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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2014, Volume 111, Issue 36, pp. 13193 - 13198
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 09/2017, Volume 491, Issue 2, pp. 329 - 336
Hyperglycemia plays a crucial role in the pathogenesis of diabetic complications; however, the mechanisms underlying diabetic cardiac fibrosis remain unclear.... 
MicroRNA | EndMT | Notch2 | Fibrosis | High glucose | TARGET | CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | MICRORNAS | DIABETIC CARDIOMYOPATHY | BIOPHYSICS | ALAGILLE-SYNDROME | EXPRESSION | Dependovirus - genetics | Cadherins - metabolism | Genetic Vectors - administration & dosage | Humans | Aortic Valve - drug effects | Diabetes Mellitus, Experimental - genetics | Male | MicroRNAs - metabolism | Diabetes Mellitus, Experimental - chemically induced | Cadherins - genetics | Endomyocardial Fibrosis - prevention & control | Dependovirus - metabolism | Genetic Vectors - chemistry | Signal Transduction | Endothelial Cells - metabolism | Genetic Vectors - metabolism | Glucose - pharmacology | Heart Ventricles - metabolism | MicroRNAs - genetics | Calcium-Binding Proteins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Streptozocin | Vimentin - metabolism | Actins - metabolism | Receptor, Notch2 - genetics | Actins - genetics | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Vimentin - genetics | Epithelial-Mesenchymal Transition | Diabetic Cardiomyopathies - prevention & control | Endomyocardial Fibrosis - genetics | Endomyocardial Fibrosis - chemically induced | Endomyocardial Fibrosis - pathology | Diabetes Mellitus, Experimental - metabolism | Heart Ventricles - pathology | Calcium-Binding Proteins - metabolism | Cell Line | Mice, Inbred C57BL | Gene Expression Regulation | Receptor, Notch2 - metabolism | Aortic Valve - cytology | Aortic Valve - metabolism | Fibronectins - metabolism | Diabetic Cardiomyopathies - genetics | Animals | Endothelial Cells - cytology | Diabetes Mellitus, Experimental - pathology | Diabetic Cardiomyopathies - pathology | Fibronectins - genetics | Diabetic Cardiomyopathies - chemically induced | Endothelial Cells - drug effects | Fibronectins | Hyperglycemia | Stem cells | Glucose | Muscle proteins | Dextrose | Endothelium | Index Medicus
Journal Article
Molecular Therapy, ISSN 1525-0016, 05/2017, Volume 25, Issue 5, pp. 1168 - 1186
CRISPR-associated protein 9 (Cas9)-mediated genome editing provides a promising cure for HIV-1/AIDS; however, gene delivery efficiency in vivo remains an... 
genome editing | humanized BLT mice | animal models | viral latency | HIV | AAV | Tg26 mice | gene therapy | Cas9 | gRNA | EFFICIENT DELIVERY | MEDICINE, RESEARCH & EXPERIMENTAL | GENE EDITING TECHNOLOGIES | PACKAGING CAPACITY | LENTIVIRAL VECTORS | ADENOASSOCIATED VIRAL VECTORS | STAPHYLOCOCCUS-AUREUS CAS9 | CRISPR-CAS9 NUCLEASES | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | CRISPR/CAS9 SYSTEM | GENETICS & HEREDITY | MODULATES PROLIFERATION | NEURAL PRECURSOR CELLS | Oligonucleotides - genetics | Dependovirus - genetics | Endonucleases - genetics | Staphylococcus aureus - enzymology | RNA, Guide - genetics | RNA, Guide - metabolism | Humans | HIV Long Terminal Repeat | pol Gene Products, Human Immunodeficiency Virus - metabolism | Endonucleases - metabolism | gag Gene Products, Human Immunodeficiency Virus - metabolism | HIV Infections - pathology | Proviruses - genetics | Base Sequence | Clustered Regularly Interspaced Short Palindromic Repeats | gag Gene Products, Human Immunodeficiency Virus - genetics | pol Gene Products, Human Immunodeficiency Virus - genetics | Disease Models, Animal | Genome, Viral | HIV-1 - metabolism | Dependovirus - metabolism | Genetic Vectors - chemistry | HIV Infections - virology | Bacterial Proteins - genetics | Genetic Vectors - metabolism | Mice, Transgenic | HIV-1 - genetics | Proviruses - metabolism | Staphylococcus aureus - chemistry | Oligonucleotides - metabolism | Animals | CRISPR-Cas Systems | HIV Infections - therapy | Bacterial Proteins - metabolism | Gene Editing - methods | Mice | Genetic Therapy - methods | Neuroimaging | Animal models | Bioluminescence | Intravenous administration | Genes | Liver | Clinical trials | Chronic infection | Infections | Genomes | Thymus | Acquired immune deficiency syndrome--AIDS | Efficiency | Transgenic animals | Human immunodeficiency virus--HIV | Bone marrow | Colon | Deoxyribonucleic acid--DNA | Spleen | CRISPR | Gene transfer | Advisors | Transgenic mice | Ribonucleic acid--RNA | Progenitor cells | Polymerase chain reaction | Studies | Genotyping | Neural stem cells | Original
Journal Article
Journal Article
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 09/2015, Volume 35, Issue 35, pp. 12137 - 12151
Journal Article
Molecular Therapy, ISSN 1525-0016, 11/2017, Volume 25, Issue 11, pp. 2513 - 2525
A causal role of hypercholesterolemia in non-ischemic heart failure has never been demonstrated. Adeno-associated viral serotype 8 (AAV8) (AAV8 ) gene transfer... 
metabolic remodeling | heart failure | gene transfer | low-density lipoprotein receptor | cholesterol-lowering therapy | adeno-associated viral vectors | gene therapy | cardiac hypertrophy | non-ischemic cardiomyopathy | hypercholesterolemia | MAMMALIAN TARGET | MEDICINE, RESEARCH & EXPERIMENTAL | PRESSURE-OVERLOAD | OXIDATIVE STRESS | LIPID-METABOLISM | FATTY-ACID-METABOLISM | PROLIFERATOR-ACTIVATED RECEPTOR | CELL-GROWTH | COENZYME Q | HYPERTROPHIED RAT-HEART | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | CARDIAC-HYPERTROPHY | GENETICS & HEREDITY | Constriction, Pathologic - metabolism | Dependovirus - genetics | Constriction, Pathologic - pathology | Genetic Vectors - administration & dosage | TOR Serine-Threonine Kinases - metabolism | Cardiomegaly - etiology | Cardiomyopathies - etiology | TOR Serine-Threonine Kinases - genetics | Cardiomegaly - mortality | Cardiomyopathies - mortality | Myocardium - metabolism | Receptors, LDL - deficiency | Female | Heart Function Tests | Biomarkers - metabolism | Receptors, LDL - genetics | Gene Expression | Dependovirus - metabolism | Genetic Vectors - chemistry | Genetic Vectors - metabolism | Myocardium - pathology | Cholesterol - metabolism | Constriction, Pathologic - complications | Cardiomyopathies - therapy | Mice, Knockout | Acetyl-CoA C-Acetyltransferase - genetics | Animals | Aorta - surgery | Cardiomyopathies - metabolism | Survival Analysis | Mice | Hemodynamics | Acetyl-CoA C-Acetyltransferase - metabolism | Cardiomegaly - metabolism | Cardiomegaly - therapy | Genetic Therapy - methods | TOR protein | Oxidative stress | Cardiomyopathy | Cardiovascular disease | Ischemia | Conflicts of interest | Aorta | Coenzyme A | Lipoprotein (low density) receptors | Heart diseases | Heart failure | Mortality | Cardiomyocytes | Rapamycin | LDLR gene | Metabolism | Pressure | Cholesterol | Lipoproteins | Hypercholesterolemia | Magnetic resonance imaging | Coronary vessels | Cardiac function | Fibrosis | Receptor density | Software | Gene therapy | Hypertrophy | Apoptosis | Original
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 12, p. e15870
Journal Article
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 10/2013, Volume 203, Issue 2, pp. 345 - 357
Although the canonical transforming growth factor. signaling pathway represses skeletal muscle growth and promotes muscle wasting, a role in muscle for the... 
DROSOPHILA NEUROMUSCULAR-JUNCTION | SYNAPTIC GROWTH | SIGNALING PATHWAY | ATROPHY | MYOSTATIN | TGF-BETA SUPERFAMILY | ACTIVIN RECEPTOR | DIFFERENTIATION | FOXO TRANSCRIPTION FACTORS | UBIQUITIN LIGASES | CELL BIOLOGY | Genetic Therapy | Phosphorylation | TOR Serine-Threonine Kinases - metabolism | Bone Morphogenetic Protein 7 - genetics | Humans | Follistatin - metabolism | Muscle, Skeletal - innervation | Muscle, Skeletal - metabolism | Muscle Development | Transfection | HEK293 Cells | Female | Muscular Atrophy - prevention & control | Disease Models, Animal | Transduction, Genetic | Muscle, Skeletal - growth & development | Muscular Atrophy - metabolism | Signal Transduction | Mice, Inbred C57BL | Muscular Atrophy - pathology | Ubiquitin-Protein Ligases - metabolism | Rats | Histone Deacetylases - metabolism | Muscular Atrophy - genetics | Bone Morphogenetic Protein Receptors, Type I - metabolism | Dependovirus | Rats, Sprague-Dawley | Animals | Bone Morphogenetic Protein 7 - metabolism | Mice | Muscle, Skeletal - pathology | Genetic Vectors | Smad Proteins - metabolism | Myogenin - metabolism | Hypertrophy | Physiological aspects | Muscles | Bone morphogenetic proteins | Transforming growth factors | Health aspects | Basic Medicine | Physiology | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Fysiologi
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2015, Volume 112, Issue 29, pp. 9046 - 9051
Journal Article