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Journal Article
Mucosal Immunology, ISSN 1933-0219, 03/2016, Volume 9, Issue 2, pp. 444 - 457
Journal Article
PLoS Genetics, ISSN 1553-7390, 09/2011, Volume 7, Issue 9, p. e1002253
Aging is characterized by the accumulation of damaged cellular macromolecules caused by declining repair and elimination pathways. An integral component... 
20S PROTEASOME | YEAST | OXIDATIVE STRESS | ENHANCES LONGEVITY | CAENORHABDITIS-ELEGANS | GENETICS & HEREDITY | GENE DISRUPTION | DEGRADATION | AUTOPHAGY | DOMAIN PROTEIN | DELETION | Saccharomyces cerevisiae - genetics | Humans | Transcriptional Activation | Ubiquitin - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Silent Information Regulator Proteins, Saccharomyces cerevisiae - genetics | Sirtuin 2 - metabolism | DNA-Binding Proteins - metabolism | Gene Expression Regulation, Fungal | Saccharomyces cerevisiae - physiology | Silent Information Regulator Proteins, Saccharomyces cerevisiae - metabolism | Oxidative Stress - genetics | Ubiquitin-Protein Ligases - metabolism | Ubiquitin - genetics | Saccharomyces cerevisiae Proteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Phosphatidylinositol 3-Kinases - genetics | Proteasome Endopeptidase Complex - genetics | Transcription Factors - metabolism | Sirtuin 2 - genetics | Saccharomyces cerevisiae Proteins - metabolism | DNA Replication - genetics | Proteasome Endopeptidase Complex - metabolism | Ubiquitin-Protein Ligases - genetics | Physiological aspects | Transcription factors | Genetic aspects | Research | Brewer's yeast | Ubiquitin-proteasome system | Protein folding | Aging | Homeostasis | Software | Experiments | Age | Cytoplasm
Journal Article
Nature, ISSN 0028-0836, 01/2002, Volume 415, Issue 6867, pp. 92 - 96
The unfolded protein response (UPR), caused by stress, matches the folding capacity of endoplasmic reticulum (ER) to the load of client proteins in the... 
Transcription Factors - chemistry | Caenorhabditis elegans Proteins - chemistry | Caenorhabditis elegans Proteins - metabolism | Endoplasmic Reticulum - metabolism | Molecular Sequence Data | RNA, Messenger - metabolism | Stem Cells - metabolism | Caenorhabditis elegans Proteins - secretion | Endoplasmic Reticulum - secretion | X-Box Binding Protein 1 | DNA-Binding Proteins - metabolism | Transcription Factors - secretion | RNA Splicing | RNA, Helminth - metabolism | Base Sequence | Protein Denaturation | Fibroblasts | Nucleic Acid Conformation | RNA, Helminth - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - secretion | Cell Line | Caenorhabditis elegans - metabolism | Introns - genetics | Caenorhabditis elegans - genetics | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mutation - genetics | DNA-Binding Proteins - chemistry | Regulatory Factor X Transcription Factors | Protein Folding | Membrane Proteins | Transcription Factors - metabolism | RNA, Helminth - chemistry | Animals | Caenorhabditis elegans - cytology | RNA, Messenger - chemistry | Mice | Caenorhabditis elegans Proteins - genetics | Physiological aspects | Genetic aspects | Messenger RNA | DNA binding proteins | Research | Endoplasmic reticulum | Proteins | Mutation | Ribonucleic acid--RNA | Ribonucleic acid | Genes | Worms
Journal Article
Journal of Immunology, ISSN 0022-1767, 08/2013, Volume 191, Issue 4, pp. 1927 - 1934
Helminthic infections protect mice from colitis in murine models of inflammatory bowel disease and also may protect people. Helminths like Heligmosomoides... 
EXCRETORY-SECRETORY PRODUCTS | HOMEOSTASIS | CYTOKINE PRODUCTION | CROHNS-DISEASE | MECHANISM | INFLAMMATION | MICE | INDUCTION | IMMUNOLOGY | PROTEOMIC ANALYSIS | EXPRESSION | T-Lymphocyte Subsets - immunology | Lymph Nodes - pathology | Spleen - immunology | Colon - parasitology | Colon - immunology | DNA-Binding Proteins - deficiency | Nematospiroides dubius - immunology | T-Lymphocytes, Regulatory - immunology | Helminthiasis, Animal - immunology | Intestinal Mucosa - immunology | Strongylida Infections - immunology | T-Lymphocyte Subsets - chemistry | T-Lymphocytes, Regulatory - chemistry | Colitis - immunology | Spleen - pathology | Interleukin-10 - analysis | Forkhead Transcription Factors - deficiency | Genes, Reporter | Disease Models, Animal | Specific Pathogen-Free Organisms | Immunotherapy, Adoptive | Mice, Inbred C57BL | Cytokines - secretion | Graft Survival | Mice, Transgenic | Mesentery | Therapy with Helminths | Lymph Nodes - immunology | T-Lymphocyte Subsets - transplantation | Intestinal Diseases, Parasitic - immunology | Colitis - parasitology | Mice, Knockout | Animals | Forkhead Transcription Factors - analysis | Mice | T-Lymphocytes, Regulatory - transplantation | Colitis - prevention & control | Cytokines - biosynthesis | Inflammatory Bowel Diseases - therapy | Intestinal Mucosa - pathology | colitis | helminths | Foxp3 | T cells | mucosa
Journal Article
Experimental Dermatology, ISSN 0906-6705, 07/2015, Volume 24, Issue 7, pp. 554 - 556
Journal Article
Immunity, ISSN 1074-7613, 2013, Volume 39, Issue 2, pp. 259 - 271
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, p. e39102
ERR alpha is an orphan nuclear receptor emerging as a novel biomarker of breast cancer. Over-expression of ERR alpha in breast tumor is considered as a... 
COACTIVATOR-1-ALPHA | ERR-ALPHA | INDEPENDENT TRANSCRIPTIONAL ACTIVATION | GROWTH | BIOLOGY | GENE-EXPRESSION | RNAS | ORPHAN RECEPTOR | MICRORNA TARGETS | DIFFERENTIATION | IDENTIFICATION | Oncogene Proteins - genetics | Receptors, Estrogen - metabolism | Cell Proliferation | Receptors, Estrogen - genetics | Signal Transduction | Humans | RNA, Messenger - genetics | Gene Expression Regulation, Neoplastic | Oncogene Proteins - metabolism | MicroRNAs - metabolism | Cyclin E - genetics | Cell Movement - genetics | Breast Neoplasms - metabolism | Breast Neoplasms - genetics | RNA Interference | Base Sequence | Cell Line, Tumor | Cell Cycle Checkpoints - genetics | Female | RNA, Messenger - chemistry | MicroRNAs - genetics | Cyclin E - metabolism | 3' Untranslated Regions | Binding Sites | Prognosis | MicroRNA | Genes | Luciferase | Estrogen | Cancer cells | Protection and preservation | Phenols | Development and progression | Breast cancer | Genetic aspects | Genetic transcription | Cell proliferation | Post-transcription | Leukocyte migration | Estrogens | Colorectal cancer | Biochemistry | Body mass index | Reporter gene | Transfection | Epidermal growth factor | Cell cycle | miRNA | Physiology | Tumorigenesis | Bioinformatics | Lymphatic system | Cloning | Metabolism | Gene expression | Ribonucleic acid--RNA | Studies | Overexpression | 3' Untranslated regions | MicroRNAs | Medical prognosis | Biomarkers | Breast | Tumor suppressor genes | Ligands | Genetic engineering | Molecular biology | Prostate cancer | Cell migration | Cancer | Tumors | RNA | Ribonucleic acid
Journal Article