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The New England Journal of Medicine, ISSN 0028-4793, 05/2009, Volume 360, Issue 22, pp. 2289 - 2301
Journal Article
International Journal of Cancer, ISSN 0020-7136, 06/2019, Volume 144, Issue 11, pp. 2887 - 2896
Gefitinib, erlotinib and afatinib are approved for first‐line treatment of advanced non‐small cell lung cancer (NSCLC) bearing an activating epidermal growth... 
EGFR mutation | EGFR TKI | T790M | NSCLC | uncommon EGFR mutation | SURVIVAL | BIBW 2992 | PROGNOSTIC-FACTORS | SOLID TUMORS | ADENOCARCINOMA | OPEN-LABEL | IRREVERSIBLE EGFR | ONCOLOGY | RESISTANCE | TYROSINE KINASE INHIBITOR | PHASE-I | Lung Neoplasms - drug therapy | Follow-Up Studies | Lung Neoplasms - mortality | Humans | Middle Aged | ErbB Receptors - genetics | Afatinib - pharmacology | Male | Liver Neoplasms - mortality | Carcinoma, Non-Small-Cell Lung - secondary | Time Factors | Female | Retrospective Studies | Liver Neoplasms - secondary | Afatinib - therapeutic use | Lung Neoplasms - genetics | Gefitinib - pharmacology | Liver Neoplasms - genetics | Carcinoma, Non-Small-Cell Lung - genetics | Liver Neoplasms - drug therapy | Gefitinib - therapeutic use | Carcinoma, Non-Small-Cell Lung - mortality | Disease Progression | Drug Resistance, Neoplasm - genetics | Protein Kinase Inhibitors - therapeutic use | Erlotinib Hydrochloride - pharmacology | Progression-Free Survival | Taiwan - epidemiology | Sex Factors | Aged | Erlotinib Hydrochloride - therapeutic use | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Drug Resistance, Neoplasm - drug effects | Tyrosine | Yuan (China) | Care and treatment | Patient outcomes | Liver | Lung cancer, Small cell | Metastasis | Lung cancer, Non-small cell | Antineoplastic agents | Antimitotic agents | Liver cancer | Epidermal growth factor | Genetic aspects | Epidermal growth factor receptors | Lung cancer | Non-small cell lung carcinoma | Gene deletion | Multivariate analysis | Kinases | Patients | Survival | Clinical outcomes | Metastases | Confidence intervals | Clonal deletion | Deletion | Gefitinib | Health risk assessment | Growth factors | Protein-tyrosine kinase | Cancer
Journal Article
Journal Article
Journal Article
Journal Article
Oncogene, ISSN 0950-9232, 09/2013, Volume 32, Issue 37, pp. 4417 - 4426
Journal Article
Journal Article
The Journal of Physiology, ISSN 0022-3751, 01/2019, Volume 597, Issue 2, pp. 543 - 560
Key points Electrophysiological characterization of Q1412X‐CFTR, a C‐terminal truncation mutation of cystic fibrosis transmembrane conductance regulator (CFTR)... 
Anion channel | ABC transporter | cystic fibrosis | PHYSIOLOGY | IVACAFTOR | PHOSPHORYLATION | STABILITY | ATP BINDING | CL-CHANNEL | NUCLEOTIDE-BINDING DOMAIN | NEUROSCIENCES | TRANSMEMBRANE CONDUCTANCE REGULATOR | IN-VITRO | DISEASE | CHLORIDE CHANNEL | Channel opening | Phosphorylation | Clonal deletion | Nonsense mutation | Channel gating | Cystic fibrosis | Stop codon | Mutation | Gene deletion | Gating | Binding sites
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 03/2014, Volume 370, Issue 10, pp. 943 - 949
Journal Article