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European Journal of Cancer, ISSN 0959-8049, 2008, Volume 45, Issue 3, pp. 365 - 373
Journal Article
Journal of the National Cancer Institute, ISSN 0027-8874, 07/2013, Volume 105, Issue 13, pp. 989 - 992
Journal Article
Gastroenterology, ISSN 0016-5085, 2014, Volume 147, Issue 5, pp. 1064 - 1072.e5
Background & Aims Lynch syndrome, a nonpolyposis form of hereditary colorectal cancer, is caused by inherited defects in DNA mismatch repair (MMR) genes. Most... 
Gastroenterology and Hepatology | Carcinogen | Defective Mismatch Repair | Cancer Risk | Dietary Factors | STEM-CELLS | NONPOLYPOSIS COLORECTAL-CANCER | COLON | MLH1 | INACTIVATION | DNA | MICE | AGENTS | MUTATIONS | METHYLATION TOLERANCE | GASTROENTEROLOGY & HEPATOLOGY | Intestines - drug effects | Adenocarcinoma - pathology | Adenoma - genetics | Neoplastic Stem Cells - drug effects | Adenocarcinoma - chemically induced | Colorectal Neoplasms, Hereditary Nonpolyposis - pathology | Male | Cell Transformation, Neoplastic - chemically induced | Intestines - metabolism | Adenoma - metabolism | Adenocarcinoma - metabolism | Neoplastic Stem Cells - metabolism | Time Factors | Cell Transformation, Neoplastic - genetics | MutS Homolog 2 Protein - deficiency | Dacarbazine - analogs & derivatives | Neoplastic Stem Cells - pathology | Female | Adenocarcinoma - genetics | Disease Models, Animal | Intestines - pathology | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Risk Factors | DNA Mismatch Repair - drug effects | MutS Homolog 2 Protein - genetics | Cell Transformation, Neoplastic - metabolism | Mice, Knockout | Colorectal Neoplasms, Hereditary Nonpolyposis - metabolism | Animals | Adenoma - pathology | Cell Proliferation - drug effects | Colorectal Neoplasms, Hereditary Nonpolyposis - chemically induced | Cell Transformation, Neoplastic - pathology | Dacarbazine - toxicity | Adenoma - chemically induced | Temozolomide | Statistics | Analysis | Colorectal cancer | Tumors
Journal Article
Molecular Cell, ISSN 1097-2765, 07/2010, Volume 39, Issue 1, pp. 36 - 47
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2009, Volume 47, Issue 1, pp. 13 - 26
Poly(ADP-ribose) polymerases (PARPs) are defined as cell signaling enzymes that catalyze the transfer of ADP-ribose units from NAD to a number of acceptor... 
BRCA1/2 | Angiogenesis | Stroke | Free radicals | Homologous recombination | Sepsis | Cardiovascular disease | PARP | Inflammation | poly(ADP-ribose) | Diabetes | Anticancer therapy | POLY(ADP-RIBOSE) POLYMERASE | NITRIC-OXIDE SYNTHASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOCAL CEREBRAL-ISCHEMIA | HEART-FAILURE | DIABETIC ENDOTHELIAL DYSFUNCTION | DNA-REPAIR DEFECT | ADP-RIBOSE POLYMERASE | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | NF-KAPPA-B | Cell Transformation, Neoplastic - radiation effects | Poly(ADP-ribose) Polymerases - immunology | Poly(ADP-ribose) Polymerases - therapeutic use | Humans | Reperfusion Injury - drug therapy | Radiotherapy - adverse effects | DNA Mismatch Repair - genetics | Breast Neoplasms - physiopathology | Radiation-Sensitizing Agents - therapeutic use | Cell Transformation, Neoplastic - genetics | Drug-Related Side Effects and Adverse Reactions | Angiogenesis Inhibitors - therapeutic use | Anti-Inflammatory Agents - therapeutic use | Female | Oxidative Stress - radiation effects | Gene Expression Regulation, Neoplastic - drug effects | BRCA1 Protein - deficiency | Autoimmune Diseases - drug therapy | Gene Expression Regulation, Neoplastic - genetics | DNA Mismatch Repair - radiation effects | Oxidative Stress - genetics | DNA Mismatch Repair - drug effects | Clinical Trials as Topic | Combined Modality Therapy | Breast Neoplasms - blood supply | Breast Neoplasms - drug therapy | Poly(ADP-ribose) Polymerase Inhibitors | Breast Neoplasms - radiotherapy | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Neovascularization, Pathologic - drug therapy | BRCA2 Protein - deficiency | Gene Expression Regulation, Neoplastic - radiation effects | Cell Transformation, Neoplastic - drug effects | Oxidative Stress - drug effects | Poly (ADP-Ribose) Polymerase-1 | Rheumatoid factor | Enzymes | Chemotherapy | Cell death | Rheumatoid arthritis | DNA | Development and progression | DNA binding proteins | Health aspects | Cancer | Index Medicus
Journal Article