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Molecular Nutrition & Food Research, ISSN 1613-4125, 09/2019, Volume 63, Issue 17, pp. e1801039 - n/a
Green and black tea extracts can regulate the disturbances of Na + /K + ‐ATPase in the intestine to reduce the plasma glucose in type 1 diabetic mice and black... 
theaflavines | green tea | diabetes mellitus | black tea | Nutrient deficiency | Theaflavins | Diabetes mellitus | Pharmacology | Diabetes mellitus (insulin dependent) | Na+/K+-exchanging ATPase | Black tea | Chemical compounds | High fat diet | Monosaccharides | Tea | Hyperglycemia | Absorption | Sodium | Intestine | Disturbance | Low fat diet | Mice | Diabetes | Green tea
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 03/2015, Volume 290, Issue 11, pp. 6763 - 6776
Journal Article
Genes and Development, ISSN 0890-9369, 10/2003, Volume 17, Issue 20, pp. 2552 - 2563
Journal Article
1995, Molecular biology intelligence unit (Unnumbered), ISBN 9781570591150, 226
Book
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2012, Volume 287, Issue 39, pp. 32717 - 32727
Journal Article
BBA - General Subjects, ISSN 0304-4165, 07/2016, Volume 1860, Issue 7, pp. 1431 - 1438
The natriuretic effect of uroguanylin (UGN) involves reduction of proximal tubule (PT) sodium reabsorption. However, the target sodium transporters as well as... 
Proximal tubule | Sodium pump | Uroguanylin | Sodium reabsorption | Cell signaling | Kidney | MAMMALIAN TARGET | ATPASE ACTIVITY | PROTEIN-KINASE | MTOR COMPLEXES | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUBULOGLOMERULAR FEEDBACK | CONVOLUTED TUBULE | NATRIURETIC-PEPTIDE | BIOPHYSICS | PROXIMAL TUBULE CELLS | HYPERTENSIVE-RATS | RENAL FANCONI SYNDROME | Natriuretic Agents - pharmacology | Phosphorylation | TOR Serine-Threonine Kinases - metabolism | Sodium - metabolism | Cyclic GMP-Dependent Protein Kinases - metabolism | Dose-Response Relationship, Drug | LLC-PK1 Cells | TOR Serine-Threonine Kinases - antagonists & inhibitors | Cyclic GMP-Dependent Protein Kinases - antagonists & inhibitors | Renal Reabsorption - drug effects | Swine | Adenosine Triphosphate - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Kidney Tubules, Proximal - enzymology | Cyclic AMP - metabolism | Cyclic AMP-Dependent Protein Kinases - metabolism | Second Messenger Systems - drug effects | Natriuresis - drug effects | Renal Elimination - drug effects | Natriuretic Peptides - pharmacology | Hydrolysis | Sodium-Potassium-Exchanging ATPase - metabolism | Animals | Cyclic GMP - metabolism | Protein Kinase Inhibitors - pharmacology | Enzyme Activation | Kidney Tubules, Proximal - drug effects | Adenosine triphosphatase
Journal Article
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, ISSN 0892-6638, 07/2019, Volume 33, Issue 9, pp. fj201900445R - 10206
Journal Article
QUARTERLY REVIEWS OF BIOPHYSICS, ISSN 0033-5835, 11/2010, Volume 43, Issue 4, pp. 501 - 566
The sarcoplasmic (SERCA 1a) Ca2+-ATPase is a membrane protein abundantly present in skeletal mucles where it functions as an indispensable component of the... 
CA2+ TRANSPORT ATPASE | ADP-INSENSITIVE PHOSPHOENZYME | BIOPHYSICS | P-TYPE ATPASES | TRANSMEMBRANE SEGMENTS 6 | RETICULUM CALCIUM-PUMP | DEPENDENT ADENOSINE-TRIPHOSPHATASE | NUCLEAR MAGNETIC-RESONANCE | NUCLEOTIDE-BINDING SITE | UNCOUPLED ATP HYDROLYSIS | CATALYZING OXIDATIVE PHOSPHORYLATION
Journal Article
BIOPOLYMERS, ISSN 0006-3525, 08/2016, Volume 105, Issue 8, pp. 483 - 491
Myosins are a large family of molecular motors that use the common P-loop, Switch 1 and Switch 2 nucleotide binding motifs to recognize ATP, to create a... 
MOLECULAR-MECHANISM | DOMAIN | STRUCTURAL MECHANISM | ACTIN | MOTOR-ACTIVITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | LOAD-DEPENDENT KINETICS | REAL-TIME DETECTION | BIOPHYSICS | transition states | ATPase mechanism | ENERGY TRANSDUCTION | POWER STROKE | molecular motor | MUSCLE-CONTRACTION
Journal Article
BBA - Bioenergetics, ISSN 0005-2728, 08/2016, Volume 1857, Issue 8, pp. 1167 - 1182
In this contribution we summarize most of the findings reported for the molecular and cellular biology of the physiological inhibitor of the mitochondrial H... 
ATPase Inhibitory Factor 1 | ATP synthase | Mitochondria | Nuclear reprogramming | ROS signaling | Cancer | RAT-LIVER | CANCER-CELLS | RESPIRATORY-CHAIN | OXIDATIVE-PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | BETA-F1-ATPASE MESSENGER-RNA | DEPENDENT PROTEIN-KINASE | CYTOCHROME-C-OXIDASE | BIOPHYSICS | BIOENERGETIC SIGNATURE | ELECTRON-TRANSPORT | MITOCHONDRIAL ADENOSINE-TRIPHOSPHATASE | Neoplasms - metabolism | Protons | Phosphorylation | Reactive Oxygen Species - metabolism | Humans | Cyclic AMP-Dependent Protein Kinase Catalytic Subunits - genetics | Protein Subunits - metabolism | Cellular Reprogramming | Hypoxia - metabolism | Cyclic AMP-Dependent Protein Kinase Catalytic Subunits - metabolism | Mitochondrial Proton-Translocating ATPases - genetics | Neoplasms - genetics | Cell Transformation, Neoplastic - genetics | Protein Subunits - genetics | Signal Transduction | Gene Expression Regulation | Oxidative Phosphorylation | Mitochondria - metabolism | Mitochondria - pathology | Cell Transformation, Neoplastic - metabolism | Serine - metabolism | Mitochondrial Proton-Translocating ATPases - metabolism | Proteins - genetics | Hypoxia - genetics | Proteins - metabolism | Hypoxia - pathology | Cell Transformation, Neoplastic - pathology | Neoplasms - pathology | Physiological aspects | Cell death | Adenosine triphosphatase
Journal Article