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Journal of Neurochemistry, ISSN 0022-3042, 12/2010, Volume 115, Issue 6, pp. 1421 - 1433
J. Neurochem. (2010) 115, 1421–1433. Type I interferons (IFNs) are known to cause neuropsychiatric side effects, which have been proposed to be mediated by... 
JAK–STAT signaling | apoptosis | PI3K/Akt pathway | human neuroblastoma cells | PKR | type I interferons | JAK-STAT signaling | PHOSPHATIDYLINOSITOL 3-KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DEPENDENT PROTEIN-KINASE | ALPHA | Akt pathway | NEUROSCIENCES | CYTOKINE-INDUCED DEPRESSION | PI3K | GROWTH | DEPHOSPHORYLATION | PROMOTES SURVIVAL | CENTRAL-NERVOUS-SYSTEM | INHIBITOR | INSULIN-RECEPTOR SUBSTRATE-1 | STAT1 Transcription Factor - physiology | Phosphatidylinositol 3-Kinase - physiology | Phosphatidylinositol 3-Kinase - antagonists & inhibitors | Humans | Neuroblastoma - enzymology | Interferon-beta - physiology | Janus Kinases - metabolism | Proto-Oncogene Proteins c-akt - physiology | Down-Regulation - physiology | STAT1 Transcription Factor - metabolism | Animals | Janus Kinases - physiology | Cell Line, Tumor | Signal Transduction - physiology | Mice | Neuroblastoma - metabolism | Apoptosis - physiology | Neuroblastoma - pathology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Index Medicus | eIF-2 kinase | Phosphorylation | Tyk2 protein | Transcription | AKT protein | Glial cells | Caspase-3 | Neuroblastoma cells | Signal transduction | Neurotoxicity | Ribose | DNA fragmentation | Janus kinase | Stat1 protein | Tyrosine | Cell survival | Cytokines | Neurons | 1-Phosphatidylinositol 3-kinase | Cytochrome c | Side effects | beta -Interferon | Protein kinase | Caspase-9 | Interferon | Apoptosis
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European Journal of Pharmaceutical Sciences, ISSN 0928-0987, 05/2017, Volume 102, pp. 220 - 229
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