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1. Plasticity and pathology
: on the formation of the neural subject
by Bates, David William editor and Bassiri, Nima, editor and Doreen B. Townsend Center for the Humanities
2016
Neuroplasticity | Brain | physiopathology | Pathophysiology | Neuronal Plasticity | Synaptic Transmission
Conference Proceeding
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2. Physiology and pathology of dendrites
: editor, Georg W. Kreutzberg
by Kreutzberg, Georg W
1975, Advances in neurology, Volume 12, xi, 511 p. ill.
Congresses | Dendrites | Diseases
Book
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3. Full Text Spreading of neurodegenerative pathology via neuron-to-neuron transmission of β-amyloid
by Nath, Sangeeta and Agholme, Lotta and Kurudenkandy, Firoz Roshan and Granseth, Björn and Marcusson, Jan and Hallbeck, Martin and Närsjukvården i centrala Östergötland and Linköpings universitet and Institutionen för klinisk och experimentell medicin and Diagnostikcentrum and Klinisk patologi och klinisk genetik and Geriatrik and Geriatriska kliniken and Östergötlands Läns Landsting and Experimentell patologi and Hälsouniversitetet and Cellbiologi
Journal of Neuroscience, ISSN 0270-6474, 06/2012, Volume 32, Issue 26, pp. 8767 - 8777
Alzheimer's disease (AD) is the major cause of dementia. During the development of AD, neurofibrillary tangles progress in a fixed pattern, starting in the... 
PRION PROTEIN | CELLS | TRANSPORT | ALZHEIMERS-DISEASE | MECHANISMS | PRECURSOR PROTEIN | NEUROSCIENCES | BRAIN | OLIGOMERS | PEPTIDE | PARKINSONS-DISEASE | Membrane Glycoproteins - metabolism | Coculture Techniques | Humans | Cell Communication - physiology | Male | Green Fluorescent Proteins - genetics | rab5 GTP-Binding Proteins - metabolism | Neurons - ultrastructure | Time Factors | Amyloid beta-Peptides - metabolism | Synaptic Transmission - drug effects | Neurons - metabolism | Animals, Newborn | Microinjections | Endocytosis - drug effects | Rats | Rats, Sprague-Dawley | Nerve Degeneration - pathology | Patch-Clamp Techniques | Cell Communication - drug effects | Lysosomal-Associated Membrane Protein 2 - metabolism | Heterocyclic Compounds, 3-Ring - metabolism | Synaptic Transmission - physiology | Peptide Fragments - toxicity | Exocytosis - physiology | Exocytosis - drug effects | Nerve Degeneration - chemically induced | Neuroglia - drug effects | Dose-Response Relationship, Drug | Tetrazolium Salts | Transfection | Thiazoles | Female | Neurons - drug effects | Neuroblastoma - pathology | Dendrites - metabolism | Green Fluorescent Proteins - metabolism | Microscopy, Electron, Transmission | Peptide Fragments - metabolism | Amyloid beta-Peptides - toxicity | Cells, Cultured | Hippocampus - cytology | Endocytosis - physiology | Nerve Tissue Proteins - metabolism | Animals | Cell Differentiation - drug effects | Heterocyclic Compounds, 3-Ring - administration & dosage | Neocortex - cytology | Neuroglia - metabolism | Index Medicus | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
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4. Full Text Sequential distribution of pTDP-43 pathology in behavioral variant frontotemporal dementia (bvFTD)
by Brettschneider, Johannes and Del Tredici, Kelly and Irwin, David J and Grossman, Murray and Robinson, John L and Toledo, Jon B and Fang, Lubin and Van Deerlin, Vivianna M and Ludolph, Albert C and Lee, Virginia M.-Y and Braak, Heiko and Trojanowski, John Q
Acta Neuropathologica, ISSN 0001-6322, 3/2014, Volume 127, Issue 3, pp. 423 - 439
We examined regional distribution patterns of phosphorylated 43-kDa TAR DNA-binding protein (pTDP-43) intraneuronal inclusions in frontotemporal lobar... 
Pathology | Neurosciences | Medicine & Public Health | Neurodegeneration | TDP-43 | FTD | Frontotemporal lobar degeneration | FTLD, frontotemporal dementia | Proteinopathies | ALS, amyotrophic lateral sclerosis | FRONTAL-LOBE | LOBAR DEGENERATION | ALZHEIMERS-DISEASE | RHESUS-MONKEY | AMYOTROPHIC-LATERAL-SCLEROSIS | PATHOLOGY | NEUROSCIENCES | PREFRONTAL CORTEX | CLINICAL NEUROLOGY | CLINICOPATHOLOGICAL CORRELATIONS | MULTIPLE SYSTEM ATROPHY | ORBITOFRONTAL CORTEX | PARKINSONS-DISEASE | Immunohistochemistry | Dendrites - pathology | Frontotemporal Lobar Degeneration - pathology | Neurons - pathology | Genetic Testing | Spinal Cord - metabolism | Humans | Middle Aged | Male | Brain - metabolism | DNA-Binding Proteins - metabolism | Frontotemporal Dementia - metabolism | DNA Repeat Expansion | Frontotemporal Lobar Degeneration - metabolism | Spinal Cord - pathology | Female | C9orf72 Protein | Neurons - metabolism | Inclusion Bodies - metabolism | Frontotemporal Dementia - pathology | Frontotemporal Dementia - genetics | Severity of Illness Index | Dendrites - metabolism | Proteins - genetics | Brain - pathology | Inclusion Bodies - pathology | Aged | Frontotemporal Lobar Degeneration - genetics | Amyotrophic lateral sclerosis | Low density lipoproteins | DNA | Dementia | Protein binding | Index Medicus | Amygdala
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5. Full Text Glucocorticoids Increase Amyloid-beta and Tau Pathology in a Mouse Model of Alzheimer's Disease
by Green, Kim N and Billings, Lauren M and Roozendaal, Benno and McGaugh, James L and LaFerla, Frank M
Journal of Neuroscience, ISSN 0270-6474, 08/2006, Volume 26, Issue 35, pp. 9047 - 9056
Various environmental and genetic factors influence the onset and progression of Alzheimer's disease (AD). Dysregulation of the hypothalamic-pituitary-adrenal... 
Tau | Aβ peptide | Glucocorticoids | Corticosterone | Alzheimer's disease | Stress | stress | tau | TRANSGENIC MODEL | NEUROSCIENCES | PEPTIDE | IN-VIVO | A-BETA | A beta peptide | glucocorticoids | PRECURSOR PROTEIN | RAT-BRAIN | corticosterone | PITUITARY-ADRENAL AXIS | COGNITIVE DEFICITS | CORTISOL | Glucocorticoids - administration & dosage | tau Proteins - metabolism | Male | Alzheimer Disease - pathology | Brain - metabolism | Dose-Response Relationship, Drug | Dexamethasone - pharmacology | Time Factors | Glucocorticoids - metabolism | Aspartic Acid Endopeptidases | Stress, Physiological - metabolism | Amyloid Precursor Protein Secretases | Amyloid beta-Peptides - metabolism | Amyloid beta-Protein Precursor - metabolism | Neurons - metabolism | Corticosterone - blood | Transgenes | Disease Models, Animal | Cell Line | Dendrites - metabolism | Endopeptidases - metabolism | Amyloid beta-Peptides - biosynthesis | Solubility | Mice, Transgenic | Amyloid beta-Protein Precursor - genetics | Animals | Alzheimer Disease - metabolism | Brain - pathology | Glucocorticoids - pharmacology | Mice | Amyloid beta-Peptides - chemistry | Alzheimer Disease - genetics | Index Medicus
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6. Full Text Preservation of Neuronal Number Despite Age-Related Cortical Brain Atrophy in Elderly Subjects Without Alzheimer Disease
by Freeman, Stefanie H and Kandel, Ruth and Cruz, Luis and Rozkalne, Anete and Newell, Kathy and Frosch, Matthew P and Hedley-Whyte, E. Tessa and Locascio, Joseph J and Lipsitz, Lewis A and Hyman, Bradley T
Journal of Neuropathology and Experimental Neurology, ISSN 0022-3069, 12/2008, Volume 67, Issue 12, pp. 1205 - 1212
Cerebral volume loss has long been associated with normal aging, but whether this is due to aging itself or to age-related diseases, including incipient... 
Aging | Cerebral cortex | Stereology | Neurons | HUMAN NEOCORTEX | MRI | RHESUS-MONKEY | LAYER 1 | PATHOLOGY | NEUROSCIENCES | CLINICAL NEUROLOGY | DECLINE | HUMAN CEREBRAL-CORTEX | IN-VIVO | GRAY-MATTER | Dendrites - pathology | Memory Disorders - physiopathology | Neurons - pathology | Cell Count | Humans | Middle Aged | Atrophy - pathology | Cerebral Cortex - pathology | Nerve Degeneration - physiopathology | Male | Neural Pathways - pathology | Alzheimer Disease - pathology | Cerebral Cortex - physiopathology | Cognition Disorders - etiology | Aged, 80 and over | Female | Neural Pathways - physiopathology | Memory Disorders - pathology | Cognition Disorders - physiopathology | Atrophy - physiopathology | Cognition Disorders - pathology | Atrophy - etiology | Disease Progression | Nerve Degeneration - pathology | Aging - pathology | Neuropsychological Tests | Memory Disorders - etiology | Aged | Nerve Degeneration - etiology | Index Medicus
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7. Full Text Dendritic Degeneration, Neurovascular Defects, and Inflammation Precede Neuronal Loss in a Mouse Model for Tau-Mediated Neurodegeneration
by Jaworski, Tomasz and Lechat, Benoit and Demedts, David and Gielis, Lies and Devijver, Herman and Borghgraef, Peter and Duimel, Hans and Verheyen, Fons and Kügler, Sebastian and Van Leuven, Fred
American Journal of Pathology, The, ISSN 0002-9440, 2011, Volume 179, Issue 4, pp. 2001 - 2015
Adeno-associated virus (AAV)–mediated expression of wild-type or mutant P301L protein tau produces massive degeneration of pyramidal neurons without protein... 
Pathology | WILD-TYPE | ENDOGENOUS TAU | PHOSPHORYLATION | ALZHEIMERS-DISEASE | AMYLOID-BETA | CELL-CYCLE REENTRY | PATHOLOGY | FRACTALKINE-RECEPTOR | BLOOD-BRAIN-BARRIER | NEUROPIL THREADS | TRANSGENIC MICE | Dendrites - pathology | Dependovirus - genetics | Inflammation - pathology | Oxidative Stress | Blood Vessels - pathology | Humans | Blood Vessels - ultrastructure | tau Proteins - metabolism | Male | Dendrites - ultrastructure | Nervous System - blood supply | Synapses - pathology | CA1 Region, Hippocampal - pathology | Inflammation - complications | Nervous System - pathology | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Axons - ultrastructure | Female | CA1 Region, Hippocampal - ultrastructure | Disease Models, Animal | Biomarkers - metabolism | Synapses - ultrastructure | Permeability | Nerve Degeneration - complications | Nerve Degeneration - pathology | Blood-Brain Barrier - pathology | Animals | Axons - pathology | Mice | Index Medicus | Abridged Index Medicus | Regular
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8. Full Text A human neurodevelopmental model for Williams syndrome
by Chailangkarn, Thanathom and Trujillo, Cleber A and Freitas, Beatriz C and Hrvoj-Mihic, Branka and Herai, Roberto H and Yu, Diana X and Brown, Timothy T and Marchetto, Maria C and Bardy, Cedric and McHenry, Lauren and Stefanacci, Lisa and Järvinen, Anna and Searcy, Yvonne M and Dewitt, Michelle and Wong, Wenny and Lai, Philip and Ard, M. Colin and Hanson, Kari L and Romero, Sarah and Jacobs, Bob and Dale, Anders M and Dai, Li and Korenberg, Julie R and Gage, Fred H and Bellugi, Ursula and Halgren, Eric and Semendeferi, Katerina and Muotri, Alysson R
Nature, ISSN 0028-0836, 08/2016, Volume 536, Issue 7616, pp. 338 - 343
Williams syndrome is a genetic neurodevelopmental disorder characterized by an uncommon hypersociability and a mosaic of retained and compromised linguistic... 
PLURIPOTENT STEM-CELLS | PROSPECTIVE MOTION CORRECTION | SURFACE-BASED ANALYSIS | HUMAN-FETUS | MRI SCANS | HUMAN CEREBRAL-CORTEX | GENE | MAGNETIC-RESONANCE IMAGES | MULTIDISCIPLINARY SCIENCES | NEURONS | GOLGI | Dendrites - pathology | Neurons - pathology | Williams Syndrome - genetics | Calcium - metabolism | Humans | Cerebral Cortex - pathology | Male | Synapses - pathology | Cellular Reprogramming | Young Adult | Cell Shape | Adult | Female | Cell Differentiation | Frizzled Receptors - deficiency | Induced Pluripotent Stem Cells - pathology | Haploinsufficiency - genetics | Reproducibility of Results | Neural Stem Cells - pathology | Phenotype | Frizzled Receptors - genetics | Adolescent | Brain - pathology | Models, Neurological | Chromosomes, Human, Pair 7 - genetics | Williams Syndrome - pathology | Apoptosis | Research | Diagnosis | Health aspects | Williams syndrome | Genetic disorders | Cellular biology | Neurons | Morphology | Stem cells | Gene expression | Index Medicus
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9. Full Text Axonal and myelinic pathology in 5xFAD Alzheimer's mouse spinal cord
by Chu, TH and Cummins, K and Sparling, JS and Tsutsui, S and Brideau, C and Nilsson, KPR and Joseph, JT and Stys, PK and Kemi and Institutionen för fysik, kemi och biologi and Linköpings universitet and Tekniska fakulteten
PLOS ONE, ISSN 1932-6203, 11/2017, Volume 12, Issue 11, pp. e0188218 - e0188218
As an extension of the brain, the spinal cord has unique properties which could allow us to gain a better understanding of CNS pathology. The brain and cord... 
DENDRITES | PROTEIN | PRECURSOR | WHITE-MATTER | MULTIDISCIPLINARY SCIENCES | TAU | DISEASE MICE | NEUROPIL THREADS | BRAIN | TRANSGENIC MICE | DEGENERATION | Myelin Sheath - pathology | Plaque, Amyloid - pathology | Neuroglia - pathology | Humans | Mice, Transgenic | Motor Neurons - pathology | Alzheimer Disease - pathology | White Matter - pathology | Animals | Spinal Cord - pathology | Axons - pathology | Amyloid beta-Protein Precursor - metabolism | Gray Matter - pathology | Aging | Axons | Neurons | Physiological aspects | Development and progression | Genetic aspects | Research | Alzheimer's disease | Index Medicus | Basic Medicine | Neurosciences | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
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10. Full Text Chronic Stress Causes Frontostriatal Reorganization and Affects Decision-Making
by Eduardo Dias-Ferreira and João C. Sousa and Irene Melo and Pedro Morgado and Ana R. Mesquita and João J. Cerqueira and Rui M. Costa and Nuno Sousa
Science, ISSN 0036-8075, 7/2009, Volume 325, Issue 5940, pp. 621 - 625
The ability to shift between different behavioral strategies is necessary for appropriate decision-making. Here, we show that chronic stress biases... 
Brain | Neurons | Habitual behavior | Reports | Dendrites | Animal training | Behavioral neuroscience | Density | Hypertrophy | Combined stress | HABIT FORMATION | CONTINGENCY | BASAL GANGLIA | MULTIDISCIPLINARY SCIENCES | HIPPOCAMPAL | MEDIAL PREFRONTAL CORTEX | DORSAL STRIATUM | SENSITIVITY | MORPHOLOGY | ADDICTION | NUCLEUS-ACCUMBENS | Decision Making | Dendrites - pathology | Neurons - pathology | Choice Behavior | Rats, Wistar | Cell Count | Rats, Long-Evans | Reinforcement (Psychology) | Rats | Neural Pathways - pathology | Stress, Psychological - psychology | Prefrontal Cortex - pathology | Atrophy | Stress, Psychological - pathology | Animals | Chronic Disease | Corpus Striatum - pathology | Frontal Lobe - pathology | Habits | Decision-making | Chronic diseases | Complications and side effects | Corpus striatum | Stress (Psychology) | Health aspects | Brain research | Cognition & reasoning | Decision making | Stress | Index Medicus
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11. Full Text Dendritic pathology in Alzheimer's disease
by Baloyannis, S.J
Journal of the Neurological Sciences, ISSN 0022-510X, 2009, Volume 283, Issue 1, pp. 153 - 157
Abstract Dendritic pathology and decrease of dendritic spine density are prominent phenomena in early cases of Alzheimer's disease, which correlate... 
Neurology | Dendrites | Golgi technique | Electron microscopy | Alzheimer's disease | APOPTOSIS | CELLS | DEMENTIA | ACOUSTIC CORTEX | GOLGI | NEUROSCIENCES | CLINICAL NEUROLOGY | SYNAPSE LOSS | SPINES | MORPHOLOGY | Dendrites - pathology | Neurons - pathology | Visual Cortex - cytology | Humans | Middle Aged | Auditory Cortex - pathology | Male | Neurons - cytology | Dendrites - ultrastructure | Mitochondria - pathology | Microscopy, Electron | Mitochondria - ultrastructure | Alzheimer Disease - pathology | Visual Cortex - pathology | Neurons - ultrastructure | Auditory Cortex - ultrastructure | Dendritic Spines - pathology | Aged, 80 and over | Auditory Cortex - cytology | Female | Visual Cortex - ultrastructure | Aged | Dendritic Spines - ultrastructure | Snow | Mitochondrial DNA | Index Medicus
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12. Full Text Multiple events lead to dendritic spine loss in triple transgenic Alzheimer's disease mice
by Bittner, Tobias and Fuhrmann, Martin and Burgold, Steffen and Ochs, Simon M and Hoffmann, Nadine and Mitteregger, Gerda and Kretzschmar, Hans and Laferla, Frank M and Herms, Jochen
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 11, pp. e15477 - e15477
The pathology of Alzheimer's disease (AD) is characterized by the accumulation of amyloid-beta (A beta) peptide, hyperphosphorylated tau protein, neuronal... 
LONG-TERM | NEUROFIBRILLARY TANGLES | NEOCORTEX IN-VIVO | MOUSE MODEL | BIOLOGY | A-BETA | AMYLOID-PRECURSOR-PROTEIN | SYNAPSE LOSS | COGNITIVE IMPAIRMENT | ADULT CORTEX | SENILE PLAQUES | Immunohistochemistry | Brain Diseases - metabolism | Neurons - pathology | Frontal Lobe - metabolism | Brain Diseases - genetics | Presenilin-1 - physiology | tau Proteins - metabolism | Male | Dendritic Spines - metabolism | Alzheimer Disease - pathology | Somatosensory Cortex - pathology | tau Proteins - genetics | Time Factors | tau Proteins - physiology | Amyloid beta-Protein Precursor - metabolism | Female | Neurons - metabolism | Amyloid beta-Protein Precursor - physiology | Plaque, Amyloid - pathology | Presenilin-1 - genetics | Mice, Transgenic | Hippocampus - pathology | Plaque, Amyloid - genetics | Hippocampus - metabolism | Microscopy, Confocal | Amyloid beta-Protein Precursor - genetics | Animals | Brain Diseases - pathology | Dendritic Spines - pathology | Presenilin-1 - metabolism | Alzheimer Disease - metabolism | Plaque, Amyloid - metabolism | Dendritic Spines - genetics | Mice | Mutation | Alzheimer Disease - genetics | Frontal Lobe - pathology | Somatosensory Cortex - metabolism | Amino Acid Substitution | Advertising executives | Alzheimer's disease | Genetic engineering | Neurosciences | Neuropathology | Spine | Pathogenesis | Confocal | Proteins | Dendritic spines | Transgenic animals | Rodents | Dendrites | Protocol | Age | Plaques | Neurodegenerative diseases | Neurons | Cloning | Transgenic mice | Pathology | Brain research | Tau protein | β-Amyloid | Intracellular | Alzheimers disease | Animal cognition | Index Medicus
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13. Full Text Early fear memory defects are associated with altered synaptic plasticity and molecular architecture in the TgCRND8 Alzheimer's disease mouse model
by Steele, John W and Brautigam, Hannah and Short, Jennifer A and Sowa, Allison and Shi, Mengxi and Yadav, Aniruddha and Weaver, Christina M and Westaway, David and Fraser, Paul E and St George‐Hyslop, Peter H and Gandy, Sam and Hof, Patrick R and Dickstein, Dara L
Journal of Comparative Neurology, ISSN 0021-9967, 07/2014, Volume 522, Issue 10, pp. 2319 - 2335
Alzheimer's disease (AD) is a complex and slowly progressing dementing disorder that results in neuronal and synaptic loss, deposition in brain of aberrantly... 
mouse model of dementia | spine pathology | amyloid beta | neuronal morphology | dendritic pathology | Spine pathology | Mouse model of dementia | Neuronal morphology | Amyloid beta | Dendritic pathology | MICROSCOPY IMAGES | DENDRITIC SPINE MORPHOLOGY | AMYLOID PRECURSOR PROTEIN | ISOTROPIC FRACTIONATOR | NEUROSCIENCES | BETA OLIGOMERS | PYRAMIDAL NEURONS | ZOOLOGY | VISUAL-CORTEX | MEDIAL PREFRONTAL CORTEX | A-BETA | TRANSGENIC MICE | Dendrites - pathology | Memory Disorders - physiopathology | Neurons - pathology | Cell Count | Humans | Male | Dendritic Spines - metabolism | Alzheimer Disease - pathology | Brain - metabolism | Neuronal Plasticity - physiology | Amyloid beta-Protein Precursor - metabolism | Neurons - metabolism | Memory Disorders - pathology | Disease Models, Animal | Cues | Dendrites - metabolism | Alzheimer Disease - physiopathology | Amyloidosis - pathology | Plaque, Amyloid - pathology | Mice, Transgenic | Hippocampus - pathology | Nerve Tissue Proteins - metabolism | Fear | Hippocampus - metabolism | Amyloid beta-Protein Precursor - genetics | Animals | Dendritic Spines - pathology | Brain - pathology | Plaque, Amyloid - metabolism | Amyloidosis - metabolism | Alzheimer's disease | Analysis | Index Medicus
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