinib | Protein-tyrosine kinase | Tyrosine | Splicing | Epidermal growth factor receptors | Tumor cells | Regression analysis | Gene expression | Polymerase chain reaction | Inhibitors | Experimental design | Cell lines | Mutation | BIM protein | Tumors | Cancer | Apoptosis | Polymorphism | Index Medicus
Journal Article
Journal Article
Science, ISSN 0036-8075, 7/2004, Volume 305, Issue 5683, pp. 525 - 528
Journal Article
Journal Article
Circulation, ISSN 0009-7322, 1995, Volume 92, Issue 6, pp. 1387 - 1388
Background An insertion (I)/deletion (D) polymorphism of the angiotensin-converting enzyme (ACE) gene has been associated with differences in the plasma levels... 
angiotensin | genetics | GENE | GENETICS | CARDIOMYOPATHY | SERUM | CARDIOVASCULAR SYSTEM | HEMATOLOGY | ANGIOTENSIN | EXPRESSION | Humans | Middle Aged | Child, Preschool | Genotype | Infant | Male | Polymorphism, Genetic | Peptidyl-Dipeptidase A - genetics | Myocardium - enzymology | Gene Deletion | Adolescent | Peptidyl-Dipeptidase A - metabolism | Adult | Female | Child | Index Medicus | Abridged Index Medicus
Journal Article