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The FEBS Journal, ISSN 1742-464X, 02/2013, Volume 280, Issue 3, pp. 840 - 854
We previously reported that nitric oxide (NO) reduces the rate of bacteremia and maternal mortality in pregnant rats with uterine infection by Escherichia coli... 
S‐nitrosylation | nitric oxide | Sp1 | decay accelerating factor | mRNA stability | S-nitrosylation | BIOCHEMISTRY & MOLECULAR BIOLOGY | NECROSIS-FACTOR-ALPHA | RICH ELEMENTS | TRANSLATION | INTESTINAL EPITHELIAL-CELLS | MESSENGER-RNA | GESTATIONAL PYELONEPHRITIS | INTRAUTERINE INFECTION | PROMOTER | EXPRESSION | FACTOR GENE | Immunoprecipitation | Fimbriae Proteins - metabolism | Humans | 3' Untranslated Regions - genetics | CD55 Antigens - genetics | Sp1 Transcription Factor - metabolism | RNA, Messenger - metabolism | Promoter Regions, Genetic - genetics | Cysteine - genetics | Nitroso Compounds - pharmacology | Nitroso Compounds - metabolism | Escherichia coli - metabolism | Cysteine - metabolism | Nitric Oxide Donors - pharmacology | Sp1 Transcription Factor - physiology | Nitric Oxide Donors - metabolism | RNA, Messenger - genetics | ELAV Proteins - metabolism | Binding Sites - genetics | Down-Regulation - drug effects | ELAV Proteins - physiology | Blotting, Western | Regulatory Sequences, Nucleic Acid - genetics | Down-Regulation - physiology | Fimbriae Proteins - genetics | Sp1 Transcription Factor - genetics | Escherichia coli - genetics | Cell Line, Tumor | Protein Binding | CD55 Antigens - metabolism | Mutation | Nitric Oxide - metabolism | ELAV Proteins - genetics | Messenger RNA | Promoters (Genetics) | Nitric oxide | Escherichia coli | Protein binding | Proteins | E coli | Ribonucleic acid--RNA | Binding sites
Journal Article
Journal Article
Molecular Immunology, ISSN 0161-5890, 08/2015, Volume 66, Issue 2, pp. 263 - 273
Atypical hemolytic uremic syndrome (aHUS) associates with complement dysregulation caused by mutations and polymorphisms in complement activators and... 
Atypical hemolytic uremic syndrome | Factor H | TED | C3 mutation | MCP | Complement C3 | ACTIVATION | FACTOR-B | MECHANISM | BIOCHEMISTRY & MOLECULAR BIOLOGY | IMMUNOLOGY | FACTOR-H MUTATIONS | CONVERTASE | POLYMORPHISMS | GENE | PREDISPOSITION | REVEALS | Haplotypes | Humans | Molecular Sequence Data | Male | Atypical Hemolytic Uremic Syndrome - immunology | Complement C3 - chemistry | Proteolysis | Base Sequence | Adult | Female | Fibrinogen - immunology | Atypical Hemolytic Uremic Syndrome - genetics | Binding Sites | Child | Complement C3 - genetics | Protein Structure, Tertiary | Severity of Illness Index | Complement Factor H - immunology | Gene Expression | Genetic Predisposition to Disease | Models, Molecular | Recombinant Proteins - genetics | Fibrinogen - genetics | Complement C3 - immunology | Atypical Hemolytic Uremic Syndrome - pathology | Recombinant Proteins - immunology | Adolescent | Protein Binding | Membrane Cofactor Protein - genetics | Mutation | Complement Factor H - genetics | Membrane Cofactor Protein - immunology | Proteins | Analysis | Genetic aspects | DDD, dense deposit disease | MAC, membrane attack complex | FB, factor B | EM, electron microscopy | KD, equilibrium dissociation constant | CR1, complement receptor 1 | FI, factor I | FD, factor D | mt, mutant | FH, factor H | aHUS, atypical hemolytic uremic syndrome | DAF, decay accelerating factor | wt, wild-type | LP, lectin pathway | CP, classical pathway | sMCP, soluble recombinant membrane cofactor protein (MCP, CD46) | AP, alternative pathway | MCP, membrane cofactor protein | SPR, surface plasmon resonance | SCR, short consensus repeat
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 04/2019, Volume 105, Issue 4, pp. 681 - 694
Complement factor and T‐cell signaling during an effective alloimmune response plays a key role in transplant‐associated injury, which leads to the progression... 
complement mediators | T‐cell activation | costimulatory signals | T-cell activation | ISCHEMIA/REPERFUSION INJURY | DENDRITIC CELLS | CARDIAC ALLOGRAFT VASCULOPATHY | IFN-GAMMA | REPERFUSION INJURY | IMMUNOLOGY | CELL BIOLOGY | C5A RECEPTOR | MEMBRANE ATTACK COMPLEX | IMMUNE-RESPONSES | MICROVASCULAR DESTRUCTION | DECAY-ACCELERATING FACTOR | HEMATOLOGY
Journal Article
Kidney International, ISSN 0085-2538, 11/2012, Volume 82, Issue 10, pp. 1084 - 1092
Journal Article
Journal Article
Kidney International, ISSN 0085-2538, 12/2015, Volume 88, Issue 6, pp. 1314 - 1322
Abnormal regulation of the complement alternative pathway is associated with C3 glomerulopathy. Complement factor H is the main plasma regulator of the... 
therapeutic | mini-FH | factor H | C3 glomerulopathy | Therapeutic | Factor H | Mini-FH | CELLS | ACTIVATION | HEMOLYTIC-UREMIC SYNDROME | PROTEIN | NEISSERIA-MENINGITIDIS | SURFACE-RECOGNITION | DENSE DEPOSIT DISEASE | ALTERNATIVE PATHWAY | UROLOGY & NEPHROLOGY | DECAY-ACCELERATING FACTOR | BINDING | Basic Research
Journal Article
Transplantation, ISSN 0041-1337, 07/2011, Volume 92, Issue 2, pp. 139 - 147
Background. Acute humoral xenograft rejection (AHXR) is an important barrier to xenograft survival. Human tumor necrosis factor-alpha (hTNF-alpha) is one of... 
shTNFRI-Fc | Acute humoral xenograft rejection | TNF-α | Xenotransplantation | Transgenic pig | MOLECULAR CHARACTERIZATION | RHEUMATOID-ARTHRITIS | SURGERY | TNF-alpha | GENE-KNOCKOUT PIGS | ALPHA-1,3-GALACTOSYLTRANSFERASE GENE | IMMUNOLOGY | TRANSPLANTATION | FACTOR THERAPY | XENOGRAFT SURVIVAL | PORCINE ENDOTHELIAL-CELLS | SKIN XENOGRAFTS | DECAY-ACCELERATING FACTOR | Endothelium, Vascular - cytology | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Cytokines - metabolism | Animals, Genetically Modified | Graft Rejection - prevention & control | Humans | E-Selectin - metabolism | Endothelium, Vascular - drug effects | Male | NF-kappa B - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Cell Adhesion Molecules - metabolism | Transplantation, Heterologous - methods | Tumor Necrosis Factor-alpha - pharmacology | Animals | Immunity, Humoral | Endothelium, Vascular - metabolism | Swine | HEK293 Cells | Swine, Miniature | Female | Models, Animal | Immunohistochemistry | Tumor necrosis factor receptors | Flow cytometry | Cytotoxicity | Inflammation | Gene expression | Endothelial cells | Western blotting | Cell adhesion molecules | Graft rejection | Polymerase chain reaction | somatic cell nuclear transfer | E-selectin | NF- Kappa B protein | Major histocompatibility complex | Green fluorescent protein | Xenografts | Fusion protein | Chemokines | Enzyme-linked immunosorbent assay | Tumor necrosis factor- alpha | Expression vectors
Journal Article
Annals of the Rheumatic Diseases, ISSN 0003-4967, 06/2018, Volume 77, Issue Suppl 2, p. 684
Journal Article