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Publication DateJournal of Neurochemistry, ISSN 0022-3042, 01/2008, Volume 104, Issue 1, pp. 254 - 263
It has previously been observed that expression of chemokine monocyte chemoattractant protein‐1 (MCP‐1/CC chemokine ligand 2 (CCL2)) and its receptor CC...
transient receptor potential vanilloid receptor subtype 1 | chemokine | neuropathic pain | transient receptor potential ankyrin 1 | neurotransmitter | dorsal root ganglion | Chemokine | Neurotransmitter | Transient receptor potential vanilloid receptor subtype 1 | Dorsal root ganglion | Transient receptor potential ankyrin 1 | Neuropathic pain | BIOCHEMISTRY & MOLECULAR BIOLOGY | PRIMARY NOCICEPTIVE NEURONS | SPINAL-CORD | NEUROSCIENCES | ADULT-RAT BRAIN | CAPSAICIN RECEPTOR | ENDOTHELIAL-CELLS | CENTRAL-NERVOUS-SYSTEM | CHEMOKINE RECEPTORS | EXPRESSION | CCR2 | Enzyme-Linked Immunosorbent Assay - methods | Receptors, CCR2 - genetics | Receptors, CCR2 - physiology | Green Fluorescent Proteins - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Humans | Gene Expression Regulation - physiology | Mice, Transgenic | Ganglia, Spinal - cytology | Green Fluorescent Proteins - genetics | Sciatic Neuropathy - chemically induced | Polysaccharides | Animals | Drug Interactions | Transfection - methods | Chemokine CCL2 - metabolism | Mice | Neurons - metabolism | Neurons - drug effects | Sciatic Neuropathy - metabolism | Cell Line, Transformed | Disease Models, Animal | Neurons | Neurosciences | Neurology | Signal transduction | Biochemistry | Pain | Gene expression | Rodents | Index Medicus | DRG | TRPV1 | TRPA1
transient receptor potential vanilloid receptor subtype 1 | chemokine | neuropathic pain | transient receptor potential ankyrin 1 | neurotransmitter | dorsal root ganglion | Chemokine | Neurotransmitter | Transient receptor potential vanilloid receptor subtype 1 | Dorsal root ganglion | Transient receptor potential ankyrin 1 | Neuropathic pain | BIOCHEMISTRY & MOLECULAR BIOLOGY | PRIMARY NOCICEPTIVE NEURONS | SPINAL-CORD | NEUROSCIENCES | ADULT-RAT BRAIN | CAPSAICIN RECEPTOR | ENDOTHELIAL-CELLS | CENTRAL-NERVOUS-SYSTEM | CHEMOKINE RECEPTORS | EXPRESSION | CCR2 | Enzyme-Linked Immunosorbent Assay - methods | Receptors, CCR2 - genetics | Receptors, CCR2 - physiology | Green Fluorescent Proteins - metabolism | Calcium - metabolism | Capsaicin - pharmacology | Humans | Gene Expression Regulation - physiology | Mice, Transgenic | Ganglia, Spinal - cytology | Green Fluorescent Proteins - genetics | Sciatic Neuropathy - chemically induced | Polysaccharides | Animals | Drug Interactions | Transfection - methods | Chemokine CCL2 - metabolism | Mice | Neurons - metabolism | Neurons - drug effects | Sciatic Neuropathy - metabolism | Cell Line, Transformed | Disease Models, Animal | Neurons | Neurosciences | Neurology | Signal transduction | Biochemistry | Pain | Gene expression | Rodents | Index Medicus | DRG | TRPV1 | TRPA1
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Loading RightsBritish Journal of Pharmacology, ISSN 0007-1188, 05/2018, Volume 175, Issue 10, pp. 1760 - 1769
BACKGROUND AND PURPOSE The P2X3 receptor is a major receptor in the processing of nociceptive information in dorsal root ganglia. We investigated the role of...
NEUROPATHIC PAIN | PUBLICATION | ANTAGONIST | CONCISE GUIDE | P2X RECEPTOR | TOLERANCE | INVOLVEMENT | PHARMACOLOGY & PHARMACY | INFLAMMATORY PAIN | HYPERALGESIA | EPAC | Phosphorylation | Protein kinase C | Pain perception | Thresholds | Neurons | Cyclic AMP | Staining | Morphine | Exposure | Ganglia | Latency | Signaling | Pain | Dorsal root ganglia | Inhibitors | Analgesics | Drug tolerance | Information processing | Peripherin | Attenuation | Position (location) | Localization | Immunofluorescence | Research Papers | Research Paper
NEUROPATHIC PAIN | PUBLICATION | ANTAGONIST | CONCISE GUIDE | P2X RECEPTOR | TOLERANCE | INVOLVEMENT | PHARMACOLOGY & PHARMACY | INFLAMMATORY PAIN | HYPERALGESIA | EPAC | Phosphorylation | Protein kinase C | Pain perception | Thresholds | Neurons | Cyclic AMP | Staining | Morphine | Exposure | Ganglia | Latency | Signaling | Pain | Dorsal root ganglia | Inhibitors | Analgesics | Drug tolerance | Information processing | Peripherin | Attenuation | Position (location) | Localization | Immunofluorescence | Research Papers | Research Paper
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Loading RightsPain, ISSN 0304-3959, 07/2018, Volume 159, Issue 7, pp. 1325 - 1345
Molecular neurobiological insight into human nervous tissues is needed to generate next-generation therapeutics for neurological disorders such as chronic...
Comparative transcriptomics | Human sensory neuroscience | Dorsal root ganglion | Target identification | TRIGEMINAL GANGLIA | PROTEIN-SYNTHESIS | NERVE INJURY | NEUROSCIENCES | CLINICAL NEUROLOGY | DIFFERENTIAL GENE | NEUROPATHIC PAIN | SENSITIZATION | GROWTH | GENE-EXPRESSION | ANESTHESIOLOGY | DIVERSITY | REVEALS | Index Medicus
Comparative transcriptomics | Human sensory neuroscience | Dorsal root ganglion | Target identification | TRIGEMINAL GANGLIA | PROTEIN-SYNTHESIS | NERVE INJURY | NEUROSCIENCES | CLINICAL NEUROLOGY | DIFFERENTIAL GENE | NEUROPATHIC PAIN | SENSITIZATION | GROWTH | GENE-EXPRESSION | ANESTHESIOLOGY | DIVERSITY | REVEALS | Index Medicus
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Loading RightsPLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e79523 - e79523
The specific functions of sensory systems depend on the tissue-specific expression of genes that code for molecular sensor proteins that are necessary for...
PERIPHERAL-NERVE INJURY | RAT TEMPOROMANDIBULAR-JOINT | ACTIVATED CHLORIDE CHANNEL | SENSITIVE K+ CHANNEL | MULTIDISCIPLINARY SCIENCES | PROTEIN-COUPLED RECEPTORS | POTENTIAL THERAPEUTIC TARGET | SMOOTH-MUSCLE-CELLS | OF-FUNCTION MUTATIONS | DORSAL-ROOT GANGLION | GATED CALCIUM-CHANNELS | Animals | In Situ Hybridization | Receptors, G-Protein-Coupled | Trigeminal Ganglion - metabolism | Male | Ganglia, Sensory - metabolism | High-Throughput Nucleotide Sequencing | Mice | Ganglia, Spinal - metabolism | Potassium Channels - genetics | Proteins | RNA sequencing | RNA | Analysis | Genes | Liver | Muscles | Ion channels | G proteins | Gene expression | Brain | Neurosciences | G protein-coupled receptors | Transcription | Genomics | Diabetic neuropathy | Genomes | Pruritus | Gene sequencing | Receptors | Pain | Dorsal root ganglia | Rodents | Animal tissues | Sensory evaluation | Sensory neurons | Physiology | Bioinformatics | Cytokines | Neurons | Sensory systems | Mammals | Epithelium | Ribonucleic acid--RNA | Ganglia | Skeletal muscle | Signaling | Ligands | Olfactory epithelium | Trigeminal ganglion | Index Medicus | Ribonucleic acid
PERIPHERAL-NERVE INJURY | RAT TEMPOROMANDIBULAR-JOINT | ACTIVATED CHLORIDE CHANNEL | SENSITIVE K+ CHANNEL | MULTIDISCIPLINARY SCIENCES | PROTEIN-COUPLED RECEPTORS | POTENTIAL THERAPEUTIC TARGET | SMOOTH-MUSCLE-CELLS | OF-FUNCTION MUTATIONS | DORSAL-ROOT GANGLION | GATED CALCIUM-CHANNELS | Animals | In Situ Hybridization | Receptors, G-Protein-Coupled | Trigeminal Ganglion - metabolism | Male | Ganglia, Sensory - metabolism | High-Throughput Nucleotide Sequencing | Mice | Ganglia, Spinal - metabolism | Potassium Channels - genetics | Proteins | RNA sequencing | RNA | Analysis | Genes | Liver | Muscles | Ion channels | G proteins | Gene expression | Brain | Neurosciences | G protein-coupled receptors | Transcription | Genomics | Diabetic neuropathy | Genomes | Pruritus | Gene sequencing | Receptors | Pain | Dorsal root ganglia | Rodents | Animal tissues | Sensory evaluation | Sensory neurons | Physiology | Bioinformatics | Cytokines | Neurons | Sensory systems | Mammals | Epithelium | Ribonucleic acid--RNA | Ganglia | Skeletal muscle | Signaling | Ligands | Olfactory epithelium | Trigeminal ganglion | Index Medicus | Ribonucleic acid
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Loading RightsJournal of Clinical Investigation, ISSN 0021-9738, 05/2017, Volume 127, Issue 5, pp. 1741 - 1756
The integration of somatosensory information is generally assumed to be a function of the central nervous system ( CNS). Here we describe fully functional...
MEDICINE, RESEARCH & EXPERIMENTAL | RAT SPINAL-CORD | SUBUNIT MESSENGER-RNAS | CHRONIC PAIN | DRG NEURONS | PATCH-CLAMP | DORSAL-ROOT GANGLION | ACTION-POTENTIAL PROPAGATION | NERVE INJURY | SATELLITE GLIAL-CELLS | GABA-INDUCED DEPOLARIZATION | Rats, Wistar | Neuralgia - metabolism | Rats | GABA Uptake Inhibitors - pharmacology | Ganglia, Spinal - physiopathology | Neuralgia - pathology | GABA Uptake Inhibitors - adverse effects | Rats, Sprague-Dawley | Animals | Ganglia, Spinal - pathology | Nociception - drug effects | Neuralgia - chemically induced | Synaptic Transmission - drug effects | GABAergic Neurons - metabolism | Neuralgia - physiopathology | Ganglia, Spinal - metabolism | Nociception | GABA | Cellular signal transduction | Research | Health aspects | Ganglia | Index Medicus | Abridged Index Medicus
MEDICINE, RESEARCH & EXPERIMENTAL | RAT SPINAL-CORD | SUBUNIT MESSENGER-RNAS | CHRONIC PAIN | DRG NEURONS | PATCH-CLAMP | DORSAL-ROOT GANGLION | ACTION-POTENTIAL PROPAGATION | NERVE INJURY | SATELLITE GLIAL-CELLS | GABA-INDUCED DEPOLARIZATION | Rats, Wistar | Neuralgia - metabolism | Rats | GABA Uptake Inhibitors - pharmacology | Ganglia, Spinal - physiopathology | Neuralgia - pathology | GABA Uptake Inhibitors - adverse effects | Rats, Sprague-Dawley | Animals | Ganglia, Spinal - pathology | Nociception - drug effects | Neuralgia - chemically induced | Synaptic Transmission - drug effects | GABAergic Neurons - metabolism | Neuralgia - physiopathology | Ganglia, Spinal - metabolism | Nociception | GABA | Cellular signal transduction | Research | Health aspects | Ganglia | Index Medicus | Abridged Index Medicus
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Loading RightsPain Medicine, ISSN 1526-2375, 10/2013, Volume 14, Issue 10, pp. 1557 - 1568
There is an unmet clinical need for novel analgesics for neuropathic pain. This study was designed to elucidate the mechanism through which EMA300, a small...
Analgesia | Type 2 Receptor | p38 Mitogen Activated Protein Kinase | p44/p42 | Angiotensin | Neuropathic Pain | MAPK | Dorsal Root Ganglia | P38 Mitogen Activated Protein Kinase (MAPK) | P44/p42 MAPK | Angiotensin II | Immunohistochemistry | Analgesics - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Neuralgia - metabolism | Molecular Sequence Data | Rats | Male | Imidazoles - pharmacology | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Neuralgia - drug therapy | Angiotensin II Type 2 Receptor Blockers - pharmacology | Mice, Knockout | Animals | Ganglia, Spinal - enzymology | Mitogen-Activated Protein Kinase 3 - metabolism | Female | Mice | Pyridines - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Ganglia, Spinal - drug effects | Disease Models, Animal | Mitogen-Activated Protein Kinase 1 - metabolism | Analgesics | Protein kinases | Mitogens | Pain | Kinases | Rodents | Index Medicus
Analgesia | Type 2 Receptor | p38 Mitogen Activated Protein Kinase | p44/p42 | Angiotensin | Neuropathic Pain | MAPK | Dorsal Root Ganglia | P38 Mitogen Activated Protein Kinase (MAPK) | P44/p42 MAPK | Angiotensin II | Immunohistochemistry | Analgesics - pharmacology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Neuralgia - metabolism | Molecular Sequence Data | Rats | Male | Imidazoles - pharmacology | Enzyme Activation - drug effects | Rats, Sprague-Dawley | Neuralgia - drug therapy | Angiotensin II Type 2 Receptor Blockers - pharmacology | Mice, Knockout | Animals | Ganglia, Spinal - enzymology | Mitogen-Activated Protein Kinase 3 - metabolism | Female | Mice | Pyridines - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Ganglia, Spinal - drug effects | Disease Models, Animal | Mitogen-Activated Protein Kinase 1 - metabolism | Analgesics | Protein kinases | Mitogens | Pain | Kinases | Rodents | Index Medicus
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Loading RightsPLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, pp. e68312 - e68312
This study examines key elements of glutamatergic transmission within sensory ganglia of the rat. We show that the soma of primary sensory neurons release...
PRIMARY AFFERENT NEURONS | PERIPHERAL NERVOUS-SYSTEM | MULTIDISCIPLINARY SCIENCES | CHRONIC CONSTRICTION INJURY | RAT DRG NEURONS | SPINAL-CORD | SUBSTANCE-P | DORSAL-ROOT GANGLION | SATELLITE GLIAL-CELLS | MOUSE TRIGEMINAL GANGLIA | EXCITATORY AMINO-ACIDS | Immunohistochemistry | Receptors, Glutamate - metabolism | Calcium - metabolism | Male | Neurons - cytology | Ganglia, Sensory - metabolism | Ganglia, Spinal - cytology | Satellite Cells, Perineuronal - metabolism | Neuroglia - cytology | Sciatic Nerve - metabolism | Neurons - physiology | Neurons - metabolism | Membrane Potentials - drug effects | Trigeminal Ganglion - cytology | Glutamic Acid - pharmacology | Cells, Cultured | Neurotransmitter Agents - metabolism | Rats | Trigeminal Ganglion - metabolism | Excitatory Amino Acid Agonists - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Membrane Potentials - physiology | Rats, Sprague-Dawley | Microscopy, Confocal | Animals | Ganglia, Sensory - cytology | Sciatic Nerve - surgery | Neuroglia - metabolism | Glutamic Acid - metabolism | Culture Media, Conditioned - metabolism | Ganglia, Spinal - metabolism | Physiological aspects | Neurotransmitters | Genetic aspects | Research | Glutamate | Ganglia | Cell culture | Neurosciences | Spinal cord | Calcium | Glutamic acid receptors (ionotropic) | Antagonists | Colorimetry | Glutamic acid receptors | Glial cells | Neuronal-glial interactions | Depolarization | Receptors | Dorsal root ganglia | Pain | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid | Chlorides | Rodents | Surgery | Sensory neurons | Enzymes | Pain perception | Neurons | Calcium imaging | Cultures | Data processing | N-Methyl-D-aspartic acid receptors | Gene expression | Potassium chloride | Axons | Glutamatergic transmission | Trigeminal ganglion | Glutamic acid receptors (metabotropic) | Sciatic nerve | Index Medicus
PRIMARY AFFERENT NEURONS | PERIPHERAL NERVOUS-SYSTEM | MULTIDISCIPLINARY SCIENCES | CHRONIC CONSTRICTION INJURY | RAT DRG NEURONS | SPINAL-CORD | SUBSTANCE-P | DORSAL-ROOT GANGLION | SATELLITE GLIAL-CELLS | MOUSE TRIGEMINAL GANGLIA | EXCITATORY AMINO-ACIDS | Immunohistochemistry | Receptors, Glutamate - metabolism | Calcium - metabolism | Male | Neurons - cytology | Ganglia, Sensory - metabolism | Ganglia, Spinal - cytology | Satellite Cells, Perineuronal - metabolism | Neuroglia - cytology | Sciatic Nerve - metabolism | Neurons - physiology | Neurons - metabolism | Membrane Potentials - drug effects | Trigeminal Ganglion - cytology | Glutamic Acid - pharmacology | Cells, Cultured | Neurotransmitter Agents - metabolism | Rats | Trigeminal Ganglion - metabolism | Excitatory Amino Acid Agonists - pharmacology | Excitatory Amino Acid Antagonists - pharmacology | Membrane Potentials - physiology | Rats, Sprague-Dawley | Microscopy, Confocal | Animals | Ganglia, Sensory - cytology | Sciatic Nerve - surgery | Neuroglia - metabolism | Glutamic Acid - metabolism | Culture Media, Conditioned - metabolism | Ganglia, Spinal - metabolism | Physiological aspects | Neurotransmitters | Genetic aspects | Research | Glutamate | Ganglia | Cell culture | Neurosciences | Spinal cord | Calcium | Glutamic acid receptors (ionotropic) | Antagonists | Colorimetry | Glutamic acid receptors | Glial cells | Neuronal-glial interactions | Depolarization | Receptors | Dorsal root ganglia | Pain | α-Amino-3-hydroxy-5-methyl-4-isoxazole propionic acid | Chlorides | Rodents | Surgery | Sensory neurons | Enzymes | Pain perception | Neurons | Calcium imaging | Cultures | Data processing | N-Methyl-D-aspartic acid receptors | Gene expression | Potassium chloride | Axons | Glutamatergic transmission | Trigeminal ganglion | Glutamic acid receptors (metabotropic) | Sciatic nerve | Index Medicus
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Loading RightsGlia, ISSN 0894-1491, 06/2019, Volume 67, Issue 6, pp. 1062 - 1075
Chronic pain is one of the most prevalent chronic diseases in the world. The plastic changes of sensory neurons in dorsal root ganglia (DRG) have been...
neurogenesis | chronic pain | dorsal root ganglia | satellite glial cells | NERVOUS-SYSTEM | PATHOBIOLOGY | INJURY | PROLIFERATION | GLIAL-CELLS | NEUROSCIENCES | SENSORY NEURONS | ENTERIC GLIA | IDENTITY | MEMORY | EXPRESSION | Care and treatment | Pain | Analysis | Chronic pain | Tetrodotoxin | Pain perception | Neurons | Capsaicin | Glial fibrillary acidic protein | Satellite cells | Glial cells | Neurogenesis | Ganglia | Neuronal-glial interactions | Proteins | Calcitonin gene-related peptide | Enteric nervous system | Dorsal root ganglia | Cells (biology) | Plasticity | Sensory neurons | Carotid body | Growth factors | Sciatic nerve | Chronic illnesses
neurogenesis | chronic pain | dorsal root ganglia | satellite glial cells | NERVOUS-SYSTEM | PATHOBIOLOGY | INJURY | PROLIFERATION | GLIAL-CELLS | NEUROSCIENCES | SENSORY NEURONS | ENTERIC GLIA | IDENTITY | MEMORY | EXPRESSION | Care and treatment | Pain | Analysis | Chronic pain | Tetrodotoxin | Pain perception | Neurons | Capsaicin | Glial fibrillary acidic protein | Satellite cells | Glial cells | Neurogenesis | Ganglia | Neuronal-glial interactions | Proteins | Calcitonin gene-related peptide | Enteric nervous system | Dorsal root ganglia | Cells (biology) | Plasticity | Sensory neurons | Carotid body | Growth factors | Sciatic nerve | Chronic illnesses
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Loading RightsBrain Research, ISSN 0006-8993, 2016, Volume 1644, pp. 183 - 191
Abstract Adrenomedullin (AM), a member of the calcitonin gene-related peptide (CGRP) family, has been demonstrated to be a pain peptide. This study...
Neurology | Adrenomedullin (AM) | Tumor necrosis factor-alpha (TNF-α) | Nuclear factor kappa B (NF-kB) | Extracellular signal-related kinase (ERK) | cAMP-responsive element binding protein (CREB) | Satellite cells | Dorsal root ganglion (DRG) | MECHANICAL ALLODYNIA | NEUTRALIZING ANTIBODIES | Tumor necrosis factor-alpha (TNF-alpha) | PROTEIN-KINASE | PAIN-RELATED BEHAVIOR | NEUROSCIENCES | SATELLITE GLIAL-CELLS | NEUROPATHIC PAIN | NEUROTROPHIC FACTOR EXPRESSION | SPINAL NERVE LIGATION | DEPENDENT UP-REGULATION | TNF-ALPHA | Receptors, Adrenomedullin - metabolism | Rats | Male | Peptide Fragments - pharmacology | Adrenomedullin - metabolism | Adrenomedullin - pharmacology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Rats, Sprague-Dawley | Receptors, Adrenomedullin - antagonists & inhibitors | Tumor Necrosis Factor-alpha - pharmacology | Matrix Metalloproteinase 9 - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Interleukin-1beta - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Chemokine CCL2 - metabolism | Cell Culture Techniques | Ganglia, Spinal - drug effects | Receptors, Adrenomedullin - agonists | Ganglia, Spinal - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
Neurology | Adrenomedullin (AM) | Tumor necrosis factor-alpha (TNF-α) | Nuclear factor kappa B (NF-kB) | Extracellular signal-related kinase (ERK) | cAMP-responsive element binding protein (CREB) | Satellite cells | Dorsal root ganglion (DRG) | MECHANICAL ALLODYNIA | NEUTRALIZING ANTIBODIES | Tumor necrosis factor-alpha (TNF-alpha) | PROTEIN-KINASE | PAIN-RELATED BEHAVIOR | NEUROSCIENCES | SATELLITE GLIAL-CELLS | NEUROPATHIC PAIN | NEUROTROPHIC FACTOR EXPRESSION | SPINAL NERVE LIGATION | DEPENDENT UP-REGULATION | TNF-ALPHA | Receptors, Adrenomedullin - metabolism | Rats | Male | Peptide Fragments - pharmacology | Adrenomedullin - metabolism | Adrenomedullin - pharmacology | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Rats, Sprague-Dawley | Receptors, Adrenomedullin - antagonists & inhibitors | Tumor Necrosis Factor-alpha - pharmacology | Matrix Metalloproteinase 9 - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Interleukin-1beta - metabolism | Cyclic AMP Response Element-Binding Protein - metabolism | Chemokine CCL2 - metabolism | Cell Culture Techniques | Ganglia, Spinal - drug effects | Receptors, Adrenomedullin - agonists | Ganglia, Spinal - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
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