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Diabetes, obesity & metabolism, ISSN 1462-8902, 1999
Journal
Journal Article
1999
eJournal
Diabetes (New York, N.Y.), ISSN 1939-327X, 2017, Volume 66, Issue 2, pp. 241 - 255
Journal Article
The New England journal of medicine, ISSN 1533-4406, 2007, Volume 356, Issue 15, pp. 1517 - 1526
.... Type 2 diabetes mellitus occurs when beta-cell function fails to compensate for insulin resistance... 
INSULIN | MEDICINE, GENERAL & INTERNAL | OBESITY | ISLETS | GLUCOSE | BETA-CELL APOPTOSIS | MUSCLE | EXPRESSION | RECEPTOR ANTAGONIST | Recombinant Proteins - therapeutic use | Insulin-Secreting Cells - secretion | Glucose Transporter Type 4 - metabolism | Glycated Hemoglobin A - metabolism | Humans | Middle Aged | Male | Diabetes Mellitus, Type 2 - metabolism | RNA, Messenger - metabolism | Heat-Shock Proteins - genetics | Insulin Resistance - physiology | Interleukin-6 - blood | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Female | Interleukin 1 Receptor Antagonist Protein - pharmacology | Body Mass Index | Glucose Tolerance Test | Double-Blind Method | Glucose Transporter Type 4 - genetics | Heat-Shock Proteins - metabolism | C-Reactive Protein - secretion | Recombinant Proteins - pharmacology | Transcription Factors - genetics | Gene Expression Regulation - drug effects | Transcription Factors - metabolism | Interleukin 1 Receptor Antagonist Protein - therapeutic use | Insulin-Secreting Cells - drug effects | Interleukin 1 Receptor Antagonist Protein - adverse effects | Blood Glucose - metabolism | Diabetes Mellitus, Type 2 - drug therapy | Receptors, Interleukin-1 - antagonists & inhibitors | Type 2 diabetes | Interleukin-1 | Research | Drug therapy | Pancreas | Diabetes | Apoptosis | Clinical Medicine | Endokrinologi och diabetes | Medical and Health Sciences | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
Journal Article
Kidney International, ISSN 0085-2538, 08/2011, Volume 80, Issue 4, pp. 358 - 368
.... Amounts of miR-200b/c were increased in glomeruli from type 1 (streptozotocin) and type 2 (db/db) diabetic mice, and in mouse mesangial cells treated with TGF... 
TGF-β | fibrosis | diabetic nephropathy | gene expression | cell signaling | Up-Regulation | Diabetes Mellitus, Type 2 - genetics | Homeodomain Proteins - metabolism | Transforming Growth Factor beta1 - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Homeostasis | MicroRNAs - metabolism | Diabetes Mellitus, Type 2 - metabolism | Transfection | Time Factors | Kruppel-Like Transcription Factors - metabolism | Upstream Stimulatory Factors - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Diabetes Mellitus, Experimental - chemically induced | 3' Untranslated Regions | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Collagen Type IV - metabolism | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Diabetic Nephropathies - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Transforming Growth Factor beta1 - genetics | Mesangial Cells - metabolism | Oligonucleotides - metabolism | Animals | Fibrosis | Diabetes Mellitus, Experimental - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice | Diabetes Mellitus, Type 2 - pathology | Mutation | Zinc Finger E-box-Binding Homeobox 1
Journal Article
Journal of Diabetes and Its Complications, ISSN 1056-8727, 2016, Volume 30, Issue 4, pp. 738 - 745
Abstract Background Vascular complications are the leading cause of morbidity and mortality among patients with type 1 and type 2 diabetes mellitus... 
Endocrinology & Metabolism | vascular complications | hypercoagulability | inflammation | endothelial dysfunction | Diabetes mellitus | FACTOR-VIII | MARKERS | MICROVASCULAR COMPLICATIONS | VON-WILLEBRAND-FACTOR | URINARY ALBUMIN EXCRETION | VONWILLEBRAND-FACTOR | ENDOCRINOLOGY & METABOLISM | CARDIOVASCULAR-DISEASE | ASSOCIATION | D-DIMER | Vasculitis - metabolism | Oxidative Stress | Vasculitis - physiopathology | Humans | Diabetes Mellitus, Type 1 - metabolism | Diabetes Mellitus, Type 1 - complications | Diabetes Mellitus, Type 2 - metabolism | Thrombophilia - complications | Vasculitis - complications | Vasculitis - immunology | Thrombophilia - immunology | Diabetic Angiopathies - physiopathology | Diabetes Mellitus, Type 2 - immunology | Diabetic Angiopathies - complications | Diabetes Mellitus, Type 1 - immunology | Diabetes Mellitus, Type 2 - complications | Endothelium, Vascular - immunology | Diabetic Angiopathies - metabolism | Thrombophilia - physiopathology | Diabetes Mellitus, Type 1 - physiopathology | Endothelium, Vascular - physiopathology | Biomarkers - blood | Animals | Diabetes Mellitus, Type 2 - physiopathology | Endothelium, Vascular - metabolism | Models, Biological | Diabetic Angiopathies - immunology | Thrombophilia - metabolism | Type 2 diabetes | Oxidative stress | Complications and side effects | Inflammation | Index Medicus
Journal Article
Physiological reviews, ISSN 1522-1210, 2013, Volume 93, Issue 1, pp. 137 - 188
.... These complications occur in the majority of individuals with both type 1 and type 2 diabetes. Among the most prevalent microvascular complications are kidney disease, blindness, and amputations, with current therapies only slowing disease progression... 
GLOMERULAR-FILTRATION-RATE | PROTEIN-KINASE-C | TUMOR-NECROSIS-FACTOR | PHYSIOLOGY | INTERCELLULAR-ADHESION MOLECULE-1 | ENDOPLASMIC-RETICULUM-STRESS | NITRIC-OXIDE SYNTHASE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | FACTOR-KAPPA-B | GLYCATION END-PRODUCTS | ENDOTHELIAL GROWTH-FACTOR | Prognosis | Diabetic Neuropathies - etiology | Oxidative Stress | Diabetic Nephropathies - etiology | Diabetes Mellitus, Type 2 - genetics | Humans | Diabetes Mellitus, Type 1 - metabolism | Diabetic Angiopathies - etiology | Diabetes Complications - therapy | Diabetes Mellitus, Type 1 - complications | Diabetes Mellitus, Type 2 - metabolism | Diabetes Mellitus, Type 1 - therapy | Diabetes Mellitus, Type 2 - therapy | Inflammation Mediators - metabolism | Diabetes Complications - genetics | Diabetes Mellitus, Type 2 - complications | Disease Models, Animal | Signal Transduction | Diabetes Complications - metabolism | Risk Factors | Gene Expression Regulation | Diabetes Mellitus, Type 1 - genetics | Diabetes Complications - etiology | Animals | Glycation End Products, Advanced - metabolism | Blood Glucose - metabolism | Kidneys | Blood vessels | Physiological aspects | Causes of | Diabetes | Kidney diseases | Cardiovascular diseases | Risk factors
Journal Article