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Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 8345 - 7
The assembly and deposition of amyloid beta protein (A beta) is a fundamental event during the early stages of Alzheimer's disease (AD) and cerebral amyloid... 
NERVOUS-SYSTEM | GM1 GANGLIOSIDE | DUTCH TYPE | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | INSECT CELLS | HEREDITARY CEREBRAL-HEMORRHAGE | MODEL | MELANOGASTER | EXPRESSION | PARKINSONS-DISEASE | Cerebral amyloid angiopathy | Molecular modelling | Insects | Transgenes | Gangliosides | Alzheimer's disease
Journal Article
Stroke, ISSN 0039-2499, 09/2018, Volume 49, Issue 9, pp. 2081 - 2087
BACKGROUND AND PURPOSE—Previous studies of symptomatic and asymptomatic hereditary cerebral amyloid angiopathy (CAA) patients offered the possibility to study... 
Alzheimer disease | HEMORRHAGE | MRI | ALZHEIMERS-DISEASE | TISSUE | CLINICAL NEUROLOGY | BETA | cerebral amyloid angiopathy | DUTCH TYPE | PERIPHERAL VASCULAR DISEASE | cerebral hemorrhage | neuropathology | point mutation | BRAIN IRON
Journal Article
Brain Pathology, ISSN 1015-6305, 07/2018, Volume 28, Issue 4, pp. 495 - 506
Hereditary cerebral hemorrhage with amyloidosis‐Dutch type (HCHWA‐D) is an early onset hereditary form of cerebral amyloid angiopathy (CAA) pathology, caused... 
TGFβ | amyloid β mutation E22Q | postmortem human brain tissue | cerebral amyloid angiopathy | phospho‐SMAD2/3 granules | hereditary cerebral hemorrhage with amyloidosis‐Dutch type | hereditary cerebral hemorrhage with amyloidosis-Dutch type | phospho-SMAD2/3 granules | phospho-SMAD2 | SMALL VESSEL DISEASE | PROTEIN | ALZHEIMERS-DISEASE | PATHOLOGY | NEUROSCIENCES | COLOCALIZES | CLINICAL NEUROLOGY | amyloid beta mutation E22Q | 3 granules | MICROVASCULAR DEGENERATION | TRANSFORMING GROWTH-FACTOR-BETA-1 | ANGIOPATHY | TGF beta | EXPRESSION | TRANSGENIC MICE | TGF-BETA-1 | Up-Regulation | Phosphorylation | Frontal Lobe - metabolism | Signal Transduction | Humans | Middle Aged | Smad2 Protein - metabolism | Cerebral Amyloid Angiopathy, Familial - metabolism | Cerebral Amyloid Angiopathy, Familial - pathology | Male | Smad3 Protein - metabolism | Occipital Lobe - pathology | Occipital Lobe - blood supply | Aged, 80 and over | Female | Aged | Frontal Lobe - blood supply | Transforming Growth Factor beta - metabolism | Frontal Lobe - pathology | Occipital Lobe - metabolism | Immunohistochemistry | Brain | Deregulation | Pathogenesis | Transforming growth factor-a | Transgenic mice | Blood vessels | Gene expression | Hemorrhage | Lobes | Pathology | Cerebral amyloid angiopathy | Autopsy | Smad2 protein | Fibrosis | Biomarkers | β-Amyloid | Amyloidosis | Microvasculature | Occipital lobes | Index Medicus
Journal Article
Journal Article
Journal Article
LWT - Food Science and Technology, ISSN 0023-6438, 09/2017, Volume 83, pp. 95 - 100
The main objective of this study was to evaluate the effect of adjunct culture LPC-37 on the physicochemical properties of ripening Dutch-type cheeses. Those... 
Adjunct culture | Lactobacillus paracasei LPC-37 | Proteolysis | Dutch-type cheese | PROBIOTIC CHEDDAR CHEESE | DEFINED-STRAIN STARTER | FOOD SCIENCE & TECHNOLOGY | SENSORY CHARACTERISTICS | TEMPERATURES | LACTIC-ACID BACTERIA | CASEI | ABILITIES | PLANTARUM | FLAVOR | Analysis
Journal Article
Molecular Psychiatry, ISSN 1359-4184, 07/2017, Volume 22, Issue 7, pp. 981 - 989
Recent data suggest that intraneuronal accumulation of metabolites of the amyloid-beta-precursor protein (APP) is neurotoxic. We observed that transgenic mice... 
FIBRIL FORMATION | CHOLINERGIC DYSFUNCTION | PSYCHIATRY | UNBIASED STEREOLOGICAL ESTIMATION | ALZHEIMERS-DISEASE | MEMORY DEFICITS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROSCIENCES | DUTCH TYPE | DOWN-SYNDROME | A-BETA | BASAL FOREBRAIN | HEREDITARY CEREBRAL-HEMORRHAGE | Peptide Fragments - metabolism | Humans | Lysosomal-Associated Membrane Protein 2 - genetics | Mice, Transgenic | Cerebral Cortex - metabolism | Brain - metabolism | Hippocampus - metabolism | Amyloid beta-Protein Precursor - genetics | Animals | Proteins - metabolism | Amyloid beta-Peptides - metabolism | Mice | Neurons - metabolism | Alzheimer Disease - genetics | alpha-Synuclein - metabolism | Lysosomal-Associated Membrane Protein 2 - metabolism | Disease Models, Animal | Brain | Research | Gene expression | Neurons | Alzheimer's disease | Amyloid beta-protein | Forebrain | Synuclein | Accumulation | Western blotting | Fragmentation | Proteins | Neurotoxicity | γ-Aminobutyric acid | Metabolites | Rodents | Cathepsin D | Deposition | Age | Neurodegenerative diseases | LAMP-2 protein | Transgenic mice | Fragments | Forebrain (basal) | Conversion | Amyloid precursor protein | Pathology | Gliosis | β-Amyloid | Mutation | Alzheimers disease | Hippocampus | Index Medicus | oligomeric amyloid β | neurodegeneration | lysosomes | carboxyl-terminal fragments of APP
Journal Article
Journal Article
Lancet Neurology, The, ISSN 1474-4422, 2016, Volume 16, Issue 2, pp. 98 - 99
  In The Lancet Neurology, Anna van Opstal and colleagues2 use data from 12 presymptomatic mutation carriers, 15 symptomatic HCHWA-D patients, and 33 healthy... 
Neurology | CEREBRAL AMYLOID ANGIOPATHY | DUTCH TYPE | HEMORRHAGE | ALZHEIMERS-DISEASE | CLINICAL NEUROLOGY | Congenital Abnormalities | Ear - abnormalities | Humans | Amyloid beta-Peptides | Cerebral Amyloid Angiopathy | Case-Control Studies | Cerebral Amyloid Angiopathy, Familial | Biomarkers | Hormone replacement therapy | Mutation | Alzheimers disease
Journal Article