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Blood, ISSN 0006-4971, 04/2014, Volume 123, Issue 16, pp. 2562 - 2572
Loss of inhibitor of apoptosis proteins (IAPs), particularly cIAP1, can promote production of tumor necrosis factor (TNF) and sensitize cancer cell lines to... 
RIP1 KINASE | ACTIVATION | NECROSIS | NECROPTOSIS | ALPHA-DEPENDENT APOPTOSIS | CASPASE-8 | TNF-ALPHA | ANTAGONISTS | HEMATOLOGY | NF-KAPPA-B | CELL-DEATH | Index Medicus | Abridged Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 03/2018, Volume 67, Issue 3, pp. 437 - 447
Polymorphism in TCF7L2, a component of the canonical Wnt signaling pathway, has a strong association with beta-cell dysfunction and type 2 diabetes through a... 
VARIANTS | GLUCOSE-METABOLISM | CLONING | TRANSCRIPTION-FACTOR-7-LIKE-2 TCF7L2 GENE | INSULIN-SECRETION | MOUSE | ENDOCRINOLOGY & METABOLISM | RISK | EXPRESSION | BETA-CATENIN/TCF-4 | DELETION | Glucose Intolerance - metabolism | Recombinant Proteins - therapeutic use | Bone Morphogenetic Protein 4 - genetics | Bacterial Proteins - chemistry | Male | Glucose Intolerance - pathology | Bone Morphogenetic Protein 4 - metabolism | Recombinant Fusion Proteins - metabolism | Transcription Factor 7-Like 2 Protein - metabolism | Glucose Intolerance - drug therapy | Transcription Factor 7-Like 2 Protein - chemistry | Insulin-Secreting Cells - metabolism | Pericytes - pathology | Luminescent Proteins - chemistry | Cell Differentiation | Insulin Secretion | Glucose Intolerance - physiopathology | Recombinant Proteins - metabolism | Bone Morphogenetic Protein 4 - therapeutic use | Transcription Factor 7-Like 2 Protein - genetics | Tissue Culture Techniques | Pericytes - metabolism | Bacterial Proteins - genetics | Gene Expression Regulation | Cell Communication | Cellular Microenvironment | Mice, Transgenic | Recombinant Fusion Proteins - chemistry | Insulin - metabolism | Animals | Glucose - metabolism | Bacterial Proteins - metabolism | Ligands | Luminescent Proteins - genetics | Insulin-Secreting Cells - pathology | Mutation | Luminescent Proteins - metabolism | Usage | Genetic aspects | Research | B cells | Diabetes | Diagnosis | Glucose tolerance tests | Genetic polymorphisms | Bone morphogenetic protein 4 | Wnt protein | Secretion | Genes | Transgenic mice | Insulin | Glucose tolerance | Beta cells | Signal transduction | Rodents | Pericytes | Islets of Langerhans | Pancreas | Diabetes mellitus (non-insulin dependent) | Polymorphism | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal Article
Aging Cell, ISSN 1474-9718, 10/2017, Volume 16, Issue 5, pp. 956 - 965
Emerging evidence suggests that cell senescence plays an important role in aging‐associated diseases including neurodegenerative diseases. HIV leads to a... 
senescence | HIV | β‐catenin | methamphetamine | astrocyte | β-catenin | beta-catenin | ALZHEIMER-DISEASE | ANTAGONIST | CELL SENESCENCE | SCHIZOPHRENIA | DISORDERS | P16(INK4A) | CELL BIOLOGY | GERIATRICS & GERONTOLOGY | TRANSPORT | EXPRESSION | METHAMPHETAMINE USERS | PARKINSONS-DISEASE | Neurons - pathology | HIV-1 - pathogenicity | Methamphetamine - adverse effects | Humans | Astrocytes - pathology | Male | Substance-Related Disorders - genetics | DNA-Binding Proteins - metabolism | HIV-1 - physiology | Central Nervous System Stimulants - adverse effects | AIDS Dementia Complex - complications | Neurons - metabolism | Astrocytes - virology | Disease Models, Animal | AIDS Dementia Complex - physiopathology | Cellular Senescence - genetics | Neurons - virology | Substance-Related Disorders - metabolism | Signal Transduction | Animals, Genetically Modified | Gene Expression Regulation | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Rats | DNA-Binding Proteins - genetics | Substance-Related Disorders - physiopathology | beta Catenin - metabolism | Substance-Related Disorders - complications | beta Catenin - genetics | AIDS Dementia Complex - genetics | AIDS Dementia Complex - metabolism | Animals | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Mice | Primary Cell Culture | Astrocytes - metabolism | Index Medicus | Original
Journal Article
Journal Article
Diabetes, Obesity and Metabolism, ISSN 1462-8902, 07/2017, Volume 19, Issue 7, pp. 997 - 1005
Aim: To test the hypothesis that, given the role of AMP-activated protein kinase (AMPK) in regulating intracellular ATP levels, AMPK may alter ATP release from... 
A | GT1 | INS | 1 | 2 | C2C12 | cortical astrocytes | BV | 769662 | H4IIE | 7 | hypothalamic astrocytes | AMPK | intracellular calcium | ATP | U373 | SH‐SY5Y | GT1-7 | SH-SY5Y | BV-2 | A-769662 | INS-1 | EXTRACELLULAR ATP | RAT | FOOD-INTAKE | DEPENDENT RELEASE | KAPPA-B | OBESITY | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | GLUCOSE-UPTAKE | RECEPTORS | HYPOTHALAMUS | Enzyme Activators - pharmacology | Hypothalamus - secretion | AMP-Activated Protein Kinases - metabolism | Humans | Adenosine Triphosphate - secretion | Cerebral Cortex - cytology | Pyrones - pharmacology | Cerebral Cortex - metabolism | Protein Processing, Post-Translational - drug effects | Adenosine Triphosphate - metabolism | Cerebral Cortex - drug effects | Hypothalamus - drug effects | Phosphorylation - drug effects | Animals, Newborn | Astrocytes - cytology | Astrocytes - drug effects | Cell Line | Nerve Tissue Proteins - agonists | Cells, Cultured | Thiophenes - pharmacology | Enzyme Activation - drug effects | Nerve Tissue Proteins - genetics | AMP-Activated Protein Kinases - chemistry | Hypoglycemic Agents - pharmacology | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Hypothalamus - metabolism | Hypothalamus - cytology | Calcium Signaling - drug effects | Embryo, Mammalian - cytology | Mice | Cerebral Cortex - secretion | AMP-Activated Protein Kinases - genetics | Acetyl-CoA Carboxylase - metabolism | Acetyl-CoA Carboxylase - chemistry | Astrocytes - metabolism | Astrocytes - secretion | Muscle proteins | Protein kinases | Analysis | Kinases | Rodents | Index Medicus
Journal Article