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Chest, ISSN 0012-3692, 2016, Volume 149, Issue 6, pp. 1445 - 1459
Journal Article
Annales d'Endocrinologie, ISSN 0003-4266, 04/2011, Volume 72, Issue 2, pp. 82 - 86
Congenital hypothyroidism (CH) is the most common congenital endocrine disorder, accounting for up to 1:1500 newborns per year. CH can be related to defects in... 
Congenital hypothyroidism | DUOX A | DUOX | Dyshormonogenesis
Journal Article
Redox Biology, ISSN 2213-2317, 01/2020, Volume 28, p. 101346
The NADPH oxidase reaction produces protons. In the case of the NADPH oxidase, NOX2, activity depends on secretion of these protons and is inhibited by... 
Hv1 | Bronchial epithelium | Duox | Zinc
Journal Article
Methods in Molecular Biology, ISSN 1064-3745, 2019, Volume 1982, pp. 173 - 190
Biosynthesis of active human dual oxidases (DUOX1 and DUOX2) requires maturation factors, a.k.a. DUOX activator proteins (DUOXA1 and DUOXA2), that form... 
Dual oxidases | DUOXA | Hydrogen peroxide | NADPH oxidases | NOX | DUOX activator | DUOX | Cell surface targeting
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 01/2020, Volume 500, p. 110635
Thyroid hormone (TH) synthesis requires extracellular hydrogen peroxide generated by the NADPH oxidases, DUOX1 and DUOX2, with maturation factors, DUOXA1 and... 
DUOXA | Zebrafish | DUOX | Thyroid hormones | VAS2870 | Congenital hypothyroidism
Journal Article
Journal Article
Experimental Cell Research, ISSN 0014-4827, 11/2019, Volume 384, Issue 1, p. 111620
Thyroid hormone synthesis requires H O , produced by two NADPH oxidases, Duox1 and Duox2. To be fully active at the apical pole of the thyrocytes, these... 
DuoxA | Reactive oxygen species | Duox | N-glycosylation | DNA damage | Nox | NADPH OXIDASE | MUTAGENESIS | MATURATION | IDENTIFICATION | CELL BIOLOGY | P22(PHOX) | GENE | ONCOLOGY | ROLES | CONGENITAL HYPOTHYROIDISM | HYDROGEN-PEROXIDE | MUTATIONS
Journal Article
Thyroid, ISSN 1050-7256, 08/2018, Volume 28, Issue 8, pp. 1052 - 1062
Dual oxidases (DUOX1 and DUOX2) were initially identified as H O sources involved in thyroid hormone synthesis. Congenital hypothyroidism (CH) resulting from... 
DUOXA | disulfide bridge | oxidative folding | DUOX | Humans | Congenital Hypothyroidism - genetics | Congenital Hypothyroidism - metabolism | HEK293 Cells | Mutation | Dual Oxidases - genetics | Hydrogen Peroxide - metabolism
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 08/2016, Volume 97, pp. 95 - 108
Amyotrophic lateral sclerosis (ALS) is an incurable neurodegenerative disease characterized by progressive loss of motor neurons, gliosis, neuroinflammation... 
SOD1G93A mice | Thioridazine | NADPH oxidase | NOX | Amyotrophic lateral sclerosis | Perphenazine | Microglia | Phenothiazine | DUOX dual oxidase | List of abbreviations ALS amyotrophic lateral sclerosis | WST-1 2-(4-Iodophenyl)-3-(4-nitrophenyl)-5-(2,4-disulfophenyl)-2H-tetrazolium, monosodium salt | PBS phosphate buffered saline | HOCl hypochlorous acid | BBB blood brain barrier | WT wild typ | 2-chloroethidium | 2-Cl-E | NOXO1 NADPH oxidase organiser type 1 | CNS central nervous system | DPI diphenyleneiodonium | ethidium | NOX NADPH oxidase | HE hydroethidine | qPCR quantitative polymerase chain reaction | MPO myeloperoxidase | LC/MS/MS Liquid chromatography-tandem mass spectrometry | GM-CSF granulocyte-macrophage colony-stimulating factor | PMA phorbol myristate acetate | TDP 43 TAR DNA binding protein 43 | DMEM Dulbecco's modified Eagle Medium | hydrogen peroxide | DNA deoxyribonucleic acid | ROS reactive oxygen species | superoxide radical anion | SOD superoxide dismutase | HBSS Hank's buffered salt solution | half maximal efficient concentration | 2-hydroxyethidium | NADPH nicotinamide adenine dinucleotide phosphate | NC non-carrier | FBS fetal bovine serum | 2-OH-E | FUS/TLS Fused in Sarcoma/Translocated in Sarcoma | DMSO Dimethylsulfoxide | SURVIVAL | SUPEROXIDE | HYDROETHIDINE | SOD1(G93A) mice | BIOCHEMISTRY & MOLECULAR BIOLOGY | SPINAL-CORD | REACTIVE OXYGEN | MOTOR-NEURON DEGENERATION | NOX FAMILY | ENDOCRINOLOGY & METABOLISM | ALS MICE | INHIBITOR | Spinal Cord - drug effects | Amyotrophic Lateral Sclerosis - genetics | Humans | Middle Aged | Male | NADPH Oxidase 1 - genetics | Thioridazine - administration & dosage | Amyotrophic Lateral Sclerosis - drug therapy | Motor Neurons - pathology | Gene Expression Regulation - drug effects | Motor Neurons - metabolism | Amyotrophic Lateral Sclerosis - pathology | Animals | NADPH Oxidase 2 - metabolism | Spinal Cord - pathology | Superoxide Dismutase-1 - genetics | Adult | Female | Aged | Mice | Perphenazine - administration & dosage | NADPH Oxidase 2 - genetics | Superoxide Dismutase-1 - antagonists & inhibitors | Disease Models, Animal | Oxidases | Nervous system diseases | Analysis | Oncology, Experimental | Superoxide | Research | Cancer
Journal Article