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Hippocampus, ISSN 1050-9631, 04/2012, Volume 22, Issue 4, pp. 723 - 736
Journal Article
Lipids in Health and Disease, ISSN 1476-511X, 2012, Volume 11, Issue 1, pp. 142 - 142
Journal Article
Oxidative Medicine and Cellular Longevity, ISSN 1942-0900, 2016, Volume 2016, pp. 8950384 - 10
Exercise training induces adaptations in mitochondrial metabolism, dynamics, and oxidative protection. Omega-3 fatty acids change membrane lipid composition... 
SKELETAL-MUSCLE | OXIDATIVE STRESS | BIOGENESIS | MONONUCLEAR-CELLS | DYNAMICS | POLYUNSATURATED FATTY-ACIDS | UNCOUPLING PROTEIN-3 | MECHANISMS | UP-REGULATION | EXERCISE | CELL BIOLOGY | Adaptation, Physiological | Leukocytes, Mononuclear - metabolism | Antioxidants - metabolism | Humans | Superoxide Dismutase - blood | Male | Protein Carbonylation - drug effects | Young Adult | Time Factors | Physical Fitness | Leukocytes, Mononuclear - drug effects | Oxidation-Reduction | Reactive Oxygen Species - blood | Soccer | Treatment Outcome | Mitochondria - metabolism | Mitochondrial Proteins - biosynthesis | Mitochondria - drug effects | Docosahexaenoic Acids - administration & dosage | Spain | Mitochondrial Dynamics - drug effects | Adolescent | Oxidative Stress - drug effects | Physical Conditioning, Human - methods | Dietary Supplements | Oxidases | Proteins | Cytochrome c | Antioxidants | Unsaturated fatty acids | Dietary supplements | Physiological aspects | Biosynthesis | Superoxide | Research | Omega-3 fatty acids | Oxidative stress | Health sciences | Colleges & universities | Cell division | Homeostasis | Mitochondrial DNA | Gene expression | Fatty acids | Signal transduction | Musculoskeletal system | Nutrition research | Mitochondria | Physical fitness | Exercise | Diet | Metabolites | Quality | College football | Physiology | Islands | Apoptosis | Index Medicus | Clinical Study
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 29, pp. 12828 - 12833
While many functions of the p53 tumor suppressor affect mitochondrial processes, the role of altered mitochondrial physiology in a modulation of p53 response... 
Up regulation | Pyrimidines | HCT116 cells | Epithelial cells | Cell lines | HeLa cells | Actins | Biosynthesis | Respiration | Apoptosis | p53 tumor suppressor | NQO1 and NQO2 | Dihydroorotate dehydrogenase | Mitochondrial electron transport chain | dihydroorotate dehydrogenase | mitochondrial electron transport chain | ACTIVATION | PROTEASOMAL DEGRADATION | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | PROTHYMOSIN-ALPHA | apoptosis | HYPOXIA | ARREST | INHIBITION | TUMOR-SUPPRESSOR | S-PHASE | STRESS | Mitochondria - enzymology | Electron Transport Complex III - metabolism | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Humans | Pyrimidines - biosynthesis | Membrane Potential, Mitochondrial - drug effects | Oxidoreductases Acting on CH-CH Group Donors - antagonists & inhibitors | Oxidoreductases Acting on CH-CH Group Donors - deficiency | Protein Processing, Post-Translational - drug effects | Cell Respiration - drug effects | Methacrylates - pharmacology | Potassium Cyanide - pharmacology | Quinone Reductases - metabolism | Tumor Suppressor Protein p53 - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Isoxazoles - pharmacology | Up-Regulation - drug effects | Electron Transport Complex III - antagonists & inhibitors | Signal Transduction - drug effects | Cell Line, Tumor | NAD(P)H Dehydrogenase (Quinone) - metabolism | Thiazoles - pharmacology | Proteasome Endopeptidase Complex - metabolism | Observations | Health aspects | Signal transduction | Tissue | Enzymes | DNA damage | Electron transfer | Organic chemicals | Tumors | Index Medicus | Phosphorylation | Carcinoma | Serine | Data processing | Nuclei | p53 protein | Electron transport chain | Mitochondria | Cytochrome bc1 | NAD(P)H dehydrogenase (quinone) | Hypoxia | Tumor suppressor genes | Intoxication | pyrimidines | Electron transport | p14ARF protein | Biological Sciences
Journal Article
Cell Metabolism, ISSN 1550-4131, 11/2013, Volume 18, Issue 5, pp. 698 - 711
mRNA translation is thought to be the most energy-consuming process in the cell. Translation and energy metabolism are dysregulated in a variety of diseases... 
MAMMALIAN TARGET | RAPAMYCIN | BINDING PARTNER | SKELETAL-MUSCLE | DIFFERENTIAL TRANSLATION | PROTEIN-KINASE | ENDOCRINOLOGY & METABOLISM | GENOME-WIDE | MOTIF PHOSPHORYLATION | CELL-GROWTH | COMPLEX 2 | CELL BIOLOGY | Protein Biosynthesis | Ribosomal Protein S6 Kinases - metabolism | TOR Serine-Threonine Kinases - metabolism | Humans | Mitochondrial Turnover | Adenosine Triphosphate - biosynthesis | Mechanistic Target of Rapamycin Complex 2 | Phosphoproteins - metabolism | RNA, Messenger - metabolism | Mechanistic Target of Rapamycin Complex 1 | DNA-Binding Proteins - metabolism | Multiprotein Complexes - metabolism | Cell Nucleus - metabolism | Cell Respiration | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mitochondrial Proteins - metabolism | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Autophagy - genetics | DNA, Mitochondrial - biosynthesis | Eukaryotic Initiation Factors - metabolism | RNA, Messenger - genetics | Gene Expression Regulation | Mitochondria - metabolism | Genome, Human - genetics | Transcription Factors - metabolism | Animals | Models, Biological | Mice | Adaptor Proteins, Signal Transducing - metabolism | RNA | Physiological aspects | Biosynthesis | Mitochondrial DNA | Diabetes | Binding proteins | Heart diseases | Genetic translation | Protein binding | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 07/2009, Volume 297, Issue 1, pp. 13 - 20
Pathways that regulate mitochondrial biogenesis are potential therapeutic targets for the amelioration of endothelial dysfunction and vascular disease.... 
Endothelial dysfunction | Histone deacetylase | Obesity | Vasoprotection | Diabetes | LIFE-SPAN | KAPPA-B ACTIVATION | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | NITRIC-OXIDE SYNTHASE | PROTEIN-KINASE | CARDIAC DYSFUNCTION | vasoprotection | NECROSIS-FACTOR-ALPHA | histone deacetylase | endothelial dysfunction | POLYPHENOLIC PHYTOALEXIN | CALORIC RESTRICTION | PERIPHERAL VASCULAR DISEASE | diabetes | obesity | SMALL-MOLECULE ACTIVATORS | RNA, Small Interfering - genetics | Electron Transport - drug effects | Diabetes Mellitus, Type 2 - genetics | Electron Transport - genetics | Nitric Oxide Synthase Type III - biosynthesis | Sirtuins - drug effects | Stilbenes - pharmacology | Endothelium, Vascular - physiology | Endothelial Cells - ultrastructure | RNA, Messenger - biosynthesis | DNA, Mitochondrial - genetics | Sirtuin 1 | RNA, Small Interfering - biosynthesis | Aorta, Thoracic - drug effects | Sirtuins - genetics | DNA, Mitochondrial - biosynthesis | Endothelial Cells - metabolism | RNA, Messenger - genetics | Cells, Cultured | Aorta, Thoracic - metabolism | Enzyme Induction | Mitochondria - metabolism | Antioxidants - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Aorta, Thoracic - cytology | Animals | Mice | Endothelial Cells - drug effects | Musculoskeletal system | Antibiotics | Rodents | Liver | Mitochondrial DNA | Biosynthesis | Cells | Metabolic disorders | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 12/2016, Volume 540, Issue 7632, pp. 270 - 275
Journal Article