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Synergy, ISSN 2213-7130, 12/2018, Volume 7, pp. 10 - 16
There are many efforts to diminish risks associated with the use of cancer chemotherapeutic agents through design and utilization of new strategies including... 
siRNA | Breast cancer | dff45 | Methotrexate | Autophagy | Apoptosis
Journal Article
Journal Article
BMB Reports, ISSN 1976-6696, 2009, Volume 42, Issue 11, p. 713
Journal Article
BMC Cancer, ISSN 1471-2407, 04/2018, Volume 18, Issue 1, pp. 418 - 418
Background: The expression of DNA fragmentation factor 45 (DFF45) and B-cell lymphoma 2 (BCL2) in glands of the normal human endometrium is related to phases... 
B-cell lymphoma 2 (BCL2) | DNA fragmentation factor 40 (DFF40) | Overall survival (OS) | Disease-free survival (DFS) | DNA fragmentation factor 45 (DFF45) | Endometrial cancers (ECs) | RISK-FACTORS | MELF-PATTERN | P53 EXPRESSION | EXPRESSION PROFILES | NODE METASTASIS | ONCOLOGY | BURKITTS-LYMPHOMA | POOR-PROGNOSIS | POSTMENOPAUSAL WOMEN | CARCINOMA | LYMPHOVASCULAR SPACE INVASION | Immunohistochemistry | Reproducibility of Results | Lymph Nodes - pathology | Poly-ADP-Ribose Binding Proteins - metabolism | Endometrial Neoplasms - mortality | Humans | Kaplan-Meier Estimate | Neoplasm Recurrence, Local | Proportional Hazards Models | Poly-ADP-Ribose Binding Proteins - genetics | Biomarkers, Tumor | Deoxyribonucleases - metabolism | Apoptosis Regulatory Proteins - metabolism | Deoxyribonucleases - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Neoplasm Grading | Endometrial Neoplasms - genetics | Apoptosis Regulatory Proteins - genetics | Endometrial Neoplasms - pathology | Female | Neoplasm Staging | Proto-Oncogene Proteins c-bcl-2 - genetics | Care and treatment | B cells | Research | Diagnosis | Endometrial cancer | Gene expression | Medical research | Prognosis | Patient outcomes | Menopause | Postmenopausal women | Non-Hodgkin's lymphomas | Diseases | Proteins | Analysis | DNA | Medicine, Experimental | Genetic research | Genetic aspects | Cancer | Index Medicus
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e57871 - e57871
We previously showed that DNA fragmentation factor, which comprises a caspase-3-activated DNase (CAD) and its inhibitor (ICAD), may influence the rate of cell... 
POLY(ADP-RIBOSE) POLYMERASE PARP | FACTOR DFF45 | ENDONUCLEASE | EPITHELIAL-CELLS | MULTIDISCIPLINARY SCIENCES | CASPASE-ACTIVATED DNASE | TUMOR-SUPPRESSOR | BUTYRATE | TNF-ALPHA | DNA-FRAGMENTATION FACTOR | EXPRESSION | Genomic Instability | Colon - enzymology | Colonic Neoplasms - genetics | Epithelial Cells - metabolism | Deoxyribonucleases - deficiency | Humans | Apoptosis - genetics | Colonic Neoplasms - metabolism | Deoxyribonucleases - metabolism | Genetic Predisposition to Disease | Colon - pathology | Mice, Inbred C57BL | Carcinogenesis - genetics | Epithelial Cells - pathology | Colon - metabolism | Carcinogenesis - pathology | Deoxyribonucleases - genetics | Poly(ADP-ribose) Polymerases - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | Colonic Neoplasms - pathology | Colonic Neoplasms - enzymology | Mice | DNA Damage | Poly (ADP-Ribose) Polymerase-1 | Immunohistochemistry | Ionizing radiation | Colon cancer | Genetically modified organisms | Cytogenetics | Development and progression | Genetic aspects | Tumor proteins | Risk factors | Tumors | Cancer | Apoptosis | Health sciences | Biotechnology | Laboratories | Epithelial cells | p53 Protein | DNA damage | Colorectal cancer | Raf protein | Malignancy | Hybridization | Caspase-3 | Carcinogenesis | Genomic instability | Animal reproduction | Carcinogens | Rodents | Animal tissues | DNA fragmentation | Fibroblasts | Tumorigenesis | Colon | Deoxyribonucleic acid--DNA | Deoxyribonuclease | Stability | Mortality | Poly(ADP-ribose) polymerase | Caspase | Stability analysis | Gene expression | Radiation dosage | Pathology | Poly(ADP-ribose) Polymerase 1 | Cell death | Irradiation | Mutation | Aberration | Radiation damage | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
BBA - General Subjects, ISSN 0304-4165, 01/2014, Volume 1840, Issue 1, pp. 191 - 198
Journal Article
Journal of Diabetes Investigation, ISSN 2040-1116, 07/2018, Volume 9, Issue 4, pp. 741 - 752
CIDEC suppressed the expression of AMPK in lung tissue and iuduced pulmonary vascular remodeling in diabetes, and the silence of CIDEC gene reversed this... 
45‐like effector C | Pulmonary vascular remodeling | Cell death‐inducing | Diabetes | DFF | Cell death-inducing DFF45-like effector C | METABOLIC SYNDROME | ACTIVATED PROTEIN-KINASE | MUSCLE-CELLS | CIDEC/FSP27 | INFLAMMATION | ARTERIAL-HYPERTENSION | ENDOCRINOLOGY & METABOLISM | AMPK | DEGRADATION | EXPRESSION | ADIPOSE-TISSUE | Protein Kinases - metabolism | Signal Transduction | Diabetes Mellitus, Type 2 - genetics | Gene Silencing | Male | Rats, Sprague-Dawley | Proteins - genetics | Pulmonary Artery - metabolism | Animals | Vascular Remodeling - genetics | Lung - metabolism | Diabetes Mellitus, Type 2 - pathology | Diabetes Mellitus, Type 2 - complications | Pulmonary Artery - pathology | Apoptosis | Disease Models, Animal | Proteins | Immunohistochemistry | Type 2 diabetes | Blood circulation disorders | RNA | Cell death | Adenoviruses | Genes | Insulin resistance | Genetic engineering | Protein kinases | Cell proliferation | Arterioles | Smooth muscle | Kinases | Western blotting | Signal transduction | Vascular diseases | Ribose | C gene | Rodents | Proliferating cell nuclear antigen | Diabetes mellitus (non-insulin dependent) | Jugular vein | Antigens | Adenosine | AMP | Pulmonary arteries | Lung diseases | Diabetes mellitus | Caspase | Insulin | Pulmonary artery | Endothelial cells | DNA nucleotidylexotransferase | Gene silencing | Protein kinase | Metabolic disorders | Index Medicus | Cell death‐inducing DFF45‐like effector C | Original
Journal Article