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1. Full Text Effect of Finerenone on Albuminuria in Patients With Diabetic Nephropathy: A Randomized Clinical Trial
by Bakris, George L and Agarwal, Rajiv and Chan, Juliana C and Cooper, Mark E and Gansevoort, Ron T and Haller, Hermann and Remuzzi, Giuseppe and Rossing, Peter and Schmieder, Roland E and Nowack, Christina and Kolkhof, Peter and Joseph, Amer and Pieper, Alexander and Kimmeskamp-Kirschbaum, Nina and Ruilope, Luis M and Mineralocorticoid Receptor and Mineralocorticoid Receptor Antagonist Tolerability Study–Diabetic Nephropathy (ARTS-DN) Study Group
JAMA : the journal of the American Medical Association, ISSN 0098-7484, 09/2015, Volume 314, Issue 9, pp. 884 - 894
Blood Pressure | Creatinine | Glomerular Filtration Rate | Diabetic Nephropathies | Double-Blind Method | Drug Administration Schedule | Naphthyridines | Administration, Oral | Angiotensin Receptor Antagonists | Humans | Middle Aged | Male | Albuminuria | Incidence | Dose-Response Relationship, Drug | Hyperkalemia | Analysis of Variance | Angiotensin-Converting Enzyme Inhibitors | Least-Squares Analysis | Female | Aged | Diabetes Mellitus, Type 2 | Mineralocorticoid Receptor Antagonists | Potassium | Medicine, General & Internal | Life Sciences & Biomedicine | General & Internal Medicine | Science & Technology | Naphthyridines - adverse effects | Diabetic Nephropathies - drug therapy | Diabetes Mellitus, Type 2 - ethnology | Glomerular Filtration Rate - drug effects | Diabetic Nephropathies - physiopathology | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Hyperkalemia - epidemiology | Blood Pressure - drug effects | Diabetes Mellitus, Type 2 - complications | Diabetic Nephropathies - urine | Diabetes Mellitus, Type 2 - urine | Diabetic Nephropathies - ethnology | Creatinine - urine | Mineralocorticoid Receptor Antagonists - therapeutic use | Albuminuria - drug therapy | Hyperkalemia - chemically induced | Potassium - blood | Angiotensin Receptor Antagonists - therapeutic use | Naphthyridines - administration & dosage | Proteins | Clinical trials | Side effects | Diabetes | Antagonist drugs | ACE inhibitors | Index Medicus | Abridged Index Medicus
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2. Full Text Inhibition of high glucose-induced inflammation and fibrosis by a novel curcumin derivative prevents renal and heart injury in diabetic mice
by Chen, Hongjin and Yang, Xi and Lu, Kongqin and Lu, Chun and Zhao, Yunjie and Zheng, Suqing and Li, Jieli and Huang, Zhangjian and Huang, Yi and Zhang, Yali and Liang, Guang
Toxicology letters, ISSN 0378-4274, 08/2017, Volume 278, pp. 48 - 58
Diabetic cardiomyopathy | Diabetic nephropathy | Curcumin | Anti-inflammation | Fibrosis | Life Sciences & Biomedicine | Toxicology | Science & Technology | Curcumin - analogs & derivatives | Diabetes Mellitus, Experimental - drug therapy | Diabetic Cardiomyopathies - metabolism | Kidney - pathology | Streptozocin | Male | Kidney - metabolism | Time Factors | Inflammation Mediators - metabolism | Diabetic Cardiomyopathies - prevention & control | Diabetes Mellitus, Experimental - chemically induced | p38 Mitogen-Activated Protein Kinases - metabolism | Diabetes Mellitus, Experimental - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Diabetic Nephropathies - prevention & control | Cell Line | Diabetic Nephropathies - pathology | Kidney - drug effects | Cytokines - metabolism | Anti-Inflammatory Agents - pharmacology | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Curcumin - pharmacology | Diabetic Nephropathies - chemically induced | Myocytes, Cardiac - pathology | Animals | Myocytes, Cardiac - drug effects | Signal Transduction - drug effects | Diabetes Mellitus, Experimental - pathology | Myocytes, Cardiac - metabolism | Diabetic Cardiomyopathies - pathology | Blood Glucose - metabolism | Diabetic Cardiomyopathies - chemically induced | Prevention | Hyperglycemia | Analysis | Diabetic nephropathies | Inflammation | Glucose | Health aspects | Dextrose | Index Medicus
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3. Full Text Mitochondrial carnitine palmitoyltransferase 2 is involved in Nε-(carboxymethyl)-lysine-mediated diabetic nephropathy
by Lee, Jangho and Hyon, Ju-Yong and Min, Jin Young and Huh, Yang Hoon and Kim, Hyo Jung and Lee, Hayoung and Yun, Sung Ho and Choi, Chi-Won and Jeong Ha, Su and Park, Joon and Chung, Young-Ho and Jeong, Hye Gwang and Ha, Sang Keun and Jung, Sung Keun and Kim, YoonSook and Han, Eun Hee
Pharmacological research, ISSN 1043-6618, 02/2020, Volume 152, pp. 104600 - 104600
Diabetic nephropathy (DN) | End-stage renal disease | Advanced glycation end products | Mitochondrial dysfunction | Nε-(carboxymethyl)-lysine | Carnitine palmitoyltransferase 2 | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Glycated Hemoglobin A - analysis | Mitochondria - enzymology | Carnitine O-Palmitoyltransferase - genetics | Kidney - pathology | Humans | Diabetes Mellitus, Experimental - genetics | Diabetes Mellitus, Type 1 - metabolism | Male | Kidney - metabolism | Diabetes Mellitus, Type 1 - chemically induced | Membrane Potential, Mitochondrial | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Cell Line | Lysine - analogs & derivatives | Diabetic Nephropathies - pathology | Blood Glucose - analysis | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetes Mellitus, Type 1 - pathology | Diabetic Nephropathies - chemically induced | Diabetes Mellitus, Type 1 - genetics | Diabetic Nephropathies - genetics | Animals | Diabetes Mellitus, Experimental - pathology | Mitochondria - physiology | Index Medicus
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4. Full Text MicroRNA-27a promotes podocyte injury via PPARγ-mediated β-catenin activation in diabetic nephropathy
by Zhou, Zhanmei and Wan, Jiao and Hou, Xiaoyan and Geng, Jian and Li, Xiao and Bai, Xiaoyan
Cell death & disease, ISSN 2041-4889, 2017, Volume 8, Issue 3, pp. e2658 - e2658
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Diabetic Nephropathies - pathology | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Humans | Transcriptional Activation | Diabetes Mellitus, Experimental - genetics | Diabetic Nephropathies - chemically induced | Gene Expression Regulation | Rats | Diabetic Nephropathies - genetics | Male | Podocytes - pathology | Cell Movement - genetics | beta Catenin - genetics | Actins - genetics | Animals | Wnt Signaling Pathway - genetics | Glucose - toxicity | Diabetes Mellitus, Experimental - chemically induced | MicroRNAs - genetics | Diabetes Mellitus, Experimental - physiopathology | PPAR gamma - genetics | Index Medicus | Original
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5. Full Text CXCL8 antagonist improves diabetic nephropathy in male mice with diabetes and attenuates high glucose-induced mesangial injury
by Cui, Siyuan and Zhu, Yujie and Du, Jianling and Khan, Muhammad Noman and Wang, Bing and Wei, Jing and Cheng, Jya-Wei and Gordon, John R and Mu, Yutian and Li, Fang
Endocrinology (Philadelphia), ISSN 0013-7227, 06/2017, Volume 158, Issue 6, pp. 1671 - 1684
Life Sciences & Biomedicine | Endocrinology & Metabolism | Science & Technology | Diabetes Mellitus, Experimental - drug therapy | Diabetic Nephropathies - pathology | Streptozocin | Mice, Inbred C57BL | Diabetic Nephropathies - chemically induced | Male | Peptide Fragments - pharmacology | Interleukin-8 - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | Mesangial Cells - drug effects | Dose-Response Relationship, Drug | Animals | Mesangial Cells - pathology | Glucose - toxicity | Interleukin-8 - pharmacology | Diabetes Mellitus, Experimental - complications | Mice | Diabetic Nephropathies - prevention & control | Glucose | Inflammatory diseases | High fat diet | Receptors | Rodents | Janus kinase 2 | Mesangial cells | Extracellular matrix | Attenuation | Inhibition | CXC chemokine receptors | Urine | Creatinine | Kidneys | CXCR2 protein | Diabetes mellitus | Stat3 protein | Extracellular signal-regulated kinase | Inflammation | Chemokine receptors | White blood cells | Urea | Signaling | Cell injury | Nephropathy | Fibrosis | Infiltration | Diabetes | Kidney transplantation | Index Medicus | Abridged Index Medicus
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6. Full Text High glucose-induced O-GlcNAcylated Carbohydrate response element-binding protein (ChREBP) mediates mesangial cell lipogenesis and fibrosis the possible role in the development of diabetic nephropathy
by Park, Min-Jung and Kim, Dong-Il and Lim, Seul-Ki and Choi, Joo-Hee and Han, Ho-Jae and Yoon, Kyung-Chul and Park, Soo-Hyun
The Journal of biological chemistry, ISSN 0021-9258, 1768, Volume 289, Issue 19, pp. 13519 - 13530
Gene Expression Regulation, Enzymologic - drug effects | Triglycerides - biosynthesis | Humans | Diabetes Mellitus, Experimental - genetics | Fibrosis - metabolism | Cholesterol - genetics | HEK293 Cells | Acetylglucosamine - analogs & derivatives | Diabetes Mellitus, Experimental - metabolism | Diabetic Nephropathies - pathology | Fibrosis - genetics | Fibrosis - chemically induced | Sweetening Agents - pharmacology | Diabetic Nephropathies - metabolism | Diabetic Nephropathies - chemically induced | Rats | Acetylglucosamine - pharmacology | Diabetic Nephropathies - genetics | Glucose - pharmacology | Mesangial Cells - metabolism | beta-N-Acetylhexosaminidases - biosynthesis | Phenylcarbamates - pharmacology | Animals | Gene Expression Regulation, Enzymologic - genetics | Lipogenesis - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Mesangial Cells - pathology | Diabetes Mellitus, Experimental - pathology | Oximes - pharmacology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Fibrosis - pathology | Triglycerides - genetics | Cholesterol - biosynthesis | Index Medicus | Molecular Bases of Disease | O-GlcNAcylation | Kidney Metabolism | Diabetes | Lipogenesis | Fibrosis
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7. Full Text Associations between persistent organic pollutants, type 2 diabetes, diabetic nephropathy and mortality
by Grice, Brian A and Nelson, Robert G and Williams, Desmond E and Knowler, William C and Mason, Clinton and Hanson, Robert L and Bullard, Kai McKeever and Pavkov, Meda E
Occupational and environmental medicine (London, England), ISSN 1351-0711, 07/2017, Volume 74, Issue 7, pp. 521 - 527
Environment | PCBs | Renal | Diabetes mellitus | Pesticides | Life Sciences & Biomedicine | Public, Environmental & Occupational Health | Science & Technology | Kidney Failure, Chronic - mortality | Environmental Pollutants - adverse effects | Humans | Male | Case-Control Studies | Cause of Death | Diabetes Mellitus, Type 2 - epidemiology | Arizona - epidemiology | Young Adult | Fungicides, Industrial - adverse effects | Kidney Failure, Chronic - chemically induced | Adult | Female | Hexachlorobenzene - adverse effects | Diabetic Nephropathies - chemically induced | Organic Chemicals - adverse effects | Logistic Models | Diabetic Nephropathies - mortality | Environmental Exposure - adverse effects | Diabetes Mellitus, Type 2 - blood | Polychlorinated Biphenyls - adverse effects | Adolescent | Pesticides - adverse effects | Diabetes Mellitus, Type 2 - chemically induced | Indians, North American | Type 2 diabetes | Logistic regression | Care and treatment | Usage | Chronic kidney failure | Pollutants | Health aspects | End-stage renal disease | Laboratories | Sex | Risk | Diabetic neuropathy | Glucose | PCB compounds | Body mass index | Herbicides | Demographics | Population | Mathematical models | Persistent organic pollutants | Hexachlorobenzene | Age | Task forces | Adjustment | Chlorine | Mortality | Principal components analysis | Regression analysis | Disease control | Studies | Nephropathy | Body mass | Polychlorinated biphenyls--PCB | Death | Insulin resistance | Scientific imaging | Models | Kidney diseases | Diabetes | Kidney transplantation | Index Medicus
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8. Full Text Protective effects of allicin on streptozotocin‐induced diabetic nephropathy in rats
by Huang, Hong and Jiang, Ying and Mao, Genxiang and Yuan, Fang and Zheng, Hexin and Ruan, Yuan and Wu, Tianfeng
Journal of the science of food and agriculture, ISSN 0022-5142, 03/2017, Volume 97, Issue 4, pp. 1359 - 1366
allicin | phosphorylated extracellular signal‐regulated kinase 1/2 | transforming growth factor β1 | diabetic nephropathy | phosphorylated extracellular signal-regulated kinase 1/2 | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Applied | Food Science & Technology | Agriculture, Multidisciplinary | Agriculture | Science & Technology | Sulfinic Acids - therapeutic use | Diabetes Mellitus, Experimental - drug therapy | Phosphorylation | Kidney - pathology | Plant Extracts - pharmacology | Streptozocin | Blood Urea Nitrogen | Transforming Growth Factor beta1 - metabolism | Diabetic Nephropathies - drug therapy | Sulfinic Acids - pharmacology | Allium - chemistry | Hyperglycemia - drug therapy | Diabetes Mellitus, Experimental - blood | Kidney - metabolism | Plant Roots | Diabetes Mellitus, Experimental - complications | Phytotherapy | Diabetic Nephropathies - pathology | Collagen Type I - metabolism | Kidney - drug effects | Albuminuria - etiology | Albumins - metabolism | Diabetic Nephropathies - metabolism | Diabetic Nephropathies - chemically induced | Rats, Sprague-Dawley | Animals | MAP Kinase Signaling System - drug effects | Albuminuria - drug therapy | Triglycerides - blood | Creatinine - blood | Diabetes Mellitus, Experimental - pathology | Hyperglycemia - etiology | Blood Glucose - metabolism | Plant Extracts - therapeutic use | Immunohistochemistry | Urea | Blood sugar | Collagen | Diabetic nephropathies | Albumin | Bone morphogenetic proteins | Transforming growth factors | Diabetic neuropathy | Garlic | Diet | Rodents | Index Medicus | Hyperglycemia | Kidneys | Collagens | Rats | United Arab Emirates | Kinases | Secretions | Blood
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9. Full Text Defective podocyte insulin signalling through p85-XBP1 promotes ATF6-dependent maladaptive ER-stress response in diabetic nephropathy
by Madhusudhan, Thati and Wang, Hongjie and Dong, Wei and Ghosh, Sanchita and Bock, Fabian and Thangapandi, Veera Raghavan and Ranjan, Satish and Wolter, Juliane and Kohli, Shrey and Shahzad, Khurrum and Heidel, Florian and Krueger, Martin and Schwenger, Vedat and Moeller, Marcus J and Kalinski, Thomas and Reiser, Jochen and Chavakis, Triantafyllos and Isermann, Berend
Nature communications, ISSN 2041-1723, 03/2015, Volume 6, Issue 1, pp. 6496 - 6496
Index Medicus
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10. Full Text Lycium barbarum polysaccharides ameliorates renal injury and inflammatory reaction in alloxan-induced diabetic nephropathy rabbits
by Zhao, Qihan and Li, Jingjing and Yan, Jun and Liu, Shuai and Guo, Yulin and Chen, Dajie and Luo, Qiong
Life sciences (1973), ISSN 0024-3205, 07/2016, Volume 157, pp. 82 - 90
Lycium barbarum polysaccharides | Diabetic nephropathy | Renal function | Inflammatory reaction | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Rabbits | Kidney - drug effects | Diabetic Nephropathies - chemically induced | Body Weight - drug effects | Male | Polysaccharides - pharmacology | Alloxan | Intercellular Adhesion Molecule-1 - metabolism | Kidney - metabolism | Organ Size - drug effects | Animals | Chemokine CCL2 - metabolism | Inflammation - prevention & control | Blood Glucose - metabolism | Diabetic Nephropathies - prevention & control | Lycium - chemistry | Pyrimidines | Polysaccharides | Diabetic nephropathies | Prevention | Urea | RNA | Analysis | Telmisartan | Index Medicus
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