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The Journal of experimental medicine, ISSN 1540-9538, 2011, Volume 208, Issue 10, pp. 2005 - 2016
Despite lack of tumor control in many models, spontaneous T cell priming occurs frequently in response to a growing tumor... 
MEDICINE, RESEARCH & EXPERIMENTAL | METASTATIC MELANOMA | INTERFERON-ALPHA | DYING CELLS | COLONY-STIMULATING FACTOR | TUMOR-ANTIGENS | REJECTION | PROSTATE-CANCER | INNATE IMMUNE-RESPONSE | RECEPTOR | IMMUNOLOGY | LYMPHOCYTES-T
Journal Article
Journal Article
Nature medicine, ISSN 1546-170X, 2006, Volume 13, Issue 1, pp. 54 - 61
Anthracyclin-treated tumor cells are particularly effective in eliciting an anticancer immune response, whereas other DNA-damaging agents such as etoposide and mitomycin C do not induce immunogenic cell death... 
MEDICINE, RESEARCH & EXPERIMENTAL | CLEARANCE | DENDRITIC CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR | CHAPERONE | CELL BIOLOGY | DYING CELLS | APOPTOTIC CELLS | SURFACE CALRETICULIN | ENDOPLASMIC-RETICULUM | BINDING | ANTIGEN | Apoptosis - drug effects | Colonic Neoplasms - drug therapy | Dendritic Cells - immunology | Electrophoresis, Gel, Two-Dimensional | Immunoblotting | Antineoplastic Agents - therapeutic use | Protein Transport - drug effects | Calreticulin - genetics | Colonic Neoplasms - metabolism | Cell Cycle Proteins - antagonists & inhibitors | RNA Interference | Anthracyclines - pharmacology | Neoplasms, Experimental - immunology | Calreticulin - metabolism | Antigens, Differentiation - metabolism | Antineoplastic Agents - pharmacology | Cell Membrane - metabolism | Protein Phosphatase 1 | Cell Membrane - drug effects | Mitomycin - therapeutic use | Cell Cycle Proteins - metabolism | Phagocytosis - immunology | Etoposide - pharmacology | Etoposide - therapeutic use | Recombinant Proteins - pharmacology | Mitomycin - pharmacology | Animals | Apoptosis - immunology | Mice, Nude | Colonic Neoplasms - pathology | Cell Line, Tumor | Cell Membrane - immunology | Mice | Mice, Inbred BALB C | Anthracyclines - therapeutic use | Calreticulin - immunology | Neoplasms, Experimental - metabolism | Neoplasms, Experimental - drug therapy | Chemotherapy | Immunology | DNA damage | Rodents | Cancer | Apoptosis | Tumors | Antigens, Differentiation | Cell Membrane | Anthracyclines | Dendritic Cells | Recombinant Proteins | Life Sciences | Calreticulin | Neoplasms, Experimental | Mitomycin | Etoposide | Antineoplastic Agents | Protein Transport | Phagocytosis | Cell Cycle Proteins | Colonic Neoplasms
Journal Article
Nature (London), ISSN 1476-4687, 2005, Volume 433, Issue 7028, pp. 887 - 892
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 02/2011, Volume 108, Issue 6, pp. 2384 - 2389
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 02/2014, Volume 95, Issue 2, pp. 325 - 336
NOD CD8+ DCs express increased CD40; targeting of autoantigen to these cells induces Th1 responses, not tolerance, unless CD40/CD40L interactions are blocked... 
type 1 diabetes | CD40 | peripheral tolerance | DEC‐205 | Peripheral tolerance | Type 1 diabetes | DEC-205 | RECEPTOR DEC-205 | IFN-GAMMA | IMMUNOLOGY | PEPTIDE | CELL BIOLOGY | PATHOGENESIS | DYING CELLS | NONOBESE DIABETIC MICE | UNRESPONSIVENESS | IN-VIVO | HEMATOLOGY | EXPRESSION | ANTIGEN | Dendritic Cells - immunology | T-Lymphocytes, Regulatory - immunology | CD4-Positive T-Lymphocytes - immunology | Lymphocyte Activation - immunology | CD40 Antigens - metabolism | CD40 Ligand - metabolism | Forkhead Transcription Factors - metabolism | Immune Tolerance - immunology | Dendritic Cells - drug effects | Toll-Like Receptors - metabolism | Immune Tolerance - drug effects | Th1 Cells - drug effects | Interferon-gamma - secretion | Peptides - immunology | Antibodies - pharmacology | Diabetes Mellitus, Experimental - immunology | Phenotype | T-Lymphocytes, Regulatory - drug effects | Animals | Lymphocyte Activation - drug effects | CD8-Positive T-Lymphocytes - drug effects | Diabetes Mellitus, Experimental - pathology | Antigens - immunology | Ligands | Mice, Inbred NOD | Cell Proliferation - drug effects | Mice | Th1 Cells - secretion | CD40 Ligand - antagonists & inhibitors | CD8-Positive T-Lymphocytes - immunology | CD4-Positive T-Lymphocytes - drug effects | Inflammation, Extracellular Mediators, & Effector Molecules
Journal Article