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Gut, ISSN 0017-5749, 05/2017, Volume 66, Issue 5, pp. 823 - 838
ObjectiveInterleukin (IL)-36R signalling plays a proinflammatory role in different organs including the skin, but the expression of IL-36R ligands and their... 
STEM-CELLS | IL-1-ALPHA | REPAIR | ANTAGONIST | ULCERATIVE-COLITIS | RECEPTOR | MICE | IL-1-BETA | LIGAND | GASTROENTEROLOGY & HEPATOLOGY | INFLAMMATORY BOWEL DISEASES | Dextran Sulfate | Intestinal Mucosa - metabolism | Cell Proliferation | Epithelial Cells - metabolism | Lipocalin-2 - biosynthesis | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Colitis - genetics | Humans | Receptors, Interleukin-1 - genetics | Cytoplasm - metabolism | Wound Healing | Inflammatory Bowel Diseases - metabolism | Receptors, Interleukin - metabolism | Cell Nucleus - metabolism | Colitis - chemically induced | Interleukin-6 - metabolism | Fibroblasts - metabolism | Calgranulin B - biosynthesis | Interleukin-1 - metabolism | Cytokines - metabolism | Myeloid Differentiation Factor 88 - genetics | Signal Transduction - genetics | Mice, Knockout | Receptors, Interleukin-1 - metabolism | Macrophages - metabolism | Animals | Colitis - metabolism | Ligands | Chemokines - metabolism | Mice | Myeloid Differentiation Factor 88 - metabolism | Fibroblasts | Intestines | Care and treatment | Cellular signal transduction | Research | Epithelial cells | Cell proliferation | Animal models | Mucosa | Genomes | CD14 antigen | Interleukin 6 | Signal transduction | Intestine | Rodents | Tumor necrosis factor-TNF | Colony-stimulating factor | Colon | Wound healing | Bacterial infections | Cytokines | Granulocyte-macrophage colony-stimulating factor | Lipocalin | Inflammation | Epithelium | Ribonucleic acid--RNA | Inflammatory bowel disease | Dextran | Sodium | Biopsy | MyD88 protein | Skin | Colitis | Chemokines | Cytoplasm | Index Medicus | Abridged Index Medicus
Journal Article
Molecular Immunology, ISSN 0161-5890, 07/2013, Volume 54, Issue 3-4, pp. 482 - 492
► Oral treatment with α,β-amyrin ameliorate disease activity in DSS-induced colitis. ► α,β-Amyrin treatment decrease leukocyte influx to the colon. ► The... 
Inflammatory bowel disease | α,β-Amyrin | Dextran sodium sulfate | Inflammatory cytokines | Cannabinoid system | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | alpha,beta-amyrin | MECHANISMS | BETA-AMYRIN | ADHESION MOLECULES | IMMUNOLOGY | PROTIUM-HEPTAPHYLLUM | ALPHA-AMYRIN | INFLAMMATORY-BOWEL-DISEASE | MOUSE MODEL | RECEPTORS | EXPRESSION | P-Selectin - metabolism | Colitis - genetics | Tumor Necrosis Factor-alpha - genetics | Colon - drug effects | Body Weight - drug effects | Ki-67 Antigen - metabolism | Male | Interleukin-1beta - genetics | Antigens, CD - genetics | Body Weight - genetics | Antigens, CD - metabolism | Lectins, C-Type - metabolism | Interleukin-1beta - metabolism | Cannabinoids - genetics | Colitis - drug therapy | Cannabinoids - metabolism | Administration, Oral | CD18 Antigens - genetics | Membrane Proteins - genetics | Asialoglycoproteins - metabolism | P-Selectin - genetics | Cell Adhesion Molecules - metabolism | Chemokines - genetics | Colon - metabolism | CD18 Antigens - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Receptor, Cannabinoid, CB1 - genetics | Ki-67 Antigen - genetics | Intercellular Adhesion Molecule-1 - genetics | Colitis - metabolism | Chemokines - metabolism | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Tumor Necrosis Factor-alpha - metabolism | Dextran Sulfate | Cell Adhesion Molecules - genetics | Oleanolic Acid - analogs & derivatives | Oleanolic Acid - pharmacology | Lectins, C-Type - genetics | Receptor, Cannabinoid, CB2 - genetics | Asialoglycoproteins - genetics | Colitis - chemically induced | Vascular Cell Adhesion Molecule-1 - genetics | Membrane Proteins - metabolism | Interleukin-4 - genetics | Amidohydrolases - metabolism | Amidohydrolases - genetics | Interleukin-4 - metabolism | RNA, Messenger - genetics | Up-Regulation - genetics | Antigens, Differentiation, Myelomonocytic - genetics | Up-Regulation - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Peroxidase - genetics | Peroxidase - metabolism | Index Medicus
Journal Article
Gut, ISSN 0017-5749, 08/2014, Volume 63, Issue 8, pp. 1300 - 1312
Objective Enteric glia activation has been reported to amplify intestinal inflammation via the enteroglial-specific S100B protein. This neurotrophin promotes... 
NITRIC-OXIDE PRODUCTION | INHIBITION | CROHNS-DISEASE | MACROPHAGES | ULCERATIVE-COLITIS | FATTY-ACID AMIDE | S100B PROTEIN | N-PALMITOYL-ETHANOLAMINE | GLIAL-CELLS | GASTROENTEROLOGY & HEPATOLOGY | BOWEL-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Dextran Sulfate | Ethanolamines - pharmacology | Endocannabinoids - pharmacology | Rectum - pathology | Humans | Middle Aged | Palmitic Acids - therapeutic use | Male | NF-kappa B - metabolism | Glial Fibrillary Acidic Protein - metabolism | Nitric Oxide - analysis | Colon, Sigmoid - chemistry | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Neuroglia - drug effects | Colitis - chemically induced | Female | Indoles - pharmacology | Colitis - drug therapy | S100 Calcium Binding Protein beta Subunit - metabolism | Colitis, Ulcerative - drug therapy | Palmitic Acids - pharmacology | Colon, Sigmoid - pathology | Endocannabinoids - therapeutic use | Neutrophil Infiltration - drug effects | Severity of Illness Index | PPAR alpha - antagonists & inhibitors | Signal Transduction | Colitis, Ulcerative - metabolism | Cells, Cultured | Colitis, Ulcerative - pathology | Toll-Like Receptor 4 - metabolism | Dinoprostone - metabolism | Nerve Tissue Proteins - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Anilides - pharmacology | Colitis - metabolism | Cyclooxygenase 2 - metabolism | Rectum - chemistry | Ethanolamines - therapeutic use | Neuroglia - metabolism | Mice | PPAR alpha - metabolism | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Influence | Amides | Inflammation | Neuroglia | Analysis | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 12/2012, Volume 131, Issue 11, pp. 2553 - 2561
Estrogen receptor‐beta (ERβ) has been suggested to exert anti‐inflammatory and anti‐tumorigenic effects in the colon, providing a translational potential to... 
dextran sodium sulfate | estrogen receptor‐beta | colon cancer | azoxymethane | inflammatory bowel disease | estrogen receptor-beta | Estradiol - blood | Mucin-2 - genetics | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Colitis - genetics | Colorectal Neoplasms - genetics | Tumor Necrosis Factor-alpha - genetics | Colitis - pathology | NF-kappa B - metabolism | Interferon-gamma - metabolism | Inflammatory Bowel Diseases - metabolism | Estrogen Receptor beta - metabolism | Cell Differentiation - genetics | Inflammation - metabolism | Estrogen Receptor beta - genetics | Proliferating Cell Nuclear Antigen - genetics | Neoplasms - genetics | Inflammatory Bowel Diseases - pathology | Inflammatory Bowel Diseases - genetics | Female | Interferon-gamma - genetics | Interleukin-6 - metabolism | Colorectal Neoplasms - metabolism | Mucin-2 - metabolism | Mucin-1 - metabolism | Interleukin-6 - genetics | Colon - pathology | Mice, Inbred C57BL | Caveolin 1 - genetics | Interleukin-17 - genetics | beta Catenin - metabolism | Colon - metabolism | beta Catenin - genetics | Mice, Knockout | Caveolin 1 - metabolism | Interleukin-17 - metabolism | Animals | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis - metabolism | Inflammation - genetics | Mice | Colorectal Neoplasms - pathology | Neoplasms - pathology | Proliferating Cell Nuclear Antigen - metabolism | Mucin-1 - genetics | Nitric Oxide Synthase Type II - metabolism | Index Medicus | Estrogen receptor-beta
Journal Article
European Journal of Neuroscience, ISSN 0953-816X, 02/2008, Volume 27, Issue 3, pp. 654 - 670
Journal Article
Journal of Ethnopharmacology, ISSN 0378-8741, 01/2012, Volume 139, Issue 2, pp. 616 - 625
Ph-ME has a potent anti-inflammatory activity mediated by Src/Syk/NF-κB, and IRAK1/AP-1 inhibitory properties ( ). L. (Polygonaceae) has been traditionally... 
Prostaglandin E2 | Tumour necrosis factor-α | Anti-inflammatory effects | Polygonaceae | Nitric oxide | Polygonum hydropiper L | Prostaglandin E | RHEUMATOID-ARTHRITIS | ACTIVATION | CHEMISTRY, MEDICINAL | MACROPHAGES | Prostaglandin E-2 | GINSENG | ETHANOL EXTRACT | PLANT SCIENCES | INTEGRATIVE & COMPLEMENTARY MEDICINE | TOLL-LIKE RECEPTORS | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | NF-KAPPA-B | Tumour necrosis factor-alpha | T-CELLS | MOLECULAR-MECHANISMS | Plant Extracts - pharmacology | Protein-Tyrosine Kinases - metabolism | Solvents - chemistry | Humans | Male | NF-kappa B - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | RNA, Messenger - metabolism | Colitis, Ulcerative - immunology | Time Factors | Cyclooxygenase 2 - genetics | Anti-Inflammatory Agents - isolation & purification | Methanol - chemistry | Syk Kinase | Colitis, Ulcerative - prevention & control | Disease Models, Animal | Anti-Inflammatory Agents - pharmacology | Colitis, Ulcerative - metabolism | Plant Extracts - isolation & purification | Polygonum - chemistry | Dinoprostone - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Macrophages - metabolism | Signal Transduction - drug effects | Interleukin-1 Receptor-Associated Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | Mice | Plants, Medicinal | Nitric Oxide Synthase Type II - metabolism | Protein-Tyrosine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - metabolism | Dextran Sulfate | Interleukin-1 Receptor-Associated Kinases - metabolism | Plant Extracts - chemistry | Transcription Factor AP-1 - genetics | Transcription Factor AP-1 - metabolism | Dose-Response Relationship, Drug | Transfection | HEK293 Cells | Inflammation Mediators - metabolism | src-Family Kinases - metabolism | Macrophages - immunology | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Mice, Inbred C57BL | src-Family Kinases - antagonists & inhibitors | Animals | Anti-Inflammatory Agents - chemistry | Mitogen-Activated Protein Kinases - antagonists & inhibitors | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Colitis, Ulcerative - chemically induced | Cyclic AMP Response Element-Binding Protein - metabolism | Cyclooxygenase 2 - metabolism | Macrophages - drug effects | Protein Kinase Inhibitors - pharmacology | Nitric Oxide - metabolism | Mitogen-Activated Protein Kinases - metabolism | Tyrosine | Enzymes | Anti-inflammatory drugs | Biological response modifiers | Dextran | Prostaglandins E | Interferon beta | Genetically modified organisms | Protein kinases | Mitogens | Enzyme-linked immunosorbent assay | Methanol | Tumors | Index Medicus
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 08/2010, Volume 207, Issue 8, pp. 1617 - 1624
The protease a disintegrin and metalloprotease (ADAM) 17 cleaves tumor necrosis factor (TNF), L-selectin, and epidermal growth factor receptor (EGF-R) ligands... 
MEDICINE, RESEARCH & EXPERIMENTAL | STAT3 ACTIVATION | EPITHELIAL-CELLS | SULFATE-INDUCED COLITIS | EPIDERMAL-GROWTH-FACTOR | IN-VIVO | DISEASE | IMMUNOLOGY | FACTOR RECEPTOR | CANCER | T-CELLS | FACTOR-ALPHA | ADAM17 Protein | Tumor Necrosis Factor-alpha - metabolism | Cyclin D1 - metabolism | Epithelial Cells - metabolism | Animal Structures - abnormalities | Gene Expression - genetics | Epithelial Cells - drug effects | Inflammatory Bowel Diseases - immunology | Gene Expression Profiling | L-Selectin - metabolism | Brain - metabolism | Mammary Glands, Animal - growth & development | Receptors, Tumor Necrosis Factor, Type II - blood | Inflammatory Bowel Diseases - enzymology | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - pathology | Intestinal Mucosa - enzymology | Female | Inflammatory Bowel Diseases - chemically induced | Phosphorylation - drug effects | Epithelial Cells - cytology | Transforming Growth Factor alpha - metabolism | STAT3 Transcription Factor - metabolism | Transforming Growth Factor alpha - pharmacology | Dextran Sulfate - pharmacology | Cytokines - metabolism | Colon - pathology | Liver - metabolism | Mice, Inbred C57BL | ADAM Proteins - deficiency | Mice, Transgenic | Permeability | Colon - metabolism | Intestinal Mucosa - physiology | Regeneration | ADAM Proteins - metabolism | Animals | Cell Proliferation - drug effects | Chemokines - metabolism | Mice | ADAM Proteins - genetics | Animal Structures - metabolism | Intestinal Mucosa - pathology | Peroxidase - metabolism | Index Medicus | Brief Definitive Report
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 6/2004, Volume 316, Issue 3, pp. 315 - 323
The purpose of this study was to determine the cellular distribution and degradation in rat liver following intravenous injection of superparamagnetic iron... 
Medicine | Contrast agents | Iron oxide particles | Magnetic resonance imaging | Rat (Wister) | Liver | Metabolism | magnetic resonance imaging | LIVER ENDOTHELIAL-CELLS | liver | TRANSVERSE RELAXATION | iron oxide particles | contrast agents | ENDOCYTOSIS | CELL BIOLOGY | PHARMACOKINETICS | ENHANCEMENT | rat (wister) | KUPFFER CELLS | metabolism | PARTICLE-SIZE | FERRITIN | MR CONTRAST AGENTS | Injections, Intravenous | Rats, Wistar | Contrast Media - pharmacokinetics | Magnetic Resonance Imaging - methods | Male | Hepatocytes - metabolism | Ferrosoferric Oxide | Dextrans | Ferritins - metabolism | Liver - drug effects | Time Factors | Iron - pharmacokinetics | Kupffer Cells - metabolism | Hepatocytes - drug effects | Magnetite Nanoparticles | Oxides - metabolism | Hemosiderin - metabolism | Endothelial Cells - metabolism | Liver - metabolism | Rats | Iron - metabolism | Metabolic Clearance Rate - physiology | Kupffer Cells - drug effects | Oxides - pharmacokinetics | Cell Compartmentation - physiology | Animals | Cell Compartmentation - drug effects | Contrast Media - metabolism | Liver - cytology | Endothelial Cells - drug effects | Index Medicus | Cell Compartmentation/drug effects/physiology | Medical and Health Sciences | Endothelial Cells/drug effects/metabolism | Rats; Wistar | Medicin och hälsovetenskap | Injections; Intravenous | Magnetic Resonance Imaging/methods | Iron/metabolism/pharmacokinetics | Hemosiderin/metabolism | Liver/cytology/drug effects/metabolism | Oxides/metabolism/pharmacokinetics | Contrast Media/metabolism/pharmacokinetics | Ferritin/metabolism | Kupffer Cells/drug effects/metabolism | Metabolic Clearance Rate/physiology | Hepatocytes/drug effects/metabolism
Journal Article
Journal Article
Neuroscience, ISSN 0306-4522, 2005, Volume 130, Issue 1, pp. 165 - 183
The circadian clock housed in the suprachiasmatic nucleus (SCN) controls various circadian rhythms including daily sleep–wake cycles. Using dual tract-tracing,... 
orexin/hypocretin | dual tract-tracing | immunohistochemistry | aminergic | cholinergic | FREELY MOVING RATS | PHASEOLUS-VULGARIS-LEUKOAGGLUTININ | DORSOMEDIAL HYPOTHALAMIC NUCLEUS | FOS EXPRESSION | NEUROSCIENCES | NOCTURNAL RODENTS | PARAVENTRICULAR NUCLEUS | VENTROLATERAL PREOPTIC NUCLEUS | LOCUS-COERULEUS | RAPHE NUCLEI | CHOLERA-TOXIN | Tyrosine 3-Monooxygenase - metabolism | Rats, Wistar | Male | Intracellular Signaling Peptides and Proteins - metabolism | Preoptic Area - metabolism | Choline O-Acetyltransferase - metabolism | Neuropeptide Y - metabolism | Biotin - metabolism | Suprachiasmatic Nucleus - metabolism | Functional Laterality - physiology | Neurons - metabolism | Paraventricular Hypothalamic Nucleus - anatomy & histology | Dextrans - metabolism | Orexins | Cholera Toxin - metabolism | Histidine Decarboxylase - metabolism | Rats | Dorsomedial Hypothalamic Nucleus - anatomy & histology | Neuropeptides - metabolism | Circadian Rhythm - physiology | Dorsomedial Hypothalamic Nucleus - metabolism | Suprachiasmatic Nucleus - anatomy & histology | Immunohistochemistry - methods | Arousal - physiology | Neural Pathways - anatomy & histology | Animals | Cell Count - methods | Serotonin - metabolism | Paraventricular Hypothalamic Nucleus - metabolism | Neural Pathways - metabolism | Preoptic Area - anatomy & histology | Biotin - analogs & derivatives | Index Medicus
Journal Article