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Immunity, ISSN 1074-7613, 12/2017, Volume 47, Issue 6, pp. 1100 - 1113.e6
Natural killer (NK) cells are present in large populations at the maternal-fetal interface during early pregnancy. However, the role of NK cells in fetal... 
growth-promoting factors | fetal growth restriction | maternal-fetal interface | aged pregnancy | tissue-resident NK | decidual NK cells | fetal development | INTERFACE | RESIDENT NK CELLS | INFLAMMATION | SENESCENCE | INNATE LYMPHOID-CELLS | DIFFERENTIATION | IMMUNOLOGY | IMMUNE TOLERANCE | HLA-G | EXPRESSION | PREGNANCY | Abortion, Habitual - pathology | Antigens, CD - immunology | Leukocyte Immunoglobulin-like Receptor B1 - genetics | Humans | Abortion, Habitual - immunology | Basic-Leucine Zipper Transcription Factors - immunology | Fetal Growth Retardation - genetics | T-Box Domain Proteins - immunology | Adoptive Transfer | Antigens, CD - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Leukocyte Immunoglobulin-like Receptor B1 - immunology | Gene Expression Regulation, Developmental | Abortion, Habitual - genetics | Killer Cells, Natural - immunology | Adult | Female | Fetus | HLA-G Antigens - immunology | HLA-G Antigens - genetics | Cytokines - genetics | Carrier Proteins - immunology | Cytokines - immunology | Killer Cells, Natural - transplantation | Cytokines - metabolism | Decidua - immunology | Signal Transduction | Mice, Inbred C57BL | Intercellular Signaling Peptides and Proteins - genetics | Cellular Microenvironment | Fetal Growth Retardation - prevention & control | Decidua - pathology | Basic-Leucine Zipper Transcription Factors - genetics | T-Box Domain Proteins - genetics | Fetal Growth Retardation - pathology | Fetal Growth Retardation - immunology | Pregnancy | Carrier Proteins - genetics | Killer Cells, Natural - cytology | Animals | Carrier Proteins - metabolism | Fetal Development - immunology | Integrin alpha1 - genetics | Integrin alpha1 - immunology | Mice | Intercellular Signaling Peptides and Proteins - immunology | Killer cells | Histocompatibility antigens | Growth | HLA histocompatibility antigens | Osteoglycin | Secretion | Crosstalk | Fetuses | Pleiotrophin | Decidua | Adoptive transfer | Cells | Interfaces | Biomedical materials | Uterus | Biocompatibility | Histocompatibility antigen HLA | Natural killer cells
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2010, Volume 25, Issue 4, pp. 604 - 615
Abstract Activation of the maternal immune system in rodent models sets in motion a cascade of molecular pathways that ultimately result in autism- and... 
Psychiatry | Allergy and Immunology | JAK/STAT3 | Autism | Maternal immune activation | Poly(I:C) | Schizophrenia | Inflammation | Growth hormone | Insulin-like growth factor | Maternal infection | INCREASED EXPRESSION | OBSTETRIC COMPLICATIONS | PSYCHIATRY | BRAIN-DEVELOPMENT | LATENT INHIBITION | IMMUNOLOGY | PRENATAL INFECTION | NEUROSCIENCES | PHARMACOLOGICAL CHANGES | IGF-BINDING PROTEIN-3 | DOPAMINERGIC HYPERFUNCTION | GROWTH-FACTOR-I | Decidua - cytology | Endophenotypes | RNA, Messenger - analysis | Male | Pregnancy Proteins - metabolism | Maternal Exposure | Signal Transduction - immunology | Killer Cells, Natural - immunology | Female | Poly I-C - immunology | Interleukin-6 - metabolism | Macrophages - immunology | Virus Diseases - immunology | Decidua - immunology | Interleukin-6 - genetics | Placenta - immunology | Mice, Inbred C57BL | Behavior, Animal - physiology | Mice, Knockout | Placenta - metabolism | Pregnancy | Animals | Prenatal Exposure Delayed Effects - immunology | Analysis of Variance | Fetal Development - immunology | Interleukin-6 - immunology | RNA, Double-Stranded - immunology | Signal Transduction - physiology | Mice | Granulocytes - immunology | Somatotropin | Messenger RNA | Interleukins | Analysis | Immune system | maternal infection | inflammation | JAK | STAT3 | poly(I:C) | maternal immune activation | insulin-like growth factor | schizophrenia | growth hormone | autism
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Journal Article