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Nature, ISSN 0028-0836, 2014, Volume 512, Issue 1, pp. 49 - 53
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2006, Volume 25, Issue 5, pp. 1126 - 1136
Replication licensing is carefully regulated to restrict replication to once in a cell cycle. In higher eukaryotes, regulation of the licensing factor Cdt1 by... 
replication | Cdt1 | Geminin | cell cycle | proteolysis | Replication | Proteolysis | Cell cycle | REREPLICATION | PROTEIN | LICENSING FACTOR CDT1 | BIOCHEMISTRY & MOLECULAR BIOLOGY | RE-REPLICATION | COMPLEXES | EUKARYOTIC DNA-REPLICATION | CELL BIOLOGY | S-PHASE | DEGRADATION | CELL-CYCLE | Cell Cycle Proteins - pharmacology | Phosphorylation | Kidney - embryology | SKP Cullin F-Box Protein Ligases - genetics | Humans | Ubiquitin - metabolism | Cell Cycle Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Kidney - metabolism | S-Phase Kinase-Associated Proteins - antagonists & inhibitors | Ultraviolet Rays | DNA Replication - radiation effects | Cell Cycle Proteins - genetics | Cullin Proteins - metabolism | Fibroblasts - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Cullin Proteins - antagonists & inhibitors | Cyclin A - metabolism | RNA, Small Interfering - pharmacology | Cell Cycle Proteins - metabolism | Cells, Cultured | Gene Silencing | SKP Cullin F-Box Protein Ligases - metabolism | DNA-Binding Proteins - genetics | S-Phase Kinase-Associated Proteins - metabolism | SKP Cullin F-Box Protein Ligases - antagonists & inhibitors | Peptide Hydrolases - chemistry | Cullin Proteins - genetics | Animals | S-Phase Kinase-Associated Proteins - genetics | Mice | Cyclin E - metabolism | DNA Damage | HeLa Cells | Proliferating Cell Nuclear Antigen - metabolism | Amino acids | Eukaryotes
Journal Article
Journal Article
Cell, ISSN 0092-8674, 12/2012, Volume 151, Issue 7, pp. 1474 - 1487
Journal Article
Nature, ISSN 0028-0836, 08/2010, Volume 466, Issue 7309, pp. 941 - 946
Journal Article
Cell, ISSN 0092-8674, 05/2018, Volume 173, Issue 4, pp. 972 - 988.e23
Repair of damaged DNA is essential for maintaining genome integrity and for preventing genome-instability-associated diseases, such as cancer. By combining... 
DNA damage repair | proximity labeling | shieldin | proteomics | 53BP1 | PARP inhibitors | telomere maintenance | antibody class-switch recombination | NHEJ | BRCA1 | PATHWAYS | CELLS | RESECTION | DAMAGE-RESPONSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CLASS-SWITCH RECOMBINATION | HOMOLOGOUS RECOMBINATION | DOUBLE-STRAND BREAK | TELOMERES | CELL BIOLOGY | Mad2 Proteins - metabolism | Humans | Ubiquitin-Protein Ligases - antagonists & inhibitors | DNA Breaks, Double-Stranded | Mad2 Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Telomere-Binding Proteins - genetics | RNA Interference | BRCA1 Protein - metabolism | Trans-Activators - genetics | Immunoglobulin Class Switching - drug effects | Nuclear Proteins - genetics | Telomere-Binding Proteins - metabolism | Tumor Suppressor p53-Binding Protein 1 - metabolism | DNA End-Joining Repair - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Mutagenesis, Site-Directed | Tumor Suppressor p53-Binding Protein 1 - genetics | Ubiquitin-Protein Ligases - metabolism | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Telomere-Binding Proteins - antagonists & inhibitors | Tumor Suppressor p53-Binding Protein 1 - antagonists & inhibitors | BRCA1 Protein - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | BRCA1 Protein - antagonists & inhibitors | Mad2 Proteins - genetics | Cell Line, Tumor | Trans-Activators - metabolism | Ubiquitin-Protein Ligases - genetics | RNA, Small Interfering - metabolism | DNA damage | Genomics | Antibodies | Genomes | DNA repair | Proteins | Viral antibodies | Telomeres | Evolutionary biology | Analysis | DNA | Genetic research | Mass spectrometry
Journal Article
Science, ISSN 0036-8075, 3/2010, Volume 327, Issue 5971, pp. 1345 - 1350
Half a century ago, thalidomide was widely prescribed to pregnant women as a sedative but was found to be teratogenic, causing multiple birth defects. Today,... 
Receptors | Messenger RNA | Delta cells | Ubiquitins | Substrate specificity | Teratogenicity | HeLa cells | Pectorals | Cellular immunity | Embryos | Research Article | MULTIPLE-MYELOMA | CELLS | LIMB DEFECTS | ORIGIN | ZEBRAFISH | UBIQUITIN LIGASE | PATHWAY | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | OUTGROWTH | EXPRESSION | Teratogens - toxicity | Thalidomide - metabolism | Peptide Hydrolases - genetics | Forelimb - embryology | Humans | Fibroblast Growth Factors - genetics | Ubiquitin-Protein Ligases - antagonists & inhibitors | Zebrafish - embryology | Embryo, Nonmammalian - drug effects | DNA-Binding Proteins - metabolism | Fibroblast Growth Factors - metabolism | Ubiquitination | Forelimb - abnormalities | Gene Expression Regulation, Developmental | Cullin Proteins - metabolism | Peptide Hydrolases - metabolism | Zebrafish Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | Mutant Proteins - metabolism | Chick Embryo | Thalidomide - toxicity | Zebrafish - genetics | Animals | Carrier Proteins - metabolism | HeLa Cells | Zebrafish Proteins - genetics | Embryonic Development - drug effects | Teratogens - metabolism | Dosage and administration | Research | Teratogenic agents | Thalidomide | Protein binding | Pharmacology | Birth defects | Developmental biology | DNA damage | Binding sites | Index Medicus
Journal Article