X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (7174) 7174
Publication (935) 935
Newsletter (340) 340
Book Chapter (21) 21
Conference Proceeding (12) 12
Book Review (8) 8
Magazine Article (4) 4
Dissertation (3) 3
Book / eBook (2) 2
Data Set (2) 2
Government Document (2) 2
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
animals (4906) 4906
mice (3398) 3398
index medicus (2963) 2963
dextran (2683) 2683
humans (2427) 2427
immunology (1797) 1797
male (1742) 1742
female (1569) 1569
dextran sulfate (1487) 1487
colitis (1400) 1400
mice, inbred c57bl (1220) 1220
inflammation (1172) 1172
colitis - chemically induced (1156) 1156
dextrans (1130) 1130
inflammatory bowel disease (1107) 1107
disease models, animal (1064) 1064
inflammatory-bowel-disease (929) 929
rats (826) 826
rodents (816) 816
sulfates (810) 810
ulcerative-colitis (806) 806
mice, inbred balb c (775) 775
research article (774) 774
analysis (752) 752
colon (739) 739
sodium (713) 713
colitis - pathology (685) 685
research (648) 648
intestine (647) 647
colitis - immunology (646) 646
dextrans - pharmacology (638) 638
expression (635) 635
medicine (624) 624
cytokines (621) 621
multidisciplinary sciences (617) 617
proteins (605) 605
dextrans - immunology (580) 580
cell biology (574) 574
mice, knockout (572) 572
inflammatory bowel diseases (565) 565
crohns-disease (552) 552
cells, cultured (551) 551
ulcerative colitis (535) 535
gastroenterology & hepatology (526) 526
macrophages (525) 525
colon - pathology (520) 520
science (506) 506
dextran sulfate - toxicity (476) 476
article (467) 467
colitis - metabolism (466) 466
gene expression (464) 464
cells (463) 463
dextran sulfate sodium (462) 462
activation (448) 448
gastrointestinal diseases (448) 448
pharmacology & pharmacy (427) 427
cancer (419) 419
biochemistry & molecular biology (418) 418
permeability (391) 391
cell line (388) 388
dendritic cells (381) 381
colitis - drug therapy (373) 373
animal models (368) 368
time factors (366) 366
apoptosis (361) 361
cytokines - metabolism (347) 347
intestinal mucosa - pathology (346) 346
health aspects (333) 333
molecular weight (328) 328
colon - metabolism (327) 327
pathogenesis (327) 327
antigens (318) 318
oncology (316) 316
antibodies (312) 312
bowel-disease (312) 312
signal transduction (311) 311
adult (301) 301
intestinal mucosa - metabolism (300) 300
intestinal inflammation (299) 299
macrophages - immunology (299) 299
dextrans - metabolism (295) 295
biology (293) 293
colon - immunology (293) 293
sulfate (290) 290
sodium sulfate (284) 284
medicine, research & experimental (282) 282
t-cells (282) 282
microbiology (280) 280
intestinal mucosa - immunology (278) 278
b-lymphocytes - immunology (274) 274
abridged index medicus (271) 271
dextran sulfate - pharmacology (268) 268
physiological aspects (267) 267
t-lymphocytes - immunology (266) 266
colitis - genetics (265) 265
immunohistochemistry (262) 262
flow cytometry (260) 260
lymphocytes (258) 258
middle aged (256) 256
medical research (255) 255
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (7350) 7350
German (60) 60
Russian (40) 40
French (25) 25
Chinese (22) 22
Italian (9) 9
Japanese (9) 9
Spanish (5) 5
Polish (4) 4
Ukrainian (2) 2
Dutch (1) 1
Norwegian (1) 1
Portuguese (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 744 - 755
The microbiota stimulates inflammation, but the signaling pathways and the members of the microbiota involved remain poorly understood. We found that the... 
DEXTRAN SULFATE | IMMUNE-SYSTEM | COLITIS | MACROPHAGES | MICE | INNATE LYMPHOID-CELLS | GUT MICROBIOTA | GERM-FREE | IMMUNOLOGY | PROTEUS-MIRABILIS | BOWEL-DISEASE | Receptors, CCR2 - genetics | Inflammation - pathology | Hemolysin Proteins - immunology | NLR Family, Pyrin Domain-Containing 3 Protein | Proteus Infections - genetics | Receptors, CCR2 - immunology | Hemolysin Proteins - genetics | Salmonella Infections - genetics | Antigens, Ly - genetics | Salmonella Infections - immunology | Symbiosis - immunology | Monocytes - immunology | Interleukin-1beta - genetics | Intestines - immunology | Proteus Infections - pathology | Monocytes - pathology | Carrier Proteins - immunology | Macrophages - immunology | Macrophages - microbiology | Proteus mirabilis - immunology | Monocytes - microbiology | Salmonella Infections - pathology | Inflammation - microbiology | Microbiota - immunology | Macrophages - pathology | Proteus Infections - microbiology | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Intestines - injuries | Interleukin-1beta - immunology | Inflammation - immunology | Proteus Infections - immunology | Inflammasomes - genetics | Mice, Knockout | Carrier Proteins - genetics | Salmonella - immunology | Animals | Intestines - microbiology | Antigens, Ly - immunology | Inflammasomes - immunology | Inflammation - genetics | Salmonella Infections - microbiology | Mice
Journal Article
Immunity, ISSN 1074-7613, 10/2014, Volume 41, Issue 4, pp. 567 - 578
Programmed necrosis or necroptosis is an inflammatory form of cell death that critically requires the receptor-interacting protein kinase 3 (RIPK3). Here we... 
PROTECTS MICE | APOPTOSIS | ACTIVATION | NECROSIS | INFLAMMATION | IN-VIVO | KINASE | INDUCED COLITIS | RECEPTOR | IMMUNOLOGY | INTESTINAL EPITHELIAL-CELLS | Dextran Sulfate | Receptors, Interleukin - biosynthesis | Colitis - genetics | Dendritic Cells - immunology | Transcription Factor RelB - immunology | Caspase 1 - metabolism | Adoptive Transfer | NF-kappa B p50 Subunit - genetics | Enzyme Activation - immunology | Lipopolysaccharides | Necrosis - immunology | NF-kappa B p50 Subunit - immunology | RNA, Messenger - biosynthesis | Signal Transduction - immunology | Bone Marrow Cells - immunology | Interleukins - immunology | Female | Interleukin-1beta - biosynthesis | Interleukins - biosynthesis | Wound Healing - genetics | Colitis - immunology | Macrophages - immunology | Transcription Factor RelB - genetics | Gene Expression Regulation - immunology | Mice, Inbred C57BL | Interleukin-1beta - immunology | Inflammation - immunology | Interleukin-23 - immunology | Mice, Knockout | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Apoptosis - immunology | Interleukin-23 - biosynthesis | Mice | Enzyme Activation - genetics | Dendritic Cells - transplantation | Dextran | Cytokines | Dendritic cells | Sulfates | Inflammatory bowel disease | Rodents | Life sciences | Histology | Kinases | Experiments | Apoptosis
Journal Article
Immunity, ISSN 1074-7613, 09/2012, Volume 37, Issue 3, pp. 563 - 573
Mice lacking junctional adhesion molecule A (JAM-A, encoded by ) exhibit enhanced intestinal epithelial permeability, bacterial translocation, and elevated... 
GROWTH-FACTOR-BETA | INFLAMMATORY-BOWEL-DISEASE | IMMUNOGLOBULIN-A | IN-VIVO | REGULATORY T-CELLS | JUNCTIONAL-ADHESION-MOLECULE | DEFICIENT MICE | E-CADHERIN | IMMUNOLOGY | ORAL ANTIGEN | EXPRESSION | Dextran Sulfate | Cell Adhesion Molecules - genetics | Intestinal Mucosa - metabolism | Colitis - genetics | Immunoglobulin A - genetics | Male | Homeodomain Proteins - immunology | Intestines - metabolism | Cell Adhesion Molecules - immunology | Intestines - immunology | Adaptive Immunity - genetics | CD4-Positive T-Lymphocytes - immunology | Epithelium - immunology | Flow Cytometry | Intestinal Mucosa - immunology | Colitis - chemically induced | Female | Adaptive Immunity - immunology | Colitis - immunology | Transforming Growth Factor beta - immunology | Gene Expression | Bacterial Translocation - genetics | Epithelium - metabolism | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Bacterial Translocation - immunology | Intestinal Mucosa - microbiology | Permeability | Reverse Transcriptase Polymerase Chain Reaction | Receptors, Cell Surface - immunology | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Intestines - microbiology | Transforming Growth Factor beta - genetics | Immunoglobulin A - immunology | Mice | Transforming Growth Factor beta - metabolism | Receptors, Cell Surface - genetics | Disease susceptibility | Colitis | T cells | Transforming growth factors | Immunoglobulin A | Inflammatory bowel disease | Studies | Medical research | Nutrition research | Disease | Antibiotics | Lymphocytes | Rodents | Small intestine | Immune system | Cell adhesion & migration | Medical and Health Sciences | Homeodomain Proteins/genetics/immunology | Medicin och hälsovetenskap | Cell Surface/genetics/immunology | Colitis/chemically induced/genetics/immunology | CD4-Positive T-Lymphocytes/immunology/metabolism | Bacterial Translocation/genetics/immunology | Receptors | Immunoglobulin A/genetics/immunology | Transforming Growth Factor beta/genetics/immunology/metabolism | Intestinal Mucosa/immunology/metabolism/microbiology | Adaptive Immunity/genetics/immunology | Basic Medicine | Intestines/immunology/metabolism/microbiology | Cell and Molecular Biology | Inbred C57BL | Knockout | Cell Adhesion Molecules/genetics/immunology | Epithelium/immunology/metabolism | Medicinska och farmaceutiska grundvetenskaper | Cell- och molekylärbiologi
Journal Article
The Journal of experimental medicine, ISSN 0022-1007, 2013, Volume 210, Issue 6, pp. 1117 - 1124
Journal Article
Nature, ISSN 0028-0836, 12/2011, Volume 480, Issue 7377, pp. 336 - 343
Variable regions 1 and 2 (V1/V2) of human immunodeficiency virus-1 (HIV-1) gp120 envelope glycoprotein are critical for viral evasion of antibody... 
SYSTEM | POTENT NEUTRALIZATION | DEXTRAN SULFATE | EPITOPE | MULTIDISCIPLINARY SCIENCES | IMMUNODEFICIENCY-VIRUS TYPE-1 | ENVELOPE GLYCOPROTEIN | RECEPTOR | BINDING | T-CELLS | SOFTWARE | AIDS Vaccines - immunology | Glycopeptides - chemistry | Molecular Sequence Data | Antibody Affinity - immunology | Crystallography, X-Ray | Epitopes - immunology | HIV Envelope Protein gp120 - immunology | Antibodies, Neutralizing - immunology | Glycopeptides - immunology | Antibody Specificity - immunology | HIV Antibodies - immunology | HIV-1 - chemistry | Protein Structure, Quaternary | Conserved Sequence | Polysaccharides - chemistry | HIV Envelope Protein gp120 - chemistry | Protein Structure, Tertiary | Amino Acid Sequence | Binding Sites, Antibody - immunology | Models, Molecular | Glycosylation | Polysaccharides - immunology | HIV Antibodies - chemistry | AIDS Vaccines - chemistry | Amino Acid Motifs | HIV-1 - immunology | Immune Evasion | Hydrogen Bonding | Antigen-Antibody Complex - chemistry | Antibodies, Neutralizing - chemistry | Epitopes - chemistry | Antigen-Antibody Complex - immunology | Viral envelopes | Genetic aspects | Research | HIV (Viruses) | Structure | Binding sites (Biochemistry) | Proteins | Crystals | Vaccines | ANTIBODIES | AIDS VIRUS | BASIC BIOLOGICAL SCIENCES | AFFINITY | BIOLOGY | GLYCOPROTEINS | VULNERABILITY | ANTIGENS | IMMUNOLOGY | 60 APPLIED LIFE SCIENCES
Journal Article
Immunity, ISSN 1074-7613, 07/2013, Volume 39, Issue 1, pp. 111 - 122
Immune cells sense microbial products through Toll-like receptors (TLR), which trigger host defense responses including type 1 interferons (IFNs) secretion. A... 
TUMOR-NECROSIS-FACTOR | ACTIVATION | I INTERFERONS | CHORIOMENINGITIS VIRUS | PROTEIN-TYROSINE PHOSPHATASES | HOST-DEFENSE | MURINE LUPUS | INFLAMMATORY ARTHRITIS | IMMUNOLOGY | NF-KAPPA-B | T-CELLS | Lymphocytic choriomeningitis virus - immunology | Colitis - genetics | Humans | Autoimmunity - genetics | Immunoblotting | Arthritis - genetics | Interferon Type I - immunology | Lymphocytic Choriomeningitis - immunology | Host-Pathogen Interactions - immunology | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - immunology | Autoimmunity - immunology | Myeloid Cells - immunology | TNF Receptor-Associated Factor 3 - genetics | Colitis - chemically induced | HEK293 Cells | Arthritis - immunology | Interferon Type I - metabolism | Lymphocytic choriomeningitis virus - physiology | Toll-Like Receptors - metabolism | Colitis - immunology | Lymphocytic Choriomeningitis - genetics | Cell Line | Lymphocytic Choriomeningitis - virology | Mice, Inbred C57BL | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - metabolism | Toll-Like Receptors - immunology | Cells, Cultured | Immunity - immunology | Mice, Transgenic | TNF Receptor-Associated Factor 3 - metabolism | Ubiquitination - immunology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Animals | Dextran Sulfate - immunology | TNF Receptor-Associated Factor 3 - immunology | Toll-Like Receptors - genetics | Interferon Type I - genetics | Myeloid Cells - metabolism | Mice | Immunity - genetics | Ubiquitin | Autoimmunity | Interferon | Biological response modifiers
Journal Article
Gastroenterology, ISSN 0016-5085, 2017, Volume 152, Issue 5, pp. 1100 - 1113.e12
Journal Article
Mucosal Immunology, ISSN 1933-0219, 03/2016, Volume 9, Issue 2, pp. 336 - 351
A crosstalk between commensals, gut immune cells, and colonic epithelia is required for a proper function of intestinal mucosal barrier. Here we investigated... 
INFLAMMATORY-BOWEL-DISEASE | INTERFERON-GAMMA | MURINE COLITIS | INDOLEAMINE 2,3-DIOXYGENASE | IN-VIVO | BARRIER FUNCTION | LY6C(HI) MONOCYTES | T-CELL | IMMUNOLOGY | IL-18 BINDING-PROTEIN | IMMUNE DEFENSE | Heparin-binding EGF-like Growth Factor - genetics | Indoleamine-Pyrrole 2,3,-Dioxygenase - genetics | Antigens, CD - immunology | Dextran Sulfate | Interleukin-12 - genetics | Colitis - genetics | Dendritic Cells - immunology | Lectins, C-Type - immunology | Dendritic Cells - pathology | Colitis - pathology | Colon - immunology | Disease Resistance - immunology | Indoleamine-Pyrrole 2,3,-Dioxygenase - immunology | Lectins, C-Type - genetics | Antigens, CD - genetics | Interleukin-15 - immunology | CD11b Antigen - genetics | Intestinal Mucosa - immunology | Colitis - chemically induced | Integrin alpha Chains - immunology | Female | Interferon-gamma - genetics | Receptors, Immunologic - immunology | Colitis - immunology | Heparin-binding EGF-like Growth Factor - immunology | CD11b Antigen - immunology | Signal Transduction | Colon - pathology | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Epithelial Cells - pathology | Mice, Transgenic | Animals | Interleukin-15 - genetics | Epithelial Cells - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Mice | Mice, Inbred BALB C | Receptors, Immunologic - genetics | Integrin alpha Chains - genetics | Intercellular Signaling Peptides and Proteins - immunology | Intestinal Mucosa - pathology
Journal Article
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2015, Volume 136, Issue 2, pp. 413 - 422
Background Thymic stromal lymphopoietin (TSLP) is a cytokine with pleiotropic functions in the immune system. It has been associated with allergic reactions in... 
Allergy and Immunology | Thymic stromal lymphopoietin | gut homeostasis | celiac disease | anti-inflammatory drugs | atopic dermatitis | ulcerative colitis | skin homeostasis | DENDRITIC CELLS | TSLP | INDUCE | RECEPTOR | IMMUNOLOGY | ATOPIC-DERMATITIS | POLYMORPHISMS | EPITHELIAL-CELLS | ALLERGY | ASTHMA | NF-KAPPA-B | EXPRESSION | Dermatitis, Atopic - genetics | Skin - microbiology | Dendritic Cells - immunology | Humans | Salmonella Infections - genetics | Colitis, Ulcerative - genetics | Salmonella Infections - immunology | Dendritic Cells - pathology | Dermatitis, Atopic - microbiology | Colitis, Ulcerative - immunology | Case-Control Studies | Intestines - immunology | Celiac Disease - microbiology | Escherichia coli Infections - genetics | Celiac Disease - immunology | Leukocytes, Mononuclear - immunology | Dermatitis, Atopic - immunology | Female | Cytokines - genetics | Leukocytes, Mononuclear - microbiology | Skin - pathology | Cytokines - immunology | Disease Models, Animal | Skin - immunology | Salmonella Infections - pathology | Intestines - pathology | Celiac Disease - genetics | Gene Expression Regulation - immunology | Escherichia coli - immunology | Escherichia coli Infections - microbiology | Colitis, Ulcerative - pathology | Dermatitis, Atopic - pathology | Celiac Disease - pathology | Leukocytes, Mononuclear - pathology | Salmonella - immunology | Animals | Intestines - microbiology | Colitis, Ulcerative - microbiology | Escherichia coli Infections - pathology | Dendritic Cells - microbiology | Escherichia coli Infections - immunology | Salmonella Infections - microbiology | Mice | Protein Isoforms - immunology | Protein Isoforms - genetics | Skin | Salmonella | Food allergies | Antigens | Inflammation | Inflammatory diseases | Asthma | Inflammatory bowel disease | E coli | Biopsy | Lymphocytes | Rodents | Software | Laboratory animals | moDC, Monocyte-derived dendritic cell | NF-κB, Nuclear factor κB | TLR, Toll-like receptor | AD, Atopic dermatitis | STAT5, Signal transducer and activator of transcription 5 | mDC, Myeloid dendritic cell | shTSLP, Short isoform thymic stromal lymphopoietin | UC, Ulcerative colitis | CD, Celiac disease | DSS, Dextran sodium sulfate | DC, Dendritic cell | poly I:C, Polyinosinic:polycytidylic acid | TSLP, Thymic stromal lymphopoietin | MAPK, Mitogen-activated protein kinase | lTSLP, Long isoform thymic stromal lymphopoietin | TSLPR, Thymic stromal lymphopoietin receptor | Mechanisms of Allergy and Clinical Immunology
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2011, Volume 108, Issue 24, pp. 9927 - 9932
Cross-presentation of IgG-containing immune complexes (ICs) is an important means by which dendritic cells (DCs) activate CD8⁺ T cells, yet it proceeds by an... 
T lymphocytes | Antigens | Fc receptors | Cross priming | Dendritic cells | Internalization | Phagosomes | Inflammation | Oxidation | MHC CLASS-I | GAMMA-RECEPTOR | CD8(+) | VIVO | MULTIDISCIPLINARY SCIENCES | DISEASE | MONOCYTES | ANTIGEN PRESENTATION | MYASTHENIA-GRAVIS | T-CELLS | CUTTING EDGE | Dextran Sulfate | Membrane Glycoproteins - metabolism | Cytosol - immunology | Dendritic Cells - immunology | Receptors, Fc - metabolism | NADPH Oxidases - metabolism | NADPH Oxidases - immunology | Vacuolar Proton-Translocating ATPases - metabolism | Receptors, IgG - metabolism | Histocompatibility Antigens Class I - metabolism | Vacuolar Proton-Translocating ATPases - immunology | Phagosomes - immunology | Flow Cytometry | Immunoglobulin G - immunology | Colitis - chemically induced | Membrane Glycoproteins - immunology | Colitis - immunology | Dendritic Cells - metabolism | rab GTP-Binding Proteins - metabolism | CD11b Antigen - immunology | Mice, Inbred C57BL | Phagosomes - metabolism | Histocompatibility Antigens Class I - immunology | Mice, Transgenic | Histocompatibility Antigens Class I - genetics | Receptors, IgG - immunology | NADPH Oxidase 2 | Blotting, Western | CD8 Antigens - immunology | Mice, Knockout | CD8 Antigens - metabolism | Animals | Immunoglobulin G - genetics | Receptors, Fc - immunology | Cross-Priming - immunology | rab GTP-Binding Proteins - immunology | Colitis - metabolism | Protein Binding | rab27 GTP-Binding Proteins | Antigens - immunology | Cytosol - metabolism | Receptors, Fc - genetics | Mice | CD11b Antigen - metabolism | Mutation | Hydrogen-Ion Concentration | Immunoglobulin G - metabolism | Biological Sciences
Journal Article