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Diabetes, obesity & metabolism, ISSN 1462-8902, 1999
Journal
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 12, p. e52013
...: An experimental diabetic rat model was induced by low dose of streptozoticin(STZ) combined with high energy intake on rats... 
FIBROSIS | CELLS | OXIDATIVE STRESS | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | GENE-EXPRESSION | CARDIAC DYSFUNCTION | PPAR-GAMMA | RECEPTOR | GLYCATION END-PRODUCTS | DIASTOLIC DYSFUNCTION | Inflammation - pathology | Diabetes Mellitus, Experimental - drug therapy | Fibrosis - drug therapy | Diabetic Cardiomyopathies - metabolism | Diabetic Cardiomyopathies - drug therapy | Rats, Wistar | Apoptosis - drug effects | Diabetes Mellitus, Experimental - genetics | Apoptosis - genetics | Glycogen Synthase Kinase 3 beta | Male | Fibrosis - metabolism | Proto-Oncogene Proteins c-akt - genetics | Inflammation - metabolism | Cell Death - genetics | Ventricular Dysfunction, Left - genetics | Inflammation - drug therapy | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Cell Death - drug effects | Phosphorylation - drug effects | Diabetes Mellitus, Experimental - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Receptor for Advanced Glycation End Products | Fibrosis - genetics | Curcumin - pharmacology | Oxidative Stress - genetics | Rats | Myocardium - pathology | Glycogen Synthase Kinase 3 - metabolism | Diabetic Cardiomyopathies - genetics | Ventricular Dysfunction, Left - metabolism | Animals | Ventricular Dysfunction, Left - drug therapy | Glycogen Synthase Kinase 3 - genetics | Diabetes Mellitus, Experimental - pathology | Heart - drug effects | Inflammation - genetics | Diabetic Cardiomyopathies - pathology | Fibrosis - pathology | Oxidative Stress - drug effects | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Oxidases | Heart | Biological products | Cardiomyopathy | Heart diseases | Enzyme-linked immunosorbent assay | Apoptosis | Oxidative stress | Phosphorylation | Bax protein | Bcl-2 protein | Science | AKT protein | Cardiovascular disease | Caspase-3 | NAD(P)H oxidase | Accumulation | Proteins | Signal transduction | Hyperglycemia | Ultrastructure | Biochemical tests | Curcumin | Age | Energy intake | Enzymes | Advanced glycosylation end products | Echocardiography | Abnormalities | Diabetes mellitus | Caspase | Rac1 protein | Pharmacology | Inflammation | Glycosylation | IL-1β | Gene expression | Metabolism | Studies | Signaling | Cell death | Fibrosis | Diabetes | Aberration | Laboratory animals | Metabolic disorders | Hypertrophy
Journal Article
Nature medicine, ISSN 1546-170X, 2009, Volume 15, Issue 8, pp. 940 - 945
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 2, p. e16556
Berberine (BBR) is a compound originally identified in a Chinese herbal medicine Huanglian (Coptis chinensis French). It improves glucose metabolism in type 2... 
ACTIVATED PROTEIN-KINASE | MECHANISM DISTINCT | INSULIN-RESISTANCE | MUSCLE ATROPHY | LIVER | BIOLOGY | CARBOHYDRATE | ELEMENT-BINDING PROTEIN | FOXO TRANSCRIPTION FACTORS | LIPID HOMEOSTASIS | EXPRESSION | Diabetes Mellitus, Experimental - drug therapy | Streptozocin | Gluconeogenesis - drug effects | Male | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Diabetes Mellitus, Experimental - blood | Liver - drug effects | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Drug Evaluation, Preclinical | Hypoglycemic Agents - therapeutic use | Blood Glucose - analysis | Liver - metabolism | Rats | Down-Regulation - drug effects | Rats, Sprague-Dawley | Hypoglycemic Agents - pharmacology | Blood Glucose - drug effects | Berberine - pharmacology | Diabetes Mellitus, Type 2 - blood | Insulin - metabolism | Animals | Glucose - metabolism | Berberine - therapeutic use | Diabetes Mellitus, Type 2 - chemically induced | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Type 2 diabetes | Phosphatases | Blood sugar | Genes | DNA binding proteins | Muscle proteins | Fatty acids | Insulin | Glucose metabolism | Synthesis | Adenylic acid | Protein kinases | Adenosine triphosphate | Protein binding | Transcription factors | Peptides | Liver | Glucose | Blood | Proteins | Signal transduction | Mitochondria | Hyperglycemia | Forkhead protein | Inhibition | Drug dosages | Gluconeogenesis | Binding | Carbohydrates | Berberine | AMP | Metabolism | Fatty-acid synthase | Hepatocytes | Protein synthesis | Glucose-6-phosphatase | Endocrinology | Biomedical research | Kinases | High fat diet | Fatty liver | FOXO1 protein | Rodents | Statins | Oxygen | Fasting | Adenosine monophosphate | Diabetes mellitus | Oxygen consumption | Steatosis | Herbal medicine | Musculoskeletal system | Signaling | Protein kinase | Adenosine kinase | Coronary vessels | Insulin resistance | Diabetes | Respiration | Laboratory animals | ATP
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 5, p. e61477
Obesity is important for the development of type-2 diabetes as a result of obesity-induced insulin resistance accompanied by impaired compensation of insulin... 
BODY-WEIGHT | MEDICAL THERAPY | DIET | FOOD-INTAKE | GLUCOSE | MULTIDISCIPLINARY SCIENCES | VASOPRESSIN | BARIATRIC SURGERY | SCHIZOPHRENIA | MICE | BLOOD-BRAIN-BARRIER | Diabetes Mellitus, Experimental - drug therapy | Obesity - drug therapy | Humans | Middle Aged | Male | Prediabetic State - drug therapy | Obesity - blood | Glucose Intolerance - blood | Diabetes Mellitus, Experimental - blood | Young Adult | Glucose Intolerance - drug therapy | Hypoglycemic Agents - administration & dosage | Adult | Female | Insulin Secretion | Prediabetic State - blood | Mice, Inbred C57BL | Diabetes Mellitus, Type 2 - prevention & control | Insulin Resistance | Blood Glucose | Oxytocin - analogs & derivatives | Administration, Intranasal | Oxytocin - administration & dosage | Diabetes Mellitus, Type 2 - blood | Insulin - metabolism | Pilot Projects | Animals | Lipid Metabolism - drug effects | Mice | Type 2 diabetes | Obesity | Medical colleges | Pancreatic beta cells | Insulin resistance | Glucose | Drug therapy | Health aspects | Insulin | Prediabetic state | Dextrose | Diabetes therapy | Animal models | Breastfeeding & lactation | Mental disorders | Peptides | Streptozocin | Schizophrenia | Homeostasis | Cardiovascular disease | Neuropeptides | Analogs | Reduction | Blood-brain barrier | Rodents | Pancreas | Oxytocin | Diabetes mellitus | Metabolism | Patients | Beta cells | Medicine | Studies | Glucose tolerance | Intolerance | Weight control | Brain research | Hospitals | Regulation | Diabetes | Gastrointestinal surgery
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 1, p. e0170971
Background Pathophysiological investigation of disease in a suitable animal model is a classical approach towards development of a credible therapeutic... 
FRUCTOSE | HIGH-FAT DIET | GLUCOSE | MULTIDISCIPLINARY SCIENCES | ANIMAL-MODELS | RESISTANCE | BETA-CELL MASS | Diabetes Mellitus, Experimental - drug therapy | Rats, Wistar | Cholesterol - blood | Humans | Diabetes Mellitus, Type 2 - diet therapy | Male | Insulin - blood | Thiazolidinediones - administration & dosage | Hyperglycemia - drug therapy | Diabetes Mellitus, Experimental - diet therapy | Diabetes Mellitus, Experimental - blood | Hypoglycemic Agents - administration & dosage | Hyperglycemia - pathology | Disease Models, Animal | Insulin Resistance | Rats | Blood Glucose | Food, Fortified | Hyperglycemia - diet therapy | Diabetes Mellitus, Type 2 - blood | Animals | Hyperglycemia - blood | Diabetes Mellitus, Experimental - pathology | Diabetes Mellitus, Type 2 - pathology | Diabetes Mellitus, Type 2 - drug therapy | Genetically modified mice | Type 2 diabetes | Streptozocin | Dosage and administration | Research | Food and nutrition | Drug therapy | Drugs | Drinking water | Animal models | Peptides | Body weight | Diabetes mellitus (insulin dependent) | Biochemistry | Metabolic syndrome | Hyperglycemia | Rodents | Glibenclamide | Diabetes mellitus (non-insulin dependent) | Research & development--R&D | Pioglitazone | Diabetes mellitus | Triglycerides | Insulin | Fructose | Cholesterol | Studies | Hypercholesterolemia | Diet | Hypertriglyceridemia | Biomarkers | Insulin resistance | Metformin | Diabetes | Margarine | Laboratory animals | Research & development | R&D
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 2017, Volume 69, Issue 2, pp. 131 - 143
Abstract Background Diabetes mellitus causes microcirculatory rarefaction and may impair the responsiveness of ischemic myocardium to proangiogenic factors... 
Cardiovascular | Internal Medicine | gene therapy | chronic myocardial ischemia | thymosin β4 | angiogenesis | CARDIAC & CARDIOVASCULAR SYSTEMS | EVENTS | RISK | thymosin beta 4 | CELL-ADHESION | IMPAIRED ANGIOGENESIS | INFLAMMATION | DISEASE | GROWTH | RETINOPATHY | EXPRESSION | DRUG-ELUTING STENTS | Genetic Therapy | Microvessels - physiopathology | Neovascularization, Physiologic - drug effects | Thymosin - administration & dosage | Diabetes Mellitus, Experimental - diagnosis | Myocardial Stunning - physiopathology | Coronary Vessels - physiopathology | Humans | Translational Medical Research | Heart Failure - physiopathology | Stroke Volume - physiology | Stroke Volume - drug effects | Coronary Disease - physiopathology | Heart Transplantation | Coronary Disease - diagnosis | Diabetic Angiopathies - physiopathology | Animals | Swine | Myocardial Stunning - drug therapy | Myocardium | Diabetic Angiopathies - diagnosis | Heart Failure - diagnosis | Vascular Endothelial Growth Factor A - administration & dosage | Diabetes Mellitus, Experimental - physiopathology | Diabetes | Endothelial growth factors | Analysis | Cardiac patients | Thymosin | Neovascularization | Health aspects | Studies | Angiogenesis | Medical research | Hogs | Rodents | Acute coronary syndromes | Gene therapy | Vascular endothelial growth factor | Insulin
Journal Article
Kidney international, ISSN 0085-2538, 2018, Volume 93, Issue 1, pp. 95 - 109
Inflammation plays a central role in the etiology of diabetic nephropathy, a global health issue. We observed a significant reduction in the renal expression... 
cytokines | fibrosis | inflammation | mesangial cells | diabetic nephropathy | PATHWAYS | ACTIVATION | JNK | KINASE | FIBROBLAST-GROWTH-FACTOR | PATHOGENESIS | OBESITY | INSULIN-RESISTANCE | UROLOGY & NEPHROLOGY | NF-KAPPA-B | EXPRESSION | Diabetes Mellitus, Experimental - drug therapy | Kidney - pathology | Rats, Wistar | Diabetic Nephropathies - etiology | Humans | JNK Mitogen-Activated Protein Kinases - metabolism | Male | NF-kappa B - metabolism | Diabetes Mellitus, Type 1 - complications | Diabetic Nephropathies - blood | Diabetes Mellitus, Experimental - blood | Kidney - metabolism | Inflammation Mediators - metabolism | Diabetes Mellitus, Experimental - complications | Diabetic Nephropathies - prevention & control | Diabetes Mellitus, Type 2 - complications | Cell Line | Diabetic Nephropathies - pathology | Kidney - drug effects | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Recombinant Proteins - pharmacology | Diabetes Mellitus, Type 1 - drug therapy | Blood Glucose - drug effects | Fibroblast Growth Factor 1 - blood | Diabetes Mellitus, Type 2 - blood | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Diabetes Mellitus, Type 1 - blood | Fibroblast Growth Factor 1 - pharmacology | Macrophages - drug effects | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy
Journal Article
Journal Article
Journal of translational medicine, ISSN 1479-5876, 2015, Volume 13, Issue 1, pp. 6 - 6
...) against type 2 diabetes mellitus (T2DM) and its associated nephropathy and cardiomyopathy in experimental rats. Methods... 
Oxidative stress | Streptozotocin-nicotinamide | Nephropathy | Type 2 diabetes mellitus | Abroma augusta | Cardiomyopathy | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | BENEFICIAL ROLE | nicotinamide | CORCHORUS-OLITORIUS LEAVES | RAT MODEL | INSULIN | HYPERGLYCEMIA | PATHWAY | ENDOTHELIAL-CELLS | Streptozotocin | NF-KAPPA-B | Inflammation - pathology | Diabetes Mellitus, Experimental - drug therapy | Phytochemicals - therapeutic use | Kidney - pathology | Rats, Wistar | Plant Extracts - pharmacology | Body Weight - drug effects | Diabetic Nephropathies - drug therapy | Male | Diabetic Nephropathies - blood | Diabetes Mellitus, Experimental - blood | Inflammation - complications | Kidney - metabolism | Adenosine Triphosphate - metabolism | Myocardium - metabolism | Diabetes Mellitus, Experimental - complications | Malvaceae - chemistry | DNA Fragmentation - drug effects | Diabetes Mellitus, Type 2 - complications | NAD - metabolism | Biomarkers - metabolism | Cardiomyopathies - drug therapy | Plant Leaves - chemistry | Diabetic Nephropathies - pathology | Glucose Tolerance Test | Cardiomyopathies - blood | Kidney - drug effects | Niacinamide - therapeutic use | Cardiomyopathies - pathology | Myocardium - pathology | Diabetic Nephropathies - complications | Diabetes Mellitus, Type 2 - blood | Organ Size - drug effects | Animals | Diabetes Mellitus, Experimental - pathology | Cardiomyopathies - complications | Diabetes Mellitus, Type 2 - pathology | Niacinamide - pharmacology | Oxidative Stress - drug effects | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Phytochemicals - pharmacology | Plant Extracts - therapeutic use | Type 2 diabetes | Niacinamide | Enzymes | Flavones | Streptozocin | Creatine kinase | Blood sugar | Bioflavonoids | Body weight | Creatine | Hyperglycemia | Flavonoids | Medicine, Botanic | Medicine, Herbal | Kidney diseases | Heart diseases | Methanol | Apoptosis | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 2, p. e56022
... and to worsen the situation, wounds inflicted with drug resistant strains represent a morbid combination... 
SOFT-TISSUE INFECTIONS | BACTERIAL-INFECTIONS | VANCOMYCIN | PHARMACOKINETICS | MULTIDISCIPLINARY SCIENCES | PENETRATION | PHAGE THERAPY | MICE | MELLITUS | KLEBSIELLA-PNEUMONIAE | COMPLICATED SKIN | Bacteriophages | Diabetic Foot - complications | Linezolid | Diabetes Mellitus, Experimental - drug therapy | Staphylococcal Skin Infections - complications | Diabetic Foot - drug therapy | Anti-Infective Agents - therapeutic use | Treatment Outcome | Diabetic Foot - microbiology | Oxazolidinones - therapeutic use | Anti-Bacterial Agents - therapeutic use | Staphylococcal Skin Infections - drug therapy | Animals | Acetamides - therapeutic use | Anti-Bacterial Agents - pharmacology | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - microbiology | Mice | Mice, Inbred BALB C | Methicillin-Resistant Staphylococcus aureus - isolation & purification | Staphylococcal Skin Infections - microbiology | Infection | Type 2 diabetes | Diabetics | Drug resistance in microorganisms | Usage | Analysis | Staphylococcus aureus infections | Diabetic foot | Health aspects | Staphylococcus aureus | Therapy | Pneumonia | Costs | Laboratories | Phages | Diabetic neuropathy | Infections | Drug resistance | Methicillin | Immunology | Bacteria | Peroxidase | Strains (organisms) | Bacterial infections | Wound healing | Mortality | Diabetes mellitus | Neutrophils | Staphylococcus infections | FDA approval | Patients | Mutants | Feet | Alloxan | Immunosuppression | Infectious diseases | Antibiotics | Ulcers | Resistant mutant | Arresting (process) | Host range | Healing | Diabetes | Foot diseases
Journal Article