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Journal Article
Nature Neuroscience, ISSN 1097-6256, 07/2011, Volume 14, Issue 7, pp. 874 - 880
UNC119 is widely expressed among vertebrates and other phyla. We found that UNC119 recognized the acylated N terminus of the rod photoreceptor transducin alpha... 
PHOTORECEPTOR PROTEIN | RETINAL DEGENERATION | TRANSDUCIN | COMPLEX | LIGHT-DRIVEN TRANSLOCATION | CAENORHABDITIS-ELEGANS | PDE-DELTA | UNC-119 | C-ELEGANS | BINDING PROTEIN | NEUROSCIENCES | Adaptor Proteins, Signal Transducing - chemistry | Protein Binding - genetics | Models, Chemical | Humans | Dark Adaptation - genetics | Green Fluorescent Proteins - genetics | GTP-Binding Protein alpha Subunits, G12-G13 - metabolism | Transducin - deficiency | Time Factors | Cattle | Transducin - metabolism | Sensory Receptor Cells - metabolism | Glycine - genetics | Transducin - genetics | GTP-Binding Protein alpha Subunits - metabolism | Animals, Genetically Modified | Gene Expression Regulation - genetics | GTP-Binding Protein alpha Subunits, G12-G13 - genetics | Gene Expression Regulation - physiology | Models, Molecular | Signal Transduction - genetics | Mutation - genetics | Protein Transport - genetics | Mice, Knockout | Caenorhabditis elegans | Animals | GTP Phosphohydrolases - metabolism | Adaptor Proteins, Signal Transducing - genetics | GTP-Binding Protein alpha Subunits, Gi-Go | Protein Structure, Quaternary - genetics | GTP-Binding Protein alpha Subunits - deficiency | Mice | Adaptor Proteins, Signal Transducing - metabolism | Caenorhabditis elegans Proteins - genetics | Physiological aspects | Genetic aspects | G proteins | Research | Neurons | Genes | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 10/2012, Volume 76, Issue 2, pp. 396 - 409
Adhesive contact between pre- and postsynaptic neurons initiates synapse formation during brain development and provides a natural means of transsynaptic... 
RELEASE PROBABILITY | AMPA RECEPTORS | TISSUE-PLASMINOGEN ACTIVATOR | NMDA RECEPTORS | VISUAL-CORTEX | MATRIX-METALLOPROTEINASE | CELL-ADHESION MOLECULES | LONG-TERM POTENTIATION | NEUROSCIENCES | INHIBITORY SYNAPSE FORMATION | PLASTICITY | Electric Stimulation | Synaptic Transmission - genetics | Dark Adaptation - genetics | Status Epilepticus - chemically induced | Dendrites - ultrastructure | Green Fluorescent Proteins - genetics | Pilocarpine - toxicity | Matrix Metalloproteinase 9 - metabolism | Muscarinic Agonists - toxicity | Excitatory Amino Acid Agents - pharmacology | Synaptic Transmission - drug effects | Excitatory Postsynaptic Potentials - genetics | Organ Culture Techniques | Disease Models, Animal | Animals, Newborn | Glutamic Acid - pharmacology | Enzyme Inhibitors - pharmacology | Mice, Transgenic | Signal Transduction - genetics | Mutation - genetics | Cell Adhesion Molecules, Neuronal - genetics | Microscopy, Confocal | Patch-Clamp Techniques | Signal Transduction - drug effects | Luminescent Proteins - genetics | Mice | Synaptic Transmission - physiology | Cercopithecus aethiops | Neurons - cytology | Threonine - metabolism | Cerebral Cortex - cytology | Excitatory Postsynaptic Potentials - drug effects | Transfection | Potassium Chloride - pharmacology | Threonine - genetics | Neurons - physiology | Female | Cell Adhesion Molecules, Neuronal - metabolism | Neurons - drug effects | Plant Lectins - genetics | Dendrites - metabolism | Biotinylation | Vesicular Glutamate Transport Protein 1 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Electroporation | Photons | Hippocampus - cytology | Pregnancy | Animals | Neural Cell Adhesion Molecules - metabolism | Quaternary Ammonium Compounds - metabolism | Signal Transduction - physiology | Plant Lectins - metabolism | Pyridinium Compounds - metabolism | Luminescent Proteins - metabolism | Methyl aspartate | Neurons | Neurosciences | Proteins | Studies | Brain | Autism | Net losses | Kinases | Index Medicus | Adhesives
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 02/2012, Volume 90, Issue 2, pp. 331 - 339
Complete congenital stationary night blindness (cCSNB) is a clinically and genetically heterogeneous group of retinal disorders characterized by nonprogressive... 
COMPLETE FORM | LIGHT RESPONSE | CHANNEL | MGLUR6 | SYNAPTIC-TRANSMISSION | MOUSE MODEL | GENETICS & HEREDITY | NYCTALOPIN | RICH REPEAT PROTEIN | MUTATIONS | CONE ELECTRORETINOGRAM | Electroretinography - methods | Night Blindness - metabolism | Humans | Dark Adaptation - genetics | Retinal Rod Photoreceptor Cells - metabolism | Gene Knockdown Techniques - methods | Myopia - metabolism | Receptors, Metabotropic Glutamate - genetics | Retinal Bipolar Cells - physiology | Chromosome Mapping - methods | Genetic Diseases, X-Linked | Night Blindness - genetics | Retinal Bipolar Cells - metabolism | Eye Diseases, Hereditary | Signal Transduction | Mice, Inbred C57BL | Zebrafish | Mice, Inbred C3H | Myopia - genetics | Animals | Myopia - physiopathology | Pedigree | Night Blindness - physiopathology | Retinal Rod Photoreceptor Cells - physiology | Heterozygote | Mice | Receptors, G-Protein-Coupled - genetics | Mutation | Chromosome mapping | Usage | Gene mutations | Retinal bipolar cells | Causes of | Physiological aspects | Night blindness | Genetic aspects | Research | Analysis | Genetic disorders | Proteins | Rodents | Blindness | Gene expression | Cells | Index Medicus | Animal models | Antibodies | Insertion | stationary night blindness | Retina | Guanine nucleotide-binding protein | Electroretinograms | Depolarization | Signal transduction | Vision | Bipolar cells | Gene mapping | DNA sequencing | Report
Journal Article
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 2011, Volume 24, Issue 3, pp. 421 - 454
Physical exercise is considered to exert a positive neurophysiological effect that helps to maintain normal brain activity in the elderly. Expectations that it... 
cognition | electrophysiology | gender | voluntary exercise | amyloid-β | PHF-tau | 3xTg-AD mouse | behavior | Alzheimer's disease | age | oxidative stress | RABBIT FOLLOWING STIMULATION | CONTROLLED-TRIAL | TRIPLE-TRANSGENIC MODEL | LONG-LASTING POTENTIATION | AMYLOID PRECURSOR PROTEIN | SYNAPTIC PLASTICITY | NEUROSCIENCES | amyloid-beta | MOUSE MODEL | A-BETA | COGNITIVE DEFICITS | Alzheimer Disease - complications | Age Factors | Glutathione - metabolism | Humans | Dark Adaptation - genetics | Conditioning, Operant - physiology | Male | Reflex, Startle - genetics | Electroencephalography | Glutathione Disulfide - metabolism | Body Weight - genetics | Alzheimer Disease - pathology | Brain - metabolism | Exploratory Behavior - physiology | Cognition Disorders - prevention & control | tau Proteins - genetics | Cognition Disorders - etiology | Amyloid beta-Peptides - metabolism | Physical Therapy Modalities | Female | Excitatory Postsynaptic Potentials - genetics | Disease Models, Animal | Neural Inhibition - genetics | Peptide Fragments - metabolism | Enzyme-Linked Immunosorbent Assay | Maze Learning - physiology | Acoustic Stimulation | Presenilin-1 - genetics | Mice, Transgenic | Mutation - genetics | Amyloid beta-Protein Precursor - genetics | Animals | Alzheimer Disease - rehabilitation | Brain - pathology | Exercise - physiology | Mice | Alzheimer Disease - genetics | Index Medicus
Journal Article
Cell, ISSN 0092-8674, 2002, Volume 109, Issue 4, pp. 485 - 495
Electrical silencing of Drosophila circadian pacemaker neurons through targeted expression of K channels causes severe deficits in free-running circadian... 
NERVOUS-SYSTEM | BEHAVIORAL RHYTHMS | AMYOTROPHIC LATERAL SCLEROSIS | AXONAL SWELLINGS | SUPRACHIASMATIC NUCLEUS | BIOCHEMISTRY & MOLECULAR BIOLOGY | PERIOD | GENE-EXPRESSION | CALCIUM REGULATION | SYNAPTIC TRANSMISSION | K+ CHANNEL | CELL BIOLOGY | Action Potentials - genetics | Synaptic Transmission - genetics | Nervous System - metabolism | Dark Adaptation - genetics | Gene Expression Regulation, Developmental - genetics | Neurons - cytology | Synapses - genetics | Drosophila melanogaster - genetics | Drosophila melanogaster - metabolism | Cell Size - genetics | Synapses - metabolism | Neurons - metabolism | Potassium Channels - deficiency | Nervous System - embryology | Nuclear Proteins - genetics | Insect Proteins - metabolism | Drosophila Proteins | Nervous System - cytology | Circadian Rhythm - genetics | Drosophila melanogaster - cytology | Insect Proteins - genetics | Nuclear Proteins - metabolism | Potassium Channels, Inwardly Rectifying - genetics | Mutation - genetics | Period Circadian Proteins | Potassium Channels - genetics | Motor Activity - genetics | Animals | Biological Clocks - genetics | Genes, Lethal - genetics | Photic Stimulation | Potassium Channels, Inwardly Rectifying - metabolism | Circadian rhythms | Cell research | Neurons | Analysis | Drosophila | Physiological aspects | Genetic aspects | Research | Genetic transcription | Potassium channels | Gene expression | Index Medicus
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 06/2011, Volume 36, Issue 7, pp. 1478 - 1488
Journal Article
Neuropsychopharmacology, ISSN 0893-133X, 06/2011, Volume 36, Issue 7, pp. 1489 - 1504
The possible role of the CB2 receptor (CB(2)r) in psychiatric disorders has been considered. Several animal models use knockout (KO) mice that display... 
cannabinoid; CB | receptor; schizophrenia; risperidone; pre-pulse inhibition; gene expression | CB2 receptor | pre-pulse inhibition | NERVOUS-SYSTEM | PSYCHIATRY | cannabinoid | PREPULSE INHIBITION DEFICITS | risperidone | schizophrenia | NEUROSCIENCES | STARTLE REFLEX | MUTANT MICE | SWEDISH CONSCRIPTS | IN-VIVO | ANTIPSYCHOTIC-DRUGS | SUSCEPTIBILITY GENE | PHARMACOLOGY & PHARMACY | TRANSPORTER KNOCKOUT MICE | RAT-BRAIN | gene expression | Memory Disorders - genetics | Dark Adaptation - genetics | Male | Schizophrenia - complications | Depression - genetics | Hindlimb Suspension - physiology | Receptors, Catecholamine - metabolism | Acoustic Stimulation - methods | Depression - etiology | Exploratory Behavior - physiology | Schizophrenia - genetics | Risperidone - therapeutic use | Time Factors | Cognition Disorders - etiology | Sensory Gating - genetics | Receptors, Catecholamine - genetics | Disease Models, Animal | Inhibition (Psychology) | Maze Learning - physiology | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Risperidone - pharmacology | Cognition Disorders - genetics | Receptors, Catecholamine - classification | Avoidance Learning - physiology | Cognition Disorders - drug therapy | Mice, Knockout | Gene Expression Regulation - drug effects | Anxiety - genetics | Animals | Memory Disorders - etiology | Analysis of Variance | Avoidance Learning - drug effects | Mice | Antipsychotic Agents - pharmacology | Schizophrenia - drug therapy | Anxiety - etiology | Receptor, Cannabinoid, CB2 - deficiency | Index Medicus | animal models | behavioral science | antipsychotics | cannabinoids | Original
Journal Article