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1. Full Text Distal convoluted tubule
by Subramanya, Arohan R and Ellison, David H
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 2014, Volume 9, Issue 12, pp. 2147 - 2163
The distal convoluted tubule is the nephron segment that lies immediately downstream of the macula densa. Although short in length, the distal convoluted... 
NA-CL COTRANSPORTER | FAMILIAL HYPERKALEMIC HYPERTENSION | CORTICAL COLLECTING DUCT | RETICULUM-ASSOCIATED DEGRADATION | PSEUDOHYPOALDOSTERONISM TYPE-II | CELL-SURFACE EXPRESSION | SENSORINEURAL DEAFNESS | UROLOGY & NEPHROLOGY | KELCH-LIKE 3 | SODIUM-CHLORIDE COTRANSPORTER | RECTIFIER K+ CHANNEL | Potassium - metabolism | Signal Transduction | Calcium - metabolism | Kidney Tubules, Distal - physiology | Humans | Magnesium - metabolism | Epithelial Sodium Channels - metabolism | Renal Reabsorption - physiology | Sodium - metabolism | Cations, Divalent - metabolism | Kidney Tubules, Distal - anatomy & histology | Potassium Channels, Inwardly Rectifying - metabolism | Sodium Chloride - metabolism | Protein-Serine-Threonine Kinases - metabolism | Solute Carrier Family 12, Member 3 - metabolism | Kidney Tubules, Distal - metabolism | distal tubule | mineral metabolism | Renal Physiology | Na transport | potassium channels
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2. Full Text Initial FGF23-mediated signaling occurs in the distal convoluted tubule
by Farrow, Emily G and Davis, Siobhan I and Summers, Lelia J and White, Kenneth E
Journal of the American Society of Nephrology, ISSN 1046-6673, 05/2009, Volume 20, Issue 5, pp. 955 - 960
Fibroblast growth factor-23 (FGF23), a hormone central to phosphate and vitamin D metabolism, reduces renal absorption of phosphate by downregulating the... 
OSTEOMALACIA | PHOSPHATE | HYPERPHOSPHATEMIA | GROWTH | MUTATION | UROLOGY & NEPHROLOGY | PHYSIOLOGICAL-ROLE | ABLATION | KLOTHO GENE | MICE | EXPRESSION | Cell Line | Kidney Tubules, Distal - enzymology | Kidney Tubules, Distal - physiology | Humans | Mice, Inbred C57BL | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Fibroblast Growth Factors - pharmacology | Sodium-Phosphate Cotransporter Proteins - physiology | Recombinant Proteins - pharmacology | DNA Primers | Receptor, Fibroblast Growth Factor, Type 1 - genetics | Reverse Transcriptase Polymerase Chain Reaction | Kidney | Actins - genetics | Glucuronidase - physiology | Animals | Early Growth Response Protein 1 - genetics | Signal Transduction - physiology | Mice | Fibroblast Growth Factors - physiology | Brief Communications
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3. Full Text KCNJ10 determines the expression of the apical Na-Cl cotransporter (NCC) in the early distal convoluted tubule (DCT1)
by Chengbiao Zhang and Lijun Wang and Junhui Zhang and Xiao-Tong Su and Dao-Hong Lin and Ute I. Scholl and Gerhard Giebisch and Richard P. Lifton and Wen-Hui Wang
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2014, Volume 111, Issue 32, pp. 11864 - 11869
The renal phenotype induced by loss-of-function mutations of inwardly rectifying potassium channel (Kir), Kcnj10 (Kir4.1), includes salt wasting,... 
Knockout mice | Kidneys | Patch clamp techniques | Cell membranes | Membrane potential | Potassium channels | Physiological regulation | Magnification | Genetic mutation | P branes | SPAK | Kir.5.1 | SeSAME/EAST syndrome | WNK | MULTIDISCIPLINARY SCIENCES | SENSORINEURAL DEAFNESS | NEW-MODEL | ANGIOTENSIN-II | KELCH-LIKE 3 | BLOOD-PRESSURE | BASOLATERAL MEMBRANE | COLLECTING DUCT | MOUSE KIDNEY | K+ CHANNEL | Immunohistochemistry | Patch-Clamp Techniques | Animals | Membrane Potentials | Potassium Channels, Inwardly Rectifying - deficiency | Models, Biological | Potassium Channels, Inwardly Rectifying - genetics | Mice | Potassium Channels, Inwardly Rectifying - metabolism | Solute Carrier Family 12, Member 3 - metabolism | Kidney Tubules, Distal - metabolism | Mice, Knockout | Physiological aspects | Protein research | Phenotype | Research | Carrier proteins | Gene expression | SeSAME | Biological Sciences | EAST syndrome
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4. Full Text SPAK and OSR1 play essential roles in potassium homeostasis through actions on the distal convoluted tubule
by Ferdaus, Mohammed Z and Barber, Karl W and López‐Cayuqueo, Karen I and Terker, Andrew S and Argaiz, Eduardo R and Gassaway, Brandon M and Chambrey, Régine and Gamba, Gerardo and Rinehart, Jesse and McCormick, James A
The Journal of Physiology, ISSN 0022-3751, 09/2016, Volume 594, Issue 17, pp. 4945 - 4966
Key points STE20 (Sterile 20)/SPS‐1 related proline/alanine‐rich kinase (SPAK) and oxidative stress‐response kinase‐1 (OSR1) phosphorylate and activate the... 
INTRACELLULAR CHLORIDE | SIGNALING PATHWAYS | PHYSIOLOGY | PHOSPHOPROTEOMIC ANALYSIS | NA+-CL-COTRANSPORTER | CORTICAL COLLECTING DUCT | PROTEIN-PHOSPHORYLATION | NA+-K+-2CL(-) COTRANSPORTER | NEUROSCIENCES | SODIUM-CHLORIDE COTRANSPORTER | THICK ASCENDING LIMB | BLOOD-PRESSURE | Blood Pressure | Potassium - metabolism | Solute Carrier Family 12, Member 1 - metabolism | Kidney Tubules, Distal - physiology | Protein-Serine-Threonine Kinases - physiology | Protein-Serine-Threonine Kinases - genetics | Homeostasis | Male | Mice, Knockout | Potassium - physiology | Animals | Mice | Protein-Serine-Threonine Kinases - metabolism | Solute Carrier Family 12, Member 3 - metabolism | Kidney Tubules, Distal - metabolism | Phosphotransferases | Analysis | Oxidative stress | Phosphorylation | Sodium | Kinases | Rodents | Renal Filtration | Research Paper | Renal Physiology | Renal and endocrine
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5. Full Text Vasopressin induces phosphorylation of the thiazide-sensitive sodium chloride cotransporter in the distal convoluted tubule
by Pedersen, Nis B and Hofmeister, Marlene V and Rosenbaek, Lena L and Nielsen, Jakob and Fenton, Robert A
Kidney International, ISSN 0085-2538, 07/2010, Volume 78, Issue 2, pp. 160 - 169
The thiazide-sensitive Na –Cl cotransporter (NCC) is important for renal electrolyte balance and its phosphorylation causes an increase in its transport... 
distal tubule | Na-Cl cotransporter | cell and transport physiology | vasopressin | RAT | DECREASES | BLOOD-PRESSURE | COLLECTING DUCT | NA+-CL-COTRANSPORTER | SIGNALING PATHWAY | UROLOGY & NEPHROLOGY | DIETARY SALT | ALDOSTERONE | AQUAPORIN-2 | APICAL MEMBRANE | Rats, Brattleboro | Phosphorylation | Animals | Rats, Wistar | Solute Carrier Family 12, Member 3 | Mice, Inbred C57BL | Rats | Mice | Vasopressins - physiology | Receptors, Drug - metabolism | Symporters - metabolism | Kidney Tubules, Distal - metabolism
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6. Full Text Wnk4 controls blood pressure and potassium homeostasis via regulation of mass and activity of the distal convoluted tubule
by Kahle, Kristopher T and Lu, Yin and Lalioti, Maria D and Lifton, Richard P and Hoffmann, Kristin E and Volkman, Heather M and Booth, Carmen J and Nelson-Williams, Carol and Toka, Hakan R and Ellison, David H and Flavell, Richard and Zhang, Junhui and Geller, David S
Nature Genetics, ISSN 1061-4036, 10/2006, Volume 38, Issue 10, pp. 1124 - 1132
The mechanisms that govern homeostasis of complex systems have been elusive but can be illuminated by mutations that disrupt system behavior. Mutations in the... 
MUTANT WNK4 | NA-CL COTRANSPORTER | SODIUM | CALCIUM-METABOLISM | HYPERKALEMIA | GENETICS & HEREDITY | TRANSPORT PATHWAYS | MICE | HUMAN HYPERTENSION | MUTATIONS | REABSORPTION | Kidney Tubules, Distal - diagnostic imaging | Potassium - metabolism | Sodium Chloride Symporters - metabolism | Sodium Chloride Symporters - genetics | Pseudohypoaldosteronism - genetics | Humans | Protein-Serine-Threonine Kinases - genetics | Homeostasis | Mice, Transgenic | Electrolytes - blood | Animals | Chromosomes, Artificial, Bacterial | Ultrasonography | Female | Blood Pressure - physiology | Mice | Mutation | Protein-Serine-Threonine Kinases - metabolism | Kidney Tubules, Distal - metabolism | Control | Gene mutations | Physiological aspects | Blood pressure | Genetic aspects | Research | Health aspects | Gene expression | Molecular biology | Potassium | Rodents
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7. Full Text Bradykinin Stimulates Renal Na+ and K+ Excretion by Inhibiting the K+ Channel (Kir4.1) in the Distal Convoluted Tubule
by Zhang, Dan-Dan and Gao, Zhong-Xiuzi and Vio, Carlos P and Xiao, Yu and Wu, Peng and Zhang, Hao and Guo, Xi-Wen and Meng, Xin-Xin and Gu, Li and Wang, Jun-Lin and Duan, Xin-Peng and Lin, Dao-Hong and Wang, Wen-Hui and Gu, Ruimin
Hypertension, ISSN 0194-911X, 06/2018, Volume 72, Issue 2, pp. 361 - 369
Stimulation of BK2R (bradykinin [BK] B2 receptor) has been shown to increase renal Na excretion. The aim of the present study is to explore the role of BK2R in... 
Hypertension | Diuretics | Ion transport | Receptor | Sodium-chloride symporter | Bradykinin B2 | sodium-chloride symporter | ion transport | bradykinin B2 | BLOOD-PRESSURE | B-2 RECEPTOR | BASOLATERAL MEMBRANE | receptor | COLLECTING DUCT | POTASSIUM INTAKE | KALLIKREIN-KININ SYSTEM | diuretics | CL-COTRANSPORTER | KCNJ10 KIR4.1 | PERIPHERAL VASCULAR DISEASE | hypertension | THICK ASCENDING LIMB | SODIUM-CHLORIDE COTRANSPORTER | Immunohistochemistry | Natriuresis - physiology | Mice, Inbred C57BL | Male | Mice, Knockout | Patch-Clamp Techniques | Animals | Kidney Tubules, Distal - drug effects | Membrane Potentials | Sodium - urine | Female | Models, Animal | Ion Transport | Mice | Potassium Channels, Inwardly Rectifying - metabolism | Bradykinin - pharmacology | Solute Carrier Family 12, Member 3 - metabolism | Kidney Tubules, Distal - metabolism | Sodium channels | Bradykinin | Physiological aspects | Hypokalemia | Development and progression | Potassium channels | Kidney tubules | Health aspects | Kcnj10 | bradykinin B2receptor | Na-Cl-Cotranspotor (NCC) | Kcnj16
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8. Full Text Uromodulin is expressed in the distal convoluted tubule, where it is critical for regulation of the sodium chloride cotransporter NCC
by Tokonami, Natsuko and Takata, Tomoaki and Beyeler, Jan and Ehrbar, Iris and Yoshifuji, Ayumi and Christensen, Erik I and Loffing, Johannes and Devuyst, Olivier and Olinger, Eric G
Kidney International, ISSN 0085-2538, 10/2018, Volume 94, Issue 4, pp. 701 - 715
Uromodulin, the most abundant protein in normal urine, is essentially produced by the cells lining the thick ascending limb. There it regulates the activity of... 
uromodulin | Na+/Cl−-cotransporter | blood pressure regulation | distal convoluted tubule | salt handling | Cl | cotransporter | PROTEIN | RAT-KIDNEY | ULTRASTRUCTURAL-LOCALIZATION | SENSITIVE NA+ | BLOOD-PRESSURE | MOUSE MODEL | IN-VIVO | GENE-EXPRESSION | UROLOGY & NEPHROLOGY | Na+/Cl--cotransporter | TAMM-HORSFALL GLYCOPROTEIN | THICK ASCENDING LIMB | Solute Carrier Family 12, Member 1 - metabolism | Gene Expression | Phosphorylation | Kidney Tubules, Distal - physiology | Sodium Potassium Chloride Symporter Inhibitors - pharmacology | Solute Carrier Family 12, Member 1 - antagonists & inhibitors | Humans | Uromodulin - metabolism | Male | Sodium - metabolism | Mice, Knockout | Solute Carrier Family 12, Member 1 - genetics | Animals | Uromodulin - biosynthesis | Hypercalciuria - chemically induced | HEK293 Cells | Hypercalciuria - genetics | Mice | Furosemide - pharmacology | RNA, Messenger | Uromodulin - genetics | Kidney Tubules, Distal - metabolism
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9. Full Text H+-ATPase B1 subunit localizes to thick ascending limb and distal convoluted tubule of rodent and human kidney
by Frische, Sebastian and Chambrey, Régine and Trepiccione, Francesco and Zamani, Reza and Marcussen, Niels and Alexander, R. Todd and Skjødt, Karsten and Svenningsen, Per and Dimke, Henrik
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 09/2018, Volume 315, Issue 3, pp. F429 - F444
The vacuolar-type H+-ATPase B1 subunit is heavily expressed in the intercalated cells of the collecting system, where it contributes to H(+)transport, but has... 
Distal tubule proton pump | Acidosis | Acid-base balance | ATP6V1B1 | PHYSIOLOGY | RAT-KIDNEY | PRINCIPAL CELLS | LOAD DEPENDENCE | PROTON | distal tubule proton pump | acidosis | UROLOGY & NEPHROLOGY | acid-base balance | RENAL BICARBONATE REABSORPTION | INTERCALATED CELLS | MUTATIONS | PROMOTER DRIVES EXPRESSION | B1-SUBUNIT | CONNECTING TUBULE
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10. Full Text A new model of the distal convoluted tubule
by Ko, Benjamin and Mistry, Abinash C and Hanson, Lauren and Mallick, Rickta and Cooke, Leslie L and Hack, Bradley K and Cunningham, Patrick and Hoover, Robert S
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 09/2012, Volume 303, Issue 5, pp. F700 - F710
Ko B, Mistry AC, Hanson L, Mallick R, Cooke LL, Hack BK, Cunningham P, Hoover RS. A new model of the distal convoluted tubule. Am J Physiol Renal Physiol 303:... 
With-no-lysine kinase 4 | STE20/SPS-1-related proline/alaninerich kinase | Sodium-chloride cotransporter | CELLS | PHYSIOLOGY | with-no-lysine kinase 4 | STIMULATION | SENSITIVE NACL COTRANSPORTER | SURFACE EXPRESSION | BLOOD-PRESSURE | NA+-CL-COTRANSPORTER | PATHWAY | UROLOGY & NEPHROLOGY | STE20/SPS-1-related proline/alanine-rich kinase | MOLECULAR PATHOGENESIS | sodium-chloride cotransporter | WNK KINASES | Cell Line | Sodium Chloride Symporters - metabolism | Animals | Kidney Tubules, Distal - physiology | Protein-Serine-Threonine Kinases - physiology | Models, Animal | Mice | Protein-Serine-Threonine Kinases - pharmacology | Thiazides | Kidney Tubules, Distal - metabolism | Physiological aspects | Models | Kidney tubules | Phosphotransferases | Membrane proteins | STE20 | SPS-1-related proline | alanine-rich kinase
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11. Full Text Hypertrophy in the distal convoluted tubule of an 11β-hydroxysteroid dehydrogenase type 2 knockout model
by Hunter, Robert W and Ivy, Jessica R and Flatman, Peter W and Kenyon, Christopher J and Craigie, Eilidh and Mullins, Linda J and Bailey, Matthew A and Mullins, John J
Journal of the American Society of Nephrology, ISSN 1046-6673, 07/2015, Volume 26, Issue 7, pp. 1537 - 1548
Na(+) transport in the renal distal convoluted tubule (DCT) by the thiazide-sensitive NaCl cotransporter (NCC) is a major determinant of total body Na(+) and... 
DNA, Complementary - analysis | Sodium Chloride Symporters - metabolism | Kidney Tubules, Distal - pathology | Transcytosis - physiology | RNA - analysis | Epithelial Cells - drug effects | Mice, Inbred C57BL | Cells, Cultured | Male | Random Allocation | Hypertrophy - pathology | Mice, Knockout | Polymerase Chain Reaction - methods | Animals | Kidney Tubules, Distal - drug effects | Analysis of Variance | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - pharmacology | 11-beta-Hydroxysteroid Dehydrogenase Type 2 - metabolism | Female | Mice | Phosphorylation - drug effects | Epithelial Cells - cytology | Disease Models, Animal | epithelial sodium transport | distal tubule | epithelial cells | hypertrophy | renal tubular | Basic Research
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12. Full Text Comparative transcriptomic analysis identifies evolutionarily conserved gene products in the vertebrate renal distal convoluted tubule
by Sugano, Yuya and Cianciolo Cosentino, Chiara and Loffing-Cueni, Dominique and Neuhauss, Stephan C. F and Loffing, Johannes
Pflügers Archiv - European Journal of Physiology, ISSN 0031-6768, 8/2017, Volume 469, Issue 7, pp. 859 - 867
Understanding the molecular basis of the complex regulatory networks controlling renal ion transports is of major physiological and clinical importance. In... 
Neurosciences | Receptors | Biomedicine | Human Physiology | Comparative transcriptomics | Zebrafish | Molecular Medicine | Distal convoluted tubule | NaCl cotransporter | Kidney | Cell Biology | PHYSIOLOGY | PHOSPHORYLATION | DARPP-32 | BLOOD-PRESSURE | TRANSPORT | COTRANSPORTER | POTASSIUM | HYPERTENSION | EXPRESSION | BINDING | Sodium Chloride Symporters - metabolism | Sodium Chloride Symporters - genetics | Receptors, Drug - genetics | Dopamine and cAMP-Regulated Phosphoprotein 32 - genetics | Transcriptome | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Receptors, Drug - metabolism | Animals | Conserved Sequence | Mice | Zebrafish Proteins - genetics | Evolution, Molecular | Kidney Tubules, Distal - metabolism | Genetic research | Animal genetic engineering | Comparative analysis | Gene expression | Genes | Transcription factors | Phosphorylation | Renal function | Kidneys | Syngeneic grafts | Phylogeny | Sodium chloride | Ribonucleic acid--RNA | Kidney transplantation
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13. Full Text Magnesium Transport in the Renal Distal Convoluted Tubule
by Long-Jun Dai and Gordon Ritchie and Dirk Kerstan and Hyung Sub Kang and David E. C. Cole and Gary A. Quamme
Physiological Reviews, ISSN 0031-9333, 01/2001, Volume 81, Issue 1, pp. 51 - 84
The distal tubule reabsorbs ∼10% of the filtered Mg2+, but this is 70–80% of that delivered from the loop of Henle. Because there is little Mg2+ reabsorption... 
PROTEIN-KINASE-C | HYPERPROSTAGLANDIN-E SYNDROME | PHYSIOLOGY | REFRACTORY POTASSIUM REPLETION | CALCIUM-SENSING RECEPTOR | NEONATAL SEVERE HYPERPARATHYROIDISM | SENSITIVE ADENYLATE-CYCLASE | PARATHYROID-HORMONE STIMULATION | FAMILIAL HYPOCALCIURIC HYPERCALCEMIA | CISPLATIN-INDUCED HYPOMAGNESEMIA | THICK ASCENDING LIMB | Hormones - pharmacology | Antibiotics, Antineoplastic - toxicity | Diuretics - pharmacology | Humans | Receptors, Cell Surface - metabolism | Magnesium - metabolism | Acid-Base Equilibrium - physiology | Immunosuppressive Agents - toxicity | Receptors, Calcium-Sensing | Kidney Tubules, Distal - cytology | Phosphates - metabolism | Animals | Cisplatin - toxicity | Kidney Tubules, Distal - drug effects | Renal Tubular Transport, Inborn Errors - metabolism | Hormones - metabolism | Aminoglycosides - toxicity | Potassium Deficiency - metabolism | Ion Transport - drug effects | Phosphates - deficiency | Ion Transport - physiology | Kidney Tubules, Distal - metabolism | Physiological aspects | Homeostasis | Kidney tubules | Magnesium in the body
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