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Journal Article
Kidney International, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
Enhanced transforming growth factor-β1 (TGF-β1) expression in renal cells promotes fibrosis and hypertrophy during the progression of diabetic nephropathy. The... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Nephrology Dialysis Transplantation, ISSN 0931-0509, 1990, Volume 5, Issue 3, pp. 220 - 225
Journal Article
Journal Article
Journal of Diabetes Investigation, ISSN 2040-1116, 11/2016, Volume 7, Issue 6, pp. 915 - 918
Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 02/2018, Volume 233, Issue 2, pp. 1585 - 1600
Increasing numbers of patients with type 1 diabetes mellitus are overweight or obese. We treated type 1 diabetic mice with a high fat diet consisting of medium... 
bone mass | type 1 diabetes mellitus | high fat diet | bone marrow adipose tissue | ENERGY | PHYSIOLOGY | GLYCATION | RECEPTOR | C57BL/6J | CELL BIOLOGY | MARROW ADIPOSITY | MINERAL DENSITY | OBESITY | INSULIN-RESISTANCE | SKELETAL | EXPRESSION | Body Composition | Streptozocin | Male | Insulin - blood | Diabetes Mellitus, Experimental - diet therapy | Diabetes Mellitus, Experimental - blood | Adiposity | Time Factors | Ketones - blood | Diet, High-Fat | Diabetes Mellitus, Type 1 - chemically induced | Fatty Acids - administration & dosage | Diabetes Mellitus, Experimental - chemically induced | Osteoporosis - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Osteoporosis - blood | Osteoporosis - chemically induced | Osteoporosis - prevention & control | Diabetes Mellitus, Type 1 - physiopathology | Mice, Inbred C57BL | Biomarkers - blood | Animals | Weight Loss | Diabetes Mellitus, Type 1 - blood | Diabetes Mellitus, Type 1 - diet therapy | Blood Glucose - metabolism | Bone Remodeling | Type 2 diabetes | Evaluation | Obesity | Adolescence | Reducing diets | Fatty acids | Density | Hyperglycemia | Diet therapy | Type 1 diabetes | Analysis | Physiological aspects | Bones | Adipose tissues | Adipose tissue | Body fat | Body weight | Chains | Bone (trabecular) | Low fat | Body composition | High fat diet | Biomedical materials | Body composition (biology) | Rodents | Bone marrow | Biocompatibility | Longitudinal studies | Bone loss | Bone (cortical) | Adolescents | Polydipsia | Osteopathy | Bone composition | Diabetes mellitus | Bone turnover | Insulin | Patients | Fractures | Bone mass | Diet | Correlation analysis | Low fat diet | Mice | Bone | Diabetes | Metabolic disorders | Index Medicus | Bone Marrow Adipose Tissue | High Fat Diet | Type 1 Diabetes Mellitus | Bone Mass
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 03/2018, Volume 314, Issue 3, pp. F439 - F444
About one-third of patients with type 1 diabetes develops kidney disease. The mechanism is largely unknown, but intrarenal hypoxia has been proposed as a... 
Oxygen consumption | Endothelin | Diabetes | Oxygen tension | Kidney | NADPH OXIDASE | HOMEOSTASIS | PHYSIOLOGY | GLOMERULAR HYPERFILTRATION | HYPOXIA | NEPHROPATHY | endothelin | kidney | ET(A) | ET(B) | INHIBITION | oxygen consumption | oxygen tension | IN-VIVO | UROLOGY & NEPHROLOGY | diabetes | VASOCONSTRICTION | Diabetes Mellitus, Experimental - drug therapy | Streptozocin | Diabetic Nephropathies - drug therapy | Male | Diabetic Nephropathies - blood | Diabetes Mellitus, Experimental - blood | Receptor, Endothelin B - agonists | Kidney - metabolism | Diabetic Nephropathies - physiopathology | Diabetes Mellitus, Type 1 - chemically induced | Renal Circulation - drug effects | Diabetes Mellitus, Experimental - chemically induced | Kidney - physiopathology | Diabetes Mellitus, Experimental - physiopathology | Kidney - drug effects | Diabetes Mellitus, Type 1 - physiopathology | Diabetic Nephropathies - chemically induced | Viper Venoms - pharmacology | Rats, Sprague-Dawley | Oxygen - blood | Diabetes Mellitus, Type 1 - drug therapy | Animals | Signal Transduction - drug effects | Diabetes Mellitus, Type 1 - blood | Hemodynamics - drug effects | Receptor, Endothelin B - metabolism | Physiological aspects | Cell receptors | Kidneys | Type 1 diabetes | Oxygen | Renal function | Renal artery | Diabetes mellitus | Vasoconstriction | Homeostasis | Diabetes mellitus (insulin dependent) | Vasodilation | Blood flow | Nephropathy | Sodium | Hypoxia | Oxygenation | Kidney transplantation | Index Medicus | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Fysiologi | Physiology
Journal Article
Cardiovascular Pathology, ISSN 1054-8807, 2015, Volume 24, Issue 6, pp. 375 - 381
Abstract Introduction Hyperglycemia-induced reactive oxygen species (ROS) generation contributes to the development of diabetic cardiomyopathy. However, little... 
Pathology | Diabetic cardiomyopathy | Oxidative stress | Nuclear factor-erythroid 2-related factor 2 | MicroRNA-144 | Streptozotocin | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTECTION | KINASE | RATS | RECEPTOR | DEATH | CARDIOMYOCYTES | PATHOLOGY | TRANSLATION | NRF2 | ENDOTHELIAL-CELLS | EXPRESSION | Diabetic Cardiomyopathies - metabolism | Reactive Oxygen Species - metabolism | Oxidative Stress | Ventricular Function, Left | Streptozocin | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Male | MicroRNAs - metabolism | Diabetes Mellitus, Type 1 - therapy | Diabetes Mellitus, Experimental - therapy | Transfection | Diabetes Mellitus, Type 1 - chemically induced | Diabetic Cardiomyopathies - prevention & control | NF-E2-Related Factor 2 - genetics | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Cardiomyopathies - genetics | Myocytes, Cardiac - pathology | Animals | NF-E2-Related Factor 2 - metabolism | Diabetes Mellitus, Experimental - pathology | Glucose - metabolism | Myocytes, Cardiac - metabolism | Diabetic Cardiomyopathies - pathology | MicroRNAs - genetics | Oligonucleotides - administration & dosage | Diabetic Cardiomyopathies - chemically induced | Apoptosis | MicroRNA | Index Medicus
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 03/2012, Volume 23, Issue 3, pp. 458 - 469
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 12/2014, Volume 281, Issue 2, pp. 211 - 220
Epidemiological studies have reported associations between particulate matter (PM) and cardiovascular effects, and diabetes mellitus (DM) patients might be... 
Rats | Air pollution | Inflammation | Particulate matter | Diabetes mellitus | ENDOTHELIAL DYSFUNCTION | LONG-TERM EXPOSURE | SUBCLINICAL ATHEROSCLEROSIS | SYSTEMIC INFLAMMATION | AIR-POLLUTION EXPOSURE | INSULIN-RESISTANCE | CARDIOVASCULAR-DISEASE | FINE PARTICLES | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | TOXICOLOGY | ASSOCIATION | Inflammation - chemically induced | Particulate Matter - toxicity | Myocarditis - chemically induced | Streptozocin | Glycated Hemoglobin A - metabolism | Homeostasis | Male | Diabetes Complications - diagnosis | Diabetic Nephropathies - blood | Inflammation - blood | Diabetes Mellitus, Experimental - blood | Time Factors | Interleukin-6 - blood | Diabetes Mellitus, Type 1 - chemically induced | Inhalation Exposure - adverse effects | Diabetes Mellitus, Experimental - chemically induced | Diabetic Cardiomyopathies - blood | Inflammation - diagnosis | Diabetic Angiopathies - chemically induced | Diabetes Mellitus, Experimental - diagnosis | Risk Factors | Diabetic Nephropathies - chemically induced | Inflammation Mediators - blood | Toxicity Tests, Subchronic | Biomarkers - blood | Diabetic Angiopathies - blood | Myocarditis - blood | Diabetes Complications - blood | Rats, Sprague-Dawley | Blood Glucose - drug effects | Particle Size | Diabetes Complications - chemically induced | Animals | Diabetes Mellitus, Type 1 - blood | Fibrinogen - metabolism | Blood Glucose - metabolism | Diabetic Cardiomyopathies - chemically induced | Glucose metabolism | Type 1 diabetes | Fibrinogen | Hemoglobin | Glucose | Health aspects | Dextrose | Index Medicus | HOMEOSTASIS | INJURIES | RATS | AIR | COMPARATIVE EVALUATIONS | 60 APPLIED LIFE SCIENCES | AIR POLLUTION | DAMAGE | KIDNEYS | STREPTOZOCIN | GLOBAL ASPECTS | HYPERGLYCEMIA | BLOOD VESSELS | GLUCOSE | INFLAMMATION | DIABETES MELLITUS | FIBRINOGEN