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Gut, ISSN 0017-5749, 01/2017, Volume 66, Issue 1, pp. 97 - 106
Objective Sessile serrated adenomas (SSAs) are the precursors of at least 15% of colorectal carcinomas, but their biology is incompletely understood. We... 
COLONIC POLYPS | COLON CARCINOGENESIS | COLORECTAL CANCER GENES | COLONIC NEOPLASMS | ISLAND METHYLATOR PHENOTYPE | COLON-CANCER | COLONOSCOPY | HYPERPLASTIC POLYPS | EARLY NEOPLASTIC PROGRESSION | BRAF MUTATION | PATHWAY | COLORECTAL-CANCER | MICROSATELLITE INSTABILITY | POOR SURVIVAL | GASTROENTEROLOGY & HEPATOLOGY | Adenoma - genetics | Proto-Oncogene Proteins p21(ras) - genetics | Colorectal Neoplasms - genetics | Humans | Middle Aged | Male | Tumor Suppressor Protein p14ARF - analysis | Carcinoma - chemistry | Young Adult | Colonic Polyps - genetics | Cell Transformation, Neoplastic - genetics | Aged, 80 and over | Neoplastic Syndromes, Hereditary - genetics | Wnt Signaling Pathway | Brain Neoplasms - genetics | Tumor Burden | DNA Repair Enzymes - analysis | Phenotype | MutL Protein Homolog 1 - genetics | CpG Islands | Carcinoma - genetics | Mutation | Age Factors | beta Catenin - analysis | Colonic Polyps - pathology | DNA Repair Enzymes - genetics | Colorectal Neoplasms - chemistry | Tumor Suppressor Protein p53 - genetics | Tumor Suppressor Protein p53 - analysis | Tumor Suppressor Proteins - genetics | Adult | Female | Carcinoma - pathology | DNA Modification Methylases - analysis | Cross-Sectional Studies | Gene Silencing | Adenoma - chemistry | Colonic Polyps - chemistry | beta Catenin - genetics | MutL Protein Homolog 1 - analysis | DNA Modification Methylases - genetics | Proto-Oncogene Proteins B-raf - genetics | Sex Factors | Adenoma - pathology | Aged | Tumor Suppressor Protein p14ARF - genetics | Cell Transformation, Neoplastic - pathology | Colorectal Neoplasms - pathology | Tumor Suppressor Proteins - analysis | Dysplasia | Usage | Carcinoma | Diagnosis | Clinical pathology | Cancer | DNA methylation | Bias | Deoxyribonucleic acid--DNA | Tumors | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Brain Pathology, ISSN 1015-6305, 10/2008, Volume 18, Issue 4, pp. 520 - 532
Silencing of O6-methylguanine-DNA methyltransferase (MGMT) protein expression because of MGMT gene promoter hypermethylation is considered to be associated... 
glioblastoma | immunohistochemistry | biomarker | MGMT | prognosis | Immunohistochemistry | Biomarker | Prognosis | Glioblastoma | METHYLATION ANALYSIS | PROMOTER HYPERMETHYLATION | MISMATCH REPAIR | PATHOLOGY | O-6-METHYLGUANINE-DNA METHYLTRANSFERASE GENE | NEUROSCIENCES | CLINICAL NEUROLOGY | BRAIN-TUMORS | DNA-DAMAGING AGENTS | INTERLABORATORY VALIDATION | O-6-ALKYLGUANINE-DNA ALKYLTRANSFERASE | MALIGNANT GLIOMAS | INTEROBSERVER REPRODUCIBILITY | Antibody Specificity | Predictive Value of Tests | Glioblastoma - enzymology | Prospective Studies | Humans | Middle Aged | Brain Neoplasms - physiopathology | DNA Repair Enzymes - genetics | Antibodies | Promoter Regions, Genetic - genetics | DNA Methylation | Tumor Suppressor Proteins - genetics | DNA Repair Enzymes - metabolism | Biomarkers, Tumor - metabolism | Adult | DNA Modification Methylases - analysis | Brain Neoplasms - enzymology | Diagnosis, Differential | Reproducibility of Results | Tumor Suppressor Proteins - metabolism | DNA Modification Methylases - metabolism | Glioblastoma - diagnosis | Biomarkers, Tumor - analysis | Brain Neoplasms - diagnosis | Survival Rate | DNA Repair Enzymes - analysis | Immunohistochemistry - methods | DNA Modification Methylases - genetics | Aged | Glioblastoma - physiopathology | Observer Variation | Cohort Studies | Tumor Suppressor Proteins - analysis | Algorithms | Methylation | Glioblastoma multiforme | Patient outcomes | Index Medicus
Journal Article
Modern Pathology, ISSN 0893-3952, 06/2013, Volume 26, Issue 6, pp. 825 - 834
KRAS-mutated carcinomas comprise 35-40% of all colorectal carcinomas but little is known about their characteristics. The aim of this study was to examine the... 
KRAS mutation | colorectal cancer | survival analysis | MGMT | colorectal polyp | molecular pathology | BRAF mutation | WILD-TYPE | PROMOTER METHYLATION | MICROSATELLITE INSTABILITY | PATHOLOGY | ISLAND METHYLATOR PHENOTYPE | COLON-CANCER | RECTAL-CANCER | BRAF MUTATIONS | LOW CIMP-LOW | KIRSTEN-RAS | KI-RAS MUTATIONS | Immunohistochemistry | ras Proteins - genetics | Microsatellite Instability | Proto-Oncogene Proteins p21(ras) | Prognosis | Prospective Studies | Colonic Polyps - pathology | Colorectal Neoplasms - genetics | Humans | Middle Aged | DNA Repair Enzymes - genetics | Carcinoma - mortality | Colorectal Neoplasms - chemistry | Male | Colonic Polyps - mortality | DNA Methylation | Colonic Polyps - genetics | Time Factors | DNA Mutational Analysis | Tumor Suppressor Proteins - genetics | Adult | Female | Cell Differentiation | Carcinoma - pathology | DNA Modification Methylases - analysis | Colorectal Neoplasms - mortality | Genetic Predisposition to Disease | Biomarkers, Tumor - analysis | Risk Factors | Victoria | Kaplan-Meier Estimate | Proportional Hazards Models | Proto-Oncogene Proteins - genetics | Chi-Square Distribution | DNA Repair Enzymes - analysis | DNA Modification Methylases - genetics | Phenotype | Proto-Oncogene Proteins B-raf - genetics | Carcinoma - genetics | Aged | Biomarkers, Tumor - genetics | Mutation | Colorectal Neoplasms - pathology | Tumor Suppressor Proteins - analysis | Index Medicus
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 11/2009, Volume 15, Issue 21, pp. 6683 - 6693
Journal Article
Analytica Chimica Acta, ISSN 0003-2670, 08/2014, Volume 840, pp. 28 - 32
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Journal Article
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