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Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 06/2013, Volume 33, Issue 6, pp. 1401 - 1408
Journal Article
Circulation Research, ISSN 0009-7330, 08/2008, Volume 103, Issue 5, pp. 545 - 553
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 10/2011, Volume 301, Issue 4, pp. H1341 - H1352
Journal Article
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 01/2018, Volume 495, Issue 1, pp. 41 - 45
Journal Article
Hypertension, ISSN 0194-911X, 02/2015, Volume 65, Issue 2, pp. 385 - 392
Nitroxyl (HNO), the reduced and protonated form of nitric oxide (NO·), confers unique physiological effects including vasorelaxation and enhanced cardiac... 
vasodilation | nitrogen oxides | guanylate cyclase | pharmacology | cardiotonic agents | cyclic GMP-dependent protein kinases | cardiovascular physiological phenomena | RESISTANCE ARTERIES | PROTEIN-KINASE | ISOPROPYLAMINE-NONOATE | CARDIAC-MUSCLE | ANGELIS SALT | CENTER-DOT | CORONARY VASCULATURE | PERIPHERAL VASCULAR DISEASE | TROPONIN-I PHOSPHORYLATION | CONTRACTILE FUNCTION | NITRIC-OXIDE NO | Cyclic GMP - physiology | Receptors, Cytoplasmic and Nuclear - deficiency | Myocardial Contraction - physiology | Myocardial Contraction - drug effects | Cyclic GMP-Dependent Protein Kinase Type I - genetics | Muscle, Smooth, Vascular - physiopathology | Guanylate Cyclase - physiology | Myocardium - metabolism | Cyclic GMP-Dependent Protein Kinase Type I - deficiency | Nitric Oxide Donors - pharmacology | Vasodilation - physiology | Cyclic GMP-Dependent Protein Kinase Type I - chemistry | Oxidation-Reduction | Aorta - drug effects | Mice, Inbred C57BL | Nitric Oxide - physiology | Receptors, Cytoplasmic and Nuclear - physiology | Soluble Guanylyl Cyclase | Receptors, Cytoplasmic and Nuclear - genetics | Cardiotonic Agents - pharmacology | Cysteine - chemistry | Sulfonamides - pharmacology | Mice, Knockout | Guanylate Cyclase - deficiency | Animals | Nitrogen Oxides - pharmacology | Guanylate Cyclase - genetics | Second Messenger Systems - physiology | Mice | Vasodilation - drug effects | Index Medicus | cardiovascular physiology | contractility
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 03/2012, Volume 46, Issue 3, pp. 372 - 379
As powerful vasodilators, prostacyclin analogues are presently the mainstay in the treatment of severe pulmonary arterial hypertension. Although the... 
Prostacyclin | Pulmonary circulation | Potassium channels | Vascular smooth muscle | PPAR | CELLS | vascular smooth muscle | prostacyclin | pulmonary circulation | BIOCHEMISTRY & MOLECULAR BIOLOGY | HYPOXIA | PPARS | BETA | CELL BIOLOGY | VESSELS | RESPIRATORY SYSTEM | potassium channels | ARTERIAL SMOOTH-MUSCLE | HYPERTENSION | EXPRESSION | Receptors, Epoprostenol | Iloprost - pharmacology | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Humans | Male | PPAR gamma - metabolism | Receptors, Prostaglandin - metabolism | Potassium Channels, Calcium-Activated - genetics | Dose-Response Relationship, Drug | Pulmonary Artery - metabolism | Potassium Channels, Calcium-Activated - metabolism | Epoprostenol - pharmacology | Myocytes, Smooth Muscle - metabolism | PPAR delta - genetics | Muscle, Smooth, Vascular - drug effects | PPAR gamma - genetics | Cyclic AMP-Dependent Protein Kinases - metabolism | Potassium Channels, Tandem Pore Domain - metabolism | Receptors, Prostaglandin - drug effects | Vasodilator Agents - pharmacology | Cells, Cultured | Gene Silencing | Potassium Channels, Tandem Pore Domain - genetics | Rats | Pulmonary Artery - drug effects | Nerve Tissue Proteins - drug effects | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Potassium Channels, Tandem Pore Domain - drug effects | Epoprostenol - analogs & derivatives | Animals | Membrane Potentials | Signal Transduction - drug effects | PPAR delta - agonists | Potassium Channels, Calcium-Activated - drug effects | PPAR gamma - agonists | PPAR delta - metabolism | Vasodilation - drug effects | Index Medicus
Journal Article
Journal Article