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Journal of Clinical Investigation, ISSN 0021-9738, 02/2016, Volume 126, Issue 2, pp. 721 - 731
Renal erythropoietin-producing cells (REPCs) remain in the kidneys of patients with chronic kidney disease, but these cells do not produce sufficient... 
FIBROSIS | MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | MOUSE MODEL | GENE-EXPRESSION | ANEMIA | HYPOXIA | FIBROBLASTS | PERICYTE-MYOFIBROBLAST TRANSITION | CHRONIC-RENAL-FAILURE | SERUM ERYTHROPOIETIN | Myofibroblasts - pathology | Basic Helix-Loop-Helix Transcription Factors - genetics | DNA Modification Methylases - metabolism | DNA Modification Methylases - antagonists & inhibitors | Pericytes - metabolism | Mice, Transgenic | Receptor, Platelet-Derived Growth Factor beta - genetics | Renal Insufficiency, Chronic - drug therapy | Renal Insufficiency, Chronic - pathology | Myofibroblasts - metabolism | Renal Insufficiency, Chronic - metabolism | Azacitidine - pharmacology | Erythropoietin - genetics | Collagen Type I - biosynthesis | DNA Modification Methylases - genetics | Animals | Collagen Type I - genetics | Basic Helix-Loop-Helix Transcription Factors - metabolism | Pericytes - pathology | Fibrosis | Renal Insufficiency, Chronic - genetics | Mice | Erythropoietin - biosynthesis | Receptor, Platelet-Derived Growth Factor beta - metabolism | Enzymes | Erythropoietin | Development and progression | Genetic aspects | Regulation | Kidney diseases | Health aspects | Medical research | Plasma | Kidneys | Laboratories | Anemia | Studies | Proteins | Hospitals | Rodents | DNA methylation | Fibroblasts | Hypoxia | Deoxyribonucleic acid--DNA | University colleges
Journal Article
STEM CELLS, ISSN 1066-5099, 05/2010, Volume 28, Issue 5, pp. 851 - 862
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 04/2007, Volume 13, Issue 7, pp. 2038 - 2045
Journal Article
Oncotarget, ISSN 1949-2553, 05/2016, Volume 7, Issue 18, pp. 24950 - 24961
Pancreatic neuroendocrine tumor (PanNET) is a neoplastic entity in which few prognostic factors are well-known. Here, we aimed to evaluate the prognostic... 
Immunohistochemistry | NDRG-1 | PHLDA-3 | MGMT | Pancreatic neuroendocrine tumor | DNA METHYLATION | MTOR PATHWAY | ENDOCRINE TUMORS | NDRG1 | CHEMOTHERAPY | TEMOZOLOMIDE | CELL BIOLOGY | pancreatic neuroendocrine tumor | RELEVANCE | CARCINOMAS | immunohistochemistry | EXPRESSION | PHLDA3 | Cell Cycle Proteins - analysis | Nuclear Proteins - analysis | Prognosis | Humans | Middle Aged | DNA Repair Enzymes - genetics | Male | Neoplasm Recurrence, Local - mortality | Neoplasm Recurrence, Local - pathology | DNA Methylation | Nuclear Proteins - biosynthesis | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Female | Pancreatic Neoplasms - mortality | Nuclear Proteins - genetics | DNA Modification Methylases - analysis | Intracellular Signaling Peptides and Proteins - genetics | Neuroendocrine Tumors - pathology | Biomarkers, Tumor - analysis | Pancreatic Neoplasms - pathology | Intracellular Signaling Peptides and Proteins - analysis | Kaplan-Meier Estimate | Cell Cycle Proteins - biosynthesis | Intracellular Signaling Peptides and Proteins - biosynthesis | DNA Repair Enzymes - analysis | Immunohistochemistry - methods | Disease-Free Survival | DNA Modification Methylases - genetics | DNA Modification Methylases - biosynthesis | DNA Repair Enzymes - biosynthesis | Neuroendocrine Tumors - mortality | Tumor Suppressor Proteins - biosynthesis | Tumor Suppressor Proteins - analysis
Journal Article
Journal Article
Anticancer Research, ISSN 0250-7005, 03/2010, Volume 30, Issue 3, pp. 945 - 952
In our primary studies, we have shown that emodin, aloe-emodin and rhein induced cytotoxic effects, including cell cycle arrest and apoptosis in SCC-4 human... 
ATM | BRCA1 | RT-PCR | Comet assay | CARCINOMA-CELLS | MITOCHONDRIAL SIGNALING PATHWAY | DEATH PATHWAY | INDUCED APOPTOSIS | ANTICANCER AGENTS | DEPENDENT PATHWAYS | ENDOPLASMIC-RETICULUM STRESS | P53 | IN-VITRO | ONCOLOGY | S-PHASE | Exoribonucleases | Gene Expression - drug effects | Tumor Suppressor Proteins - antagonists & inhibitors | Humans | DNA Repair Enzymes - genetics | DNA Repair - genetics | Tongue Neoplasms - drug therapy | DNA-Activated Protein Kinase - genetics | Tumor Suppressor Proteins - genetics | Exonucleases - genetics | Polymerase Chain Reaction | Cell Cycle Proteins - genetics | DNA Repair Enzymes - antagonists & inhibitors | Neoplasm Proteins - genetics | Tongue Neoplasms - metabolism | DNA Repair - drug effects | Emodin - pharmacology | Neoplasm Proteins - biosynthesis | Tongue Neoplasms - genetics | Enzyme Inhibitors - pharmacology | Exonucleases - biosynthesis | Protein-Serine-Threonine Kinases - genetics | Cell Cycle Proteins - biosynthesis | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | BRCA1 Protein - biosynthesis | Comet Assay | Protein-Serine-Threonine Kinases - biosynthesis | BRCA1 Protein - genetics | DNA Modification Methylases - genetics | DNA Modification Methylases - biosynthesis | DNA Repair Enzymes - biosynthesis | Cell Line, Tumor | 14-3-3 Proteins | Anthraquinones - pharmacology | Biomarkers, Tumor - genetics | DNA Damage | Tumor Suppressor Proteins - biosynthesis | DNA-Binding Proteins - biosynthesis | Biomarkers, Tumor - biosynthesis | DNA-Activated Protein Kinase - biosynthesis
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 12/2010, Volume 285, Issue 52, pp. 40461 - 40471
Journal Article
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 1, p. e16146
O-6-methylguanine DNA-methyltransferase (MGMT) promoter methylation has been identified as a potential prognostic marker for glioblastoma patients. The... 
SURVIVAL | GLIOBLASTOMA | HYPERMETHYLATION | ASSAY | O6-METHYLGUANINE-DNA METHYLTRANSFERASE | MULTIDISCIPLINARY SCIENCES | CONCOMITANT | GENE-EXPRESSION | PROGNOSTIC-SIGNIFICANCE | RADIOTHERAPY | ADJUVANT TEMOZOLOMIDE | Glioblastoma - enzymology | Prognosis | Humans | DNA Repair Enzymes - genetics | Gene Silencing | Treatment Outcome | DNA Methylation - genetics | Promoter Regions, Genetic - genetics | Disease-Free Survival | DNA Modification Methylases - genetics | DNA Modification Methylases - biosynthesis | Glioblastoma - genetics | Base Sequence | DNA Repair Enzymes - biosynthesis | Glioblastoma - pathology | Tumor Suppressor Proteins - genetics | CpG Islands | Tumor Suppressor Proteins - biosynthesis | Glioblastoma - mortality | Purines | Sulfites | Pyrimidines | RNA | Genes | Genetic engineering | Methylation | Multiplexing | Therapy | Neurosciences | Bisulfite | Transcription factors | Laboratories | Brain cancer | Genomics | Glioblastoma | Cancer therapies | DNA repair | Guanine | O6-methylguanine-DNA methyltransferase | Proteins | Classification | DNA methylation | Cytosine | Deoxyribonucleic acid--DNA | CpG islands | Medical research | Methylguanine | Gene expression | Patients | Survival | Pathology | Gene silencing | Chemotherapy | Brain research | Correlation analysis | DNA methyltransferase | Clinical medicine | Added value | Binding sites | Apoptosis | Deoxyribonucleic acid | DNA
Journal Article
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