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The New England journal of medicine, ISSN 1533-4406, 10/2013, Volume 369, Issue 14, pp. 1317 - 1326
.... Type 2 diabetes mellitus doubles the risk of major cardiovascular complications in patients with and in patients without established cardiovascular disease, 1... 
Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Dipeptidyl-Peptidase IV Inhibitors - adverse effects | Dipeptidyl-Peptidase IV Inhibitors - therapeutic use | Humans | Middle Aged | Hospitalization - statistics & numerical data | Male | Adamantane - therapeutic use | Aged, 80 and over | Cardiovascular Diseases - epidemiology | Cardiovascular Diseases - mortality | Female | Hypoglycemia - chemically induced | Heart Failure - epidemiology | Diabetes Mellitus, Type 2 - complications | Adamantane - adverse effects | Adamantane - analogs & derivatives | Hypoglycemic Agents - therapeutic use | Cardiovascular Diseases - etiology | Dipeptides - therapeutic use | Double-Blind Method | Pancreatitis - epidemiology | Dipeptides - adverse effects | Kaplan-Meier Estimate | Pancreatitis - chemically induced | Aged | Diabetes Mellitus, Type 2 - drug therapy | Hypoglycemic Agents - adverse effects | Type 2 diabetes | Complications and side effects | Patient outcomes | Dosage and administration | Drug therapy | Heart diseases | Risk factors | Saxagliptin | Myocardial infarction | Cerebral infarction | Heart attacks | Peptidase | Diabetes mellitus | Pancreatitis | Cardiovascular disease | Angina | Patients | Heart rate | Ischemia | Diabetes | Cardiovascular diseases | Diabetes mellitus (non-insulin dependent) | Index Medicus | Abridged Index Medicus
Journal Article
Kidney international, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
...Diabetes mellitus is associated with several debilitating complications including kidney disease or diabetic nephropathy (DN), a main cause for patients... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Journal of cellular physiology, ISSN 0021-9541, 02/2018, Volume 233, Issue 2, pp. 1585 - 1600
...)‐induced model using male C57BL/6J mice at 8 weeks of age. Dietary intervention was initiated... 
bone mass | type 1 diabetes mellitus | high fat diet | bone marrow adipose tissue | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Body Composition | Streptozocin | Male | Insulin - blood | Diabetes Mellitus, Experimental - diet therapy | Diabetes Mellitus, Experimental - blood | Adiposity | Time Factors | Ketones - blood | Diet, High-Fat | Diabetes Mellitus, Type 1 - chemically induced | Fatty Acids - administration & dosage | Diabetes Mellitus, Experimental - chemically induced | Osteoporosis - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Osteoporosis - blood | Osteoporosis - chemically induced | Osteoporosis - prevention & control | Diabetes Mellitus, Type 1 - physiopathology | Mice, Inbred C57BL | Biomarkers - blood | Animals | Weight Loss | Diabetes Mellitus, Type 1 - blood | Diabetes Mellitus, Type 1 - diet therapy | Blood Glucose - metabolism | Bone Remodeling | Type 2 diabetes | Evaluation | Obesity | Adolescence | Reducing diets | Fatty acids | Density | Hyperglycemia | Diet therapy | Type 1 diabetes | Analysis | Physiological aspects | Bones | Adipose tissue | Body fat | Body weight | Chains | Bone (trabecular) | Low fat | Body composition | High fat diet | Biomedical materials | Body composition (biology) | Rodents | Bone marrow | Biocompatibility | Longitudinal studies | Bone loss | Bone (cortical) | Adolescents | Polydipsia | Osteopathy | Bone composition | Diabetes mellitus | Bone turnover | Insulin | Patients | Fractures | Bone mass | Diet | Correlation analysis | Low fat diet | Mice | Diabetes | Metabolic disorders | Index Medicus | Bone Marrow Adipose Tissue | High Fat Diet | Type 1 Diabetes Mellitus | Bone Mass
Journal Article
Diabetes (New York, N.Y.), ISSN 0012-1797, 11/2011, Volume 60, Issue 11, pp. 2872 - 2882
OBJECTIVE-To evaluate whether healthy or diabetic adult mice can tolerate an extreme loss of pancreatic a-cells and how this sudden massive depletion affects... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Insulin-Secreting Cells - secretion | Apoptosis - drug effects | Cell Count | Glucagon - genetics | Male | Diphtheria Toxin - toxicity | Insulin - blood | Glucagon - blood | Diabetes Mellitus, Experimental - blood | Hypoglycemia - prevention & control | Intercellular Signaling Peptides and Proteins - metabolism | Glucagon-Secreting Cells - drug effects | Insulin-Secreting Cells - metabolism | Hyperglycemia - chemically induced | Glucagon-Secreting Cells - metabolism | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Glucagon-Secreting Cells - secretion | Hyperglycemia - prevention & control | Promoter Regions, Genetic | Signal Transduction | Glucagon-Secreting Cells - pathology | Intercellular Signaling Peptides and Proteins - genetics | Pancreas - drug effects | Pancreas - pathology | Receptors, Glucagon - metabolism | Mice, Transgenic | Pancreas - metabolism | Heparin-binding EGF-like Growth Factor | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Tamoxifen - pharmacology | Diabetes Mellitus, Experimental - pathology | Glucagon - metabolism | Mice | Streptozocin - toxicity | Insulin-Secreting Cells - pathology | Selective Estrogen Receptor Modulators - pharmacology | Index Medicus | Abridged Index Medicus | Islet Studies
Journal Article
American journal of transplantation, ISSN 1600-6143, 06/2010, Volume 10, Issue 6, pp. 1420 - 1427
Journal Article