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Kidney international, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
...Diabetes mellitus is associated with several debilitating complications including kidney disease or diabetic nephropathy (DN), a main cause for patients... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Cardiovascular Pathology, ISSN 1054-8807, 2015, Volume 24, Issue 6, pp. 375 - 381
Abstract Introduction Hyperglycemia-induced reactive oxygen species (ROS) generation contributes to the development of diabetic cardiomyopathy... 
Pathology | Diabetic cardiomyopathy | Oxidative stress | Nuclear factor-erythroid 2-related factor 2 | MicroRNA-144 | Streptozotocin | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Ventricular Function, Left | Streptozocin | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Male | MicroRNAs - metabolism | Diabetes Mellitus, Type 1 - therapy | Diabetes Mellitus, Experimental - therapy | Transfection | Diabetes Mellitus, Type 1 - chemically induced | Diabetic Cardiomyopathies - prevention & control | NF-E2-Related Factor 2 - genetics | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Cardiomyopathies - genetics | Myocytes, Cardiac - pathology | Animals | NF-E2-Related Factor 2 - metabolism | Diabetes Mellitus, Experimental - pathology | Glucose - metabolism | Myocytes, Cardiac - metabolism | Diabetic Cardiomyopathies - pathology | MicroRNAs - genetics | Oligonucleotides - administration & dosage | Diabetic Cardiomyopathies - chemically induced | Apoptosis | MicroRNA | Index Medicus
Journal Article
Diabetes (New York, N.Y.), ISSN 0012-1797, 11/2011, Volume 60, Issue 11, pp. 2872 - 2882
OBJECTIVE-To evaluate whether healthy or diabetic adult mice can tolerate an extreme loss of pancreatic a-cells and how this sudden massive depletion affects... 
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Insulin-Secreting Cells - secretion | Apoptosis - drug effects | Cell Count | Glucagon - genetics | Male | Diphtheria Toxin - toxicity | Insulin - blood | Glucagon - blood | Diabetes Mellitus, Experimental - blood | Hypoglycemia - prevention & control | Intercellular Signaling Peptides and Proteins - metabolism | Glucagon-Secreting Cells - drug effects | Insulin-Secreting Cells - metabolism | Hyperglycemia - chemically induced | Glucagon-Secreting Cells - metabolism | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Glucagon-Secreting Cells - secretion | Hyperglycemia - prevention & control | Promoter Regions, Genetic | Signal Transduction | Glucagon-Secreting Cells - pathology | Intercellular Signaling Peptides and Proteins - genetics | Pancreas - drug effects | Pancreas - pathology | Receptors, Glucagon - metabolism | Mice, Transgenic | Pancreas - metabolism | Heparin-binding EGF-like Growth Factor | Insulin - metabolism | Animals | Insulin-Secreting Cells - drug effects | Tamoxifen - pharmacology | Diabetes Mellitus, Experimental - pathology | Glucagon - metabolism | Mice | Streptozocin - toxicity | Insulin-Secreting Cells - pathology | Selective Estrogen Receptor Modulators - pharmacology | Index Medicus | Abridged Index Medicus | Islet Studies
Journal Article
Toxicology and applied pharmacology, ISSN 0041-008X, 12/2014, Volume 281, Issue 2, pp. 211 - 220
Epidemiological studies have reported associations between particulate matter (PM) and cardiovascular effects, and diabetes mellitus... 
Rats | Air pollution | Inflammation | Particulate matter | Diabetes mellitus | Pharmacology & Pharmacy | Toxicology | Life Sciences & Biomedicine | Science & Technology | Inflammation - chemically induced | Particulate Matter - toxicity | Myocarditis - chemically induced | Streptozocin | Glycated Hemoglobin A - metabolism | Homeostasis | Male | Diabetes Complications - diagnosis | Diabetic Nephropathies - blood | Inflammation - blood | Diabetes Mellitus, Experimental - blood | Time Factors | Interleukin-6 - blood | Diabetes Mellitus, Type 1 - chemically induced | Inhalation Exposure - adverse effects | Diabetes Mellitus, Experimental - chemically induced | Diabetic Cardiomyopathies - blood | Inflammation - diagnosis | Diabetic Angiopathies - chemically induced | Diabetes Mellitus, Experimental - diagnosis | Risk Factors | Diabetic Nephropathies - chemically induced | Inflammation Mediators - blood | Toxicity Tests, Subchronic | Biomarkers - blood | Diabetic Angiopathies - blood | Myocarditis - blood | Diabetes Complications - blood | Rats, Sprague-Dawley | Blood Glucose - drug effects | Particle Size | Diabetes Complications - chemically induced | Animals | Diabetes Mellitus, Type 1 - blood | Fibrinogen - metabolism | Blood Glucose - metabolism | Diabetic Cardiomyopathies - chemically induced | Glucose metabolism | Type 1 diabetes | Fibrinogen | Hemoglobin | Glucose | Health aspects | Dextrose | Index Medicus | HOMEOSTASIS | INJURIES | RATS | AIR | COMPARATIVE EVALUATIONS | 60 APPLIED LIFE SCIENCES | AIR POLLUTION | DAMAGE | KIDNEYS | STREPTOZOCIN | GLOBAL ASPECTS | HYPERGLYCEMIA | BLOOD VESSELS | GLUCOSE | INFLAMMATION | DIABETES MELLITUS | FIBRINOGEN
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 11/2017, Volume 21, Issue 11, pp. 2732 - 2747
...)‐induced diabetic nephropathy, and dynamically regulated in cultured mouse podocytes stimulated with high glucose, which showed a trend from rise to decline... 
MALAT1 | β‐catenin | high glucose | diabetic nephropathy | SRSF1 | podocyte | β-catenin | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | RNA, Small Interfering - genetics | Diabetes Mellitus, Experimental - genetics | Male | Serine-Arginine Splicing Factors - metabolism | Serine-Arginine Splicing Factors - genetics | Glucose - toxicity | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Wnt Signaling Pathway | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetic Nephropathies - chemically induced | Gene Expression Regulation | Diabetic Nephropathies - genetics | RNA, Long Noncoding - genetics | Podocytes - pathology | beta Catenin - metabolism | beta Catenin - genetics | Protein Transport | Serine-Arginine Splicing Factors - antagonists & inhibitors | Feedback, Physiological | Animals | Podocytes - drug effects | beta Catenin - antagonists & inhibitors | Diabetes Mellitus, Experimental - pathology | Protein Binding | Mice | Streptozocin - toxicity | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | Cell Line, Transformed | RNA, Small Interfering - metabolism | Index Medicus | Original
Journal Article
Journal of cellular physiology, ISSN 0021-9541, 02/2018, Volume 233, Issue 2, pp. 1585 - 1600
...)‐induced model using male C57BL/6J mice at 8 weeks of age. Dietary intervention was initiated... 
bone mass | type 1 diabetes mellitus | high fat diet | bone marrow adipose tissue | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Body Composition | Streptozocin | Male | Insulin - blood | Diabetes Mellitus, Experimental - diet therapy | Diabetes Mellitus, Experimental - blood | Adiposity | Time Factors | Ketones - blood | Diet, High-Fat | Diabetes Mellitus, Type 1 - chemically induced | Fatty Acids - administration & dosage | Diabetes Mellitus, Experimental - chemically induced | Osteoporosis - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Osteoporosis - blood | Osteoporosis - chemically induced | Osteoporosis - prevention & control | Diabetes Mellitus, Type 1 - physiopathology | Mice, Inbred C57BL | Biomarkers - blood | Animals | Weight Loss | Diabetes Mellitus, Type 1 - blood | Diabetes Mellitus, Type 1 - diet therapy | Blood Glucose - metabolism | Bone Remodeling | Type 2 diabetes | Evaluation | Obesity | Adolescence | Reducing diets | Fatty acids | Density | Hyperglycemia | Diet therapy | Type 1 diabetes | Analysis | Physiological aspects | Bones | Adipose tissue | Body fat | Body weight | Chains | Bone (trabecular) | Low fat | Body composition | High fat diet | Biomedical materials | Body composition (biology) | Rodents | Bone marrow | Biocompatibility | Longitudinal studies | Bone loss | Bone (cortical) | Adolescents | Polydipsia | Osteopathy | Bone composition | Diabetes mellitus | Bone turnover | Insulin | Patients | Fractures | Bone mass | Diet | Correlation analysis | Low fat diet | Mice | Diabetes | Metabolic disorders | Index Medicus | Bone Marrow Adipose Tissue | High Fat Diet | Type 1 Diabetes Mellitus | Bone Mass
Journal Article