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Kidney International, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
Enhanced transforming growth factor-β1 (TGF-β1) expression in renal cells promotes fibrosis and hypertrophy during the progression of diabetic nephropathy. The... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2014, Volume 74, pp. 139 - 150
Abstract Exosomes, nano-vesicles naturally released from living cells, have been well recognized to play critical roles in mediating cell-to-cell... 
Cardiovascular | Type 2 diabetes | Cardiomyocytes | Exosomes | miR-320 | Myocardial angiogenesis | MiR-320 | CARDIAC & CARDIOVASCULAR SYSTEMS | HYPERGLYCEMIA | DYSFUNCTION | EXPRESSION | MICRORNA-320 | CELL BIOLOGY | Exosomes - drug effects | Exosomes - metabolism | Cell Proliferation | Rats, Wistar | Coculture Techniques | Diabetes Mellitus, Type 2 - genetics | Humans | Diabetes Mellitus, Experimental - genetics | Exosomes - pathology | HSP20 Heat-Shock Proteins - genetics | MicroRNAs - metabolism | Insulin-Like Growth Factor I - genetics | Diabetes Mellitus, Type 2 - metabolism | HSP20 Heat-Shock Proteins - metabolism | Biological Transport | Proto-Oncogene Protein c-ets-2 - genetics | Proto-Oncogene Protein c-ets-2 - metabolism | Diabetes Mellitus, Experimental - metabolism | Biomarkers - metabolism | Aniline Compounds - pharmacology | Signal Transduction | Endothelial Cells - metabolism | Gene Expression Regulation | Rats | Myocytes, Cardiac - pathology | Animals | Benzylidene Compounds - pharmacology | Myocytes, Cardiac - drug effects | Diabetes Mellitus, Experimental - pathology | Myocytes, Cardiac - metabolism | MicroRNAs - genetics | Diabetes Mellitus, Type 2 - pathology | Endothelial Cells - pathology | Insulin-Like Growth Factor I - metabolism | Neovascularization, Physiologic | Cell Movement | Endothelial Cells - drug effects | Antimitotic agents | Heat shock proteins | Diabetes | Antineoplastic agents | Heart cells | Endothelium | Index Medicus
Journal Article
Circulation, ISSN 0009-7322, 10/2017, Volume 136, Issue 17, pp. 1629 - 1642
Journal Article
Cell Metabolism, ISSN 1550-4131, 05/2013, Volume 17, Issue 5, pp. 695 - 708
Diabetes is a major risk factor for atherosclerosis. Although atherosclerosis is initiated by deposition of cholesterol-rich lipoproteins in the artery wall,... 
DIABETES-MELLITUS | PLAQUE REGRESSION | INSULIN-RESISTANCE | ADVANCED GLYCATION ENDPRODUCTS | ENDOCRINOLOGY & METABOLISM | LEUKOCYTE COUNT | RECEPTOR | STEM-CELL PROLIFERATION | APOE(-/-) MICE | DEFICIENT MICE | G-CSF | CELL BIOLOGY | Leukocytes - pathology | Humans | Diabetes Mellitus, Type 1 - metabolism | Male | Monocytes - metabolism | NF-kappa B - metabolism | Leukocytosis - metabolism | Coronary Disease - metabolism | Bone Marrow - metabolism | Hyperglycemia - pathology | Monocytes - pathology | Diabetes Mellitus, Experimental - metabolism | Neutrophils - metabolism | Receptor for Advanced Glycation End Products | Leukocytosis - pathology | Neutrophils - pathology | Myeloid Progenitor Cells - metabolism | Atherosclerosis - pathology | Myelopoiesis - physiology | Cytokines - metabolism | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - pathology | Myeloid Progenitor Cells - pathology | Atherosclerosis - metabolism | Hyperglycemia - metabolism | Coronary Disease - pathology | Animals | Bone Marrow - pathology | Glycation End Products, Advanced - metabolism | Glucose - metabolism | Mice | Leukocytes - metabolism | Receptors, Immunologic - metabolism | Hyperglycemia | Type 1 diabetes | Atherosclerosis | Development and progression | Universities and colleges | Blood lipids | Coronary heart disease | Medicine, Preventive | Cholesterol | Preventive health services | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2009, Volume 106, Issue 32, pp. 13505 - 13510
Diabetes is associated with poor outcomes following acute vascular occlusive events. This results in part from a failure to form adequate compensatory... 
Diabetic angiopathies | Wound healing | Diabetes complications | Diabetes mellitus | Fibroblasts | Hypoxia | Mice | Diabetes | Type 2 diabetes mellitus | Transactivation | HIF-1 | Hyperglycemia | Ischemia | Deferoxamine | MOBILIZATION | STABILIZATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | VESSEL FORMATION | MELLITUS | ischemia | HYDROXYLASES | hyperglycemia | deferoxamine | METABOLISM | COMPLICATIONS | INDUCIBLE FACTOR-1 | ENDOTHELIAL GROWTH-FACTOR | Diabetes Mellitus - pathology | Reactive Oxygen Species - metabolism | Humans | Transcriptional Activation - drug effects | Hyperglycemia - complications | Vascular Endothelial Growth Factor A - metabolism | Neovascularization, Pathologic - complications | Neovascularization, Pathologic - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Protein Binding - drug effects | Hyperglycemia - pathology | Diabetes Mellitus, Experimental - complications | Diabetes Complications - pathology | Diabetes Mellitus, Experimental - metabolism | Wound Healing - drug effects | Disease Models, Animal | Deferoxamine - pharmacology | Pyruvaldehyde - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Diabetes Complications - metabolism | Cells, Cultured | Diabetes Mellitus - metabolism | Glucose - pharmacology | Hypoxia - complications | Up-Regulation - drug effects | p300-CBP Transcription Factors - metabolism | Animals | Diabetes Mellitus, Experimental - pathology | Research | Neovascularization | Vascular endothelial growth factor | Glucose | Rodents | Index Medicus | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 2, pp. e16556 - e16556
Berberine (BBR) is a compound originally identified in a Chinese herbal medicine Huanglian (Coptis chinensis French). It improves glucose metabolism in type 2... 
ACTIVATED PROTEIN-KINASE | MECHANISM DISTINCT | INSULIN-RESISTANCE | MUSCLE ATROPHY | LIVER | BIOLOGY | CARBOHYDRATE | ELEMENT-BINDING PROTEIN | FOXO TRANSCRIPTION FACTORS | LIPID HOMEOSTASIS | EXPRESSION | Diabetes Mellitus, Experimental - drug therapy | Streptozocin | Gluconeogenesis - drug effects | Male | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Diabetes Mellitus, Experimental - blood | Liver - drug effects | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Drug Evaluation, Preclinical | Hypoglycemic Agents - therapeutic use | Blood Glucose - analysis | Liver - metabolism | Rats | Down-Regulation - drug effects | Rats, Sprague-Dawley | Hypoglycemic Agents - pharmacology | Blood Glucose - drug effects | Berberine - pharmacology | Diabetes Mellitus, Type 2 - blood | Insulin - metabolism | Animals | Glucose - metabolism | Berberine - therapeutic use | Diabetes Mellitus, Type 2 - chemically induced | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Type 2 diabetes | Phosphatases | Blood sugar | Genes | DNA binding proteins | Muscle proteins | Fatty acids | Insulin | Glucose metabolism | Synthesis | Adenylic acid | Protein kinases | Adenosine triphosphate | Protein binding | Transcription factors | Peptides | Liver | Glucose | Blood | Proteins | Signal transduction | Mitochondria | Hyperglycemia | Forkhead protein | Inhibition | Drug dosages | Gluconeogenesis | Binding | Carbohydrates | Berberine | AMP | Metabolism | Fatty-acid synthase | Hepatocytes | Protein synthesis | Glucose-6-phosphatase | Endocrinology | Biomedical research | Kinases | High fat diet | Fatty liver | FOXO1 protein | Rodents | Statins | Fasting | Adenosine monophosphate | Diabetes mellitus | Oxygen consumption | Steatosis | Herbal medicine | Musculoskeletal system | Signaling | Protein kinase | Adenosine kinase | Coronary vessels | Insulin resistance | Diabetes | Respiration | Laboratory animals | ATP | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2012, Volume 109, Issue 39, pp. 15888 - 15893
The epidemics of insulin resistance (IR) and type 2 diabetes (T2D) affect the first world as well as less-developed countries, and now affect children as well.... 
Receptors | 3T3 L1 cells | Myasthenia gravis | Diet | Insulin resistance | Mice | Adipocytes | Type 2 diabetes mellitus | Diabetes | Insulin | Aging | Glycotoxins | Obesity | LIFE-SPAN | OXIDATIVE STRESS | METABOLIC SYNDROME | OXIDANT STRESS | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | RISK-FACTOR | glycotoxins | MESANGIAL CELLS | aging | INFLAMMATORY RESPONSE | END-PRODUCTS | obesity | METHYLGLYOXAL | Sirtuin 1 - metabolism | Deoxyglucose - metabolism | Diabetes Mellitus, Type 2 - genetics | Humans | Diabetes Mellitus, Experimental - genetics | Metabolic Syndrome - metabolism | Diabetes Mellitus, Type 2 - metabolism | Insulin Receptor Substrate Proteins - metabolism | Metabolic Syndrome - chemically induced | Sirtuin 1 - genetics | Inflammation - drug therapy | Glycation End Products, Advanced - adverse effects | Diabetes Mellitus, Experimental - metabolism | Insulin Receptor Substrate Proteins - genetics | Receptor for Advanced Glycation End Products | Macrophages - pathology | Administration, Oral | Oxidative Stress - genetics | 3T3-L1 Cells | Adipocytes - pathology | Proto-Oncogene Proteins c-akt | Macrophages - metabolism | Animals | Glycation End Products, Advanced - pharmacology | Metabolic Syndrome - genetics | Adipocytes - metabolism | Deoxyglucose - genetics | Diabetes Mellitus, Experimental - pathology | Inflammation - genetics | Diabetes Mellitus, Type 2 - pathology | Metabolic Syndrome - pathology | Oxidative Stress - drug effects | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Type 2 diabetes | Antioxidants | Cell receptors | Physiological aspects | Glycosylation | Properties | Proteins | Oxidative stress | Genotype & phenotype | Epidemics | Index Medicus | Biological Sciences
Journal Article