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Atherosclerosis, ISSN 0021-9150, 2015, Volume 243, Issue 2, pp. 438 - 448
Abstract Objective The circulating irisin increases energy expenditure and improves insulin resistance in mice and humans. The improvement of insulin... 
Cardiovascular | Irisin | AMPK | Diabetes mellitus | Atherosclerosis | Endothelium | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | CHOLESTEROL | PHOSPHORYLATION | NITRIC-OXIDE SYNTHASE | CIRCULATING IRISIN | KINASE-C | CORONARY-ARTERY | PERIPHERAL VASCULAR DISEASE | DYSFUNCTION | CELL | ASSOCIATION | Diabetes Mellitus, Experimental - drug therapy | Apolipoproteins E - deficiency | Human Umbilical Vein Endothelial Cells - metabolism | Apoptosis - drug effects | Atherosclerosis - genetics | Diabetes Mellitus, Experimental - genetics | Endothelium, Vascular - drug effects | Male | Diabetic Angiopathies - physiopathology | RNA Interference | T-Lymphocytes - metabolism | T-Lymphocytes - drug effects | Diabetic Angiopathies - prevention & control | Proto-Oncogene Proteins c-akt - metabolism | Cytokines - genetics | Cytoprotection | Diabetes Mellitus, Experimental - physiopathology | Atherosclerosis - pathology | Fibronectins - pharmacology | Genetic Predisposition to Disease | Human Umbilical Vein Endothelial Cells - drug effects | Atherosclerosis - physiopathology | Endothelial Cells - metabolism | Plaque, Atherosclerotic | Nitric Oxide Synthase Type III - genetics | Atherosclerosis - metabolism | Mice, Knockout | Nitric Oxide Synthase Type III - antagonists & inhibitors | Macrophages - metabolism | Phenotype | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Endothelium, Vascular - pathology | Human Umbilical Vein Endothelial Cells - pathology | Oxidative Stress - drug effects | Vasodilation - drug effects | Endothelial Cells - pathology | AMP-Activated Protein Kinases - genetics | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Diabetic Angiopathies - pathology | Transfection | Nitric Oxide Synthase Type III - metabolism | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Diabetic Angiopathies - metabolism | Cytokines - metabolism | AMP-Activated Protein Kinases - antagonists & inhibitors | Mice, Inbred C57BL | Cells, Cultured | Endothelium, Vascular - physiopathology | Animals | Apolipoproteins E - genetics | Diabetes Mellitus, Experimental - pathology | Macrophages - drug effects | Protein Kinase Inhibitors - pharmacology | Atherosclerosis - prevention & control | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Endothelial Cells - drug effects | Insulin resistance | Diabetes | Apolipoproteins
Journal Article
Diabetes care, ISSN 1935-5548, 2009, Volume 32, Issue suppl_2, pp. S314 - S321
Journal Article
Vascular Pharmacology, ISSN 1537-1891, 2016, Volume 82, pp. 30 - 40
Abstract Vascular calcification (VC), a disorder that causes blood vessel hardening and dysfunction, is a significant risk factor for type-2 diabetes mellitus... 
Cardiovascular | Vascular calcification | RANKL | TRAIL | Type-2 diabetes mellitus | OPG | SERUM OSTEOPROTEGERIN | AORTIC CALCIFICATION | PLASMA OSTEOPROTEGERIN | APOPTOSIS-INDUCING LIGAND | CORONARY-ARTERY CALCIFICATION | RECEPTOR ACTIVATOR | OSTEOCLASTIC DIFFERENTIATION | PHARMACOLOGY & PHARMACY | SMOOTH-MUSCLE-CELLS | CHRONIC KIDNEY-DISEASE | KAPPA-B LIGAND | RANK Ligand - metabolism | Humans | Diabetic Angiopathies - etiology | Arteries - metabolism | Atherosclerosis - etiology | Vascular Calcification - metabolism | Diabetic Angiopathies - pathology | Vascular Calcification - etiology | Osteoprotegerin - therapeutic use | Diabetes Mellitus, Type 2 - complications | Diabetic Angiopathies - metabolism | Atherosclerosis - pathology | Osteoclasts - pathology | Atherosclerosis - drug therapy | Signal Transduction | Osteogenesis - drug effects | Arteries - drug effects | Arteries - pathology | Diabetic Angiopathies - drug therapy | Vascular Calcification - drug therapy | Plaque, Atherosclerotic | TNF-Related Apoptosis-Inducing Ligand - metabolism | Vascular Calcification - pathology | Osteoclasts - metabolism | TNF-Related Apoptosis-Inducing Ligand - therapeutic use | Atherosclerosis - metabolism | Cardiovascular Agents - therapeutic use | Osteoblasts - pathology | Animals | RANK Ligand - antagonists & inhibitors | Osteoprotegerin - metabolism | Osteoblasts - metabolism | Type 2 diabetes | Analysis | Atherosclerosis | Venture capital | Physiological aspects | Calcification | Cardiovascular diseases | Health aspects
Journal Article
Peritoneal dialysis international, ISSN 0896-8608, 1989, Volume 9, Issue 1, pp. 41 - 45
Journal Article
The American journal of cardiology, ISSN 0002-9149, 2017, Volume 120, Issue 1, pp. S37 - S47
Epidemiologic and clinical data from the last 2 decades have shown that the prevalence of heart failure in diabetes is very high, and the prognosis for... 
Cardiovascular | ALL-CAUSE MORTALITY | 2013 ACCF/AHA GUIDELINE | DIPEPTIDYL PEPTIDASE-4 INHIBITORS | CARDIAC & CARDIOVASCULAR SYSTEMS | MYOCARDIAL-INFARCTION | CARDIOVASCULAR OUTCOMES | CARDIAC METABOLISM | ASSOCIATION TASK-FORCE | GLUCOSE ABNORMALITIES | INDEPENDENT RISK-FACTOR | ATRIAL-FIBRILLATION | Hypoglycemic Agents - therapeutic use | Prevalence | Prognosis | Comorbidity | Humans | Heart Failure - physiopathology | Diabetic Angiopathies - drug therapy | Heart Failure - pathology | Diabetes Mellitus, Type 2 - epidemiology | Heart Failure - drug therapy | Incidence | Diabetic Angiopathies - physiopathology | Diabetes Mellitus, Type 2 - physiopathology | Diabetic Angiopathies - pathology | Diabetic Angiopathies - epidemiology | Glucosides - therapeutic use | Diabetes Mellitus, Type 2 - pathology | Diabetes Mellitus, Type 2 - drug therapy | Benzhydryl Compounds - therapeutic use | Heart Failure - epidemiology | Heart failure | Type 2 diabetes | Periodical publishing | Diabetics | Cardiac patients | Pharmaceutical industry | Coronary heart disease | Risk factors | Glucose | Dextrose | Heart | Heart attacks | Cardiomyopathy | Cardiovascular disease | Metabolic syndrome | Epidemiology | Population | Heart diseases | Mortality | Diabetes mellitus | Coronary artery | Health risks | Hospitalization | Risk analysis | Coronary artery disease | Patients | Sodium | Medical prognosis | Coronary vessels | Diabetes | Acute coronary syndromes | Cardiovascular diseases | Health risk assessment
Journal Article
Journal of vascular surgery, ISSN 0741-5214, 2013, Volume 58, Issue 3, pp. 766 - 775.e12
Objective The present study is designed to understand the contribution of peripheral vascular disease and peripheral neuropathy to the wound-healing impairment... 
Surgery | CONNECTION | SURGERY | CELLS | REPAIR | INHIBITION | NEUROPATHY | INFLAMMATION | EAR | ANIMAL-MODELS | PERIPHERAL VASCULAR DISEASE | SUBSTANCE-P | RECEPTORS | Up-Regulation | Diabetic Neuropathies - etiology | Skin - metabolism | Diabetes Mellitus, Experimental - genetics | Diabetic Angiopathies - etiology | Ischemia - genetics | Skin Ulcer - etiology | Skin Ulcer - immunology | Wound Healing | Skin Ulcer - metabolism | Diabetic Neuropathies - genetics | Diabetic Angiopathies - pathology | Ischemia - immunology | Time Factors | Inflammation Mediators - metabolism | Diabetic Neuropathies - immunology | Diabetes Mellitus, Experimental - complications | Neuropeptides - genetics | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Cytokines - genetics | Ischemia - pathology | Macrophages - immunology | Skin - pathology | Skin - immunology | Diabetic Angiopathies - metabolism | Rabbits | Cytokines - metabolism | Down-Regulation | Skin Ulcer - genetics | Ischemia - metabolism | Neuropeptides - metabolism | Diabetes Mellitus, Experimental - immunology | Skin Ulcer - pathology | Animals | Diabetic Angiopathies - immunology | Diabetic Neuropathies - pathology | Diabetic Neuropathies - metabolism | Ischemia - etiology | Neuropeptide Y | Blood circulation disorders | Wound healing | Diabetes | Cytokines | Analysis
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2008, Volume 57, Issue 9, pp. 2461 - 2469
Journal Article
Atherosclerosis, ISSN 0021-9150, 2012, Volume 221, Issue 2, pp. 387 - 396
Highlights ► STZ–CML–HFD for 4 months could induce atherosclerotic calcification of diabetes. ► We constructed a new calcification model in a high-lipid,... 
Cardiovascular | Advanced glycation end-products | Phenotype transformation | OSTEOGENIC DIFFERENTIATION | RECEPTOR | MECHANISMS | RAGE | CORONARY-ARTERY CALCIFICATION | Atherosclerosis | DISEASE | Vascular calcification | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | INTRAVASCULAR ULTRASOUND | APOPTOTIC BODIES | Diabetes | Vascular smooth muscle cells | Apoptosis | Immunohistochemistry | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Alkaline Phosphatase - metabolism | Diabetic Angiopathies - etiology | Male | Vascular Calcification - metabolism | Time Factors | Vascular Calcification - etiology | Diet, High-Fat | Cell Differentiation | Diabetes Mellitus, Experimental - complications | Lipoproteins, LDL - metabolism | Lysine - metabolism | Receptor for Advanced Glycation End Products | Lysine - analogs & derivatives | Atherosclerosis - pathology | Core Binding Factor Alpha 2 Subunit - metabolism | Macrophages - pathology | Lysine - administration & dosage | Rats | Vascular Calcification - pathology | Atherosclerosis - metabolism | Blotting, Western | Disease Progression | Mice, Knockout | Macrophages - metabolism | Staining and Labeling | Glucose - metabolism | Mice | Injections, Intravenous | Muscle, Smooth, Vascular - metabolism | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | Atherosclerosis - etiology | Diabetic Angiopathies - pathology | Bone Morphogenetic Protein 2 - metabolism | Diabetes Mellitus, Experimental - metabolism | Diabetic Angiopathies - genetics | Myocytes, Smooth Muscle - metabolism | Cell Line | Diabetic Angiopathies - metabolism | Mice, Inbred C57BL | Muscle, Smooth, Vascular - pathology | Animals | Apolipoproteins E - genetics | Osteogenesis | Receptors, Immunologic - metabolism
Journal Article
Diabetes (New York, N.Y.), ISSN 1939-327X, 2015, Volume 64, Issue 1, pp. 266 - 278
Journal Article
Circulation research, ISSN 0009-7330, 02/2013, Volume 112, Issue 3, pp. 510 - 522
RATIONALE:The impact of diabetes mellitus on bone marrow (BM) structure is incompletely understood. OBJECTIVE:Investigate the effect of type-2 diabetes... 
microangiopathy | stem cells | macroangiopathy | diabetes mellitus type 2 | bone marrow | CARDIAC & CARDIOVASCULAR SYSTEMS | TRANSCRIPTION | MICRORNAS | TRANSPLANTATION | HEART | ENDOTHELIAL PROGENITOR CELLS | DISEASE | PERIPHERAL VASCULAR DISEASE | COMPLICATIONS | NEOVASCULARIZATION | DIFFERENTIATION | HEMATOLOGY | EXPRESSION | Immunohistochemistry | Humans | Middle Aged | Microvessels - metabolism | Hematopoietic Stem Cells - pathology | Microvessels - pathology | Male | MicroRNAs - metabolism | Ischemia - genetics | Adipose Tissue - metabolism | Cyclin-Dependent Kinase Inhibitor p27 - metabolism | Forkhead Transcription Factors - metabolism | Bone Marrow Cells - immunology | Aged, 80 and over | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Ischemia - pathology | Biomarkers - metabolism | Peripheral Arterial Disease - metabolism | Stem Cell Niche | Signal Transduction | Endothelial Cells - metabolism | Adipose Tissue - pathology | Bone Marrow Cells - pathology | Bone Marrow Examination | Cell Lineage | MicroRNAs - genetics | Diabetes Mellitus, Type 2 - pathology | Endothelial Cells - pathology | Forkhead Box Protein O3 | Antigens, CD34 - metabolism | Peripheral Arterial Disease - pathology | Diabetes Mellitus, Type 2 - genetics | Diabetes Mellitus, Type 2 - metabolism | Microvessels - immunology | Case-Control Studies | Hematopoietic Stem Cells - immunology | Diabetic Angiopathies - pathology | Flow Cytometry | Transfection | Peripheral Arterial Disease - genetics | Adult | Female | Diabetic Angiopathies - genetics | Diabetic Angiopathies - metabolism | Cells, Cultured | Gene Expression Regulation | Ischemia - metabolism | Hematopoietic Stem Cells - metabolism | Forkhead Transcription Factors - genetics | Aged | Bone Marrow Cells - metabolism | Apoptosis
Journal Article
Atherosclerosis, ISSN 0021-9150, 2016, Volume 247, pp. 207 - 217
Journal Article