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The EMBO Journal, ISSN 0261-4189, 12/2006, Volume 25, Issue 24, pp. 5775 - 5782
The phosphatidyl inositol 3‐kinase‐like kinases (PIKKs), ataxia‐telangiectasia mutated (ATM) and ATM‐ and Rad3‐related (ATR) regulate parallel damage response... 
ataxia telangiectasia‐mutated protein | PIKKs | phosphorylation | DNA damage responses | Phosphorylation | Ataxia telangiectasia-mutated protein | RECRUITMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | ataxia telangiectasia-mutated protein | DNA-DAMAGE | COMPLEXES | PROTEIN-KINASES | CELL BIOLOGY | CHK1 | SECKEL-SYNDROME | CHECKPOINT | IONIZING-RADIATION | ATAXIA-TELANGIECTASIA | BINDING | Protein Kinases - metabolism | Hydroxyurea - pharmacology | Protein-Serine-Threonine Kinases - deficiency | DNA Replication - drug effects | Humans | G2 Phase - drug effects | DNA-Binding Proteins - metabolism | Ultraviolet Rays | DNA Replication - radiation effects | Phosphorylation - drug effects | Protein-Serine-Threonine Kinases - metabolism | Protein Structure, Tertiary | Tumor Suppressor Proteins - metabolism | Mitosis - radiation effects | G2 Phase - radiation effects | Cell Cycle Proteins - metabolism | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | Enzyme Activation - drug effects | Fibroblasts - pathology | Phosphoserine - metabolism | Enzyme Activation - radiation effects | Histones - deficiency | Phosphorylation - radiation effects | Animals | Mitosis - drug effects | Fibroblasts - radiation effects | Models, Biological | Fibroblasts - drug effects | Phosphoproteins - deficiency | Checkpoint Kinase 2 | Checkpoint Kinase 1 | Fibroblasts - cytology | Mice | Ions | Ultraviolet radiation | Molecular biology | Kinases | DNA damage
Journal Article
Plant Physiology, ISSN 0032-0889, 12/2009, Volume 151, Issue 4, pp. 2162 - 2173
Replication protein A (RPA), a highly conserved single-stranded DNA-binding protein in eukaryotes, is a stable complex comprising three subunits termed RPA1,... 
Somatic cells | DNA | Irradiation | Systems Biology, Molecular Biology, and Gene Regulation | Meiosis | Megasporocytes | Plants | Embryo sac | DNA repair | Chromosomes | Rice | PATHWAYS | CELLS | STRAND BREAK REPAIR | GENE | HIGHER-PLANTS | MEIOSIS | ARABIDOPSIS | GENOME | DAMAGE | NUCLEOTIDE EXCISION-REPAIR | PLANT SCIENCES | RNA Interference - drug effects | Germ Cells, Plant - radiation effects | DNA Replication - drug effects | DNA Repair - radiation effects | Genetic Complementation Test | DNA Fragmentation - radiation effects | Meiosis - drug effects | Chromosomes, Plant - radiation effects | Ultraviolet Rays | Oryza - genetics | DNA Replication - radiation effects | Plants, Genetically Modified | Plant Proteins - metabolism | DNA Fragmentation - drug effects | Oryza - embryology | Pollen - cytology | Pollen - drug effects | Germ Cells, Plant - drug effects | Chromosomes, Plant - drug effects | DNA Repair - drug effects | Mitosis - radiation effects | Mutagens - pharmacology | Replication Protein A - metabolism | Oryza - drug effects | RNA Interference - radiation effects | Chromosomes, Plant - metabolism | Mutation - genetics | Germ Cells, Plant - growth & development | Pollen - radiation effects | Mitomycin - pharmacology | Phenotype | DNA, Bacterial - genetics | Meiosis - radiation effects | Mitosis - drug effects | Genes, Plant - genetics | Recombination, Genetic - radiation effects | Methyl Methanesulfonate - pharmacology | Oryza - cytology | Recombination, Genetic - drug effects | Arabidopsis thaliana | DNA replication | Physiological aspects | Environmental aspects | Genetic aspects | DNA binding proteins | Properties
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2012, Volume 109, Issue 33, pp. 13302 - 13307
In postmitotic mammalian cells, protein p53R2 substitutes for protein R2 as a subunit of ribonucleotide reductase. In human patients with mutations in RRM2B,... 
Ribonucleotides | DNA damage | DNA | Cell lines | Fibroblasts | Irradiation | Fluorescence | Mitochondrial DNA | DNA repair | Genetic mutation | Mitochondrial disease | Cell cycle | DNA precursors | dNTP de novo synthesis | cell cycle | MULTIDISCIPLINARY SCIENCES | DEPLETION | mitochondrial disease | DAMAGE | FIBROBLASTS | NUCLEOTIDE EXCISION-REPAIR | STRAND BREAKS | INHIBITION | HISTONE H2AX PHOSPHORYLATION | GENE | RRM2B | MUTATIONS | Hydroxyurea - pharmacology | Fibroblasts - enzymology | DNA Replication - drug effects | Humans | DNA Repair - radiation effects | Ethidium - metabolism | DNA Repair - genetics | Fluorometry | Protein Subunits - metabolism | DNA, Mitochondrial - genetics | Ultraviolet Rays | Mitochondria - genetics | DNA Replication - radiation effects | Nucleotides - pharmacology | Mitochondria - radiation effects | Phosphorylation - drug effects | Nucleic Acid Conformation | Mammals - metabolism | DNA Repair - drug effects | Cell Cycle Proteins - metabolism | Mitochondria - drug effects | Fibroblasts - pathology | Mutation - genetics | Phosphorylation - radiation effects | Animals | Fibroblasts - radiation effects | Fibroblasts - drug effects | Ribonucleotide Reductases - metabolism | DNA Replication - genetics | Histones - metabolism | Hydroxyurea - metabolism | DNA replication | Physiological aspects | Research | Health aspects | Biological Sciences
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 24, pp. 10004 - 10009
Genome integrity is continuously threatened by external stresses and endogenous hazards such as DNA replication errors and reactive oxygen species. The DNA... 
Epidermal cells | DNA damage | Ploidies | DNA | Cell cycle | Stem cells | Giant cells | Root tips | Cell cycle proteins | Plant cells | Protein degradation | Root meristem | EARLY EMBRYONIC LETHALITY | MULTIDISCIPLINARY SCIENCES | KINASE | DAMAGE | root meristem | ENDOCYCLE | CELL-CYCLE CHECKPOINT | THALIANA | GENE-EXPRESSION | PLANTS | IONIZING-RADIATION | protein degradation | ATR | Gamma Rays | Mutagens - toxicity | Arabidopsis - growth & development | DNA Replication - drug effects | Plant Roots - genetics | Gene Expression Profiling | DNA Breaks, Double-Stranded - radiation effects | Cisplatin - toxicity | Ultraviolet Rays | DNA Breaks, Double-Stranded - drug effects | DNA Replication - radiation effects | Cell Cycle Proteins - genetics | Plant Roots - growth & development | DNA, Plant - genetics | Bleomycin - toxicity | Arabidopsis Proteins - genetics | Gene Expression Regulation, Plant - radiation effects | Arabidopsis - cytology | Cells, Cultured | Gene Expression Regulation, Developmental - drug effects | Protein-Serine-Threonine Kinases - genetics | Signal Transduction - genetics | Ataxia Telangiectasia Mutated Proteins | Transcription Factors - genetics | Arabidopsis - genetics | Gene Expression Regulation, Plant - drug effects | Gene Expression Regulation, Developmental - radiation effects | DNA Damage | Mutation | Methyl Methanesulfonate - toxicity | Arabidopsis thaliana | DNA replication | Genetic aspects | Research | Biological Sciences
Journal Article
Journal Article
DNA Repair, ISSN 1568-7864, 02/2014, Volume 14, Issue 1, pp. 27 - 38
Ultraviolet (UV)-induced DNA damage are removed by nucleotide excision repair (NER) or can be tolerated by specialized translesion synthesis (TLS) polymerases,... 
Cell cycle progression | Replication fork | UV damage bypass | γH2AX | TLS DNA polymerase η | NER | DNA strand breaks | UV-IRRADIATION | TLS DNA polymerase eta | TRANSLESION SYNTHESIS | DOUBLE-STRAND BREAKS | MAMMALIAN-CELLS | POLYMERASE ETA | NUCLEOTIDE EXCISION-REPAIR | HISTONE H2AX PHOSPHORYLATION | XERODERMA-PIGMENTOSUM | gamma H2AX | GENETICS & HEREDITY | CELL-CYCLE | TOXICOLOGY | CHECKPOINT ACTIVATION | G2 Phase - genetics | Genome, Human - radiation effects | DNA Replication - drug effects | Humans | DNA Repair - radiation effects | DNA-Directed DNA Polymerase - deficiency | Genome, Human - drug effects | DNA-Binding Proteins - deficiency | G2 Phase - drug effects | DNA Breaks, Double-Stranded - radiation effects | DNA, Single-Stranded - genetics | DNA Breaks, Single-Stranded - drug effects | Phosphorylation - genetics | DNA Breaks, Double-Stranded - drug effects | DNA Replication - radiation effects | Caffeine - pharmacology | Cell Cycle Checkpoints - genetics | Phosphorylation - drug effects | Cell Line | DNA Repair - drug effects | G2 Phase - radiation effects | DNA Breaks, Single-Stranded - radiation effects | DNA, Single-Stranded - metabolism | S Phase - genetics | Genome, Human - genetics | Ultraviolet Rays - adverse effects | DNA, Single-Stranded - biosynthesis | Phosphorylation - radiation effects | S Phase - radiation effects | Cell Cycle Checkpoints - radiation effects | Cell Cycle Checkpoints - drug effects | DNA Replication - genetics | S Phase - drug effects | DNA Damage | Histones - metabolism | Dose-Response Relationship, Radiation | Proteins | DNA damage | Genomics | DNA
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 04/2007, Volume 27, Issue 8, pp. 3131 - 3142
Journal Article