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Journal Article
PloS one, ISSN 1932-6203, 2019, Volume 14, Issue 12, p. e0225475
Rapid sample preparation is one of the leading bottlenecks to low-cost and efficient sample component detection. To overcome this setback, a technology known... 
CELLS | EXTRACTION | ASSAY | DNA | MULTIDISCIPLINARY SCIENCES | METAL-ENHANCED FLUORESCENCE | MICROWAVE-TRIGGERED CHEMILUMINESCENCE | ROS | PROBES | Staphylococcus aureus - radiation effects | Temperature | Reactive Oxygen Species - metabolism | Bacteriological Techniques - methods | Oxidation-Reduction - radiation effects | DNA, Bacterial - chemistry | DNA, Bacterial - radiation effects | RNA Stability - drug effects | DNA Fragmentation - radiation effects | Oxygen - metabolism | DNA, Bacterial - drug effects | Oxygen - analysis | Proteolysis - drug effects | Time Factors | Analytic Sample Preparation Methods - methods | Oxidation-Reduction - drug effects | RNA, Bacterial - radiation effects | DNA Fragmentation - drug effects | Proteolysis - radiation effects | Microwaves | Staphylococcus aureus - genetics | Detergents - pharmacology | Vibrio cholerae - genetics | Reactive Oxygen Species - analysis | Listeria monocytogenes - radiation effects | RNA, Bacterial - drug effects | RNA, Bacterial - chemistry | Listeria monocytogenes - genetics | Vibrio cholerae - radiation effects | Hydrolysis - radiation effects | RNA Stability - radiation effects | Staphylococcus aureus - drug effects | Vibrio cholerae - drug effects | Listeria monocytogenes - drug effects | Proteins | Proteolysis | Oxidative stress | Reactive oxygen species | Fluorescence | Electron transfer | Biochemistry | Biology | Fluorescent indicators | Fragmentation | Degradation | Sample preparation | Listeria | Technology | DNA fragmentation | Hydroxyl radicals | Biomolecules | Oxidation | Spinach | Deoxyribonucleic acid--DNA | Biodegradation | Enzymes | Oxygen | Free radicals | DNA probes | Superoxide | Ribonucleic acid--RNA | Chemistry | Singlet oxygen | Irradiation | RNA | Deoxyribonucleic acid | Ribonucleic acid
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2017, Volume 58, Issue 1, pp. 147 - 162
The purpose our study was to determine the protective effects of mitochondria division inhibitor 1 (Mdivi1... 
mitochondrial division inhibitor 1 | synaptic pathology | dynamin-related protein 1 | mitochondrial dysfunction | Amyloid-β | mitochondrial fission | mitochondrial dynamics | OXIDATIVE DAMAGE | NEUROSCIENCES | NEURONAL DAMAGE | DRP1 | DEGENERATION | DEFECTIVE AXONAL-TRANSPORT | DYNAMIN-RELATED PROTEIN-1 | PRECURSOR PROTEIN | DYSFUNCTION | FISSION | HUNTINGTONS-DISEASE | Amyloid-beta | Gene Expression Regulation, Enzymologic - drug effects | Mitochondrial Dynamics - genetics | Amyloid beta-Peptides - pharmacology | Peptide Fragments - pharmacology | Mitochondrial Proteins - genetics | Mitochondria - drug effects | RNA, Messenger - metabolism | Nerve Tissue Proteins - genetics | Electron Transport Complex IV - metabolism | Hydrogen Peroxide - metabolism | Nerve Tissue Proteins - metabolism | Animals | Drug Interactions | Mitochondrial Dynamics - drug effects | Neuroblastoma - ultrastructure | Mitochondrial Proteins - metabolism | Adenosine Triphosphate - metabolism | Lipid Peroxidation - drug effects | Cell Line, Tumor | Mice | Neuroblastoma - pathology | Quinazolinones - pharmacology | Cytochrome | Hydrogen peroxide | Neurodegenerative diseases | Neurons | Lipid peroxidation | Immunoblotting | Biosynthesis | Mitochondrial DNA | mRNA | Fission | Fragmentation | Polymerase chain reaction | Proteins | Mitochondria | Inhibitors | Dynamin | Amyloid | Alzheimer's disease | Peroxidation | Synaptic pathology | Mitochondrial dysfunction | Dynamin-related protein 1 | Mitochondrial dynamics | Mitochondrial division inhibitor 1 | Mitochondrial fission
Journal Article
PloS one, ISSN 1932-6203, 2019, Volume 14, Issue 9, p. e0223008
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 4, p. e0175195
In addition to their anti-bacterial action, tetracyclines also have complex biological effects, including the modification of mitochondrial protein synthesis, metabolism and gene-expression... 
RHEUMATOID-ARTHRITIS | OXIDATIVE STRESS | MINOCYCLINE | INHIBITION | THERAPY | ASSAY | MULTIDISCIPLINARY SCIENCES | DISEASE | REPERFUSION | DYSFUNCTION | Cell Line | Phosphorylation | Mitochondria, Heart - metabolism | Reactive Oxygen Species - metabolism | Rats, Wistar | Rats | Male | Heart Failure - metabolism | Mitochondria, Heart - drug effects | Membrane Potential, Mitochondrial - drug effects | Heart Failure - prevention & control | Collagen - metabolism | Animals | Muscle Proteins - metabolism | Doxycycline - pharmacology | Oxidative Stress - drug effects | Adrenergic beta-Agonists - adverse effects | Heart Failure - chemically induced | Isoproterenol - adverse effects | Microscopy, Fluorescence | Natriuretic Peptide, Brain - blood | Heart failure | Treatment outcome | Mitochondria | Analysis | Doxycycline | Dosage and administration | Research | Cytochrome | Myocardial infarction | Heart | Cell culture | Heart attacks | Aluminum | Aneurysm | Carbon dioxide | Recovery of function | Cytotoxicity | Cardiovascular disease | Biochemistry | Mitochondrial DNA | Antiinfectives and antibacterials | Density | Anticancer properties | Fragmentation | Metastases | Proteins | Degradation | Ischemia | Temperature effects | Aorta | Genetics | Blood pressure | Inhibition | ADAM protein | Cardiac muscle | Mortality | Metabolism | Atenolol | Chemistry | Atmosphere | Collection | Acetic acid | Endoplasmic reticulum | Viability | Metabolic disorders | Drugs | Oxidative stress | Toxicity | Heart function | Muscular dystrophy | Antagonism | Stains | Cardiology | Heart diseases | Oxygen | Incubation | Diabetes mellitus | Cardiomyocytes | Pharmacology | Permeability | Coronary artery disease | Chemical compounds | Medicine | Absorbance | Antibiotics | Microscopy | Lungs | Dystrophy | ATP | Apoptosis
Journal Article
Toxicology and applied pharmacology, ISSN 0041-008X, 2011, Volume 251, Issue 3, pp. 226 - 233
Acetaminophen (APAP) hepatotoxicity is the most frequent cause of acute liver failure in many countries. The mechanism of cell death is initiated by formation... 
Manganese-SOD | Bax | Oxidative stress | c-jun-N-terminal kinase | Acetaminophen | Apoptosis-inducing factor | C-jun-N-terminal kinase | HETEROZYGOUS SOD2(+/-) MICE | OXIDATIVE DAMAGE | COVALENT BINDING | N-ACETYLCYSTEINE | KNOCKOUT MICE | TERMINAL KINASE | CELL-DEATH | REACTIVE NITROGEN | INDUCED HEPATIC-NECROSIS | IN-VIVO | PHARMACOLOGY & PHARMACY | TOXICOLOGY | In Situ Nick-End Labeling | Superoxide Dismutase - genetics | Acetaminophen - toxicity | Drug Overdose | Glutathione - metabolism | Mitochondria, Liver - metabolism | Mice, Inbred C57BL | Acetaminophen - administration & dosage | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Analgesics, Non-Narcotic - administration & dosage | Glutathione Disulfide - metabolism | Necrosis - chemically induced | Mitochondria, Liver - drug effects | Animals | Analgesics, Non-Narcotic - toxicity | Signal Transduction - drug effects | Mice | Alanine Transaminase - metabolism | DNA Fragmentation - drug effects | Oxidative Stress - drug effects | Chemical and Drug Induced Liver Injury - etiology | Drugs | Proteins | Liver diseases | Liver | Genetic research | Overdose | Mitochondrial DNA | Superoxide | Protein binding | Index Medicus | APOPTOSIS | SUPEROXIDE DISMUTASE | DIGESTIVE SYSTEM | MITOCHONDRIA | TRANSITION ELEMENTS | 60 APPLIED LIFE SCIENCES | NUCLEIC ACIDS | STRESSES | ELEMENTS | NECROSIS | GLANDS | METALS | MANGANESE | OXIDIZERS | ENZYMES | DRUGS | VERTEBRATES | MICE | RADIOPROTECTIVE SUBSTANCES | MAMMALS | ANIMALS | INJURIES | RODENTS | RESPONSE MODIFYING FACTORS | ORGANIC COMPOUNDS | FRAGMENTATION | POLYPEPTIDES | ORGANS | DISEASES | PEPTIDES | GLUTATHIONE | DNA | LIVER | PROTEINS | BODY | PATHOLOGICAL CHANGES | CELL CONSTITUENTS | OXIDOREDUCTASES | manganese-SOD | apoptosis-inducing factor | oxidative stress
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