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Circulation Research, ISSN 0009-7330, 10/2004, Volume 95, Issue 7, pp. 726 - 733
Altered gap junction coupling of cardiac myocytes during ischemia may contribute to development of lethal arrhythmias. The phosphoprotein connexin 43 (Cx43) is... 
Ischemia | Gap junctions | Glycolysis | Okadaic acid | Protein phosphatases | Connexin43 | Protein phosphorylation | AMP kinase | gap junctions | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | connexin43 | PHOSPHORYLATION | GAP JUNCTIONAL COMMUNICATION | glycolysis | CARDIAC MYOCYTES | ACTIN GENE | protein phosphorylation | okadaic acid | ischemia | IN-VITRO | INTERCELLULAR COMMUNICATION | PERIPHERAL VASCULAR DISEASE | JUN KINASE | protein phosphatases | RAT VENTRICULAR MYOCYTES | HEMATOLOGY | ADRENERGIC REGULATION | Okadaic Acid - pharmacology | Tetradecanoylphorbol Acetate - pharmacology | Maleimides - pharmacology | Recombinant Fusion Proteins - physiology | Myocardial Contraction - drug effects | Brefeldin A - pharmacology | Phenanthridines - pharmacology | Antimycin A - pharmacology | Aminoimidazole Carboxamide - pharmacology | Cell Hypoxia | Ribonucleotides - pharmacology | Alkaloids | Protein Processing, Post-Translational - drug effects | Adenosine Triphosphate - metabolism | Deoxyglucose - pharmacology | Tacrolimus - pharmacology | Indoles - pharmacology | Flavonoids - pharmacology | Potassium Cyanide - pharmacology | JNK Mitogen-Activated Protein Kinases - genetics | Phosphorylation - drug effects | Ouabain - pharmacology | Cells, Cultured - drug effects | Connexin 43 - metabolism | Rats | Imidazoles - pharmacology | Cycloheximide - pharmacology | Pyrroles - pharmacology | Animals | Myocytes, Cardiac - drug effects | Aminoimidazole Carboxamide - analogs & derivatives | Benzophenanthridines | JNK Mitogen-Activated Protein Kinases - physiology | Myocytes, Cardiac - metabolism | Cells, Cultured - metabolism | Pyridines - pharmacology | Carbazoles - pharmacology | Staurosporine - pharmacology | Index Medicus
Journal Article
Journal Article
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 08/2002, Volume 174, Issue 2, pp. 233 - 246
Glucose-dependent insulinotropic polypeptide (GIP) acts as a glucose-dependent growth factor for beta-cells. Here we show that GIP and glucose also act... 
PATHWAYS | IGF-I | RECEPTOR TYROSINE KINASES | ACTIVATED PROTEIN-KINASE | PHOSPHATIDYLINOSITOL 3-KINASE | MAP KINASE | ENDOCRINOLOGY & METABOLISM | ELEMENT-BINDING PROTEIN | INS-1 CELLS | GROWTH-FACTOR | FACTOR-I | Apoptosis - drug effects | Calcium - metabolism | Humans | Maleimides - pharmacology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | MAP Kinase Kinase 1 | Isoquinolines - pharmacology | Deoxyglucose - pharmacology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Indoles - pharmacology | Flavonoids - pharmacology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Gastric Inhibitory Polypeptide - pharmacology | B-Lymphocytes - metabolism | Cell Line | Enzyme Inhibitors - pharmacology | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Arginase - genetics | Protein Kinase C - antagonists & inhibitors | Chelating Agents - pharmacology | Calcium-Calmodulin-Dependent Protein Kinases - antagonists & inhibitors | Sulfonamides - pharmacology | Egtazic Acid - pharmacology | MAP Kinase Signaling System - drug effects | Mitosis - drug effects | Androstadienes - pharmacology | Signal Transduction - drug effects | Glucokinase - antagonists & inhibitors | Plasmids | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Genistein - pharmacology | Glucose - metabolism | Alloxan - pharmacology | Benzylamines - pharmacology | Protein-Tyrosine Kinases - antagonists & inhibitors | Index Medicus
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 19, pp. 7338 - 7343
Chromosomal loci jiggle in place between segregation events in prokaryotic cells and during interphase in eukaryotic nuclei. This motion seems random and is... 
Viscosity | Azides | Enzymes | Yeasts | Sodium | Cell walls | DNA | Genetic loci | Prokaryotic cells | Cytoplasm | Nonthermal fluctuations | Intracellular transport | Active diffusion | Macromolecular motion | COMPLEX FLUIDS | PEPTIDOGLYCAN BIOSYNTHESIS | active diffusion | MULTIDISCIPLINARY SCIENCES | ESCHERICHIA-COLI | macromolecular motion | VISCOELASTIC MODULI | SACCHAROMYCES-CEREVISIAE | LOW-TEMPERATURE | intracellular transport | nonthermal fluctuations | MYOSIN I | DIFFUSION | CELL | RNA-POLYMERASE | Temperature | Chromosomes, Bacterial - genetics | Nucleic Acid Synthesis Inhibitors - pharmacology | Uncoupling Agents - pharmacology | Saccharomyces cerevisiae - genetics | Escherichia coli - drug effects | Saccharomyces cerevisiae - drug effects | Microscopy, Video | Genetic Loci | Time-Lapse Imaging | Saccharomyces cerevisiae - metabolism | Chromosomes, Bacterial - metabolism | Chromosomes, Fungal - metabolism | Adenosine Triphosphate - metabolism | Deoxyglucose - pharmacology | Escherichia coli - metabolism | Diffusion | Antimetabolites - pharmacology | Motion | Enzyme Inhibitors - pharmacology | Algorithms | Chromosomes, Fungal - genetics | Escherichia coli - genetics | 2,4-Dinitrophenol - pharmacology | Kinetics | Rifampin - pharmacology | Sodium Azide - pharmacology | Physiological aspects | Genetic aspects | Research | Chromosomes | Cells | Adenosine triphosphate | Gene loci | Metabolism | Brownian movements | Adenosine triphosphatase | Index Medicus | Biological Sciences
Journal Article
Oncogene, ISSN 0950-9232, 03/2010, Volume 29, Issue 11, pp. 1641 - 1652
Most cancer cells exhibit increased glycolysis for generation of their energy supply. This specificity could be used to preferentially kill these cells. In... 
Glycolysis | Mcl-1 | AMPK | Protein translation | MTOR | Apoptosis | SURVIVAL | protein translation | TRAIL | BIOCHEMISTRY & MOLECULAR BIOLOGY | glycolysis | apoptosis | AKT | CANCER-THERAPY | CELL BIOLOGY | POSITRON-EMISSION-TOMOGRAPHY | NECROSIS | ONCOLOGY | GLUCOSE-METABOLISM | GENETICS & HEREDITY | mTOR | EXPRESSION | MODULATION | AMP-Activated Protein Kinases - metabolism | Receptors, Death Domain - immunology | Apoptosis - drug effects | Humans | Intracellular Signaling Peptides and Proteins - metabolism | fas Receptor - metabolism | Glycolysis - drug effects | Aminoimidazole Carboxamide - pharmacology | Receptors, Death Domain - metabolism | Ribonucleotides - pharmacology | Glycolysis - physiology | Proto-Oncogene Proteins c-bcl-2 - metabolism | fas Receptor - immunology | TNF-Related Apoptosis-Inducing Ligand - pharmacology | RNA Interference | U937 Cells | Deoxyglucose - pharmacology | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Antibodies - immunology | Protein-Serine-Threonine Kinases - metabolism | Pyrazoles - pharmacology | Jurkat Cells | AMP-Activated Protein Kinases - antagonists & inhibitors | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Glucose - pharmacology | Pyrimidines - pharmacology | Recombinant Proteins - pharmacology | Enzyme Activation - drug effects | Reverse Transcriptase Polymerase Chain Reaction | TNF-Related Apoptosis-Inducing Ligand - metabolism | Sirolimus - pharmacology | Blotting, Western | Antibodies - pharmacology | Aminoimidazole Carboxamide - analogs & derivatives | Models, Biological | Myeloid Cell Leukemia Sequence 1 Protein | Protein Biosynthesis - drug effects | TOR Serine-Threonine Kinases | Apoptosis - physiology | HeLa Cells | Proto-Oncogene Proteins c-bcl-2 - genetics | TNF-Related Apoptosis-Inducing Ligand - genetics | Enzyme inhibitors | Physiological aspects | Genetic aspects | Research | Drug therapy | Health aspects | Protein kinases | Cancer | Oncology | Genetics | Glucose | Cellular biology | Index Medicus
Journal Article
Molecular Human Reproduction, ISSN 1360-9947, 02/2015, Volume 21, Issue 10, pp. 803 - 815
In this study, we aimed to investigate modulation of glucose uptake by the HTR-8/SVneo human first-trimester extravillous trophoblast cell line by a series of... 
Glucose uptake | Proliferation | Xanthohumol | Viability | Trophoblast cells | GLUT1 | proliferation | trophoblast cells | 2-DEOXY-D-GLUCOSE | DEVELOPMENTAL BIOLOGY | viability | OBSTETRICS & GYNECOLOGY | BREAST-CANCER CELLS | HEXOSE TRANSPORTER | QUERCETIN | REPRODUCTIVE BIOLOGY | INHIBITION | glucose uptake | METABOLISM | FETAL | xanthohumol | BINDING | CATECHIN | Deoxyglucose - metabolism | Humans | Propiophenones - pharmacology | Polyphenols - pharmacology | Dexamethasone - toxicity | Stilbenes - pharmacology | Pregnancy Trimester, First | Protein-Tyrosine Kinases - physiology | Trophoblasts - drug effects | Stilbenes - toxicity | Dexamethasone - pharmacology | Phloretin - pharmacology | Female | Polyphenols - toxicity | TOR Serine-Threonine Kinases - physiology | Biological Transport - drug effects | Flavonoids - pharmacology | Propiophenones - toxicity | Trophoblasts - cytology | Phlorhizin - pharmacology | Cytochalasin B - pharmacology | Placentation - drug effects | Phloretin - toxicity | Cytochalasin B - toxicity | Glucose - pharmacology | Cell Division - drug effects | Pregnancy | Cell Movement - drug effects | Flavonoids - toxicity | Melatonin - toxicity | Signal Transduction - drug effects | Glucose Transport Proteins, Facilitative - physiology | Street Drugs - toxicity | Melatonin - pharmacology | Street Drugs - pharmacology | Cell Line, Transformed | Glucose Transport Proteins, Facilitative - antagonists & inhibitors | Phlorhizin - toxicity | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, pp. e8996 - e8996
Long Term Potentiation (LTP) is a leading candidate mechanism for learning and memory and is also thought to play a role in the progression of seizures to... 
MAMMALIAN TARGET | ACTIVATED PROTEIN-KINASE | TUBEROUS SCLEROSIS GENE | LATE-PHASE | SIGNALING PATHWAY | BIOLOGY | GLUCOSE-CONCENTRATION | LONG-TERM POTENTIATION | RAT HIPPOCAMPAL SLICES | RAPAMYCIN PATHWAY | SYNAPTIC PLASTICITY | AMP-Activated Protein Kinases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Aminoimidazole Carboxamide - pharmacology | Ribonucleotides - pharmacology | Neuronal Plasticity - physiology | Long-Term Potentiation - drug effects | Deoxyglucose - pharmacology | Long-Term Potentiation - physiology | Hippocampus - enzymology | Energy Metabolism - physiology | Protein-Serine-Threonine Kinases - metabolism | Antimetabolites - pharmacology | Pyrazoles - pharmacology | AMP-Activated Protein Kinases - antagonists & inhibitors | Metformin - pharmacology | Pyrimidines - pharmacology | Enzyme Activation - drug effects | Blotting, Western | Hypoglycemic Agents - pharmacology | Vidarabine - pharmacology | Animals | Aminoimidazole Carboxamide - analogs & derivatives | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Mice, Inbred BALB C | TOR Serine-Threonine Kinases | Hippocampus - physiology | In Vitro Techniques | Energy Metabolism - drug effects | Microscopy, Fluorescence | Neurons | Epilepsy | Physiological aspects | Hypoglycemic agents | Glucose | Genetic translation | Protein kinases | Dextrose | Potentiation | Brain | Energy metabolism | Energy use | Brain slice preparation | Memory | Neuromodulation | Proteins | Consortia | Learning | Signal transduction | Mitochondria | Inhibition | Sensors | Chromosomes | Animal care | Young adults | AMP | Long-term potentiation | Rapamycin | Metabolism | Mammals | Energy balance | Neurology | Inhibitors | Protein synthesis | Metformin | Aberration | Hippocampus | TOR protein | Huntingtons disease | Transcription | Homeostasis | Kinases | AMP-activated protein kinase | Modulators | Plasticity (neural) | Energy | Rodents | Seizures | Kindling | Diabetes mellitus | Ribonucleic acid--RNA | Signaling | Brain-derived neurotrophic factor | Psychopharmacology | Glycolysis | Index Medicus | RNA | Ribonucleic acid
Journal Article
Journal Article
Journal Article