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Journal of cell death, ISSN 1179-0660, 12/2016, Volume 9, Issue 9, pp. JCD.S39824 - 29
Hepatocyte apoptosis plays important roles in both the removal of external microorganisms and the occurrence and development of liver diseases... 
Signaling pathways | Viral hepatitis | Drug-induced liver injury | Fatty liver disease | Hepatocyte apoptosis | Hepatic ischemia reperfusion injury | Complications and side effects | Liver diseases | Research | Liver cells | Health aspects | Risk factors | Apoptosis
Journal Article
Toxicology and applied pharmacology, ISSN 0041-008X, 12/2015, Volume 289, Issue 2, pp. 163 - 176
...-β1 also resulted in a significant suppression of SIRT1 protein. Nevertheless, TGF-β1-induced LX-2 cell activation was inhibited by SIRT1 plasmid, and this was accompanied by up-regulation of cell apoptosis-related proteins... 
Reversion | MALAT1 | Liver fibrosis | SIRT1 | Apoptosis | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Sirtuin 1 - metabolism | Liver - pathology | Liver - enzymology | Apoptosis - drug effects | Carbon Tetrachloride | Humans | Actins - metabolism | Myofibroblasts - enzymology | Male | RNA, Messenger - metabolism | Xanthines - pharmacology | Sirtuin 1 - genetics | Actins - genetics | Dose-Response Relationship, Drug | Liver Cirrhosis, Experimental - prevention & control | Liver Cirrhosis, Experimental - enzymology | Collagen Type I - genetics | Dexamethasone - pharmacology | Transfection | Liver - drug effects | RNA Interference | Time Factors | Liver Cirrhosis, Experimental - pathology | Female | Chemical and Drug Induced Liver Injury - pathology | Chemical and Drug Induced Liver Injury - enzymology | Chemical and Drug Induced Liver Injury - etiology | Hepatic Stellate Cells - pathology | Transforming Growth Factor beta1 - pharmacology | Hepatic Stellate Cells - drug effects | Cell Line | Chemical and Drug Induced Liver Injury - prevention & control | Insulin - pharmacology | Myofibroblasts - pathology | Collagen Type I - metabolism | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Hepatic Stellate Cells - enzymology | Chemical and Drug Induced Liver Injury - genetics | Liver Cirrhosis, Experimental - genetics | Phenotype | Animals | Liver Cirrhosis, Experimental - chemically induced | RNA, Long Noncoding - metabolism | Corticosteroids | Liver | Analysis | Fibrosis | Bone morphogenetic proteins | Transforming growth factors | Liver cirrhosis | Index Medicus | APOPTOSIS | DEXAMETHASONE | COLLAGEN | RNA | LUNGS | METASTASES | 60 APPLIED LIFE SCIENCES | HISTONES | MICE | VIRUSES | FIBROSIS | ONCOGENES | TRANSCRIPTION FACTORS | ACTIN | MATERIALS RECOVERY | STEROLS | TRANSCRIPTION | PHENOTYPE | INSULIN | NAD | GROWTH FACTORS | CARBON TETRACHLORIDE | LIVER | CARCINOMAS | RECEPTORS | MUSCLES
Journal Article
PloS one, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, pp. e90443 - e90443
.... Increased levels of ALT and enhanced cell apoptosis were observed in the liver of rat offspring at 15 and 21 weeks, and significantly increased activity of caspase-3 and caspase-9 and elevated levels... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Liver - pathology | Rats, Wistar | Apoptosis - drug effects | Benzhydryl Compounds - toxicity | Mitochondria, Liver - metabolism | Phenols - toxicity | Body Weight - drug effects | Male | Alanine Transaminase - blood | Liver - drug effects | Time Factors | Mitochondrial Proteins - metabolism | Female | DNA Fragmentation - drug effects | Chemical and Drug Induced Liver Injury - blood | Chemical and Drug Induced Liver Injury - etiology | Animals, Newborn | Liver - metabolism | Prenatal Exposure Delayed Effects - metabolism | Triglycerides - metabolism | Mitochondria, Liver - drug effects | Pregnancy | Organ Size - drug effects | Animals | Chemical and Drug Induced Liver Injury - metabolism | Estrogens, Non-Steroidal - toxicity | Lipid Metabolism - drug effects | Prenatal Exposure Delayed Effects - chemically induced | Cytochrome c | Animal experimentation | Bisphenol-A | Polycarbonates | Liver | Phenols | Apoptosis | Cytochrome | Neonates | Perinatal exposure | Bax protein | Bcl-2 protein | Laboratories | Pathogenesis | Science | Caspase-3 | Nuclei | Proteins | Mitochondria | Education | Rodents | DNA methylation | Polymers | Hepatotoxicity | Polycarbonate | Public health | Abnormalities | Environmental health | Rats | Caspase | Exposure | Permeability | Metabolism | Studies | Bisphenol A | Progeny | Offspring | Caspase-9 | Environmental protection | Index Medicus
Journal Article