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Nature reviews cardiology, ISSN 1759-5010, 2009, Volume 6, Issue 6, pp. 399 - 409
..., which interact with one another and with cells of the arterial wall. The pathological mechanisms of obesity recapitulate many features of the inflammatory processes at work in atherosclerosis... 
C-REACTIVE PROTEIN | RECEPTOR-DEFICIENT MICE | DIET-INDUCED ATHEROSCLEROSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | E-KNOCKOUT MICE | LOW-DENSITY-LIPOPROTEIN | NECROSIS-FACTOR-ALPHA | SMOOTH-MUSCLE-CELLS | RANDOMIZED CONTROLLED-TRIAL | CORONARY-HEART-DISEASE | INDUCED INSULIN-RESISTANCE | Inflammation - pathology | Prognosis | Obesity - drug therapy | Obesity - immunology | Humans | Atherosclerosis - etiology | Inflammation - metabolism | T-Lymphocytes - metabolism | Inflammation - drug therapy | Inflammation Mediators - metabolism | Anti-Inflammatory Agents - therapeutic use | Adipocytes - immunology | Macrophages - immunology | Atherosclerosis - pathology | Atherosclerosis - drug therapy | Atherosclerosis - immunology | Risk Assessment | Obesity - complications | Risk Factors | Insulin Resistance | Rupture | Lipid Metabolism | Inflammation - immunology | Immunity, Innate | Inflammation - etiology | Atherosclerosis - metabolism | Obesity - metabolism | Obesity - pathology | Cardiovascular Agents - therapeutic use | Macrophages - metabolism | Animals | Adipocytes - metabolism | T-Lymphocytes - immunology | Apoptosis | Immunologic Factors - therapeutic use | Obesity | Care and treatment | Inflammation | Research | Macrophages | T cells | Risk factors | Complications and side effects | Atherosclerosis | Physiological aspects | Diagnosis | Health aspects
Journal Article
Nature medicine, ISSN 1546-170X, 2013, Volume 20, Issue 1, pp. 54 - 61
Journal Article
Journal Article
Journal Article
Hypertension (Dallas, Tex. 1979), ISSN 0194-911X, 01/2005, Volume 45, Issue 1, pp. 9 - 14
Obesity is strongly associated with hypertension and cardiovascular disease. Several central and peripheral abnormalities that can explain the development or maintenance of high arterial pressure in obesity have been identified... 
Nervous system, sympathetic renal | Kidney | Hypertension, obesity | Vasculature | vasculature | LEPTIN SENSITIVITY | OXIDATIVE STRESS | FOOD-INTAKE | VISCERAL OBESITY | nervous system, sympathetic renal | DIET-INDUCED OBESITY | BLOOD-PRESSURE | hypertension, obesity | kidney | PLASMA GHRELIN | TYROSINE-PHOSPHATASE 1B | INSULIN-RESISTANCE | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | Humans | Sympathetic Nervous System - physiopathology | Suppressor of Cytokine Signaling Proteins | Adiponectin | Hypertension - etiology | Intercellular Signaling Peptides and Proteins - physiology | Leptin - physiology | Repressor Proteins - physiology | Ghrelin | Mice, Mutant Strains | Mineralocorticoid Receptor Antagonists - pharmacology | Appetite - physiology | Receptors, Cell Surface - physiology | Energy Metabolism - physiology | Peptide Hormones - physiology | Kidney - physiopathology | Hyperinsulinism - physiopathology | Receptors, Leptin | Transcription Factors - physiology | Obesity - complications | Aldosterone - physiology | Endothelium, Vascular - physiopathology | Obesity - physiopathology | Renin-Angiotensin System - physiology | Peptide YY - physiology | Hypertension - physiopathology | Satiation - physiology | Suppressor of Cytokine Signaling 3 Protein | Animals | Mice
Journal Article
Gut, ISSN 0017-5749, 03/2017, Volume 66, Issue 3, pp. 429 - 437
ObjectiveThe gut microbiota has been implicated as an environmental factor that modulates obesity, and recent evidence suggests that microbiota-mediated changes in bile acid profiles and signalling... 
Bile acid | obesity | FXR | BILE-ACID METABOLISM | EXTRACTION | INSULIN-RESISTANCE | MOUSE | LIVER | FAT | GUT MICROBIOTA | DIET-INDUCED OBESITY | GASTROENTEROLOGY & HEPATOLOGY | NUCLEAR RECEPTOR | Male | Cecum - microbiology | Macrophages | Obesity - microbiology | Feces - microbiology | Dietary Fats - administration & dosage | Weight Gain | Gene Expression | Fatty Liver - metabolism | Obesity - complications | Mice, Inbred C57BL | Adipose Tissue - pathology | Bile Acids and Salts - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Gastrointestinal Microbiome - genetics | Inflammation - etiology | Fecal Microbiota Transplantation | Mice, Knockout | Obesity - metabolism | Phenotype | Animals | Germ-Free Life | Glucose - metabolism | Mice | Insulin-Secreting Cells - pathology | Fatty Liver - etiology | Receptors, Cytoplasmic and Nuclear - metabolism | Obesity | Research | Microbiota (Symbiotic organisms) | Bile acid metabolism | OBESITY | 1506 | Gut Microbiota | BILE ACID | microbiota | dtvedt t | gut | p1341 | Kardiologi | mouse | p497 | 1974 | Endokrinologi och diabetes | vldl receptor | 1957 | Endocrinology and Diabetes | bile-acid metabolism | fxr | american journal of clinical nutrition | fatty liver-disease | extraction | diet-induced obesity | journal of biological chemistry | Gastroenterology & Hepatology | Cardiac and Cardiovascular Systems | insulin-resistance | v226 | nuclear receptor | v27 | lch j
Journal Article